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index medicus (19) 19
humans (18) 18
female (17) 17
breast cancer (16) 16
breast neoplasms - genetics (11) 11
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tumors (11) 11
oncology (9) 9
cancer (8) 8
metastasis (8) 8
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gene expression (6) 6
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disease progression (5) 5
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article (4) 4
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health aspects (3) 3
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immunohistochemistry (3) 3
invasion (3) 3
medical prognosis (3) 3
membrane proteins - genetics (3) 3
metalloproteinase (3) 3
neoplasms. tumors. oncology. including cancer and carcinogens (3) 3
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proportional hazards models (3) 3
research article (3) 3
survival (3) 3
triple negative breast neoplasms - genetics (3) 3
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activation (2) 2
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animals (2) 2
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biology (2) 2
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biomarkers, tumor - genetics (2) 2
carcinogenesis (2) 2
carcinoma, adenoid cystic - genetics (2) 2
carcinoma, adenoid cystic - pathology (2) 2
cells (2) 2
church history (2) 2
class i phosphatidylinositol 3-kinases - genetics (2) 2
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complex (2) 2
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gene (2) 2
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missense mutations (2) 2
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nerve tissue proteins - genetics (2) 2
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1. Full Text Genomic landscape of adenoid cystic carcinoma of the breast
by Martelotto, Luciano G and De Filippo, Maria R and Ng, Charlotte KY and Natrajan, Rachael and Fuhrmann, Laetitia and Cyrta, Joanna and Piscuoglio, Salvatore and Wen, Huei‐Chi and Lim, Raymond S and Shen, Ronglai and Schultheis, Anne M and Wen, Y Hannah and Edelweiss, Marcia and Mariani, Odette and Stenman, Göran and Chan, Timothy A and Colombo, Pierre‐Emmanuel and Norton, Larry and Vincent‐Salomon, Anne and Reis‐Filho, Jorge S and Weigelt, Britta and Institutionen för biomedicin, avdelningen för patologi and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institute of Biomedicine, Department of Pathology and Sahlgrenska Academy
The Journal of Pathology, ISSN 0022-3417, 10/2015, Volume 237, Issue 2, pp. 179 - 189
Adenoid cystic carcinoma (AdCC) is a rare type of triple‐negative breast cancer (TNBC) characterized by the presence of the MYB–NFIB fusion gene. The molecular... 
genomics | MYB–NFIB | adenoid cystic carcinoma | triple‐negative breast cancer | massively parallel sequencing | triple-negative breast cancer | MYB-NFIB | HEAD | HISTOLOGICAL SPECIAL TYPES | MYB EXPRESSION | PATHOLOGY | SALIVARY-GLANDS | MISSENSE MUTATIONS | SOMATIC MUTATIONS | ONCOLOGY | MUTATIONAL LANDSCAPE | COPY-NUMBER ALTERATION | NFIB GENE FUSION | HUMAN CANCER | Genetic Predisposition to Disease | Carcinoma, Adenoid Cystic - pathology | Biomarkers, Tumor - analysis | Gene Frequency | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Adenoid Cystic - chemistry | In Situ Hybridization, Fluorescence | Gene Dosage | Genes, myb | Reverse Transcriptase Polymerase Chain Reaction | DNA Copy Number Variations | Phenotype | Triple Negative Breast Neoplasms - chemistry | Triple Negative Breast Neoplasms - genetics | Carcinoma, Adenoid Cystic - genetics | DNA Mutational Analysis | Oncogene Proteins, Fusion - genetics | Triple Negative Breast Neoplasms - pathology | Female | Biomarkers, Tumor - genetics | Mutation | Genomics - methods | Real-Time Polymerase Chain Reaction | Ubiquitin | Carcinoma | Multiprocessing | Genomics | Genetic aspects | Breast cancer | Tumor proteins | Chromosomes | Cancer | Adenoid cystic carcinoma | massively parallel | Oncology | SOMATIC | Pathology | MUTATIONS | Cancer and Oncology | Cancer och onkologi
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2. Full Text Control of MT1-MMP transport by atypical PKC during breast-cancer progression
by Carine Rossé and Catalina Lodillinsky and Laetitia Fuhrmann and Maya Nourieh and Pedro Monteiro and Marie Irondelle and Emilie Lagoutte and Sophie Vacher and François Waharte and Perrine Paul-Gilloteaux and Maryse Romao and Lucie Sengmanivong and Mark Linch and Johan van Lint and Graça Raposo and Anne Vincent-Salomon and Ivan Bièche and Peter J. Parker and Philippe Chavrier
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 05/2014, Volume 111, Issue 18, pp. E1872 - E1879
Dissemination of carcinoma cells requires the pericellular degradation of the extracellular matrix, which is mediated by membrane type 1-matrix... 
Membrane traffic | MMP14 | Actin cytoskeleton | Multi-vesicular body | METASTASIS | POLARITY | CORTACTIN | METALLOPROTEINASE | MULTIDISCIPLINARY SCIENCES | CELL INVASION | multi-vesicular body | TUMOR-CELLS | membrane traffic | actin cytoskeleton | OVEREXPRESSION | INVADOPODIA FORMATION | EXTRACELLULAR-MATRIX DEGRADATION | EXPRESSION | Carcinoma, Ductal, Breast - genetics | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Up-Regulation | Phosphorylation | Adenocarcinoma - pathology | Humans | Middle Aged | Extracellular Matrix - metabolism | RNA, Messenger - metabolism | RNA, Neoplasm - metabolism | Endosomes - metabolism | Breast Neoplasms - metabolism | Biological Transport, Active | Adenocarcinoma - metabolism | RNA Interference | Isoenzymes - metabolism | Protein Kinase C - metabolism | Cytoplasmic Granules - metabolism | Adult | Carcinoma, Ductal, Breast - pathology | Female | Adenocarcinoma - genetics | Dynamin II - metabolism | Carcinoma, Ductal, Breast - metabolism | Isoenzymes - genetics | Neoplasm Invasiveness | RNA, Messenger - genetics | Protein Kinase C - antagonists & inhibitors | Cortactin - metabolism | Matrix Metalloproteinase 14 - metabolism | Disease Progression | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Matrix Metalloproteinase 14 - genetics | Cell Line, Tumor | RNA, Neoplasm - genetics | Aged | Isoenzymes - antagonists & inhibitors | Life Sciences | Biological Sciences | PNAS Plus
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3. Full Text Coronin 1C promotes triple-negative breast cancer invasiveness through regulation of MT1-MMP traffic and invadopodia function
by Castagnino, Alessia and Castro-Castro, Antonio and Irondelle, Marie and Guichard, Alan and Lodillinsky, Catalina and Fuhrmann, Laetitia and Vacher, Sophie and Agüera-González, Sonia and Zagryazhskaya-Masson, Anna and Romao, Maryse and El Kesrouani, Carole and Noegel, Angelika A and Dubois, Thierry and Raposo, Graça and Bear, James E and Clemen, Christoph S and Vincent-Salomon, Anne and Bièche, Ivan and Chavrier, Philippe
Oncogene, ISSN 0950-9232, 12/2018, Volume 37, Issue 50, pp. 6425 - 6441
Membrane type 1-matrix metalloproteinase (MT1-MMP), a membrane-tethered protease, is key for matrix breakdown during cancer invasion and metastasis. Assembly... 
CORTACTIN PROMOTES | METASTASIS | MATRIX-METALLOPROTEINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACTIN-BINDING | ARP2/3 COMPLEX | CELL BIOLOGY | INVASION | ONCOLOGY | EXOSOME SECRETION | WASH | GENETICS & HEREDITY | EXPRESSION | MOLECULAR-MECHANISMS | Research | Binding proteins | Gene expression | Cancer cells | Actin | Invasiveness | Collagen | Fibrils | Xenografts | Membrane trafficking | Breast cancer | Metalloproteinase | Matrix metalloproteinase | Spheroids | Metastases
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4. Full Text Fibroblast Heterogeneity and Immunosuppressive Environment in Human Breast Cancer
by Costa, Ana and Kieffer, Yann and Scholer-Dahirel, Alix and Pelon, Floriane and Bourachot, Brigitte and Cardon, Melissa and Sirven, Philemon and Magagna, Ilaria and Fuhrmann, Laetitia and Bernard, Charles and Bonneau, Claire and Kondratova, Maria and Kuperstein, Inna and Zinovyev, Andrei and Givel, Anne-Marie and Parrini, Maria-Carla and Soumelis, Vassili and Vincent-Salomon, Anne and Mechta-Grigoriou, Fatima
Cancer Cell, ISSN 1535-6108, 03/2018, Volume 33, Issue 3, pp. 463 - 479.e10
Carcinoma-associated fibroblasts (CAF) are key players in the tumor microenvironment. Here, we characterize four CAF subsets in breast cancer with distinct... 
T lymphocytes | fibroblasts | stroma | Treg | CAF | FOXP3 | triple-negative | heterogeneity | breast cancers | regulatory T cell | regulatory T cell | CARCINOMA-ASSOCIATED FIBROBLASTS | METASTASIS | ONCOLOGY | ACTIVATION PROTEIN | PANCREATIC-CANCER | REGULATORY T-CELLS | PROGNOSTIC-SIGNIFICANCE | ANTITUMOR IMMUNITY | STROMAL CELLS | EXPRESSION | TUMOR MICROENVIRONMENT | CELL BIOLOGY | Forkhead Transcription Factors - immunology | Tumor Microenvironment - immunology | Breast Neoplasms - immunology | Immune Tolerance - immunology | Humans | Cell Proliferation - physiology | Fibroblasts - immunology | Cell Differentiation - physiology | Lymphocyte Activation - physiology | Lymphocytes, Tumor-Infiltrating - immunology | T-Lymphocytes, Regulatory - immunology | Breast cancer | Dendritic cells | T cells | Analysis
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5. Full Text Combinatorial expression of microtubule-associated EB1 and ATIP3 biomarkers improves breast cancer prognosis
by Rodrigues-Ferreira, Sylvie and Nehlig, Anne and Monchecourt, Clarisse and Nasr, Sarah and Fuhrmann, Laetitia and Lacroix-Triki, Magali and Garberis, Ingrid and Scott, Véronique and Delaloge, Suzette and Pistilli, Barbara and Vielh, Philippe and Dubois, Thierry and Vincent-Salomon, Anne and André, Fabrice and Nahmias, Clara
Breast Cancer Research and Treatment, ISSN 0167-6806, 2/2019, Volume 173, Issue 3, pp. 573 - 583
The identification of molecular biomarkers for classification of breast cancer is needed to better stratify the patients and guide therapeutic decisions. The... 
Prognosis | Medicine & Public Health | Biomarkers combination | MTUS1 | Oncology | Breast cancer | Diagnosis | MAPRE1 | SURVIVAL | MARKER | OVEREXPRESSION | ONCOLOGY | TRACKING | GENES | PROGRESSION | END-BINDING PROTEIN-1 | Recurrence | Gene Expression | Microtubule-Associated Proteins - genetics | Humans | Biomarkers, Tumor | Breast Neoplasms - genetics | Neoplasm Grading | Tumor Suppressor Proteins - genetics | Survival Analysis | Breast Neoplasms - mortality | Female | Breast Neoplasms - diagnosis | Ethylenediaminetetraacetic acid | Medical research | Analysis | Medicine, Experimental | Binding proteins | Biological markers | Health aspects | Protein binding | Cancer
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6. Full Text Genetic events in the progression of adenoid cystic carcinoma of the breast to high-grade triple-negative breast cancer
by Fusco, Nicola and Geyer, Felipe C and De Filippo, Maria R and Martelotto, Luciano G and Ng, Charlotte K. Y and Piscuoglio, Salvatore and Guerini-Rocco, Elena and Schultheis, Anne M and Fuhrmann, Laetitia and Wang, Lu and Jungbluth, Achim A and Burke, Kathleen A and Lim, Raymond S and Vincent-Salomon, Anne and Bamba, Masamichi and Moritani, Suzuko and Badve, Sunil S and Ichihara, Shu and Ellis, Ian O and Reis-Filho, Jorge S and Weigelt, Britta
Modern Pathology, ISSN 0893-3952, 11/2016, Volume 29, Issue 11, pp. 1292 - 1305
Adenoid cystic carcinoma of the breast is a rare histological type of triple-negative breast cancer with an indolent clinical behavior, often driven by the... 
TRANSFORMATION | FUSION | RECOMMENDATIONS | MYB | MUTATIONAL LANDSCAPE | BASALOID FEATURES | PATHOLOGY | TUMORS | MISSENSE MUTATIONS | SOLID VARIANT | REVEALS | Triple Negative Breast Neoplasms - genetics | Carcinoma, Adenoid Cystic - pathology | Carcinoma, Adenoid Cystic - genetics | Humans | Triple Negative Breast Neoplasms - pathology | Adult | Female | Disease Progression | breast cancer | tumor progression | triple-negative | Adenoid cystic carcinoma | high-grade
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7. Full Text Comprehensive clinical and molecular analyses of neuroendocrine carcinomas of the breast
by Lavigne, Marion and Menet, Emmanuelle and Tille, Jean-Christophe and Lae, Marick and Fuhrmann, Laetitia and Bonneau, Claire and Deniziaut, Gabrielle and Melaabi, Samia and Ng, Charlotte C K and Marchiò, Caterina and Rouzier, Roman and Bièche, Ivan and Vincent-Salomon, Anne
Modern Pathology, ISSN 0893-3952, 01/2018, Volume 31, Issue 1, pp. 68 - 82
Neuroendocrine breast carcinomas represent a rare subtype of breast cancer. Their definition, prevalence, and prognosis remain controversial in the literature.... 
IMPACT | PIK3CA | SMALL-CELL CARCINOMA | ENDOCRINE DIFFERENTIATION | MAMMARY-CARCINOMA | TERM-FOLLOW-UP | PATHOLOGY | CANCER | EXPRESSION | FEATURES | REVEALS | Carcinoma, Neuroendocrine - mortality | Prognosis | Humans | Middle Aged | Kaplan-Meier Estimate | Proportional Hazards Models | Carcinoma, Neuroendocrine - genetics | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Carcinoma, Neuroendocrine - pathology | DNA Mutational Analysis | Aged, 80 and over | Adult | Breast Neoplasms - mortality | Female | Aged | Statistical analysis | Aggressive behavior | Invasiveness | p53 Protein | Estrogen | Breast cancer | Survival | Breast carcinoma | Medical prognosis | GATA-3 protein | Mutation | Progesterone | Neuroendocrine tumors | Tumors
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8. Full Text High rate of PIK3CA mutations but no TP53 mutations in low‐grade adenosquamous carcinoma of the breast
by Bataillon, Guillaume and Fuhrmann, Laetitia and Girard, Elodie and Menet, Emanuelle and Laé, Marick and Capovilla, Mathieu and Treilleux, Isabelle and Arnould, Laurent and Penault‐Llorca, Frederique and Rouzier, Roman and Marchiò, Caterina and Bieche, Ivan and Vincent‐Salomon, Anne
Histopathology, ISSN 0309-0167, 08/2018, Volume 73, Issue 2, pp. 273 - 283
Aims Low‐grade adenosquamous carcinoma of the breast (LGASC) is a rare variant of metaplastic carcinoma characterised by a favourable outcome and... 
metaplastic carcinoma | breast cancer | androgen receptor | PIK3CA mutations | low‐grade adenosquamous carcinoma | low-grade adenosquamous carcinoma | PRECURSOR | MAMMARY-CARCINOMA | RECEPTOR | PROLIFERATION | PATHOLOGY | CANCER | TUMORS | CELL BIOLOGY | NIPPLE | SCLEROSING LESIONS | DIFFERENTIATION | EXPRESSION | Class I Phosphatidylinositol 3-Kinases - genetics | Triple Negative Breast Neoplasms - genetics | Humans | Middle Aged | Aged, 80 and over | Adult | Female | Aged | Retrospective Studies | Carcinoma, Adenosquamous - genetics | Mutation | Tumor Suppressor Protein p53 - genetics | Carcinoma | Analysis | Genetic research | Breast cancer | Genetic aspects | Nucleotide sequencing | Tumor proteins | DNA sequencing | Cancer | Adenocarcinoma | Squamous cell carcinoma | Androgens | Phenotypes | Epidermal growth factor | Missense mutation | p53 Protein | Adenosquamous | Tumors | Life Sciences
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9. Full Text LIN7A is a major determinant of cell-polarity defects in breast carcinomas
by Gruel, Nadège and Fuhrmann, Laetitia and Lodillinsky, Catalina and Benhamo, Vanessa and Mariani, Odette and Cédenot, Aurélie and Arnould, Laurent and Macgrogan, Gaëtan and Sastre-Garau, Xavier and Chavrier, Philippe and Delattre, Olivier and Vincent-Salomon, Anne
Breast Cancer Research, ISSN 1465-5411, 02/2016, Volume 18, Issue 1, p. 23
Background: Polarity defects are a hallmark of most carcinomas. Cells from invasive micropapillary carcinomas (IMPCs) of the breast are characterized by a... 
Breast cancer | Cell polarity | LIN7A | Micropapillary carcinomas | INVASIVE MICROPAPILLARY | ORGANIZATION | COMPLEX | PROTEIN | EPITHELIAL-CELLS | ONCOLOGY | CANCER | PROMOTES MAMMARY TUMORIGENESIS | Cell Proliferation - genetics | Membrane Proteins - genetics | Humans | Carcinogenesis - genetics | Gene Expression Regulation, Neoplastic | Lymphatic Metastasis | Cell Polarity - genetics | Membrane Proteins - biosynthesis | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Neoplasm Invasiveness - pathology | Cell Line, Tumor | Female | Neoplasm Invasiveness - genetics | Genetic aspects | Polarity (Biology) | Gene expression | Analysis
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10. Full Text ARF6-JIP3/4 regulate endosomal tubules for MT1-MMP exocytosis in cancer invasion
by Marchesin, Valentina and Castro-Castro, Antonio and Lodillinsky, Catalina and Castagnino, Alessia and Cyrta, Joanna and Bonsang-Kitzis, Hélène and Fuhrmann, Laetitia and Irondelle, Marie and Infante, Elvira and Montagnac, Guillaume and Reyal, Fabien and Vincent-Salomon, Anne and Chavrier, Philippe
Journal of Cell Biology, ISSN 0021-9525, 10/2015, Volume 211, Issue 2, pp. 339 - 358
Invasion of cancer cells into collagen-rich extracellular matrix requires membrane-tethered membrane type 1 matrix metalloproteinase (MT1-MMP) as the key... 
INVADOPODIA FORMATION | PROTEIN | SIGNALING PATHWAY | METALLOPROTEINASE | TRAFFICKING | TUMOR-CELL INVASION | EXTRACELLULAR-MATRIX | AXONAL-TRANSPORT | DYNEIN MOTOR | ARF6 | CELL BIOLOGY | Neoplasm Invasiveness | Humans | Matrix Metalloproteinase 14 - metabolism | Exocytosis - physiology | Nerve Tissue Proteins - genetics | Endosomes - metabolism | Kinesin - metabolism | Protein Transport | Nerve Tissue Proteins - metabolism | Collagen - metabolism | RNA Interference | Adaptor Proteins, Signal Transducing - genetics | HEK293 Cells | Matrix Metalloproteinase 14 - genetics | Triple Negative Breast Neoplasms - pathology | Female | Microfilament Proteins - metabolism | RNA, Small Interfering | ADP-Ribosylation Factors - metabolism | Tumor Cells, Cultured | Adaptor Proteins, Signal Transducing - metabolism | Spheroids, Cellular | ADP-Ribosylation Factors - genetics | Cellular proteins | Genetic aspects | Properties | Proteases | Carcinogenesis | Index Medicus
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11. Full Text Distinct expression profiles and functions of Kindlins in breast cancer
by Azorin, Paula and Azorin, Paula and Bonin, Florian and Bonin, Florian and Moukachar, Ahmad and Moukachar, Ahmad and Ponceau, Aurélie and Ponceau, Aurélie and Vacher, Sophie and Vacher, Sophie and Bièche, Ivan and Bièche, Ivan and Marangoni, Elisabetta and Marangoni, Elisabetta and Fuhrmann, Laetitia and Fuhrmann, Laetitia and Vincent-Salomon, Anne and Vincent-Salomon, Anne and Lidereau, Rosette and Lidereau, Rosette and Driouch, Keltouma and Driouch, Keltouma
Journal of Experimental and Clinical Cancer Research, ISSN 1756-9966, 11/2018, Volume 37, Issue 1, pp. 1 - 15
BackgroundKindlin-1, -2, and-3 are the three members of the Kindlin family. They are best known as regulators of integrin functions, contributing to... 
Redundant functions | Kindlins | Breast cancer | Prognosis | Invasion | MIGRATION | METASTASIS | PATTERNS | IDENTIFICATION | ADHESION | MOLECULAR PORTRAITS | GENE | ONCOLOGY | PROMOTES | PROGRESSION | Immunohistochemistry | Membrane Proteins - genetics | Neoplasm Proteins - biosynthesis | Humans | Breast Neoplasms - metabolism | Membrane Proteins - biosynthesis | Breast Neoplasms - genetics | Transfection | Lymphocytes, Tumor-Infiltrating - pathology | MCF-7 Cells | Breast Neoplasms - pathology | Cell Line, Tumor | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Neoplasm Proteins - genetics | Women | Patient outcomes | Analysis | Genetic aspects | Diagnosis | Research | Gene expression | Health aspects | Proteins | Liver cancer | Medical prognosis | Morphology | Metastasis | Patients | Tumors | Cell adhesion & migration
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12. Full Text VOPP1 promotes breast tumorigenesis by interacting with the tumor suppressor WWOX
by Bonin, Florian and Taouis, Karim and Azorin, Paula and Petitalot, Ambre and Tariq, Zakia and Nola, Sebastien and Bouteille, Nadège and Tury, Sandrine and Vacher, Sophie and Bièche, Ivan and Rais, Khadija Ait and Pierron, Gaelle and Fuhrmann, Laetitia and Vincent-Salomon, Anne and Formstecher, Etienne and Camonis, Jacques and Lidereau, Rosette and Lallemand, François and Driouch, Keltouma
BMC Biology, ISSN 1741-7007, 10/2018, Volume 16, Issue 1, pp. 109 - 16
Background: The WW domain-containing oxidoreductase (WWOX) gene, frequently altered in breast cancer, encodes a tumor suppressor whose function is mediated... 
WWOX | VOPP1 | Breast tumors | TRANSCRIPTIONAL ACTIVITY | ACTIVATION | PROTEIN | FRA16D GENE | IDENTIFICATION | CANCER GROWTH | BIOLOGY | GROWTH IN-VITRO | NF-KAPPA-B | ASSOCIATION | GENE-PRODUCT | Transcription Factors - metabolism | Tumor Suppressor Proteins - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Transformation, Neoplastic - genetics | Humans | Tumor Suppressor Proteins - genetics | Cell Line, Tumor | WW Domain-Containing Oxidoreductase - metabolism | Female | Transcription Factors - genetics | WW Domain-Containing Oxidoreductase - genetics | Care and treatment | Development and progression | Tumor suppressor genes | Breast cancer | Genetic aspects | Health aspects | Oncogenes | Transformation | Yeast | Immunoprecipitation | Oxidoreductase | Lysosomes | Metabolism | Kinases | Survival | Carcinogenesis | Anticancer properties | Proteins | Carcinogens | Breast | Tumor necrosis factor-TNF | Tumorigenesis | Mutation | Cancer | Apoptosis | Tumors
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