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Experimental Gerontology, ISSN 0531-5565, 07/2018, Volume 107, pp. 55 - 58
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 12/2017, Volume 8, p. 1745
Journal Article
Journal Article
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 08/2017, Volume 8
Aging is the greatest risk factor for developing chronic diseases. Inflamm-aging, the age-related increase in low-grade chronic inflammation, may be a common... 
Aging | Obesity | Antibody responses | Inflammation | Immunity | INDUCED CYTIDINE DEAMINASE | NECROSIS-FACTOR-ALPHA | immunity | INNATE LYMPHOID-CELLS | AGED MICE | IMMUNOLOGY | PROMOTE INSULIN-RESISTANCE | INDUCED OBESE MICE | IMPAIRED AFFINITY MATURATION | INFLUENZA-VIRUS INFECTION | inflammation | BODY-MASS INDEX | aging | IG CLASS SWITCH | obesity | antibody responses
Journal Article
Journal Article
FRONTIERS IN ENDOCRINOLOGY, ISSN 1664-2392, 02/2019, Volume 10, p. 71
Journal Article
Frontiers in immunology, ISSN 1664-3224, 2017, Volume 8, p. 1003
Aging is the greatest risk factor for developing chronic diseases. Inflamm-aging, the age-related increase in low-grade chronic inflammation, may be a common... 
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, p. e0197472
The adipose tissue (AT) contributes to systemic and B cell intrinsic inflammation, reduced B cell responses and secretion of autoimmune antibodies. In this... 
B-CELLS | TUMOR-NECROSIS-FACTOR | CLASS SWITCH RECOMBINATION | T-BET EXPRESSION | INSULIN-RESISTANCE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | BODY-MASS INDEX | HUMAN OBESITY | TRANSCRIPTION FACTOR | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Autoantibodies - metabolism | Cytokines - metabolism | Obesity - immunology | Humans | Oxidative Stress - physiology | Obesity - metabolism | Subcutaneous Fat - immunology | Transcription Factors - metabolism | Autoimmunity - physiology | Inflammation - metabolism | Subcutaneous Fat - metabolism | Adipocytes - metabolism | Chemokines - metabolism | Lipolysis - physiology | Autoimmunity | Adipose tissues | Autoantibodies | Physiological aspects | Development and progression | Genetic aspects | Research | Transcription factors | Adipose tissue | Toxicity | Immunoglobulin G | DNA damage | Differentiation (biology) | Antibodies | Cytotoxicity | Lipids | Cardiovascular disease | Adipocytes | Proteins | Receptors | Immunology | Aging | Deoxyribonucleic acid--DNA | Chronic illnesses | Immune system | Antigens | Obesity | CD11c antigen | Immunoglobulins | Immune response | Cytokines | Autoantigens | Plastic surgery | Inflammation | Metabolism | Gene expression | Cell differentiation | Ribonucleic acid--RNA | Class switching | Medicine | Lymphocytes B | Cell death | Hypoxia | Human behavior | Chemokines | Cancer | RNA | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article