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Nature, ISSN 0028-0836, 10/2011, Volume 478, Issue 7367, pp. 114 - 118
Left ventricular mass (LVM) is a highly heritable trait(1) and an independent risk factor for all-cause mortality(2). So far, genome-wide association studies... 
PATHOGENESIS | OXIDATIVE STRESS | REPLICATION | BIOGENESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | DISEASE | DEATH | MECHANISMS | LEFT-VENTRICULAR MASS | EXPRESSION | Receptors, Estrogen - metabolism | Hypertrophy, Left Ventricular - enzymology | Reactive Oxygen Species - metabolism | Chromosomes, Mammalian - genetics | Cardiomegaly - pathology | Male | Rats, Inbred Strains | Body Weight - genetics | Hypertrophy, Left Ventricular - genetics | Cell Respiration | Mitochondria - genetics | Hypertrophy, Left Ventricular - pathology | Organ Size - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Endodeoxyribonucleases - metabolism | Female | Endodeoxyribonucleases - deficiency | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Lipid Metabolism | Mitochondria - metabolism | Cardiomegaly - enzymology | Mitochondria - pathology | Transcription Factors - metabolism | Genes, Mitochondrial - genetics | Endodeoxyribonucleases - genetics | Animals | Hypertrophy, Left Ventricular - physiopathology | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Apoptosis | Crosses, Genetic | Quantitative Trait Loci - genetics | Nucleases | Mitochondria | Heart enlargement | Physiological aspects | Genetic aspects | Research | Diagnosis | Risk factors | Studies | Rodents | Biomarkers | Genetics | Cardiomyocytes | Genomes | Blood pressure | Kinases
Journal Article
Experimental Physiology, ISSN 0958-0670, 02/2013, Volume 98, Issue 2, pp. 425 - 434
New findings •  What is the central question of this study? Ischaemia–reperfusion of peripheral tissues protects the heart from subsequent myocardial... 
INFARCT SIZE | ADENOSINE RECEPTORS | PROTEIN-KINASE-C | DISTANCE | PHYSIOLOGY | STIMULATION | MECHANISM | REPERFUSION INJURY | MYOCARDIAL-ISCHEMIA | CHANNELS | OCCLUSION | Hindlimb | Electric Stimulation | Ventricular Function, Left | Afferent Pathways - physiopathology | Femoral Nerve - surgery | Vagus Nerve - drug effects | Male | Muscle, Skeletal - innervation | Receptors, Muscarinic - metabolism | Sciatic Nerve - physiopathology | Ischemic Preconditioning - methods | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Heart - physiopathology | Rabbits | Vagus Nerve - metabolism | Atropine - pharmacology | Vagus Nerve - surgery | Myocardial Infarction - metabolism | Vagotomy | Myocardial Reperfusion Injury - physiopathology | Spinal Cord - surgery | Efferent Pathways - physiopathology | Vagus Nerve - physiopathology | Sensory Receptor Cells | Myocardial Reperfusion Injury - metabolism | Femoral Nerve - physiopathology | Animals | Sciatic Nerve - surgery | Myocardial Infarction - prevention & control | Spinal Cord - physiopathology | Ventricular Pressure | Myocardial Reperfusion Injury - prevention & control | Cardiac patients
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 05/2016, Volume 20, Issue 5, pp. 794 - 803
Connexin 43 (Cx43), the gap junction protein involved in cell‐to‐cell coupling in the heart, is also present in the subsarcolemmal fraction of cardiomyocyte... 
connexin 43 | apoptosis‐inducing factor | mitochondria | electron‐transfer protein | cardiomyocyte | Electron-transfer protein | Mitochondria | Connexin 43 | Apoptosis-inducing factor | Cardiomyocyte | CARDIOMYOCYTE MITOCHONDRIA | MEDICINE, RESEARCH & EXPERIMENTAL | PHOSPHORYLATION | CX43 | MICROSCOPY | CELL BIOLOGY | electron-transfer protein | OXYGEN | GAP-JUNCTIONS | ELECTRON-TRANSFER FLAVOPROTEIN | MICE | apoptosis-inducing factor | CONTRIBUTES | Electron-Transferring Flavoproteins - genetics | Immunoprecipitation | Mitochondria, Heart - metabolism | Connexin 43 - metabolism | Myocytes, Cardiac - cytology | Oxidation-Reduction | Signal Transduction | Gene Expression Regulation | Male | Mice, Transgenic | Electron-Transferring Flavoproteins - metabolism | Myocytes, Cardiac - chemistry | Protein Subunits - metabolism | Apoptosis Inducing Factor - genetics | Protein Interaction Mapping | Animals | Myocytes, Cardiac - metabolism | Protein Binding | Female | Mice | Mitochondria, Heart - genetics | Apoptosis Inducing Factor - metabolism | Connexin 43 - genetics | Protein Subunits - genetics | Proteins | Physiological aspects | Mitochondrial DNA | Analysis | Protein-protein interactions | Heart | Cardiomyocytes | Dehydrogenases | Kinases | Coupling (molecular) | Signal transduction | Localization | Redox properties | Communication | Enzymes | Molecular interactions | Mass spectroscopy | Metabolism | Fatty acids | Studies | Electron transport | Respiration | Laboratory animals | Protein interaction | Mass spectrometry | Preconditioning | Potassium | Apoptosis | Original
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 9/2018, Volume 113, Issue 5, pp. 1 - 73
Journal Article
The FASEB Journal, ISSN 0892-6638, 09/2017, Volume 31, Issue 9, pp. 3787 - 3799
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 07/2019, Volume 74, Issue 2, pp. 238 - 256
Journal Article
Journal Article