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Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, pp. 16105 - 16105
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2012, Volume 52, Issue 6, pp. 1299 - 1307
Abstract Hypertrophic cardiomyopathy (HCM) is frequently caused by mutations in MYBPC3 encoding cardiac myosin-binding protein C (cMyBP-C). The mechanisms... 
Cardiovascular | Diastolic dysfunction | Ca2+ sensitivity | Ca2+ transient | Mouse model | Hypertrophy | MYOCARDIUM | BINDING-PROTEIN-C | CARDIAC & CARDIOVASCULAR SYSTEMS | FAMILIAL HYPERTROPHIC CARDIOMYOPATHY | UBIQUITIN-PROTEASOME SYSTEM | MYOCYTES | PHOSPHORYLATION | MECHANISMS | CELL BIOLOGY | GENE | MESSENGER-RNA DECAY | EXPRESSION | Cardiomyopathy, Hypertrophic - genetics | Echocardiography | Diastole | Calcium - metabolism | Cardiomyopathy, Hypertrophic - metabolism | Mice, Transgenic | Gene Knock-In Techniques | Carrier Proteins - genetics | Animals | Heart Ventricles - physiopathology | Cardiomyopathy, Hypertrophic - physiopathology | Myocytes, Cardiac - metabolism | Heterozygote | Heart Ventricles - metabolism | Mice | Myofibrils - metabolism | Mutation | Gene Order | Index Medicus | cTnI, cardiac troponin I | Mybpc3, mouse cardiac myosin-binding protein C gene | PKA, cAMP-dependent protein kinase A | max F, maximal Ca2+-activated force | KI, homozygous Mybpc3-targeted knock-in mice | MYBPC3, human cardiac myosin-binding protein C gene | Het, heterozygous Mybpc3-targeted knock-in mice | PLB, phospholamban | KO, homozygous Mybpc3-targeted knock-out mice | Ca2+ exchanger | SERCA2, SR-Ca2+ ATPase | HCM, hypertrophic cardiomyopathy | NCX, Na+ | SL, sarcomere length | SR, sarcoplasmic reticulum | cMyBP-C, cardiac myosin-binding protein C | nH, Hill coefficient | pCa50, log of [Ca2+] required for 50% of maximal activation | LVH, left ventricular hypertrophy | CSQ, calsequestrin | Original
Journal Article
BMC Musculoskeletal Disorders, ISSN 1471-2474, 04/2017, Volume 18, Issue 1, pp. 153 - 153
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 06/2012, Volume 52, Issue 6, pp. 1299 - 1307
Hypertrophic cardiomyopathy (HCM) is frequently caused by mutations in MYBPC3 encoding cardiac myosin-binding protein C (cMyBP-C). The mechanisms leading from... 
Diastolic dysfunction | Mouse model | sensitivity | transient | Hypertrophy
Journal Article
Biology Open, ISSN 2046-6390, 11/2016, Volume 5, Issue 11, pp. 1691 - 1696
Autosomal dominant centronuclear myopathy (CNM) is a rare congenital myopathy characterized by centrally located nuclei in muscle fibers. CNM results from... 
Dynamin2 | Centronuclear myopathy | Calcium | Knockin mouse model | PHOSPHATASE | INVOLVEMENT | PHENOTYPE | SKELETAL-MUSCLE | FIBERS | Knock-in mouse model | CHANNEL | DEPENDENT INTERNALIZATION | BIOLOGY | MICE | AMPHIPHYSIN-2 BIN1 | MUTATIONS | Dynamin 2 | Life Sciences
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 12/2010, Volume 19, Issue 24, pp. 4820 - 4836
Journal Article
Endocrinology, ISSN 0013-7227, 10/2013, Volume 154, Issue 10, pp. 3764 - 3775
The orexigenic and anabolic effects induced by ghrelin and the synthetic GH secretagogues (GHSs) are thought to positively contribute to therapeutic approaches... 
GHRELIN RECEPTOR LIGANDS | OLDER-ADULTS | ENDOCRINOLOGY & METABOLISM | PHARMACOLOGICAL IN-VITRO | GH SECRETAGOGUES | VENTRICULAR MYOCYTES | VIVO EVALUATIONS | CELL-LINES | UBIQUITIN LIGASES | EXPRESSION | DES-ACYL GHRELIN | Rats, Wistar | Receptors, Ghrelin - agonists | Sarcoplasmic Reticulum - drug effects | Ghrelin - analogs & derivatives | Mitochondria, Muscle - metabolism | Cytosol - drug effects | Male | Muscle, Skeletal - metabolism | Structure-Activity Relationship | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Oligopeptides - adverse effects | Spiro Compounds - adverse effects | Piperidines - pharmacology | Pituitary Gland, Anterior - secretion | Muscle, Skeletal - drug effects | Receptors, Ghrelin - metabolism | Growth Hormone - secretion | Sarcoplasmic Reticulum - metabolism | Sarcolemma - metabolism | Appetite Stimulants - pharmacology | Cell Line | Cell Survival - drug effects | Rats | Receptors, Ghrelin - antagonists & inhibitors | Ghrelin - metabolism | Animals | Calcium Signaling - drug effects | Cell Membrane Permeability - drug effects | Piperidines - adverse effects | Sarcolemma - drug effects | Cytosol - metabolism | Pituitary Gland, Anterior - drug effects | Mitochondria, Muscle - drug effects | Oligopeptides - pharmacology | Spiro Compounds - pharmacology | Appetite Stimulants - adverse effects | Index Medicus | Abridged Index Medicus
Journal Article
Annals of Neurology, ISSN 0364-5134, 06/2009, Volume 65, Issue 6, pp. 677 - 686
Journal Article
American Journal of Physiology, ISSN 0363-6143, 09/2010, Volume 299, Issue 3, p. C706
  Muscular dystrophies are often associated with significant cardiac disease that can be the prominent feature associated with gene mutations in sarcoglycan.... 
Transmission electron microscopy | Calcium | Mutation | Gene expression | Muscular dystrophy | Cells | Apoptosis
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 01/2001, Volume 280, Issue 1, pp. 146 - 154
The expression of the Na+/Ca2+ exchanger was studied in differentiating muscle fibers in rats. NCX1 and NCX3 isoform (Na+/Ca2+ exchanger isoform) expression... 
Gene expression | Sodium/calcium exchanger isoforms | CARDIAC NA+-CA2+ EXCHANGER | PHYSIOLOGY | ISOFORMS | RAT | SODIUM-CALCIUM EXCHANGER | MEMBRANE | SARCOPLASMIC-RETICULUM | RELEASE | IDENTIFICATION | CA2+-BINDING DOMAIN | sodium/calcium exchanger isoforms | CELL BIOLOGY | FIBER TYPES | gene expression
Journal Article