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Apoptosis - An International Journal on Programmed Cell Death, ISSN 1360-8185, 11/2017, Volume 22, Issue 11, p. 1344
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s10495-017-1415-x The... 
Prevention | Proteins | Phosphates | Tyrosine | Colon cancer | Cell death | Phospholipids | Biochemistry | B cells
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 11/2018, Volume 217, Issue 11, p. 3839
Finely tuned regulation of epithelial cell death maintains tissue integrity and homeostasis. At the cellular level, life and death decisions are controlled by... 
Tissue engineering | Epithelial cells | Mortality | Homeostasis | E-cadherin | Cells | Environmental effects | Signal transduction | Signaling | Receptors | FasL protein | Cell death | Actin | Polarity | Death receptors | Binding sites | Apoptosis
Journal Article
Apoptosis, ISSN 1360-8185, 11/2017, Volume 22, Issue 11, pp. 1344 - 1352
The Fas/FasL system plays a critical role in death by apoptosis and immune escape of cancer cells. The Fas receptor being ubiquitously expressed in tissues,... 
Biochemistry, general | Biomedicine | Cell death | Btk | Cancer Research | Oncology | Fas | PI(4,5)P 2 | PIP5K1γ | Cell Biology | Virology | PI(4,5)P | TRANSFORMATION | APOPTOSIS | PROTEIN | CANCER CELLS | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PI(4,5)P-2 | RECEPTOR | SIGNALING COMPLEX | CELL BIOLOGY | IMMUNE-SYSTEM | INHIBITION | PIP5K1 gamma | BRUTONS-TYROSINE-KINASE | RNA, Small Interfering - genetics | Fas Ligand Protein - metabolism | Epithelial Cells - metabolism | Killer Cells, Natural | Protein-Tyrosine Kinases - metabolism | Epithelial Cells - drug effects | Humans | fas Receptor - metabolism | Protein-Tyrosine Kinases - genetics | Fas Ligand Protein - pharmacology | Cell Death - genetics | fas Receptor - genetics | Cell Death - drug effects | Epithelial Cells - cytology | Lymphocytes - metabolism | Cell Line | Signal Transduction | Gene Expression Regulation | Phosphotransferases (Alcohol Group Acceptor) - genetics | Lymphocytes - cytology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | HT29 Cells | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Lymphocytes - drug effects | Cell Line, Tumor | Fas Ligand Protein - genetics | Phosphatidylinositol 4,5-Diphosphate - biosynthesis | Protein-Tyrosine Kinases - antagonists & inhibitors | RNA, Small Interfering - metabolism | Tyrosine | Phosphates | Biotechnology | Epithelial cells | Mortality | Phosphatidylinositol 4,5-diphosphate | Tumor cell lines | Kinases | Tissues | Bruton's tyrosine kinase | 1-Phosphatidylinositol-4-phosphate 5-kinase | Proteins | Escape systems | Colon cancer | FasL protein | Lymphocytes B | Regulatory mechanisms (biology) | Protein-tyrosine kinase receptors | Lipid kinase | Colon | Protein-tyrosine kinase | Cancer | Apoptosis
Journal Article
The Journal of cell biology, ISSN 0021-9525, 11/2018, Volume 217, Issue 11, pp. 3839 - 3852
Finely tuned regulation of epithelial cell death maintains tissue integrity and homeostasis. At the cellular level, life and death decisions are controlled by... 
CADHERIN | ADHESION | APOPTOSIS | ACTIVATION | COMPLEX | PROTEIN | CYTOSKELETON | ALPHA-CATENIN | MECHANISMS | EXPRESSION | CELL BIOLOGY
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 9/2003, Volume 162, Issue 7, pp. 1189 - 1196
Integrin α6β4 signaling proceeds through Src family kinase (SFK)-mediated phosphorylation of the cytoplasmic tail of β4, recruitment of Shc, and activation of... 
Phosphorylation | Ligation | Rafts | Hemidesmosomes | Keratinocytes | Cell biology | Antibodies | Lipids | Reports | Cells | Integrins | Keratinocyte | Cysteine | Proliferation | Palmitoylation | Hemidesmosome | Report | keratinocyte; proliferation; palmitoylation; hemidesmosome; cysteine
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 5/2001, Volume 153, Issue 4, pp. 835 - 849
Laminin 5 is a basement membrane component that actively promotes adhesion and migration of epithelial cells. Laminin 5 undergoes extracellular proteolysis of... 
Cell culture techniques | Molecules | Complementary DNA | Plasmids | Epithelial cells | Cell lines | Cell adhesion | Keratinocytes | Cells | Basement membrane | Epithelial adhesion | Keratinocyte | Epidermolysis bullosa | Cell migration
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2012, Volume 125, Issue 18, pp. 4241 - 4252
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 10/2001, Volume 155, Issue 3, pp. 447 - 457
We have examined the mechanism and functional significance of hemidesmosome disassembly during normal epithelial cell migration and squamous carcinoma... 
T lymphocytes | Squamous cell carcinoma | Phosphorylation | Hemidesmosomes | Cell lines | Antibodies | Keratinocytes | Immunoblotting | Cells | Integrins | α6β4 | Fyn | EGF-R | Carcinoma invasion | α6β4; fyn; EGF-R; hemidesmosomes; carcinoma invasion
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 09/2003, Volume 162, Issue 7, p. 1189
  Integrin alpha6beta4 signaling proceeds through Src family kinase (SFK)-mediated phosphorylation of the cytoplasmic tail of beta4, recruitment of Shc, and... 
Cellular biology | Cell division | Lipids | Mutation
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 12/2003, Volume 121, Issue 6, pp. 1336 - 1343
Genetic mutations in α6β4 integrin cause junctional epidermolysis bullosa with pyloric atresia, a genodermatosis characterized by blistering of the skin and... 
integrin α6β4 | proteasome | skin blistering disease | β-propeller | lysosome | Integrin α6β4 | Skin blistering disease | Lysosome | Proteasome
Journal Article
The Journal of Investigative Dermatology, ISSN 0022-202X, 12/2003, Volume 121, Issue 6, p. 1336
  Genetic mutations in alpha6beta4 integrin cause junctional epidermolysis bullosa with pyloric atresia, a genodermatosis characterized by blistering of the... 
Journal Article
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