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Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 11309
.... Mechanistic analyses indicate that miR-3188 directly targets mTOR to inactivate p-PI3K/p-AKT/c-JUN and induces its own expression... 
EPITHELIAL-MESENCHYMAL TRANSITION | CANCER-CELLS | ACTIVATION | INHIBITION | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | C-MYC | TUMOR-SUPPRESSOR | CISPLATIN RESISTANCE | RECEPTOR | PROMOTES | Nasopharyngeal Neoplasms - genetics | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Prognosis | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | JNK Mitogen-Activated Protein Kinases - metabolism | Male | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Nasopharyngeal Neoplasms - pathology | TOR Serine-Threonine Kinases - genetics | Cell Transformation, Neoplastic - genetics | Forkhead Transcription Factors - metabolism | Aged, 80 and over | Adult | Female | JNK Mitogen-Activated Protein Kinases - genetics | Proto-Oncogene Proteins c-akt - metabolism | Nasopharyngeal Neoplasms - diagnosis | Nasopharyngeal Carcinoma | Signal Transduction | Forkhead Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Survival Analysis | Cell Line, Tumor | Aged | Forkhead Box Protein O1 | Mice | MicroRNAs - genetics | Cell Transformation, Neoplastic - pathology | Neoplasm Staging
Journal Article
2013, ISBN 7552901020, 1 v.
Book
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 2015, Volume 14, Issue 7, pp. 1046 - 1058
Journal Article
2013, ISBN 9787506565196, 1 v.
Book
Journal Article
Journal Article
Pain (Amsterdam), ISSN 0304-3959, 2010, Volume 148, Issue 2, pp. 309 - 319
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, p. 41795
Journal Article
Journal of virology, ISSN 0022-538X, 08/2017, Volume 91, Issue 15
Autophagy is closely associated with the regulation of hepatitis B virus (HBV) replication. HBV X protein (HBx), a multifunctional regulator in HBV-associated... 
CELLS | APOPTOSIS | autophagy | JNK | signaling pathway | HBX PROTEIN | CANCER | viral replication | INHIBITION | REPLICATION | VIROLOGY | CORE PROMOTER | UP-REGULATION | HBV X protein | MODULATION
Journal Article
Journal Article
Frontiers in oncology, ISSN 2234-943X, 2019, Volume 9, p. 33
Hypoxia is a predominant feature in glioblastoma (GBM) and contributes greatly to its drug resistance. However, the molecular mechanisms which are responsible... 
Hypoxia | CEBPB | JUN | HDAC3 | Drug resistance | Glioblastoma | hypoxia | MECHANISMS | MULTIFORME | CANCER | TEMOZOLOMIDE | glioblastoma | HISTONE DEACETYLASES | ONCOLOGY | GROWTH | GENE-EXPRESSION | drug resistance | DNA-REPAIR | BRAIN | PROGRESSION | Genetic aspects | Research | Gene expression | Glioblastoma multiforme
Journal Article