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Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 16, pp. C23 - C23
Consistently, melatonin administration did not further increase autophagy, decrease apoptosis, or alleviate mitochondrial integrity and biogenesis in Mst1... 
Cardiology | Medical colleges | Melatonin | Biosynthesis | Autophagy | Heart diseases | Rodents | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 08/2018, Volume 123, Issue Suppl_1
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2016, Volume 113, Issue 52, pp. 15126 - 15131
Following cardiac injury, early immune cell responses are essential for initiating cardiac remodeling and tissue repair. We previously demonstrated the... 
C-chemokine receptor 2 | Cardiac injury | β-arrestin | Leukocyte | β2-adrenergic receptor | Index Medicus | Biological Sciences | cardiac injury | leukocyte
Journal Article
Cell Cycle, ISSN 1538-4101, 01/2012, Volume 11, Issue 14, pp. 2747 - 2755
Triple-negative breast cancer (TNBC) is an aggressive disease that lacks established markers to direct therapeutic intervention. Thus, these tumors are... 
triple-negative breast cancer | retinoblastoma tumor suppressor | anthracyclines | cyclin-dependent kinase | CDK4/6 inhibition | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Cyclin-dependent kinase | Retinoblastoma tumor suppressor | Anthracyclines | Triple-negative breast cancer | PROTECTION | DNA-DAMAGE | MECHANISMS | CHECKPOINT RESPONSE | CHEMOTHERAPY | NEGATIVE BREAST-CANCER | CELL BIOLOGY | RETINOBLASTOMA PROTEIN | ASSOCIATION | XENOGRAFTS | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | Doxorubicin - therapeutic use | Antibiotics, Antineoplastic - toxicity | Humans | Transplantation, Heterologous | Female | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Pyridines - therapeutic use | Cell Survival - drug effects | Retinoblastoma Protein - metabolism | Cyclin-Dependent Kinase 6 - metabolism | Piperazines - therapeutic use | Cyclin-Dependent Kinase 4 - metabolism | Breast Neoplasms - drug therapy | Piperazines - pharmacology | Doxorubicin - toxicity | Animals | Antibiotics, Antineoplastic - therapeutic use | Cell Cycle Checkpoints - drug effects | Mice, Nude | Protein Kinase Inhibitors - therapeutic use | Retinoblastoma Protein - genetics | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Index Medicus | Report | CDK4 | 6 inhibition
Journal Article
Life Sciences, ISSN 0024-3205, 06/2019, Volume 226, pp. 22 - 32
Estrogen plays an important role in cardioprotection. Animal experiments showed that the G-protein coupled estrogen receptor 30 (GPR30) specific agonist G1... 
Cardiac fibroblasts | Cardioprotection | G-protein coupled estrogen receptor 30 (GPR30) | Hypoxia/serum deprivation (H/SD) | Ovariectomy | Myocardial infarction (MI) | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | ESTROGEN-RECEPTOR GPER | MESENCHYMAL STEM-CELLS | ISCHEMIA-REPERFUSION INJURY | INHIBITION | INFLAMMATION | IN-VIVO | PHARMACOLOGY & PHARMACY | CARDIAC FIBROSIS | MODULATION | Quinolines | Receptors, Estrogen - metabolism | Cell Proliferation | Receptors, G-Protein-Coupled - metabolism | Signal Transduction | Heart - physiology | Mice, Inbred C57BL | Myocardial Infarction - metabolism | Receptors, G-Protein-Coupled - agonists | Fibrosis - metabolism | Phosphatidylinositol 3-Kinases | Proto-Oncogene Proteins c-akt | Animals | Benzodioxoles - pharmacokinetics | Myocardium - metabolism | Female | Mice | Disease Models, Animal | Fibroblasts - metabolism | Receptors, Estrogen - physiology | Receptors, G-Protein-Coupled - physiology | Animal experimentation | Proteins | Heart | Interleukins | Analysis | Fibrosis | Estrogen | Myocardial infarction | Deprivation | Animal models | Echocardiography | Computer simulation | Estrogens | Interleukin | AKT protein | Tumor necrosis factor-α | Western blotting | 1-Phosphatidylinositol 3-kinase | Ischemia | Morphology | Interleukin 10 | Fibroblasts | Hypoxia | In vivo methods and tests | Hemodynamics | Immunofluorescence | Apoptosis | Index Medicus
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2015, Volume 19, Issue 6, pp. 1296 - 1307
Oncostatin M ( OSM ) exhibits many unique biological activities by activating Oβ receptor. However, its role in myocardial I/R injury in diabetic mice remains... 
inositol pyrophosphate 7 | oncostatin M | diabetes | ischaemia/Reperfusion injury | Inositol pyrophosphate 7 | Ischaemia/Reperfusion injury | Diabetes | Oncostatin M | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | MYOCARDIAL-INFARCTION | MYOCYTES | MEDIATOR | Reperfusion injury | CELL BIOLOGY | HEART | SKELETAL-MUSCLE | ischaemia | INHIBITION | REDUCTION | DYSFUNCTION | TANSHINONE IIA PRETREATMENT | Inositol Phosphates - antagonists & inhibitors | Apoptosis - drug effects | Receptors, Oncostatin M - metabolism | Diabetes Mellitus, Experimental - genetics | Insulin Resistance | Apoptosis - genetics | Myocardium - pathology | Mice, 129 Strain | Oncostatin M - pharmacology | Blotting, Western | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Organelle Biogenesis | Animals | Receptors, Oncostatin M - genetics | Mice, Mutant Strains | Protective Agents - pharmacology | Myocardium - metabolism | Glucose - metabolism | Inositol Phosphates - metabolism | Myocardial Reperfusion Injury - genetics | Homeostasis - genetics | Myocardial Reperfusion Injury - prevention & control | Diabetes Mellitus, Experimental - physiopathology | Phosphates | Inositol | Biosynthesis | Glucose | Insulin | Dextrose | Apoptosis | Rodents | Cardiomyocytes | Kinases | Statins | Heart | Bax protein | Diabetes mellitus | Adenine | AKT protein | 1-Phosphatidylinositol 3-kinase | Mitochondria | Sensitivity | Reperfusion | Ischemia | Protein kinase | Insulin resistance | Mice | Inhibition | Injuries | Index Medicus | Original
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2017, Volume 114, Issue 5, pp. E859 - E868
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2018, Volume 72, Issue 16, p. C74
Journal Article
Circulation, ISSN 0009-7322, 11/2018, Volume 138, Issue Suppl_1 Suppl 1, pp. A10751 - A10751
The inflammatory response after myocardial infarction (MI) can aggravate tissue damage to progress to heart failure. Autotaxin (ATX) produces pro-inflammatory... 
Journal Article
Apoptosis, ISSN 1360-8185, 4/2014, Volume 19, Issue 4, pp. 567 - 580
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 12/2016, Volume 113, Issue 52, p. 15126
  Following cardiac injury, early immune cell responses are essential for initiating cardiac remodeling and tissue repair. We previously demonstrated the... 
Proteins | Tissue | Leucocytes | Heart attacks | Rodents | Bone marrow | Chemokines
Journal Article