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American journal of respiratory cell and molecular biology, 01/2019, Volume 60, Issue 6, p. 613
There is increasing evidence that autoimmunity plays an important role in the progression of chronic lung diseases such as chronic obstructive pulmonary... 
Immunoglobulin M | Antigens | Immunoglobulins | Autoantibodies | Transplants & implants | Syngeneic grafts | Lung diseases | Immunoglobulin G | Lung transplantation | Lymphocytes T | Smoke | Studies | Pulmonary fibrosis | Immunology | Ischemia | Obstructive lung disease | Fibrosis | Chronic obstructive pulmonary disease | Autoimmune diseases | Emphysema | Injuries
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 12/2018, Volume 198, Issue 12, pp. 1465 - 1466
In this issue of the Journal, Wang and colleagues (pp. 1500-1518) describe a mediator, ADAM9 (a disintegrin and metalloprotease domain-9), that was previously... 
RESPIRATORY SYSTEM | CRITICAL CARE MEDICINE | Enzymes | Airway management | Lungs | Lung diseases | Extracellular matrix | Tumor necrosis factor-TNF | Bronchitis | Inflammation | Chronic obstructive pulmonary disease | Growth factors | Emphysema | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2016, Volume 311, Issue 4, pp. L714 - L718
Journal Article
Journal Article
Science, ISSN 0036-8075, 10/2010, Volume 330, Issue 6000, pp. 90 - 94
Leukotriene A₄ hydrolase (LTA₄H) is a proinflammatory enzyme that generates the inflammatory mediator leukotriene B₄ (LTB₄). LTA₄H also possesses... 
Enzymes | Lungs | Leukotrienes | REPORTS | Neutrophils | Collagens | Mass spectroscopy | Infections | Inflammation | Chronic obstructive pulmonary disease | Cigarettes | Chemotaxis, Leukocyte | Pneumococcal Infections - metabolism | Pneumonia - therapy | Proline - metabolism | Humans | Pneumonia - pathology | Pulmonary Disease, Chronic Obstructive - pathology | Smoke | Epoxide Hydrolases - isolation & purification | Pneumococcal Infections - immunology | Leukotriene B4 - metabolism | Pneumonia - immunology | Female | Lung - metabolism | Acetylation | Pulmonary Disease, Chronic Obstructive - metabolism | Lung - pathology | Neutrophils - enzymology | Proline - analogs & derivatives | Orthomyxoviridae Infections - metabolism | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Cells, Cultured | Neutrophils - immunology | Neutrophils - physiology | Pneumococcal Infections - pathology | Oligopeptides - metabolism | Tobacco | Animals | Chemokines, CXC - metabolism | Mice | Mice, Inbred BALB C | Bronchoalveolar Lavage Fluid - chemistry | Epoxide Hydrolases - metabolism | Pneumonia - metabolism | Pulmonary Disease, Chronic Obstructive - immunology | Epoxide Hydrolases - antagonists & inhibitors | Lung - immunology | Orthomyxoviridae Infections - immunology | Leucocytes | Cellular biology | Inflammatory diseases | Immune system | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, pp. e15781 - e15781
Background: Chronic neutrophilic inflammation is a poorly understood feature in a variety of diseases with notable worldwide morbidity and mortality. We have... 
SIGNALING PATHWAYS | CHEMOATTRACTANT | GELATINASE-B | BIOLOGY | P38 MAPK | MATRIX-METALLOPROTEINASE-9 | SMOOTH-MUSCLE-CELLS | CYSTIC-FIBROSIS | TNF-ALPHA | KAPPA-B | PROTEIN-KINASES | Neutrophil Activation | Proline - analogs & derivatives | Proline - biosynthesis | Humans | Inflammation - etiology | MAP Kinase Signaling System | Oligopeptides - biosynthesis | Proline - physiology | Collagen - metabolism | Feedback, Physiological | Matrix Metalloproteinase 9 - metabolism | Oligopeptides - physiology | Receptors, Interleukin-8A - metabolism | Receptors, Interleukin-8B - metabolism | Chemotactic Factors - physiology | Chronic Disease | Neutrophils - metabolism | Neutrophils - pathology | Collagen | Mortality | Hostages | Liquid chromatography | Inflammation | Glycine | Mass spectrometry | Health aspects | Disease | Pathogenesis | Critical care | Intracellular signalling | Activation | Arthritis | Matrix metalloproteinase | Kinases | Proteins | Signal transduction | Receptors | Granulocytes | Breakdowns | Proteolysis | Extracellular matrix | Chronic obstructive pulmonary disease | Metalloproteinase | Antigens | Cytokines | CXCR2 protein | Neutrophils | Extracellular signal-regulated kinase | MAP kinase | Cystic fibrosis | Mass spectroscopy | Leukocytes (neutrophilic) | Morbidity | Gelatinase B | Medicine | Inhibitors | Correlation analysis | Chemokines | Index Medicus
Journal Article
Journal Article
Annals of the American Thoracic Society, ISSN 2325-6621, 04/2016, Volume 13, Issue Suppl 2, pp. S123 - S129
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2016, Volume 311, Issue 5, pp. L868 - L880
Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are diseases with high mortality. Macrophages and neutrophils are responsible for... 
Acute lung injury | Suppressor of cytokine signaling- 3 | STAT3 inhibitor | JAK/STAT pathway | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ACTIVATION | PHYSIOLOGY | acute lung injury | RESPIRATORY-DISTRESS-SYNDROME | SYSTEMIC INFLAMMATION | suppressor of cytokine signaling-3 | SIGNALING PATHWAY | RESPIRATORY SYSTEM | IN-VIVO | GENE-EXPRESSION | ALVEOLAR MACROPHAGES | NF-KAPPA-B | INNATE IMMUNITY | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Humans | Pneumonia - pathology | Lipopolysaccharides | Respiratory Distress Syndrome, Adult - blood | Acute Lung Injury - prevention & control | Gene Deletion | Inflammation Mediators - metabolism | Integrases - metabolism | Myeloid Cells - drug effects | Acute Lung Injury - metabolism | Bronchoalveolar Lavage Fluid - cytology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Pneumonia - genetics | Cell Separation | Mice, Inbred C57BL | Sulfonamides - pharmacology | Acute Lung Injury - pathology | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Acute Lung Injury - chemically induced | Inflammation - genetics | Myeloid Cells - metabolism | Macrophages - drug effects | Anthraquinones - pharmacology | Chemokines - metabolism | Suppressor of Cytokine Signaling 3 Protein - metabolism | Acute respiratory distress syndrome | Prevention | Causes of | Health aspects | Plasma | Molecules | Phosphorylation | Respiratory distress syndrome | Mortality | Genes | Cells | Index Medicus | Call for Papers | STAT pathway | suppressor of cytokine signaling-3, STAT3 inhibitor | JAK
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 2, pp. 413 - 424.e15
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 10/2015, Volume 192, Issue 8, pp. 934 - 942
Journal Article
Journal Article