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1. Histological typing of tumours of the exocrine pancreas
by Gibson, J. B. (James Blackburn) and Klöppel, Günter and World Health Organization
1996, 2nd ed., International histological classification of tumours / World Health Organization, ISBN 9783540602804, x, 61
pathology | Pancreas | Pancreatic Neoplasms | Classification | Tumors
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2. Full Text Classification and pathology of gastroenteropancreatic neuroendocrine neoplasms
by Klöppel, Günter
Endocrine-Related Cancer, ISSN 1351-0088, 10/2011, Volume 18, Issue 1, pp. S1 - S16
Gastroenteropancreatic neuroendocrine neoplasms (GEP-NENs) are composed of cells with a neuroendocrine phenotype. The old and the new WHO classifications... 
HORMONE-RELEASING-FACTOR | ONCOLOGY | IMMUNOHISTOCHEMICAL ANALYSIS | LONG-TERM SURVIVAL | GASTROINTESTINAL-TRACT | ENDOCRINOLOGY & METABOLISM | PANCREATIC-ENDOCRINE-TUMORS | ZOLLINGER-ELLISON SYNDROME | MULTIPLE GASTRIC CARCINOIDS | STATISTICAL EVALUATION | SMALL-CELL CARCINOMAS | CLINICOPATHOLOGICAL FEATURES | Neuroendocrine Tumors - pathology | Gastrointestinal Neoplasms - classification | Gastrointestinal Neoplasms - pathology | Neuroendocrine Tumors - classification | World Health Organization | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - classification | Neuroendocrine Tumors - therapy | Gastrointestinal Neoplasms - therapy | Pancreatic Neoplasms - therapy
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3. Histological typing of endocrine tumours
by Solcia, Enrico and Klöppel, Günter and Sobin, L. H
2000, 2nd ed., International histological classification of tumours, ISBN 9783540661696, viii, 160
Endocrine glands | Classification | Tumors
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4. Full Text The Gastroenteropancreatic Neuroendocrine Cell System and Its Tumors: The WHO Classification
by KLÖPPEL, GÜNTER and PERREN, AUREL and HEITZ, PHILIPP U
Annals of the New York Academy of Sciences, ISSN 0077-8923, 04/2004, Volume 1014, Issue 1, pp. 13 - 27
: Although well established in medical terminology, the term carcinoid is no longer adequate to cover the entire morphological and biological spectrum of... 
diagnosis | classification | prognosis | neuroendocrine tumors | carcinoids | Carcinoids | Prognosis | Diagnosis | Classification | Neuroendocrine tumors | Neuroendocrine Tumors - pathology | Gastrointestinal Neoplasms - classification | Gastrointestinal Neoplasms - pathology | Neuroendocrine Tumors - classification | World Health Organization | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - classification
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5. Full Text Frequency of K-ras Mutations in Pancreatic Intraductal Neoplasias Associated with Pancreatic Ductal Adenocarcinoma and Chronic Pancreatitis: A Meta-Analysis
by Löhr, Matthias and Klöppel, Gunter and Maisonneuve, Patrick and Lowenfels, Albert B and Lüttges, Jutta
Neoplasia, ISSN 1476-5586, 01/2005, Volume 7, Issue 1, pp. 17 - 23
Molecular analyses have demonstrated mutations in the K-ras gene at codon 12 in the majority of pancreatic ductal adenocarcinomas (PDACs). In order to... 
meta-analysis | chronic pancreatitis | pancreatic intraepithelial neoplasia | pancreatic ductal carcinoma | K-ras mutation | Pancreatic ductal carcinoma | Pancreatic intraepithelial neoplasia | Chronic pancreatitis | Meta-analysis | Pancreatitis - genetics | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Adenocarcinoma - genetics | Pancreatic Ducts - pathology | Mutation - genetics | Carcinoma, Pancreatic Ductal - genetics | Genes, ras - genetics
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6. Full Text Chronic pancreatitis, pseudotumors and other tumor-like lesions
by Klöppel, Günter
Modern Pathology, ISSN 0893-3952, 02/2007, Volume 20, Issue 1, pp. S113 - S131
Chronic pancreatitis is a fibroinflammatory disease of the pancreas. Etiologically, most cases are related to alcohol abuse and smoking. Recently, gene... 
Types | Histopathology | Etiology | Pathogenesis | Chronic pancreatitis | LONG-TERM | types | AUTOIMMUNE-RELATED PANCREATITIS | chronic pancreatitis | GROOVE PANCREATITIS | ALCOHOLIC CHRONIC-PANCREATITIS | LYMPHOPLASMACYTIC SCLEROSING PANCREATITIS | histopathology | PATHOLOGY | NATURAL-HISTORY | etiology | pathogenesis | RETROPERITONEAL FIBROSIS | INFLAMMATORY MYOFIBROBLASTIC TUMOR | DESTRUCTIVE CHRONIC-PANCREATITIS | STEROID-THERAPY | Pancreatitis, Chronic - pathology | Pancreatitis, Chronic - etiology | Duodenal Diseases - pathology | Genetic Predisposition to Disease | Autoimmune Diseases | Humans | Choristoma | Fibrosis | Mutation | Pancreas | Pancreatitis, Chronic - classification
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7. Full Text StellaTUM: current consensus and discussion on pancreatic stellate cell research
by Erkan, Mert and Adler, Guido and Apte, Minoti V and Bachem, Max G and Buchholz, Malte and Detlefsen, Sönke and Esposito, Irene and Friess, Helmut and Gress, Thomas M and Habisch, Hans-Joerg and Hwang, Rosa F and Jaster, Robert and Kleeff, Jörg and Klöppel, Günter and Kordes, Claus and Logsdon, Craig D and Masamune, Atsushi and Michalski, Christoph W and Oh, Junseo and Phillips, Phoebe A and Pinzani, Massimo and Reiser-Erkan, Carolin and Tsukamoto, Hidekazu and Wilson, Jeremy
Gut, ISSN 0017-5749, 02/2012, Volume 61, Issue 2, pp. 172 - 178
  In 2006, it was reported that BM is a source of myofibroblast-like cells in fibrotic liver tissue, but the involvement of these cells in the progression of... 
FAT-STORING CELLS | PROGENITOR CELLS | CANCER CELLS | DUCTAL ADENOCARCINOMA | ALCOHOLIC CHRONIC-PANCREATITIS | MOUSE | RATS | LIVER DEVELOPMENT | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | INDUCE FIBROSIS | Liver Regeneration | Pancreatic Diseases - metabolism | Pancreatic Neoplasms - metabolism | Signal Transduction | Humans | Pancreas - cytology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Stellate Cells - physiology | Pancreatic Diseases - pathology | Pancreatitis, Chronic - pathology | Regeneration | Pancreatic Stellate Cells - cytology | Fibrosis | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Pancreas - physiology | Pancreatic Stellate Cells - pathology | Pancreatitis, Chronic - metabolism | Studies | Rodents | Morphology | Pancreatic cancer | Fibroblasts | Research | Gene expression | molecular biology | chronic pancreatitis | experimental pancreatitis | stellate cells | pancreatic cancer | cancer genetics | Helicobacter pylori | pancreas | cell migration | hepatic surgery | pancreatic surgery | pancreatic physiology | hepatic stellate cell | signalling | Leading | Abdominal surgery | alcohol | pancreatic stellate cells | stem cells | liver | signal transduction | pancreatic pathology | adenocarcinoma | endoscopy | pancreatic enzymes | consensus | pancreatic function | cell biology | pancreatic fibrosis | marker | acute pancreatitis | cancer | fibrosis | pancreatitis | adjuvant treatment | pancreatic disease | hepatic fibrosis | 1506 | extracellular matrix | gene expression
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8. Full Text Correction to: Ki67 labeling index: assessment and prognostic role in gastroenteropancreatic neuroendocrine neoplasms (Virchows Archiv, (2018), 472, 3, (341-349), 10.1007/s00428-017-2258-0)
by Klöppel, Günter and La Rosa, Stefano
Virchows Archiv, ISSN 0945-6317, 03/2018, Volume 472, Issue 3, p. 515
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9. Full Text A common classification framework for neuroendocrine neoplasms: an International Agency for Research on Cancer (IARC) and World Health Organization (WHO) expert consensus proposal
by Rindi, Guido and Klimstra, David S and Abedi-Ardekani, Behnoush and Asa, Sylvia L and Bosman, Frederik T and Brambilla, Elisabeth and Krieken, J.H. van and Cree, I.A
Modern Pathology, ISSN 0893-3952, 2018, Volume 31, Issue 12, pp. 1770 - 1786
The classification of neuroendocrine neoplasms (NENs) differs between organ systems and currently causes considerable confusion. A uniform classification... 
GENOMIC LANDSCAPE | AGGRESSIVE PITUITARY-TUMORS | HIGH-GRADE | PROSTATE-CANCER | SMALL-CELL CARCINOMA | SMALL-INTESTINE | MEDULLARY-THYROID CARCINOMA | PROGNOSTIC-SIGNIFICANCE | PATHOLOGY | KI-67 PROLIFERATIVE INDEX | EUROPEAN-SOCIETY | Nets | Taxonomy | Classification | Neuroendocrine tumors | Tumors | Cancer
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10. Full Text Notch2 is required for progression of pancreatic intraepithelial neoplasia and development of pancreatic ductal adenocarcinoma
by Pawel K. Mazur and Henrik Einwächter and Marcel Lee and Bence Sipos and Hassan Nakhai and Roland Rad and Ursula Zimber-Strobl and Lothar J. Strobl and Freddy Radtke and Günter Klöppel and Roland M. Schmid and Jens T. Siveke and Elliott Kieff
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 30, pp. 13438 - 13443
Pancreatic cancer is one of the most fatal malignancies lacking effective therapies. Notch signaling is a key regulator of cell fate specification and... 
Acinar cells | Developmental biology | Cell lines | Pancreatic cells | Mice | Lesions | Carcinogenesis | Pancreatic neoplasms | Tumors | Notch receptors | K-Ras | Genetically engineered mice | Myc | Pancreatic cancer | Notch | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MOUSE | C-MYC | ADULT MICE | CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | pancreatic cancer | SIGNALING PATHWAY | GROWTH | ACINAR-CELLS | EXPRESSION | genetically engineered mice | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Male | Receptor, Notch2 - genetics | Gene Expression Profiling | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | Carcinoma in Situ - genetics | Female | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Signal Transduction | Carcinoma in Situ - pathology | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Receptor, Notch2 - metabolism | Pancreas - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Pancreatic Ductal - pathology | Proto-Oncogene Proteins c-myc - metabolism | Blotting, Western | Disease Progression | Mice, Knockout | Animals | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics | Carcinoma in Situ - metabolism | Receptor, Notch1 - genetics | Biological Sciences
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11. Full Text Stat3/Socs3 Activation by IL-6 Transsignaling Promotes Progression of Pancreatic Intraepithelial Neoplasia and Development of Pancreatic Cancer
by Lesina, Marina and Kurkowski, Magdalena U and Ludes, Katharina and Rose-John, Stefan and Treiber, Matthias and Klöppel, Günter and Yoshimura, Akihiko and Reindl, Wolfgang and Sipos, Bence and Akira, Shizuo and Schmid, Roland M and Algül, Hana
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 4, pp. 456 - 469
Physiological levels of are sufficient to induce pancreatic intraepithelial neoplasias (PanINs); the mechanisms that drive PanIN progression are unknown. Here,... 
KAPPA-B ACTIVATION | K-RAS | ONCOLOGY | DUCTAL ADENOCARCINOMA | SMALL-MOLECULE | CYCLIN D1 | STAT3 PHOSPHORYLATION | CELL-LINES | EXPRESSION | GROWTH-SUPPRESSIVE ACTIVITY | SIGNAL TRANSDUCER | CELL BIOLOGY | Signal Transduction | Pancreatic Neoplasms - etiology | Proto-Oncogene Proteins p21(ras) - genetics | Adenocarcinoma - etiology | Humans | Mice, Inbred C57BL | Middle Aged | Male | Disease Progression | Suppressor of Cytokine Signaling 3 Protein | Animals | STAT3 Transcription Factor - physiology | Interleukin-6 - blood | Interleukin-6 - physiology | Aged, 80 and over | Adult | Female | Suppressor of Cytokine Signaling Proteins - physiology | Aged | Mice | Carcinoma, Pancreatic Ductal - etiology | Genes, ras | Development and progression | Pancreatic cancer
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12. Pancreatic pathology
by Heitz, Philipp U and Klöppel, Günter
1984, ISBN 0443024367, 239
Pancreatic diseases | Histology, Pathological | Pancreas | Diseases
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13. Full Text ENETS Consensus Guidelines for the Management of Patients with Digestive Neuroendocrine Neoplasms of the Digestive System: Well-Differentiated Pancreatic Non-Functioning Tumors
by Falconi, Massimo and Bartsch, Detlef Klaus and Eriksson, Barbro and Klöppel, Günter and Lopes, José M and O'Connor, Juan M and Salazar, Ramón and Taal, Babs G and Vullierme, Marie Pierre and O'Toole, Dermot and Barcelona Consensus Conference and Barcelona Consensus Conference participants and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för medicinska vetenskaper
Neuroendocrinology, ISSN 0028-3835, 02/2012, Volume 95, Issue 2, pp. 120 - 134
  No abstract available Copyright © 2010 S. Karger AG, Basel [PUBLICATION ABSTRACT] 
ENETS Consensus Guidelines | CONTRAST-ENHANCED ULTRASONOGRAPHY | POSITRON-EMISSION-TOMOGRAPHY | FINE-NEEDLE-ASPIRATION | LONG-TERM SURVIVAL | NEURON-SPECIFIC ENOLASE | MEAL STIMULATION TEST | HIPPEL-LINDAU-DISEASE | ENDOCRINOLOGY & METABOLISM | ISLET-CELL TUMORS | SOMATOSTATIN RECEPTOR SCINTIGRAPHY | RADIOFREQUENCY THERMAL ABLATION | NEUROSCIENCES | Pancreatic Neoplasms - epidemiology | Pancreatic Neoplasms - diagnosis | Humans | Neuroendocrine Tumors - diagnosis | Neuroendocrine Tumors - epidemiology | Neuroendocrine Tumors - therapy | Pancreatic Neoplasms - therapy | Pancreas | Tumors | Medical and Health Sciences | Medicin och hälsovetenskap
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