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Nephrology, ISSN 1320-5358, 08/2015, Volume 20, Issue 8, pp. 582 - 583
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 01/2014, Volume 466, Issue 1, p. 61
Issue Title: Sodium-dependent transporters in health and disease The SLC9 gene family encodes Na^sup +^/H^sup +^ exchangers (NHEs). These transmembrane... 
Journal Article
Pflugers Archiv: European Journal of Physiology, ISSN 0031-6768, 01/2014, Volume 466, Issue 1, p. 61
The SLC9 gene family encodes Na.sup.+/H.sup.+ exchangers (NHEs). These transmembrane proteins transport ions across lipid bilayers in a diverse array of... 
Hypertension | Nervous system diseases | Membrane proteins
Journal Article
Canadian Journal of Kidney Health and Disease, ISSN 2054-3581, 7/2018, Volume 5, p. 2054358118787480
Background: Thiazide diuretics are commonly prescribed to prevent kidney stones. However, it is unclear whether higher doses confer greater benefit. Objective:... 
Diuretics | Cohort analysis | Regression analysis | Drug dosages | Older people | Kidney stones
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2013, Volume 110, Issue 24, p. 1004
  NHA2 is a sodium/hydrogen exchanger with unknown physiological function. Here we show that NHA2 is present in rodent and human β-cells, as well as β-cell... 
Sodium | Hydrogen | Rodents | Homeostasis | Insulin | Cells
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 1/2014, Volume 466, Issue 1, pp. 61 - 76
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2013, Volume 123, Issue 12, pp. 5052 - 5060
Children conceived by assisted reproductive technologies (ART) display a level of vascular dysfunction similar to that seen in children of mothers with... 
PATERNAL METHYLATION | PULMONARY | MEDICINE, RESEARCH & EXPERIMENTAL | EPIGENETIC INHERITANCE | DNA METHYLATION | NITRIC-OXIDE SYNTHASE | INSULIN-RESISTANCE | MOUSE | DISEASE | IMPRINTED GENE | ASSISTED REPRODUCTIVE TECHNOLOGIES | Butyrates - pharmacology | Nitric Oxide Synthase Type III - biosynthesis | Male | Vascular Resistance - physiology | Hypertension - etiology | DNA Methylation | Cardiovascular Abnormalities - etiology | Gene Expression Regulation, Developmental | Butyrates - therapeutic use | Fertilization in Vitro - methods | Female | Nitric Oxide Synthase Type III - physiology | Vasodilation - physiology | Aorta - enzymology | Ovulation Induction - adverse effects | Endothelium, Vascular - embryology | Fertilization in Vitro - adverse effects | Promoter Regions, Genetic | Disease Susceptibility | Down-Regulation | Endothelium, Vascular - physiopathology | Cardiovascular Abnormalities - embryology | Longevity | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Gene Expression Regulation, Enzymologic | Diet, Atherogenic | Animals | Hypertension - embryology | Histone Deacetylase Inhibitors - pharmacology | Histone Deacetylase Inhibitors - therapeutic use | Mice | Nitric Oxide - metabolism | Infants (Newborn) | Blood circulation disorders | Research | Risk factors | Reproductive technology | Diseases | Studies | Blood pressure | Rodents | Metabolic disorders
Journal Article
Kidney International, ISSN 0085-2538, 04/2018, Volume 93, Issue 4, pp. 871 - 880
In the kidney, final urinary acidification is achieved by V-ATPases expressed in type A intercalated cells. The B1 subunit of the V-ATPase is required for... 
renal tubular acidosis | distal tubule | urinary exosome | BASE TRANSPORT | SJOGRENS-SYNDROME | ACIDIFICATION | VACUOLAR H+-ATPASE | COLLECTING DUCT | UROLOGY & NEPHROLOGY | INTERCALATED CELLS | B1 SUBUNIT | STONE FORMERS | KIDNEY | AMMONIUM-CHLORIDE | Acidosis, Renal Tubular - enzymology | Exosomes - drug effects | Kidney Tubules - physiopathology | Vacuolar Proton-Translocating ATPases - genetics | Saccharomyces cerevisiae - genetics | Humans | Middle Aged | Acidosis, Renal Tubular - genetics | Male | Water-Electrolyte Balance - drug effects | Vacuolar Proton-Translocating ATPases - metabolism | Young Adult | Adult | Vacuolar Proton-Translocating ATPases - urine | Kidney Tubules - drug effects | Acidosis, Renal Tubular - urine | Ammonium Chloride - administration & dosage | Mice, Knockout | Animals | Bicarbonates - administration & dosage | Kidney Tubules - enzymology | Exosomes - enzymology | Saccharomyces cerevisiae - enzymology | Acidosis, Renal Tubular - physiopathology | Mutation | Hydrogen-Ion Concentration | Urine | H+-transporting ATPase | Plasma | Antigens | Ammonium | Excretion | Immunoglobulins | Kidneys | Acidification | Homeostasis | Metabolism | Exosomes | pH effects | Patients | Renal tubular acidosis | Ammonium chloride | Lysates | Acids | Sodium bicarbonate | Physiology | Acidosis | Adenosine triphosphatase | Methods
Journal Article
Scientific reports, 02/2018, Volume 8, Issue 1, p. 3590
Fibroblast growth factor 23 (FGF23) participates in the orchestration of mineral metabolism by inducing phosphaturia and decreasing the production of 1,25(OH)... 
Journal Article
Advances in Chronic Kidney Disease, ISSN 1548-5595, 07/2018, Volume 25, Issue 4, pp. 366 - 374
Renal tubular acidosis (RTA) is comprised of a diverse group of congenital or acquired diseases with the common denominator of defective renal acid excretion... 
Acidification test | Renal tubular acidosis | Alkali therapy | Kidney stones | CALCIUM NEPHROLITHIASIS | AUTOSOMAL RECESSIVE SYNDROME | HUMAN URINARY EXOSOMES | RISK-FACTORS | SJOGRENS-SYNDROME | ANHYDRASE-II DEFICIENCY | HYPERCHLOREMIC METABOLIC-ACIDOSIS | ADD-ON DRUG | POTASSIUM CITRATE | MEDULLARY SPONGE KIDNEY | UROLOGY & NEPHROLOGY
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 03/2019, Volume 294, Issue 20, pp. 8046 - 8063
The transport and ion-coupling mechanisms of ZIP transporters remain largely uncharacterized. Previous work in our laboratory has revealed that solute carrier... 
site-directed mutagenesis | ZIP | PROTEIN | RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | structure-function | SLC | MULTIPLE SEQUENCE ALIGNMENT | FAMILY | homology modeling | ROLES | PROSTATE-CANCER | membrane transport | metal homeostasis | zinc | EXPRESSION | Membrane Biology | structure–function
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 05/2016, Volume 27, Issue 5, pp. 1544 - 1554
Mutations in the vacuolar-type H(+)-ATPase B1 subunit gene ATP6V1B1 cause autosomal-recessive distal renal tubular acidosis (dRTA). We previously identified a... 
Urine | Recurrence | Vacuolar Proton-Translocating ATPases - genetics | Humans | Middle Aged | Adult | Female | Male | Kidney Calculi - metabolism | Kidney Calculi - genetics | Hydrogen-Ion Concentration | Polymorphism, Genetic | human genetics | Clinical Research | renal tubular acidosis | kidney stones
Journal Article