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Neoplasia, ISSN 1476-5586, 04/2018, Volume 20, Issue 4, pp. 335 - 350
Neuroblastoma is one of the commonest and deadliest solid tumours of childhood, and is thought to result from disrupted differentiation of the developing... 
SH-SY5Y CELLS | HIGH-RISK NEUROBLASTOMAS | TRANSCRIPTIONAL ACTIVATION | RETINOIC ACID | GENE | ONCOLOGY | PATHWAY | NEURONAL DIFFERENTIATION | EXPRESSION PROFILES | SCHWANN-CELLS | BETA-CATENIN
Journal Article
Cancer Letters, ISSN 0304-3835, 2017, Volume 403, pp. 74 - 85
Abstract Neuroblastoma is a biologically and clinically heterogeneous pediatric malignancy that includes a high-risk subset for which new therapeutic agents... 
Hematology, Oncology and Palliative Medicine | YK-4-279 | Chemotherapy | Drug resistance/synergy | Neuroblastoma | Mitosis | PROTEIN | KINASE | REARRANGEMENTS | LINES | TRIAL | THERAPY | ONCOLOGY | VINCRISTINE | GENES | HIGH-RISK NEUROBLASTOMA | PHASE-I | Vincristine - pharmacology | Paclitaxel - pharmacology | Apoptosis - drug effects | Drug Resistance, Multiple | Humans | Drug Resistance, Neoplasm | Aurora Kinase A - antagonists & inhibitors | Aurora Kinase A - metabolism | Tumor Suppressor Protein p53 - genetics | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Dose-Response Relationship, Drug | Transfection | RNA Interference | Time Factors | Inhibitory Concentration 50 | Prometaphase - drug effects | Indoles - pharmacology | Neuroblastoma - pathology | Neuroblastoma - genetics | Tumor Suppressor Protein p53 - metabolism | Spindle Apparatus - pathology | Pyrimidines - pharmacology | Azepines - pharmacology | Drug Synergism | Kinetochores - pathology | Mitosis - drug effects | Signal Transduction - drug effects | Neuroblastoma - drug therapy | Cell Line, Tumor | Antimitotic Agents - pharmacology | Kinetochores - drug effects | Neuroblastoma - metabolism | Protein Kinase Inhibitors - pharmacology | Spindle Apparatus - drug effects | Cell Cycle - drug effects | Cell death | Analysis | Development and progression | Lymphomas | Drug resistance | Tumor proteins | Mitogens | Cancer | Cell culture | Flow cytometry | Biotechnology | Transcription factors | Risk | Aurora kinase | Genomes | Malignancy | Kinases | Cancer therapies | Neuroblastoma cells | Proteins | Poisons | Molecules | Cell cycle | Paclitaxel | Acetylation | Drug dosages | Risk groups | Abnormalities | Extracellular signal-regulated kinase | Cell division | Pharmacology | Gene expression | Chemical compounds | Vincristine | Gene amplification | Inhibitors | Microtubules | Cell lines | Mutation | Spindles | Apoptosis | Index Medicus | TERT, Telomerase reverse transcriptase | GFP, green fluorescent protein | synergy | EGF, Epidermal growth factor | ERK, extracellular signal-regulated kinases | kDa, kilodaltons | QVD, quinolyl-valyl-O-methylaspartyl-(-2,6-difluorophenoxy)- methyl ketone | SDS-PAGE, sodium-dodecyl sulphate-polyacrylamide gel electrophoresis | RNase A, ribonuclease A | MTT, 3-(4,5-Dimethylthiazol-2-Yl)-2,5-Diphenyltetrazolium Bromide | ALK, Anaplastic Lymphoma kinase | pHH3, phospho-histone H3 (ser10) | PBS, Phosphate-buffered saline | Original | MAPK, mitogen-activated protein kinase | MEK, Mitogen-activated protein kinase kinase
Journal Article
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