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Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue 19
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 03/2012, Volume 52, Issue 3, pp. 578 - 583
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A20279 - A20279
IntroductionReceptor-mediated, intracellular signaling leading to cardioprotection and inotropy is orchestrated by signalosome. The caveolar vesicles bud off... 
Journal Article
Biophysical Journal, ISSN 0006-3495, 01/2014, Volume 106, Issue 2, pp. 306a - 306a
Journal Article
Handbook of Experimental Pharmacology, ISSN 0171-2004, 2017, Volume 240, pp. 377 - 401
Understanding the complex involvement of mitochondrial biology in disease development often requires the acquisition, analysis, and integration of large-scale... 
Visualization | Computation | Omics | Data science | Metadata | FAIR data | Open access | Animals | Humans | Computational Biology | Electronic Data Processing | Physiology | Statistics as Topic | Mitochondria - physiology | Mitochondrial Diseases - physiopathology | Information Dissemination | Mitochondrial Diseases - etiology
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2008, Volume 295, Issue 2, pp. 874 - 882
Activation of protein kinase Cε (PKCε), opening of mitochondrial ATP-sensitive K+ channels (mitoKATP), and increased mitochondrial reactive oxygen species... 
Signaling pathways | channel | Mitochondrial ATP-sensitive K | Reactive oxygen species | Protein kinase | Preconditioning
Journal Article
American Journal of Physiology, ISSN 0363-6135, 10/2013, Volume 305, Issue 7, p. H960
  Mitochondria are the major effectors of cardioprotection by procedures that open the mitochondrial ATP-sensitive potassium channel (mitoK^sub ATP^),... 
Cardiovascular system | Molecules | Mitochondria | Oxygen | Pharmacology | Potassium
Journal Article
Circulation Research, ISSN 0009-7330, 08/2012, Volume 111, Issue 4, pp. 446 - 454
RATIONALE:Activation of the mitochondrial ATP-sensitive potassium channel (mitoKATP) has been implicated in the mechanism of cardiac ischemic preconditioning,... 
oprotection | mitochondria | apoptosis | renal outer medullary potassium channel | reconditioning | ATP-sensitive potassium channel | ischemia | cytoprotection | preconditioning
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/1998, Volume 273, Issue 22, pp. 13578 - 13582
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2012, Volume 53, pp. S23 - S23
Journal Article
Biochimica et Biophysica Acta (BBA) - Bioenergetics, ISSN 0005-2728, 07/2010, Volume 1797, p. 120
Journal Article
Antioxidants and Redox Signaling, ISSN 1523-0864, 09/2018, Volume 29, Issue 7, pp. 667 - 714
Journal Article
CIRCULATION RESEARCH, ISSN 0009-7330, 08/2012, Volume 111, Issue 4, pp. 446 - 454
Rationale: Activation of the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)) has been implicated in the mechanism of cardiac ischemic... 
SENSITIVE POTASSIUM CHANNELS | CARDIAC & CARDIOVASCULAR SYSTEMS | cytoprotection | mitochondria | K-ATP CHANNELS | apoptosis | IDENTIFICATION | ATP-sensitive potassium channel | preconditioning | ischemia | INFARCT SIZE | ACTION-POTENTIAL DURATION | HIGH-AFFINITY INHIBITOR | DISEASE | renal outer medullary potassium channel | PERIPHERAL VASCULAR DISEASE | MATRIX VOLUME | CARDIOPROTECTION | HEMATOLOGY | EXPRESSION | Diazoxide - pharmacology | Mitochondria, Heart - metabolism | Cricetulus | Mitochondria, Heart - pathology | Humans | Thallium - metabolism | Mitochondria, Heart - drug effects | RNA, Messenger - metabolism | Necrosis | Mitochondrial Membranes - drug effects | Potassium Channels - metabolism | Transfection | RNA Interference | Time Factors | Cattle | Adenosine Triphosphate - metabolism | Mass Spectrometry | Proteomics - methods | Potassium Channel Blockers - pharmacology | CHO Cells | Cytoprotection | Animals, Newborn | Cricetinae | Potassium Channels, Inwardly Rectifying - drug effects | Bee Venoms - pharmacology | Gene Expression Regulation | Rats | Potassium Channels, Inwardly Rectifying - genetics | Potassium Channels - drug effects | Mitochondrial Proton-Translocating ATPases - metabolism | Mitochondrial Membranes - metabolism | Potassium Channels - genetics | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Apoptosis
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2013, Volume 305, Issue 7, pp. H960 - H968
Mitochondria are the major effectors of cardioprotection by procedures that open the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)), including... 
Cardiac ischemia | Mitochondria | Reactive oxygen species | Cardioprotection | channels | ROS signaling | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | mitochondria | REPERFUSION INJURY | cardiac ischemia | PERFUSED RAT HEARTS | N-2-MERCAPTOPROPIONYL GLYCINE | reactive oxygen species | MITOK(ATP) | cardioprotection | K-ATP channels | K-ATP CHANNEL | PERIPHERAL VASCULAR DISEASE | PKC-EPSILON | HYDROGEN-PEROXIDE | FREE-RADICAL GENERATION | ISCHEMIA-REPERFUSION | Free Radical Scavengers - pharmacology | Phosphorylation | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Mitochondria, Heart - pathology | Male | Mitochondria, Heart - drug effects | Potassium Channels - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Adenosine Triphosphate - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | Superoxides - metabolism | Phospholipids - metabolism | Disease Models, Animal | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Myocardium - pathology | Hydroxyl Radical - metabolism | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Signal Transduction - drug effects | Ischemic Preconditioning, Myocardial | Perfusion | Protein Kinase C-epsilon - metabolism | Myocardial Infarction - prevention & control | Oxidative Stress - drug effects | In Vitro Techniques | Ion Channel Gating | Myocardial Reperfusion Injury - prevention & control | Physiological aspects | Properties | Cardiotonic agents | Cardiac glycosides | Call for Papers | KATP channels
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 01/2006, Volume 290, Issue 1, pp. 406 - 415
The mitochondrial ATP-sensitive K+ channel (mitoKATP) has been assigned multiple roles in cell physiology and in cardioprotection. Each of these roles must... 
Cytochrome c | Uncoupling | Volume regulation | ATP-sensitive potassium channels | Cardiac ischemia | Diazoxide | Potassium channel openers | 5-hydroxydecanoate
Journal Article
BASIC RESEARCH IN CARDIOLOGY, ISSN 0300-8428, 08/2000, Volume 95, Issue 4, pp. 275 - 279
There is considerable evidence that opening the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)) is cardioprotective in ischemia-reperfusion. Two... 
POTASSIUM CHANNEL OPENER | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | mitochondria | RAT-LIVER MITOCHONDRIA | MUSCLE-CELLS | heart | ATP-sensitive potassium channel | ischemia | IN-VITRO | RESPIRATION | cardioprotection | MATRIX VOLUME | COMPARTMENTATION
Journal Article
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