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Journal of Hepatology, ISSN 0168-8278, 11/2017, Volume 67, Issue 5, pp. 1104 - 1105
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 05/2015, Volume 372, Issue 19, pp. 1791 - 1800
Patients with myocardial infarction 1 to 3 years previously were assigned to ticagrelor, 90 or 60 mg twice daily, or to placebo, in addition to low-dose... 
MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | CLOPIDOGREL | MANAGEMENT | ESC GUIDELINES | AMERICAN-COLLEGE | PREVENTION | ASSOCIATION TASK-FORCE | ACUTE CORONARY SYNDROMES | PRASUGREL | ASPIRIN | Purinergic P2Y Receptor Antagonists - administration & dosage | Double-Blind Method | Drug Administration Schedule | Cardiovascular Diseases - prevention & control | Humans | Middle Aged | Kaplan-Meier Estimate | Male | Risk | Secondary Prevention | Adenosine - administration & dosage | Aspirin - administration & dosage | Purinergic P2Y Receptor Antagonists - adverse effects | Myocardial Infarction - drug therapy | Adenosine - adverse effects | Adenosine - analogs & derivatives | Intracranial Hemorrhages - chemically induced | Platelet Aggregation Inhibitors - administration & dosage | Cardiovascular Diseases - mortality | Female | Aged | Hemorrhage - chemically induced | Drug Therapy, Combination | Platelet Aggregation Inhibitors - adverse effects | Dose-response relationship (Biochemistry) | Treatment outcome | Care and treatment | Safety and security measures | Analysis | Dosage and administration | Ticagrelor | Heart attack | Risk factors | Myocardial infarction | Cerebral infarction | Aspirin | Stroke | Heart attacks | Hemorrhage | Long term | Patients | Thrombolysis | Bleeding | Blood platelets | Antagonist drugs | Cardiovascular diseases | Drug therapy | Life Sciences | Human health and pathology
Journal Article
Fibrogenesis and Tissue Repair, ISSN 1755-1536, 07/2012, Volume 5, Issue 1, pp. 10 - 10
'Augmenter of liver regeneration' (ALR) (also known as hepatic stimulatory substance or hepatopoietin) was originally found to promote growth of hepatocytes in... 
Regeneration | Hepatocytes | Growth | Liver | ALR | GFER | Survival | Oxidases | Cytochrome c | Nitric oxide | Amino acids | DNA synthesis | Transforming growth factors | Interleukin-6 | Proteins | Pathology | Enzymes | Epidermal growth factor | Rodents | Insulin-like growth factors
Journal Article
Journal of Hepatology, ISSN 0168-8278, 11/2017, Volume 67, Issue 5, p. 1104
Regardless of etiology, hepatic stellate cells (HSCs) are the major contributors to fibrosis/cirrhosis. Bone marrow-derived fibrocytes,... 
Stellate cells | Cirrhosis | Cell activation | Liver diseases | Mesenchyme | Hepatocytes | Etiology | Contractility | Fibrosis | Fibroblasts | Bone marrow | Cells
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, p. e0147864
Journal Article
Hepatology, ISSN 0270-9139, 04/2018, Volume 67, Issue 4, pp. 1216 - 1218
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0176106
The NAD+-dependent 15-hydroxyprostaglandin dehydrogenase (15-PGDH) catalyzes the oxidation of the 15(S)-hydroxyl group of prostaglandin E-2 (PGE(2)),... 
NUCLEAR RECEPTORS | MULTIDISCIPLINARY SCIENCES | GROWTH | ACTIVATED-RECEPTOR-GAMMA | MECHANISMS | TNF-ALPHA | INFLAMMATORY RESPONSE | Tumor Necrosis Factor-alpha - metabolism | Liver - pathology | Humans | Lipopolysaccharides - administration & dosage | Tumor Necrosis Factor-alpha - genetics | Culture Media, Conditioned - pharmacology | Hepatocytes - metabolism | PPAR gamma - metabolism | Hydroxyprostaglandin Dehydrogenases - metabolism | Hepatocytes - cytology | Liver - drug effects | Kupffer Cells - metabolism | Chemical and Drug Induced Liver Injury - pathology | Transgenes | Hepatocytes - drug effects | PPAR gamma - genetics | Chemical and Drug Induced Liver Injury - prevention & control | Dinoprostone - pharmacology | Kupffer Cells - cytology | Macrophages - pathology | Paracrine Communication | Signal Transduction | Liver - metabolism | Gene Expression Regulation | Mice, Transgenic | Chemical and Drug Induced Liver Injury - genetics | Hydroxyprostaglandin Dehydrogenases - genetics | Aspartate Aminotransferases - metabolism | Kupffer Cells - drug effects | Dinoprostone - metabolism | Macrophages - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Chemical and Drug Induced Liver Injury - metabolism | Anilides - pharmacology | Dinoprostone - analogs & derivatives | Macrophages - drug effects | Mice | Alanine Transaminase - metabolism | Primary Cell Culture | Macrophage Activation - drug effects | Prevention | Care and treatment | Liver diseases | Physiological aspects | Genetic aspects | Research | Oxidoreductases | Gene expression | Medical research | Dehydrogenases | Cytokines | Tissue engineering | Laboratories | Inflammation | Macrophages | Medicine | Liver cancer | Pathology | Hepatitis | Immunology | Transgenic animals | Rodents | Ligands | Apoptosis
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 04/2006, Volume 354, Issue 16, pp. 1706 - 1717
Journal Article