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Atherosclerosis, ISSN 0021-9150, 08/2017, Volume 263, pp. e97 - e97
Journal Article
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 11, pp. 2053 - 2063
OBJECTIVE—Experimental studies suggest that maternal hypercholesterolemia may be relevant for the early onset of cardiovascular disease in offspring. We... 
Apolipoproteins E | Hypercholesterolemia | Atherosclerosis | Trimethylamine-N-oxide | Bile | atherosclerosis | ATHEROGENESIS | RISK-FACTORS | SUSCEPTIBILITY | LOW-DENSITY-LIPOPROTEIN | DEFICIENT MICE | bile | hypercholesterolemia | PREGNANCY | trimethylamine-N-oxide | MATERNAL-HYPERCHOLESTEROLEMIA | apolipoproteins E | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | DYSLIPIDEMIA | HEMATOLOGY | EXPRESSION | Prenatal Exposure Delayed Effects | Age Factors | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Aorta - metabolism | Aortic Diseases - metabolism | Atherosclerosis - etiology | DNA Methylation | Oxygenases - metabolism | Female | Aortic Diseases - pathology | Disease Models, Animal | Methylamines - metabolism | Animals, Newborn | Promoter Regions, Genetic | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Gallbladder - metabolism | Liver - metabolism | Receptors, LDL - metabolism | Bile Acids and Salts - metabolism | Scavenger Receptors, Class B - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Cholesterol - metabolism | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Pregnancy | Cholesterol 7-alpha-Hydroxylase - genetics | Aortic Diseases - genetics | Phenotype | Animals | Cholesterol 7-alpha-Hydroxylase - metabolism | Apolipoproteins E - genetics | Hypercholesterolemia - complications | Hypercholesterolemia - genetics | Oxygenases - genetics | Scavenger Receptors, Class B - metabolism | Hypercholesterolemia - metabolism | Aortic Diseases - etiology | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article
European Heart Journal, ISSN 0195-668X, 03/2019, Volume 40, Issue 11, pp. 928 - 937
Abstract Aims Inflammatory mediators, including blood cells and their products, contribute critically to atherogenesis, but the igniting triggers of... 
Atherogenesis | Breaches | Flow disturbance | Endothelial cells | ATHEROSCLEROTIC LESIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | SHEAR-STRESS | IN-VIVO | NEUTROPHILS | FLOW
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2007, Volume 50, Issue 6, pp. 540 - 546
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2014, Volume 111, Issue 12, p. E1101
  CD31 is a transhomophilic tyrosine-based inhibitory motif receptor and is expressed by both dendritic cells (DCs) and T lymphocytes. Previous studies have... 
Immunohistochemistry | Immunology | Cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2014, Volume 111, Issue 12, pp. 4361 - 4361
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 03/2014, Volume 111, Issue 12, pp. E1101 - E1101
CD31 is a transhomophilic tyrosine-based inhibitory motif receptor and is expressed by both dendritic cells (DCs) and T lymphocytes. Previous studies have... 
Journal Article
Journal of Immunology, ISSN 0022-1767, 05/2010, Volume 184, Issue 10, pp. 5485 - 5492
Journal Article
Cardiovascular Research, ISSN 0008-6363, 04/2012, Volume 94, Issue 1, pp. 30 - 37
The loss of the inhibitory receptor CD31 on peripheral T lymphocytes is associated with the incidence of atherosclerotic complications such as abdominal aortic... 
CD31 | Peptides | Angiotensin II | Aneurysm | Atherosclerosis | PECAM-1 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PLATELET | MECHANISMS | INDUCED HYPERTENSION | ABDOMINAL AORTIC-ANEURYSM | INFLAMMATION | REGULATORY T-CELLS | BINDING-SITES | E-DEFICIENT MICE | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Aortic Aneurysm, Abdominal - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Aortic Diseases - chemically induced | Aortic Diseases - metabolism | Aortic Aneurysm, Abdominal - metabolism | Dose-Response Relationship, Drug | Chemotaxis, Leukocyte - drug effects | Time Factors | T-Lymphocytes - drug effects | Aortic Diseases - prevention & control | Receptors, Antigen, T-Cell - agonists | Aortic Aneurysm, Abdominal - pathology | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Atherosclerosis - immunology | Receptors, Antigen, T-Cell - metabolism | Anti-Inflammatory Agents - pharmacology | Aortic Diseases - immunology | Atherosclerosis - chemically induced | Cells, Cultured | Aortic Aneurysm, Abdominal - genetics | Atherosclerosis - metabolism | Mice, Knockout | Peptides - pharmacology | Aortic Diseases - genetics | Animals | Lymphocyte Activation - drug effects | Apolipoproteins E - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - pharmacology | T-Lymphocytes - immunology | Mice | Macrophage Activation - drug effects | Atherosclerosis - prevention & control | Chemotaxis, Leukocyte | Antigens, CD31 | Aortic Aneurysm, Abdominal | T-Lymphocytes | Lymphocyte Activation | Anti-Inflammatory Agents | Receptors, Antigen, T-Cell | Macrophage Activation | Life Sciences | Human health and pathology | Apolipoproteins E | Cardiology and cardiovascular system | Aortic Diseases | pathology | genetics | pharmacology | immunology | chemically induced | drug effects | prevention & control | agonists | deficiency | metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e116295
Journal Article
Journal Article