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The EMBO Journal, ISSN 0261-4189, 06/2016, Volume 35, Issue 12, pp. 1251 - 1253
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2018, Volume 27, Issue 8, pp. 1382 - 1395
Journal Article
Nature, ISSN 0028-0836, 09/2015, Volume 525, Issue 7567, pp. 129 - 133
The GGGGCC (G(4)C(2)) repeat expansion in a noncoding region of C9orf72 is the most common cause of sporadic and familial forms of amyotrophic lateral... 
C9FTD/ALS | MULTIDISCIPLINARY SCIENCES | NUCLEAR-PORE COMPLEX | FRONTOTEMPORAL DEMENTIA | DISEASE | MESSENGER-RNA EXPORT | AMYOTROPHIC-LATERAL-SCLEROSIS | MUTATIONS | PROTEINS | HEXANUCLEOTIDE REPEAT | STEM-CELL MODELS | Protein Biosynthesis | Salivary Glands - pathology | DNA Repeat Expansion - genetics | Humans | Male | Neurons - cytology | Drosophila melanogaster - genetics | Salivary Glands - cytology | RNA - genetics | Drosophila melanogaster - metabolism | RNA Transport - genetics | Female | C9orf72 Protein | Neurons - metabolism | Induced Pluripotent Stem Cells - cytology | Muscles - metabolism | Active Transport, Cell Nucleus - genetics | Frontotemporal Dementia - pathology | Induced Pluripotent Stem Cells - metabolism | RNA - metabolism | Eye - metabolism | Frontotemporal Dementia - genetics | Animals, Genetically Modified | Amyotrophic Lateral Sclerosis - genetics | Nuclear Pore - pathology | Drosophila melanogaster - cytology | Nuclear Pore - genetics | Nuclear Pore - metabolism | Open Reading Frames - genetics | Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Phenotype | Animals | HeLa Cells | Salivary Glands - metabolism | Muscles - cytology | Trinucleotide repeats | Genetic research | Genetic aspects | Research | Biological transport | Cytoplasm | Proteins | Insects | Toxicity | Pathogenesis | Amyotrophic lateral sclerosis | Chromosomes | Dementia | RAN translation | genetic screen | repeat expansion | Drosophila | DPR | FTD | ALS | C9ORF72 | nuclear pore
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2018, Volume 27, Issue 8, pp. 1382 - 1395
Abstract Frontotemporal dementia (FTD) is one of the most prevalent forms of early-onset dementia. However, the pathological mechanisms driving neuronal... 
Index Medicus
Journal Article
Human molecular genetics, 04/2018, Volume 27, Issue 8, p. 1382
Frontotemporal dementia (FTD) is one of the most prevalent forms of early-onset dementia. However, the pathological mechanisms driving neuronal atrophy in FTD... 
Neurons - pathology | RNA, Small Interfering - genetics | Cytoskeletal Proteins - antagonists & inhibitors | Cytoskeletal Proteins - genetics | Vesicular Transport Proteins - metabolism | Humans | Nervous System - metabolism | Apoptosis - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Proto-Oncogene Proteins c-akt - genetics | Drosophila melanogaster - metabolism | Frontotemporal Dementia - metabolism | Nervous System - pathology | Cytoskeletal Proteins - metabolism | Larva - genetics | Neurons - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Proto-Oncogene Proteins c-akt - metabolism | Drosophila Proteins - antagonists & inhibitors | Disease Models, Animal | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Nerve Tissue Proteins - antagonists & inhibitors | Signal Transduction | Animals, Genetically Modified | Introns | Larva - metabolism | Vesicular Transport Proteins - genetics | Carrier Proteins - antagonists & inhibitors | Gene Expression Regulation | Rats | Longevity - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Mice | Drosophila Proteins - genetics | Primary Cell Culture | RNA, Small Interfering - metabolism
Journal Article