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Cancer Research and Treatment, ISSN 1598-2998, 2012, Volume 44, Issue 3, pp. 151 - 156
Among all types of cancer, incidence of lung cancer remains the highest with regard to cancer-related mortality. Problems contributing to recurrence of the... 
Epithelial-mesenchymal transition | Drug resistance | Neoplasm metastasis | Lung neoplasms | STEM-CELLS | SENSITIVITY | EMT | TYROSINE KINASE INHIBITORS | IN-VITRO | ZEB1 | ONCOLOGY | PROTEIN EXPRESSION | TUMOR-METASTASIS | TRANSCRIPTION FACTOR | SNAIL | Review | 의약학
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e102436 - e102436
Background: We previously reported the role of IL-6 in a murine model of cancer cachexia and currently documented a patient in whom tocilizumab, anti-IL-6... 
PROGNOSIS | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | WEIGHT-LOSS | CASTLEMAN-DISEASE | MECHANISMS | INTERLEUKIN-6 | BEARING MICE | TUMOR-GROWTH | ANTI-INTERLEUKIN-6 RECEPTOR ANTIBODY | T-CELLS | Cachexia - drug therapy | Humans | Middle Aged | Male | Antibodies, Monoclonal, Humanized - administration & dosage | Interleukin-6 - blood | Aged, 80 and over | Female | Gene Expression Regulation, Neoplastic - drug effects | Receptors, Interleukin-6 - blood | Carcinoma, Non-Small-Cell Lung - pathology | Kaplan-Meier Estimate | Carcinoma, Lewis Lung - blood | Cachexia - blood | Carcinoma, Lewis Lung - drug therapy | Biomarkers, Tumor - blood | Animals | Cachexia - pathology | Carcinoma, Lewis Lung - pathology | Interleukin-6 - biosynthesis | Carcinoma, Non-Small-Cell Lung - blood | Aged | Mice | Carcinoma, Non-Small-Cell Lung - drug therapy | Biomarkers, Tumor - biosynthesis | Chemotherapy | Cachexia | Analysis | Lung cancer | Cancer | Animal models | Anorexia | Prognosis | Disease | Lung transplantation | Clinical trials | Transplantation | Metastasis | Medical diagnosis | Metastases | Interleukin 6 | Lymphocytes | Consent | Interleukin 6 receptors | Tumor necrosis factor-TNF | Measurement methods | Water intakes | Medical research | Cytokines | Anemia | Lung carcinoma | Patients | Survival | Quality of life | Medicine | Food intake | Medical prognosis | Rheumatoid arthritis | Water intake | University faculty | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e86459
Accumulating evidence indicates that a small population of cancer stem cells (CSCs) is involved in intrinsic resistance to cancer treatment. The hypoxic... 
TYROSINE KINASE INHIBITORS | MUTANT | MET AMPLIFICATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | ACQUIRED-RESISTANCE | PHASE-II | MUTATIONS | MESENCHYMAL TRANSITION | FACTOR-I RECEPTOR | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Neoplasm Transplantation | Receptor, Epidermal Growth Factor - genetics | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Spheroids, Cellular - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Lung Neoplasms - pathology | Insulin-Like Growth Factor I - genetics | Antigens, CD - metabolism | Gene Knockdown Techniques | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Cell Hypoxia - drug effects | Carcinoma, Non-Small-Cell Lung - genetics | Cell Separation | Carcinogenesis - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Mutation - genetics | AC133 Antigen | Receptor, IGF Type 1 - genetics | Carcinogenesis - drug effects | Carcinogenesis - pathology | Up-Regulation - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Quinazolines - therapeutic use | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice, Inbred NOD | Carcinoma, Non-Small-Cell Lung - drug therapy | Cell Hypoxia - genetics | Quinazolines - pharmacology | Insulin-Like Growth Factor I - metabolism | Drug Resistance, Neoplasm - drug effects | Care and treatment | Epidermal growth factor | Genes | Stem cells | Lung cancer, Non-small cell | Gefitinib | Cancer | Insulin-like growth factor I | Oct-4 protein | Lung cancer | Insulin-like growth factors | Activation | Kinases | Drug resistance | Cancer therapies | Clonal deletion | Deletion | Hypoxia-inducible factors | Parasitology | Epidermal growth factor receptors | Non-small cell lung carcinoma | Exposure | Tumorigenicity | Insulin | CXCR4 protein | Medicine | Chemotherapy | Medical prognosis | Cell lines | Hypoxia | Mutation | Tumors | Pharmaceuticals
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 08/2019, Volume 9, Issue 1, pp. 12062 - 9
Nausea and diarrhea are the most common adverse effects of nintedanib in patients with idiopathic pulmonary fibrosis (IPF). However, the clinical risk factors... 
BIBF 1120 | MANAGEMENT | EFFICACY | SAFETY | MULTIDISCIPLINARY SCIENCES | TRIPLE ANGIOKINASE INHIBITOR | TYROSINE KINASE INHIBITOR | Toxicity | Lung diseases | Diarrhea | Nausea | Patients | Risk factors | Body mass index | Side effects | Pulmonary fibrosis | Dyspnea | Fibrosis | Respiration | Health risk assessment | Prednisolone
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3874 - 3882
Autocrine and paracrine factors, including glutamate and epidermal growth factor (EGF), are potent inducers of brain tumor cell invasion, a pathological... 
xCT | glioma | epidermal growth factor receptor | GluN2B | glutamate | MIGRATION | CELLS | TYROSINE PHOSPHORYLATION | NMDA RECEPTORS | EGFR | INVASION | ONCOLOGY | BIOLOGY | DEFICIENT | EXPRESSION | MODULATION | Neoplasm Transplantation | Phosphorylation | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Sulfasalazine - pharmacology | Brain Neoplasms - metabolism | Glioma - metabolism | Amino Acid Transport System y+ - metabolism | Receptors, N-Methyl-D-Aspartate - chemistry | Dizocilpine Maleate - administration & dosage | Protein Domains | Cell Survival - drug effects | ErbB Receptors - metabolism | Brain Neoplasms - drug therapy | Disease Progression | Drug Synergism | Sulfasalazine - administration & dosage | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Glutamic Acid - metabolism | Mice | Epidermal Growth Factor - pharmacology | Dizocilpine Maleate - pharmacology | Glioma - drug therapy | Methyl aspartate | Epidermal growth factor | Gliomas | Brain tumors | Development and progression | Aspartate | Glutamate | Immunoglobulins | Cell survival | Epidermal growth factor receptors | Paracrine signalling | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Kinases | Cell adhesion & migration | Glioma cells | Conflicts of interest | Sulfasalazine | Autocrine signalling | Cell migration | Tumors | Original
Journal Article
Cancer Science, ISSN 1347-9032, 11/2012, Volume 103, Issue 11, pp. 1946 - 1954
Somatic mutations in the epidermal growth factor receptor (EGFR) gene, such as exon 19 deletion mutations, are important factors in determining therapeutic... 
TYROSINE KINASE INHIBITORS | NONSMALL CELL | OSTEOPONTIN | GENE | ONCOLOGY | PATHWAY | ADENOCARCINOMA | EXPRESSION | ACTIVATING MUTATIONS | EGFR | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Cell Hypoxia - physiology | Humans | Lung Neoplasms - metabolism | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Proto-Oncogene Proteins c-akt - genetics | Phosphorylation - genetics | Receptor, Epidermal Growth Factor - metabolism | MAP Kinase Signaling System - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Transforming Growth Factor alpha - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Up-Regulation - genetics | Transforming Growth Factor alpha - genetics | Up-Regulation - drug effects | MAP Kinase Signaling System - drug effects | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | Epidermal growth factor | Lung cancer, Non-small cell | Transforming growth factors | Gefitinib | Genes
Journal Article
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 04/2019, Volume 110, Issue 4, pp. 1464 - 1479
Journal Article
Cancer Letters, ISSN 0304-3835, 2007, Volume 252, Issue 2, pp. 225 - 234
Journal Article