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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2015, Volume 112, Issue 16, pp. 5051 - 5056
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0181340
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 03/2013, Volume 56, pp. 216 - 225
The dual oxidases (DUOX) 1and 2constitute the major components of the thyroid H O -generating system required for thyroid hormone synthesis. With their... 
DUOX regulation | Oxidative stress | Cytokines | Caco-2 | NADPH oxidase | Inflammation | H2O2 | Thyroid | Oxidases | Hydrogen peroxide | RNA | Interleukins | Analysis | Thyrotropin | Thyroid gland | Biological response modifiers | Gene expression
Journal Article
Cell Research, ISSN 1001-0602, 05/2013, Volume 23, Issue 5, pp. 673 - 690
Airway epithelial cells are key initial innate immune responders in the fight against respiratory viruses, primarily via the secretion of antiviral and... 
innate immunity | interferon | inflammation | NADPH oxidase | antiviral | cell signaling | DOUBLE-STRANDED-RNA | TRACT EPITHELIUM | INTERFERON-RESPONSE | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | EPITHELIAL-CELLS | GENE-EXPRESSION | RESPIRATORY-SYNCYTIAL-VIRUS | CYSTIC-FIBROSIS | NF-KAPPA-B | DUAL OXIDASE | Tumor Necrosis Factor-alpha - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | NADPH Oxidases - metabolism | Autocrine Communication - drug effects | Recombinant Proteins - biosynthesis | RNA, Messenger - metabolism | Sendai virus - metabolism | Receptor, Interferon alpha-beta - antagonists & inhibitors | STAT1 Transcription Factor - metabolism | RNA Interference | RNA, Small Cytoplasmic - metabolism | Interferon-beta - genetics | NADPH Oxidases - genetics | Receptor, Interferon alpha-beta - genetics | Respiratory Syncytial Viruses - metabolism | Receptor, Interferon alpha-beta - metabolism | Cell Line | Antiviral Agents - pharmacology | Immunity, Innate - drug effects | Signal Transduction | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Hydrogen Peroxide - metabolism | Drug Synergism | Tumor Necrosis Factor-alpha - pharmacology | Interferon-beta - metabolism | Interferon-beta - pharmacology | Paracrine Communication - drug effects | STAT2 Transcription Factor - metabolism | Dual Oxidases | Original
Journal Article
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 02/2019, Volume 481, pp. 62 - 70
We studied the mechanism that may explain the relative resistance of thyrocytes to H O compared to other cell types. Ability to degrade H O , glutathione... 
Hydrogen peroxide | DNA damage | Antioxidant enzymes | Thyroid | NADPH OXIDASE | CELLS | OXIDATIVE STRESS | DNA-DAMAGE | H2O2 | RADIATION | CELL BIOLOGY | REPAIR | THYROID-CANCER | ENDOCRINOLOGY & METABOLISM | EXPRESSION | EXPOSURE
Journal Article
Experimental Cell Research, ISSN 0014-4827, 11/2019, Volume 384, Issue 1, p. 111620
Thyroid hormone synthesis requires H O , produced by two NADPH oxidases, Duox1 and Duox2. To be fully active at the apical pole of the thyrocytes, these... 
DuoxA | Reactive oxygen species | Duox | N-glycosylation | DNA damage | Nox
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e56007
Stroma cell-derived factor-1 alpha (SDF-1 alpha) is a cardioprotective chemokine, acting through its G-protein coupled receptor CXCR4. In experimental acute... 
HEART | THERAPY | MYOCARDIAL-INFARCTION | MYOCYTES | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | CXCR4 | CA2+ TRANSIENTS | EXPRESSION | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Male | Ryanodine Receptor Calcium Release Channel - metabolism | Myocytes, Cardiac - enzymology | Heart Rate - drug effects | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Troponin I - genetics | Heart Rate - physiology | Animals, Newborn | Myocytes, Cardiac - cytology | Colforsin - pharmacology | Cells, Cultured | Rats | Receptors, CXCR4 - metabolism | Gene Expression Regulation - drug effects | Phenotype | Animals | Chemokine CXCL12 - metabolism | Myocytes, Cardiac - drug effects | Calcium Signaling - drug effects | Chemokine CXCL12 - pharmacology | Myocytes, Cardiac - metabolism | Troponin I - metabolism | Inositol 1,4,5-Trisphosphate Receptors - genetics | Vimentin | Heart | Myocardial infarction | Neonates | Cell culture | G protein-coupled receptors | Calcium | Ryanodine receptors | Fluorescence | Kinases | Calcium signalling | Angiogenesis | Rodents | Calcium-binding protein | Inositol 1,4,5-trisphosphate | Inositol 1,4,5-trisphosphate receptors | Caffeine | Heart failure | Inositol trisphosphate | Stroma | Cardiomyocytes | Fluxes | CXCR4 protein | Medicine | Troponin | Troponin I | Stem cells | Forskolin | Infarction | Ventricle | Laboratory animals | Immunofluorescence | Chemokines | Fluorescence microscopy | Cancer | Apoptosis
Journal Article