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1. Full Text Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance
by Ge, Dongliang and Fellay, Jacques and Thompson, Alexander J and Simon, Jason S and Shianna, Kevin V and Urban, Thomas J and Heinzen, Erin L and Qiu, Ping and Bertelsen, Arthur H and Muir, Andrew J and Sulkowski, Mark and McHutchison, John G and Goldstein, David B
Nature, ISSN 0028-0836, 09/2009, Volume 461, Issue 7262, pp. 399 - 401
Chronic infection with hepatitis C virus (HCV) affects 170 million people worldwide and is the leading cause of cirrhosis in North America(1). Although the... 
WHOLE-GENOME ASSOCIATION | GENOTYPE 1 INFECTION | RIBAVIRIN | PEGINTERFERON ALPHA-2B | MULTIDISCIPLINARY SCIENCES | Interferon-alpha - pharmacology | Far East - ethnology | Humans | Hepatitis C, Chronic - virology | Polyethylene Glycols - adverse effects | Hepatitis C, Chronic - ethnology | Recombinant Proteins | Polyethylene Glycols - therapeutic use | Europe - ethnology | Viral Load | Chromosomes, Human, Pair 19 - genetics | Interleukins - genetics | Polyethylene Glycols - pharmacology | Hepacivirus - drug effects | Genome-Wide Association Study | Hispanic Americans - genetics | Pharmacogenetics | Gene Frequency | Interferon-alpha - therapeutic use | African Americans - genetics | Genotype | Clinical Trials as Topic | Hepatitis C, Chronic - drug therapy | Genome, Human - genetics | Interferon alpha-2 | Polymorphism, Single Nucleotide - genetics | Genetic Variation - genetics | Interferon-alpha - adverse effects | Hepatitis C, Chronic - genetics | Demographic aspects | Development and progression | Genetic aspects | Hepatitis C | Hepatitis C virus | Identification and classification | Genetic polymorphisms | Confidence intervals | Hepatitis | Human immunodeficiency virus--HIV | African Americans | Infections | Drug therapy | Gene expression | Minority & ethnic groups | Index Medicus
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2. Full Text Genetic variation in IL28B and spontaneous clearance of hepatitis C virus
by Thomas, David L and Thio, Chloe L and Martin, Maureen P and Qi, Ying and Ge, Dongliang and Ohuigin, Colm and Kidd, Judith and Kidd, Kenneth and Khakoo, Salim I and Alexander, Graeme and Goedert, James J and Kirk, Gregory D and Donfield, Sharyne M and Rosen, Hugo R and Tobler, Leslie H and Busch, Michael P and McHutchison, John G and Goldstein, David B and Carrington, Mary
Nature, ISSN 0028-0836, 10/2009, Volume 461, Issue 7265, pp. 798 - 801
Hepatitis C virus (HCV) infection is the most common bloodborne infection in the United States, with estimates of 4 million HCV-infected individuals in the... 
ALPHA-2A | MULTIDISCIPLINARY SCIENCES | HOST | INFECTION | NATURAL-HISTORY | HEMOPHILIA | FAMILY | Hepacivirus - drug effects | Genome-Wide Association Study | Hepatitis C - drug therapy | Hepacivirus - immunology | Gene Frequency | Humans | Genotype | Hepatitis C - genetics | Male | Europe - ethnology | Hepatitis C - immunology | Interleukins - genetics | Interleukins - immunology | Africa - ethnology | Hepatitis C - virology | Polymorphism, Single Nucleotide - genetics | Adult | Female | Hepacivirus - physiology | Genetic Variation - genetics | Viral genetics | Genetic variation | Genetic aspects | Research | Hepatitis C | Hepatitis C virus | Health aspects | Hepatitis | Substance abuse treatment | Ethnicity | Human immunodeficiency virus--HIV | Hemophilia | Genetics | Frequency distribution | Tropical diseases | Drug use
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3. Full Text G3viz: an R package to interactively visualize genetic mutation data using a lollipop-diagram
by Guo, Xin and Zhang, Bo and Zeng, Wenqi and Zhao, Shuting and Ge, Dongliang
Bioinformatics (Oxford, England), ISSN 1367-4803, 08/2019
The lollipop-diagram is one of the widely used graphical representations to visualize and explore translational effects of genetic mutations in cancer... 
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4. Full Text ITPA gene variants protect against anaemia in patients treated for chronic hepatitis C
by Ge, Dongliang and Bertelsen, Arthur H and Watson, Mark and Goldstein, David B and Albrecht, Janice and Brass, Clifford and Warner, Amelia and Gumbs, Curtis E and Thompson, Alexander J and McHutchison, John G and Shianna, Kevin V and Qiu, Ping and Sulkowski, Mark and Little, Latasha D and Fellay, Jacques and Urban, Thomas J and Muir, Andrew J
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7287, pp. 405 - 408
Chronic infection with the hepatitis C virus (HCV) affects 170 million people worldwide and is an important cause of liver-related morbidity and mortality. The... 
POPULATION | MULTIDISCIPLINARY SCIENCES | MISSENSE MUTATION | PHENOTYPE | WHOLE-GENOME ASSOCIATION | HEXOKINASE DEFICIENCY | RIBAVIRIN | TRIPHOSPHATE PYROPHOSPHOHYDROLASE DEFICIENCY | NONSPHEROCYTIC HEMOLYTIC-ANEMIA | INOSINE TRIPHOSPHATASE | Genome-Wide Association Study | United States | Humans | Pyrophosphatases - deficiency | Ribavirin - therapeutic use | Pyrophosphatases - genetics | Hemoglobins - metabolism | Anemia, Hemolytic - genetics | Antiviral Agents | Continental Population Groups - genetics | Hepatitis C, Chronic - complications | Hepatitis C, Chronic - drug therapy | Europe - ethnology | Pyrophosphatases - metabolism | Hemoglobins - deficiency | Anemia, Hemolytic - complications | Alleles | Polymorphism, Single Nucleotide - genetics | Anemia, Hemolytic - chemically induced | Genetic Variation - genetics | Chromosomes, Human, Pair 20 | Prevention | Anemia | Genetic variation | Development and progression | Genetic aspects | Hepatitis C | Health aspects | Risk factors | Genotype & phenotype | Genes | Quality control | African Americans | Genetics | Population | Mutation | Chromosomes | Hispanics | Blood
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5. Full Text Personal receptor repertoires: olfaction as a model
by Olender, Tsviya and Waszak, Sebastian M and Viavant, Maya and Khen, Miriam and Ben-Asher, Edna and Reyes, Alejandro and Nativ, Noam and Wysocki, Charles J and Ge, Dongliang and Lancet, Doron
BMC Genomics, ISSN 1471-2164, 08/2012, Volume 13, Issue 1, pp. 414 - 414
Background: Information on nucleotide diversity along completely sequenced human genomes has increased tremendously over the last few years. This makes it... 
Haplotypes | Copy number variation | Genetic polymorphism | Olfaction | Gene family | Olfactory receptor | Single nucleotide polymorphism | MAJOR HISTOCOMPATIBILITY COMPLEX | COPY-NUMBER VARIATION | GENETIC DIVERSITY | HUMAN GENOME | EVOLUTION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MOLECULAR RECOGNITION | ODORANT RECEPTOR | NATURAL-SELECTION | GENETICS & HEREDITY | POPULATION DIFFERENCES | WIDE | Haplotypes - genetics | Smell - physiology | Smell - genetics | Humans | Receptors, Odorant - genetics | Polymorphism, Single Nucleotide - genetics | DNA Copy Number Variations - genetics | Genome, Human - genetics | Genetic aspects | Research | Olfactory receptors | Genetic variation | Smell | Proteins | Analysis | Genomics | Genomes | Single nucleotide polymorphisms | Data mining | Biological diversity | Signal transduction | Reading | Protein folding | Cloning | Genetics | Life sciences | Gene loci | Data bases
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6. Full Text Big data analytics for genomic medicine
by He, Karen Y and Ge, Dongliang and He, Max M
International Journal of Molecular Sciences, ISSN 1661-6596, 02/2017, Volume 18, Issue 2, pp. 412 - 412
Genomic medicine attempts to build individualized strategies for diagnostic or therapeutic decision-making by utilizing patients' genomic information. Big Data... 
Clinically actionable genetic variants | Big data analytics | Healthcare | Next-generation sequencing | Electronic health records | COHORT PROFILE | next-generation sequencing | HIGH-THROUGHPUT | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMERICAN-COLLEGE | electronic health records | CHEMISTRY, MULTIDISCIPLINARY | MISSENSE MUTATIONS | healthcare | Big Data analytics | DE-NOVO MUTATIONS | PHARMACOGENOMICS | PERSONALIZED MEDICINE | WHOLE-EXOME | WIDE ASSOCIATION | HEALTH | clinically actionable genetic variants | Genetic Variation | Computational Biology - methods | Cloud Computing | Humans | High-Throughput Nucleotide Sequencing | Genomics - methods | Medical Informatics - methods | Electronic Health Records | Data Mining - methods | Precision Medicine - methods | Databases, Factual | Medicine | Big Data | Data analysis | Electronic medical records
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7. Full Text A Whole-Genome Association Study of Major Determinants for Host Control of HIV-1
by Jacques Fellay and Kevin V. Shianna and Dongliang Ge and Sara Colombo and Bruno Ledergerber and Mike Weale and Kunlin Zhang and Curtis Gumbs and Antonella Castagna and Andrea Cossarizza and Alessandro Cozzi-Lepri and Andrea De Luca and Philippa Easterbrook and Patrick Francioli and Simon Mallal and Javier Martinez-Picado and José M. Miro and Niels Obel and Jason P. Smith and Josiane Wyniger and Patrick Descombes and Stylianos E. Antonarakis and Norman L. Letvin and Andrew J. McMichael and Barton F. Haynes and Amalio Telenti and David B. Goldstein
Science, ISSN 0036-8075, 8/2007, Volume 317, Issue 5840, pp. 944 - 947
Understanding why some people establish and maintain effective control of HIV-1 and others do not is a priority in the effort to develop new treatments for... 
HLA antigens | T lymphocytes | Infectious diseases | RNA | Genetic variation | Genes | P values | Reports | HLA C antigens | Human genetics | HIV 1 | MULTICOPY GENE FAMILY | RESPONSES | MULTIDISCIPLINARY SCIENCES | ESCAPE MUTATIONS | SEQUENCE | IMMUNODEFICIENCY-VIRUS TYPE-1 | AIDS | MHC | INFECTION | Haplotypes | Major Histocompatibility Complex - genetics | HIV Infections - genetics | HIV Infections - virology | Humans | RNA, Untranslated | Male | DNA-Binding Proteins - genetics | Disease Progression | Viral Load | HIV Infections - immunology | Regression Analysis | HIV-1 - physiology | Immediate-Early Proteins - genetics | HIV Infections - therapy | Female | Genes, MHC Class I | Polymorphism, Single Nucleotide | HLA-C Antigens - genetics | Genome, Human | HLA-B Antigens - genetics | Cohort Studies | Genetic aspects | Research | Identification and classification | HIV infection | Genetic polymorphisms | Histocompatibility testing | Medical research | HIV | Acquired immune deficiency syndrome | Genomics | Infections | Human immunodeficiency virus | RNA polymerase
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8. Full Text A Genome-Wide Association Study in Chronic Obstructive Pulmonary Disease (COPD): Identification of Two Major Susceptibility Loci
by Pillai, S.G and Ge, D.L and Zhu, G.H and Kong, X.Y and Shianna, K.V and Need, A.C and Feng, S and Hersh, C.P and Bakke, P and Gulsvik, A and Ruppert, A and Carlsen, K.C.L and Roses, A and Anderson, W and Rennard, S.I and Lomas, D.A and Silverman, E.K and Goldstein, D.B and ICGN Investigators incl. Wouters, E.F.M and ICGN Investigators
Plos Genetics, ISSN 1553-7390, 01/2009, Volume 5, Issue 3, pp. e1000421 - e1000421
There is considerable variability in the susceptibility of smokers to develop chronic obstructive pulmonary disease (COPD). The only known genetic risk factor... 
LUNG-CANCER | NICOTINE DEPENDENCE | CIGARETTE-SMOKING | TWINS | SURFACTANT PROTEIN-B | ACETYLCHOLINE | GENES | GENETICS & HEREDITY | RISK | HEALTH | EXPRESSION | Smoking - adverse effects | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Humans | Risk Factors | European Continental Ancestry Group | Pulmonary Disease, Chronic Obstructive - etiology | Case-Control Studies | Chromosomes, Human, Pair 4 | Membrane Glycoproteins - genetics | Carrier Proteins - genetics | Regression Analysis | Norway | Pedigree | Polymorphism, Single Nucleotide | Pulmonary Disease, Chronic Obstructive - genetics | Quantitative trait loci | Lung diseases, Obstructive | Genetic aspects | Disease susceptibility | Diagnosis | Research | Health aspects | Identification and classification | Malalties pulmonars obstructives cròniques | Chronic obstructive pulmonary diseases | Genomics | Lung cancer | Genòmica | Càncer de pulmó | Hospitals | Aging | Genetics | Population | Chronic obstructive pulmonary disease | Genomes | Health risk assessment | Epidemiology | Emphysema | Risk factors | Smoking
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9. Full Text Common variants conferring risk of schizophrenia
by Stefansson, Hreinn and Ophoff, Roel A and Steinberg, Stacy and Aneassen, Ole A and Cichon, Sven and Rujescu, Dan and Werge, Thomas and Pietiläinen, Olli P. H and Mors, Ole and Mortensen, Preben B and Sigurdsson, Engilbert and Gustafsson, Omar and Nyegaard, Mette and Tuulio-Henriksson, Annamari and Ingason, Anes and Hansen, Thomas and Suvisaari, Jaana and Lonnqvist, Jouko and Paunio, Tiina and Børglum, Anders D and Hartmann, Annette and Fink-Jensen, Anders and Nordentoft, Merete and Hougaard, David and Norgaard-Pedersen, Bent and Böttcher, Yvonne and Olesen, Jes and Breuer, René and Möller, Hans-Jürgen and Giegling, Ina and Rasmussen, Henrik B and Timm, Sally and Mattheisen, Manuel and Bitter, István and Réthelyi, János M and Magnusdottir, Brynja B and Sigmundsson, Thordur and Olason, Pall and Masson, Gisli and Gulcher, Jeffrey R and Haraldsson, Magnus and Fossdal, Ragnheidur and Thorgeirsson, Thorgeir E and Thorsteinsdottir, Unnur and Ruggeri, Mirella and Tosato, Sarah and Franke, Barbara and Strengman, Eric and Kiemeney, Lambertus A and Melle, Ingrid and Djurovic, Srdjan and Abramova, Lilia and Kaleda, Vasily and Sanjuan, Julio and de Frutos, Rosa and Bramon, Elvira and Vassos, Evangelos and Fraser, Gillian and Ettinger, Ulrich and Picchioni, Marco and Walker, Nicholas and Toulopoulou, Timi and Need, Anna C and Ge, Dongliang and Yoon, Joeng Lim and Shianna, Kevin V and Freimer, Nelson B and Cantor, Rita M and Murray, Robin and Kong, Augustine and Golimbet, Vera and Carracedo, Angel and Arango, Celso and Costas, Javier and Jönsson, Erik G and Terenius, Lars and Agartz, Ingrid and Petursson, Hannes and Nöthen, Markus M and Rietschel, Marcella and Matthews, Paul M and Muglia, Pieranea and Peltonen, Leena and St Clair, David and Goldstein, David B and Stefansson, Kari and Collier, David A and Kahn, René S and Linszen, Don H and van Os, Jim and Wiersma, Durk and Bruggeman, Richard and Cahn, Wiepke and de Haan, Lieuwe and Krabbendam, Lydia and Myin-Germeys, Inez and GROUP and Genetic Risk and Outcome in Psychosis (GROUP)
Nature, ISSN 0028-0836, 2009, Volume 460, Issue 7256, pp. 744 - 747
Schizophrenia is a complex disorder, caused by both genetic and environmental factors and their interactions. Research on pathogenesis has traditionally... 
NEUROGRANIN | POPULATION | DISEASES | MENTAL-RETARDATION | GENE | MEMORY | MULTIDISCIPLINARY SCIENCES | 1Q21.1 | SCAN | DELETIONS | GENOME-WIDE ASSOCIATION | Neurogranin - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Transcription Factor 4 | Major Histocompatibility Complex - genetics | Humans | Genotype | Chromosomes, Human, Pair 11 - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Chromosomes, Human, Pair 6 - genetics | Genome, Human - genetics | Schizophrenia - immunology | Schizophrenia - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors | Genetic Markers - genetics | Polymorphism, Single Nucleotide - genetics | Chromosomes, Human, Pair 18 - genetics | Major histocompatibility complex | Schizophrenia | Development and progression | Genetic aspects | Research | Single nucleotide polymorphisms | Health aspects | Risk factors | Celiac disease | Genetics | Disease | Autoimmune diseases | Mass spectrometry
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