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1. Full Text Using a two-sample Mendelian randomization design to investigate a possible causal effect of maternal lipid concentrations on offspring birth weight
by Hwang, Liang-Dar and Lawlor, Deborah A and Freathy, Rachel M and Evans, David M and Warrington, Nicole M
International Journal of Epidemiology, ISSN 0300-5771, 07/2019
BACKGROUNDThe intrauterine environment is critical for fetal growth and development. However, observational associations between maternal gestational lipid... 
Index Medicus
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2. Full Text Can genetic evidence help us to understand the fetal origins of type 2 diabetes?
by Freathy, Rachel M
Diabetologia, ISSN 0012-186X, 9/2016, Volume 59, Issue 9, pp. 1850 - 1854
Lower birthweight is consistently associated with a higher risk of type 2 diabetes in observational studies, but the mechanisms underlying this association are... 
Fetal growth | Instrumental variable | Type 2 diabetes | Pleiotropy | Medicine & Public Health | Human Physiology | Genome-wide association study | Metabolic Diseases | Birthweight | Internal Medicine | Genetics | Mendelian randomisation | LOW-BIRTH-WEIGHT | ENDOCRINOLOGY & METABOLISM | ASSOCIATION | GLUCOSE-TOLERANCE | Infant, Low Birth Weight | Mendelian Randomization Analysis - methods | Genome-Wide Association Study | Diabetes Mellitus, Type 2 - genetics | Fetal Development - physiology | Humans | Birth Weight - physiology | Diabetes Mellitus, Type 2 - etiology | Fetal Development - genetics | Diabetes Mellitus, Type 2 - metabolism | Birth Weight - genetics | Medical colleges | Growth | Analysis | Genomics | Genetic research | Fetus | Epidemiology | Famines | Index Medicus | Mendelian randomization
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3. Full Text Genome-wide associations for birth weight and correlations with adult disease
by Horikoshi, Momoko and Beaumont, R.N and Day, Felix and Warrington, Nicole and Kooijman, Marjolein and Fernandez-Tajes, Juan and Feenstra, Bjarke and Van Zuydam, Natalie R and Gaulton, Kyle and Grarup, Niels and Bradfield, Jonathan and Strachan, David and Li-Gao, R and Ahluwalia, Tarunveer Singh and Kreiner, E and Rueedi, R and Lyytikäinen, Leo-Pekka and Cousminer, Diana and Wu, Ying and Thiering, Elisabeth and Wang, Carol A and Have, Christian T and Hottenga, Jouke Jan and Vilor-Tejedor, Natàlia and Joshi, Peter and Boh, E.T.H and Ntalla, Ioanna and Pitkanen, Niina and Mahajan, Anubha and Leeuwen, Elisa and Joro, Raimo and Lagou, Vasiliki and Nodzenski, Michael and Diver, L.A and Zondervan, Krina and Bustamante, Mariona and Marques-Vidal, Peo and Mercader, Josep and Bennett, Amanda and Rahmioglu, Nilufer and Nyholt, Dale and Ma, R.C.W and Tam, C.H.T and Tam, W.H and Ganesh, Santhi and Rooij, Frank and Jones, Samuel E and Loh, Po-Ru and Ruth, Katherine S and Tuke, M.A and Tyrrell, A.W.R and Wood, Anew and Yaghootkar, Hanieh and Scholtens, Denise M and Paternoster, Lavinia and Prokopenko, Inga and Kovacs, Peter and Atalay, Mustafa and Willems, Sara and Panoutsopoulou, K and Wang, Xu and Carstensen, Lisbeth and Geller, Frank and Schraut, K.E and Murcia, Mario and Beijsterveldt, Toos and Willemsen, Gonneke and Appel, E.V.R and Fonvig, C.E and Trier, C and Tiesler, C.M.T and Standl, Eberhard and Kutalik, Zoltán and Bonàs-Guarch, Silvia and Hougaard, David and Sánchez, F and Torrents, David and Waage, Johannes and Hollegaard, Mads V and De Haan, Hugoline G and Rosendaal, Frits and Medina-Gomez, C and Ring, Susan and Hemani, G and Mcmahon, George and Robertson, Neil and Groves, Christopher and Langenberg, Claudia and Luan, Jian'An and Scott, Robert and Zhao, Jing Hua and Mentch, Frank and MacKenzie, S.M and Reynolds, Rebecca and Lowe Jr., William and Tönjes, Anke and Stumvoll, Michael and Lindi, Virpi and Lakka, Timo and Duijn, Cornelia and ... and EGG Consortium and CHARGE Consortium Hematology Worki and CHARGE Consortium Hematology Working Group and Early Growth Genetics (EGG) Consortium
Nature: international weekly journal of science, ISSN 0028-0836, 01/2016, Volume 538, Issue 7624, pp. 248 - 252
textabstractBirth weight (BW) has been shown to be influenced by both fetal and maternal factors and in observational studies is reproducibly associated with... 
HUMAN HEIGHT | POPULATION | ALTITUDE ADAPTATION | METAANALYSIS | VARIANTS | GENOTYPE IMPUTATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LOCI | TRAITS | BLOOD-PRESSURE | Blood Pressure - genetics | Anthropometry | Diabetes Mellitus, Type 2 - genetics | Humans | Fetus - metabolism | Genetic Loci - genetics | Male | Aging - genetics | Glycogen - biosynthesis | Adult | Female | Genetic Predisposition to Disease | Genome-Wide Association Study | Datasets as Topic | Signal Transduction | Chromatin Assembly and Disassembly | Genotype | Birth Weight - genetics | Genomic Imprinting - genetics | Insulin - metabolism | Phenotype | Coronary Artery Disease - genetics | Glucose - metabolism | Genetic Variation - genetics | Cohort Studies | Genome-wide association studies | Birth size | Metabolic diseases | Genetic aspects | Birth weight | Cardiovascular diseases | Health aspects | Risk factors | Studies | Genomes | Disease | Correlation analysis | Index Medicus
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4. Full Text How Can Genetic Studies Help Us to Understand Links Between Birth Weight and Type 2 Diabetes?
by Beaumont, Robin N and Horikoshi, Momoko and McCarthy, Mark I and Freathy, Rachel M
Current diabetes reports, 04/2017, Volume 17, Issue 4, p. 22
In observational epidemiology, both low and high birth weights are associated with later type 2 diabetes. The mechanisms underlying the associations are poorly... 
Birth Weight | Genome-Wide Association Study | Diabetes Mellitus, Type 2 - genetics | Humans | Fetal Development | Diabetes Mellitus, Type 2 - etiology | Genotype | Genetic Loci
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5. Full Text Genome-Wide Association Analyses in 128,266 Individuals Identifies New Morningness and Sleep Duration Loci
by Jones, Samuel E and Tyrrell, Jessica and Wood, Andrew R and Beaumont, Robin N and Ruth, Katherine S and Tuke, Marcus A and Yaghootkar, Hanieh and Hu, Youna and Teder-Laving, Maris and Hayward, Caroline and Roenneberg, Till and Wilson, James F and Del Greco, Fabiola and Hicks, Andrew A and Shin, Chol and Yun, Chang-Ho and Lee, Seung Ku and Metspalu, Andres and Byrne, Enda M and Gehrman, Philip R and Tiemeier, Henning and Allebrandt, Karla V and Freathy, Rachel M and Murray, Anna and Hinds, David A and Frayling, Timothy M and Weedon, Michael N
PLoS Genetics, ISSN 1553-7390, 08/2016, Volume 12, Issue 8, pp. e1006125 - e1006125
Disrupted circadian rhythms and reduced sleep duration are associated with several human diseases, particularly obesity and type 2 diabetes, but until... 
CHRONOTYPE | OBESITY | METAANALYSIS | GWAS | VARIANTS | GENES | GENETICS & HEREDITY | COMPONENT | MENDELIAN RANDOMIZATION | DIURNAL PREFERENCE | INSIGHTS | Body Mass Index | PAX8 Transcription Factor - genetics | Genome-Wide Association Study | Diabetes Mellitus, Type 2 - genetics | Humans | Circadian Rhythm - genetics | Protein-Serine-Threonine Kinases - genetics | European Continental Ancestry Group | Male | Sleep - genetics | Obesity - genetics | Obesity - pathology | Mendelian Randomization Analysis | Female | Diabetes Mellitus, Type 2 - pathology | Sleep - physiology | Quantitative trait loci | Genome-wide association studies | Genetic aspects | Sleep | Observations | Studies | Obesity | Body mass index | Funding | Genomes | Circadian rhythm | Grants | Diabetes | Epidemiology | Disease control | Index Medicus
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6. Full Text A Common Variant in the FTO Gene Is Associated with Body Mass Index and Predisposes to Childhood and Adult Obesity
by Timothy M. Frayling and Nicholas J. Timpson and Michael N. Weedon and Eleftheria Zeggini and Rachel M. Freathy and Cecilia M. Lindgren and John R. B. Perry and Katherine S. Elliott and Hana Lango and Nigel W. Rayner and Beverley Shields and Lorna W. Harries and Jeffrey C. Barrett and Sian Ellard and Christopher J. Groves and Bridget Knight and Ann-Marie Patch and Andrew R. Ness and Shah Ebrahim and Debbie A. Lawlor and Susan M. Ring and Yoav Ben-Shlomo and Marjo-Riitta Jarvelin and Ulla Sovio and Amanda J. Bennett and David Melzer and Luigi Ferrucci and Ruth J. F. Loos and Inês Barroso and Nicholas J. Wareham and Fredrik Karpe and Katharine R. Owen and Lon R. Cardon and Mark Walker and Graham A. Hitman and Colin N. A. Palmer and Alex S. F. Doney and Andrew D. Morris and George Davey Smith and Andrew T. Hattersley and Mark I. McCarthy and Wellcome Trust Case Control and The Wellcome Trust Case Control Consortium
Science, ISSN 0036-8075, 5/2007, Volume 316, Issue 5826, pp. 889 - 894
Obesity is a serious international health problem that increases the risk of several common diseases. The genetic factors predisposing to obesity are poorly... 
Mass | Body mass index | Obesity | Average linear density | Reports | Adults | Type 2 diabetes mellitus | Children | Population genetics | Genotypes | Overweight | ENPP1 | GAD2 | MULTIDISCIPLINARY SCIENCES | Body Mass Index | Genetic Predisposition to Disease | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Overweight - genetics | Male | United Kingdom | Obesity - genetics | Case-Control Studies | Birth Weight | Homozygote | Adolescent | Alleles | Adult | Female | Aged | Polymorphism, Single Nucleotide | Adipose Tissue | Child | Infant, Newborn | Cohort Studies | Obesity gene | Research | Diabetes | Body fat | Genomics | Risk factors | Index Medicus
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7. Full Text Common Variation in the FTO Gene Alters Diabetes-Related Metabolic Traits to the Extent Expected Given Its Effect on BMI
by Rachel M. Freathy and Nicholas J. Timpson and Debbie A. Lawlor and Anneli Pouta and Yoav Ben-Shlomo and Aimo Ruokonen and Shah Ebrahim and Beverley Shields and Eleftheria Zeggini and Michael N. Weedon and Cecilia M. Lindgren and Hana Lango and David Melzer and Luigi Ferrucci and Giuseppe Paolisso and Matthew J. Neville and Fredrik Karpe and Colin N.A. Palmer and Andrew D. Morris and Paul Elliott and Marjo-Riitta Jarvelin and George Davey Smith and Mark I. McCarthy and Andrew T. Hattersley and Timothy M. Frayling
Diabetes, ISSN 0012-1797, 05/2008, Volume 57, Issue 5, pp. 1419 - 1426
Common Variation in the FTO Gene Alters Diabetes-Related Metabolic Traits to the Extent Expected Given Its Effect on BMI Rachel M. Freathy 1 , Nicholas J.... 
DIAGNOSIS | OBESITY | POLYMORPHISMS | VARIANTS | ENDOCRINOLOGY & METABOLISM | RISK | MENDELIAN RANDOMIZATION | TYPE-2 | CHILDHOOD | EPIDEMIOLOGY | GENOME-WIDE ASSOCIATION | Body Mass Index | European Continental Ancestry Group - genetics | Meta-Analysis as Topic | Blood Glucose - analysis | Diabetes Mellitus, Type 2 - genetics | Europe | Humans | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Genotype | Reference Values | Insulin - blood | Obesity - genetics | Genetic Variation | Proteins - genetics | Adult | Type 2 diabetes | Quantitative trait loci | Obesity | Body mass index | Genetic aspects | Diabetes | Gene expression | Health aspects | Risk factors | Index Medicus | Abridged Index Medicus
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8. Full Text Genome-wide association analysis identifies 20 loci that influence adult height
by Weedon, Michael N and Lango, Hana and Lindgren, Cecilia M and Wallace, Chris and Evans, David M and Mangino, Massimo and Freathy, Rachel M and Perry, John R. B and Stevens, Suzanne and Hall, Alistair S and Samani, Nilesh J and Shields, Beverly and Prokopenko, Inga and Farrall, Martin and Dominiczak, Anna and Johnson, Toby and Bergmann, Sven and Beckmann, Jacques S and Vollenweider, Peter and Waterworth, Dawn M and Mooser, Vincent and Palmer, Colin N. A and Morris, Andrew D and Ouwehand, Willem H and Zhao, Jing Hua and Li, Shengxu and Loos, Ruth J. F and Barroso, Inês and Deloukas, Panagiotis and Sandhu, Manjinder S and Wheeler, Eleanor and Soranzo, Nicole and Inouye, Michael and Wareham, Nicholas J and Caulfield, Mark and Munroe, Patricia B and Hattersley, Andrew T and McCarthy, Mark I and Frayling, Timothy M and Diabet Genetics Initiat and Cambridge GEM Consortium and Wellcome Trust Case Control Consor and Wellcome Trust Case Control Consortium and Diabetes Genetics Initiative and The Wellcome Trust Case Control Consortium
Nature Genetics, ISSN 1061-4036, 05/2008, Volume 40, Issue 5, pp. 575 - 583
Adult height is a model polygenic trait, but there has been limited success in identifying the genes underlying its normal variation. To identify genetic... 
POPULATION | COHORTS | STATURE | GENETIC ASSOCIATION | SUSCEPTIBILITY | GENETICS & HEREDITY | OSTEOARTHRITIS | BODY HEIGHT | EXPRESSION | STRATIFICATION | HERITABILITY | Extracellular Matrix Proteins - genetics | Humans | Middle Aged | Genotype | Male | Body Height - genetics | Hedgehog Proteins - genetics | Adult | Female | Aged | Polymorphism, Single Nucleotide | Genome, Human | Neoplasm Proteins - genetics | Stature | Quantitative trait loci | Genetic variation | Physiological aspects | Genetic aspects | Research | Identification and classification | Studies | Genetic research | Genetics | Gene loci | Height | Genes | Index Medicus
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9. Full Text A common variant of HMGA2 is associated with adult and childhood height in the general population
by Shields, Beverley and Salomaa, Veikko and Burtt, Noel P and Weedon, Michael N and Voight, Benjamin F and Tobias, Jonathan H and Guiducci, Candace and Morris, Andrew D and Perry, John R B and Ness, Andrew R and Lango, Hana and Frayling, Timothy M and Lettre, Guillaume and Elliott, Katherine S and Groop, Leif C and Palmer, Colin N A and Hirschhorn, Joel N and Zeggini, Eleftheria and Lindgren, Cecilia M and Freathy, Rachel M and Timpson, Nicholas J and Peltonen, Leena and Hackett, Rachel and Lyssenko, Valeriya and Rayner, Nigel W and McCarthy, Mark I and Ardlie, Kristin and Ring, Susan M and Saxena, Richa and Smith, George Davey and Hattersley, Andrew T and Diabet Genetics Initiative and Wellcome Trust Case Control Con and Wellcome Trust Case Control Consortium and Diabetes Genetics Initiative and The Diabetes Genetics Initiative and The Wellcome Trust Case Control Consortium and Genomik, diabetes och endokrinologi and Lund University and Genomics, Diabetes and Endocrinology and Lunds universitet
Nature Genetics, ISSN 1061-4036, 10/2007, Volume 39, Issue 10, pp. 1245 - 1250
Human height is a classic, highly heritable quantitative trait. To begin to identify genetic variants influencing height, we examined genome-wide association... 
METHODOLOGY | DISEASE | SUSCEPTIBILITY | GENETICS & HEREDITY | PHENOTYPE | RISK | C GENE | BODY HEIGHT | HERITABILITY | STRATIFICATION | GENOME-WIDE ASSOCIATION | Genetic Variation | Humans | HMGA2 Protein - genetics | Adult | Genotype | Polymorphism, Single Nucleotide | Child | Body Height - genetics | Case-Control Studies | Linkage Disequilibrium | Quantitative trait loci | Ubiquitin | Usage | Genetic variation | Physiological aspects | Genetic aspects | Research | Identification and classification | Height | Mutation | Genomics | Rodents | Index Medicus | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
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10. Full Text Common variants near MC4R are associated with fat mass, weight and risk of obesity
by Day, Ian and Dermitzakis, Emmanouil T and Randall, Joshua C and Weedon, Michael N and Hinney, Anke and Evans, David M and Qi, Lu and Bennett, Amanda J and Munroe, Patricia B and Herrera, Blanca and Johnson, Toby and Elliott, Katherine S and Song, Kijoung and Zeggini, Eleftheria and Cauchi, Stéphane and Teichmann, Sarah A and Lindgren, Cecilia M and Brown, Morris and Sims, Matthew A and Karpe, Fredrik and O'Rahilly, Stephen and Hattersley, Andrew T and Scherag, André and Samani, Nilesh J and Keniry, Andrew and Li, Shengxu and Beckmann, Jacques S and Cooper, Cyrus and Caulfield, Mark J and Owen, Katharine R and Luben, Robert N and McGinnis, Ralph and Ghori, Jilur and Ness, Andrew R and Bochud, Murielle and Jarvelin, Marjo-Riitta and Hebebrand, Johannes and Ong, Ken K and Freathy, Rachel M and McArdle, Wendy L and Potter, Simon and Wareham, Nicholas J and Ganz Vogel, Carla I and Mooser, Vincent and Barroso, Inês and Deloukas, Panos and Waterworth, Dawn M and Bergmann, Sven and Elliott, Paul and Gwilliam, Rhian and Tobias, Jonathan H and Jolley, Jennifer D M and Morris, Andrew D and Loos, Ruth J F and Yuan, Xin and Smith, George Davey and Palmer, Colin N A and Groves, Christopher J and Dina, Christian and Syddall, Holly E and Waeber, Gerard and Zhao, Jing Hua and Sandhu, Manjinder S and Doney, Alex S F and Hunt, Sarah E and Pouta, Anneli and Speliotes, Elizabeth K and Connell, John M and Timpson, Nicholas J and Wallace, Chris and Attwood, Antony P and Hall, Alistair S and Khaw, Kay-Tee and Meyre, David and Vollenweider, Peter and Bingham, Sheila A and Berndt, Sonja I and Hadley, David and Rayner, Nigel W and Frayling, Timothy M and Sadaf Farooqi, I and Hirschhorn, Joel N and Strachan, David P and Inouye, Michael and Papadakis, Konstantinos and Ring, Susan M and Wraight, Vicki L and Willer, Cristen J and Marchini, Jonathan and Uda, Manuela and Nica, Alexandra C and Mangino, Massimo and Soranzo, Nicole and McCarthy, Mark I and De, Subhajyoti and Neville, Matthew J and Prokopenko, Inga and Wheeler, Eleanor and Heid, Iris M and Froguel, Philippe and ... and Diabetes Genetics Initiative and SardiNIA Study and FUSION and Prostate Lung Colorectal Ovarian and KORA and Wellcome Trust Case Control Consor and Nurses' Health Study and Wellcome Trust Case Control Consortium and Prostate, Lung, Colorectal, and Ovarian (PLCO) Cancer Screening Trial and The Prostate, Lung, Colorectal, and Ovarian (PLCO) Cancer Screening Trial and The Wellcome Trust Case Control Consortium and The SardiNIA Study and Diabetes - klinisk obesitasforskning and Genomik, diabetes och endokrinologi and Lund University and Genomics, Diabetes and Endocrinology and Diabetes - Clinical Obesity and Lunds universitet
Nature Genetics, ISSN 1061-4036, 06/2008, Volume 40, Issue 6, pp. 768 - 775
To identify common variants influencing body mass index (BMI), we analyzed genome-wide association data from 16,876 individuals of European descent. After... 
FTO GENE | GENETICS & HEREDITY | MELANOCORTIN-4 RECEPTOR GENE | EARLY-ONSET OBESITY | FRAMESHIFT MUTATION | DOMINANT | ADULT OBESITY | EXPRESSION | CHILDHOOD | DEFICIENCY | GENOME-WIDE ASSOCIATION | Meta-Analysis as Topic | Humans | Middle Aged | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Male | Body Weight - genetics | Obesity - genetics | Case-Control Studies | Genetic Variation | Proteins | Adult | Female | Child | Body Mass Index | Genetic Predisposition to Disease | Cross-Sectional Studies | Receptor, Melanocortin, Type 4 - genetics | Adiposity - genetics | Linkage Disequilibrium | Obesity - pathology | Randomized Controlled Trials as Topic | Obesity - epidemiology | Adolescent | Alleles | Aged | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 18 - genetics | Genome, Human | Quantitative Trait Loci | Cohort Studies | Obesity | Body mass index | Genetic variation | Physiological aspects | Genetic aspects | Diagnosis | Research | Risk factors | Medical research | Biomedical research | Body fat | Health services | Genetics | Birth weight | Adults | Progeny | Integration | Pedigree | Data processing | Melanocortin MC4 receptors | Single-nucleotide polymorphism | Children | Mutation | Population levels | Index Medicus | Clinical Medicine | Näringslära | Hälsovetenskap | Endokrinologi och diabetes | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Nutrition and Dietetics | Endocrinology and Diabetes | Health Sciences
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