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Oncogene, ISSN 0950-9232, 06/2014, Volume 33, Issue 25, pp. 3256 - 3266
Signal transducer and activator of transcription 3 (STAT3) is altered in several epithelial cancers and represents a potential therapeutic target. Here, STAT3... 
cell migration | inhibition | esophageal cancer | cell proliferation | STAT3 | PROTEIN | TRANSCRIPTION 3 | MULTIPOLAR MITOSES | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | TYROSINE-PHOSPHORYLATED STAT3 | GROWTH-FACTOR RECEPTOR | ONCOLOGY | PATHWAY | COLORECTAL-CANCER | IN-VIVO | GENETICS & HEREDITY | GENE AMPLIFICATION | ACTIVATED SIGNAL TRANSDUCER | Cell Cycle - genetics | Up-Regulation | Phosphorylation | Adenocarcinoma - pathology | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Cell Movement - genetics | Esophageal Neoplasms - pathology | Gene Knockdown Techniques | Adenocarcinoma - metabolism | Barrett Esophagus - pathology | Esophageal Neoplasms - metabolism | Adenocarcinoma - genetics | Cell Growth Processes - genetics | Barrett Esophagus - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Barrett Esophagus - metabolism | Signal Transduction | Down-Regulation | STAT3 Transcription Factor - biosynthesis | Esophageal Neoplasms - genetics | Cell Line, Tumor | STAT3 Transcription Factor - antagonists & inhibitors | Transcription factors | Care and treatment | Oncology, Experimental | Genetic research | Genetic aspects | Research | Properties | Gene expression | Esophageal cancer | Cancer | Signal transduction | Protein expression | Cell growth | Cell cycle
Journal Article
AMERICAN JOURNAL OF SURGICAL PATHOLOGY, ISSN 0147-5185, 04/2017, Volume 41, Issue 4, pp. 458 - 471
To more fully characterize the clinical and pathologic spectrum of a recently described tumor entity of the sinonasal tract characterized by loss of nuclear... 
SURGERY | basaloid carcinoma | PBRM1 | SMARCB1 | NEOPLASMS | rhabdoid carcinoma | PATHOLOGY | ARID1A | SMARCA2 | TUMORS | CANCER | INI1 | SMARCA4 | FEATURES | FAMILY | sinonasal tract | SMARCB1-deficient sinonasal carcinoma | GENE | INI1 EXPRESSION | SMARCA4/BRG1 | UNDIFFERENTIATED CARCINOMA | Immunohistochemistry | Nose Neoplasms - chemistry | Predictive Value of Tests | United States | Biomarkers, Tumor - deficiency | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | SMARCB1 Protein - genetics | Male | Chemoradiotherapy, Adjuvant | Maxillary Sinus Neoplasms - genetics | Carcinoma - chemistry | Maxillary Sinus Neoplasms - chemistry | Young Adult | Nasal Surgical Procedures | Time Factors | Polymerase Chain Reaction | Aged, 80 and over | Adult | Female | Cell Differentiation | Retrospective Studies | SMARCB1 Protein - deficiency | Carcinoma - pathology | Maxillary Sinus Neoplasms - pathology | Nose Neoplasms - pathology | Nose Neoplasms - therapy | Carcinoma, Squamous Cell - therapy | Carcinoma, Squamous Cell - chemistry | In Situ Hybridization, Fluorescence | Treatment Outcome | Nose Neoplasms - genetics | Paranasal Sinuses - chemistry | Magnetic Resonance Imaging | Biopsy | Carcinoma - genetics | Carcinoma - therapy | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Paranasal Sinuses - pathology | Germany | Maxillary Sinus Neoplasms - therapy
Journal Article
Zentralblatt für Chirurgie - Zeitschrift für Allgemeine, Viszeral-, Thorax- und Gefäßchirurgie, ISSN 0044-409X, 09/2016, Volume 141, Issue S 01
a) In den Jahren 1998 – 2016 wurden an unsere Klinik 179 Chondrohamartome von 175 Patienten operiert. b) Die überwiegend männlichen Patienten (57%) waren im... 
Conference Proceeding
Journal Article
International journal of cancer, ISSN 0020-7136, 2014, Volume 135, Issue 7, pp. 1517 - 1530
Receptor tyrosine kinases (RTKs) are in the focus of targeted therapy for epithelial tumors. Our study addressed the role of EGFR, HER2 and HER3 expression and... 
clinical inhibitors | EGFR/HER2/HER3‐dimers | esophageal carcinomas | EGFR/HER2/HER3-dimers | ESOPHAGOGASTRIC CANCER | JUNCTION CANCER | BARRETTS-ESOPHAGUS | II TRIAL | LABEL PHASE-3 TRIAL | ARBEITSGEMEINSCHAFT INTERNISTISCHE ONKOLOGIE | GROWTH-FACTOR RECEPTOR | ONCOLOGY | GASTRIC-CANCER | SQUAMOUS-CELL CARCINOMA | HER-2/NEU GENE AMPLIFICATION | Erlotinib Hydrochloride | Receptor, ErbB-3 - metabolism | Adenocarcinoma - pathology | Apoptosis - drug effects | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Protein Multimerization | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Immunoenzyme Techniques | Esophageal Neoplasms - pathology | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | Esophageal Neoplasms - metabolism | Receptor, ErbB-2 - antagonists & inhibitors | Tumor Cells, Cultured | Antibody-Dependent Cell Cytotoxicity | Receptor, ErbB-3 - antagonists & inhibitors | Adenocarcinoma - drug therapy | Blotting, Western | Carcinoma, Squamous Cell - drug therapy | Cell Cycle Checkpoints - drug effects | Fluorescent Antibody Technique | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Quinazolines - pharmacology | Esophageal Neoplasms - drug therapy | Phosphotransferases | Squamous cell carcinoma | Esophageal cancer | Medical research | Rodents | Cell cycle
Journal Article
New England Journal of Medicine, ISSN 0028-4793, 12/2011, Volume 365, Issue 25, pp. e36 - E46
A 23-year-old man presented with a 1-year history of diabetes mellitus, hypertension, and hypogonadism and a weight gain of 22 kg. Abdominal striae were... 
MEDICINE, GENERAL & INTERNAL | Medical research | Nervous system diseases | Adrenocorticotropic hormone | Dexamethasone | Pituitary | Diabetes mellitus | Cushing's syndrome | Diabetes | Hydrocortisone | Hypogonadism
Journal Article
Cancer Cytopathology, ISSN 0008-543X, 04/2006, Volume 108, Issue 2, pp. 129 - 134
BACKGROUND Promoter hypermethylation is an important mechanism for silencing tumor‐suppressor genes in cancer and a promising tool for development of molecular... 
lung cancer | specificity | molecular diagnostic techniques | sensitivity | promoter methylation | Promoter methylation | Sensitivity | Specificity | Lung cancer | Molecular diagnostic techniques | BRONCHIAL EPITHELIUM | DNA HYPERMETHYLATION | RASSF1A | CPG ISLAND | PATHOLOGY | CELL-LINES | ABERRANT PROMOTER METHYLATION | MULTIPLE GENES | ONCOLOGY | EPIGENETIC INACTIVATION | TUMOR-SUPPRESSOR | CHRONIC SMOKERS | Molecular Diagnostic Techniques - methods | Adenocarcinoma - pathology | Carcinoma, Non-Small-Cell Lung - chemistry | Carcinoma, Small Cell - genetics | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Carcinoma, Non-Small-Cell Lung - diagnosis | Case-Control Studies | Carcinoma, Small Cell - chemistry | DNA Methylation | Sensitivity and Specificity | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | DNA, Neoplasm - analysis | Retrospective Studies | Adenocarcinoma - chemistry | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Promoter Regions, Genetic | Diagnosis, Differential | Lung Neoplasms - chemistry | Biomarkers, Tumor - analysis | Carcinoma, Non-Small-Cell Lung - genetics | Gene Silencing | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Small Cell - diagnosis | Adenocarcinoma - diagnosis | Cell Line, Tumor | Aged | Biomarkers, Tumor - genetics | Biopsy, Needle | DNA, Neoplasm - genetics | Carcinoma, Small Cell - pathology | Lung Neoplasms - diagnosis | Tumor Suppressor Proteins - analysis | Development and progression | Tumor markers | Diagnosis | Research | Methylation
Journal Article
Journal Article
Journal Article
The American journal of surgical pathology, ISSN 0147-5185, 4/2017, Volume 41, Issue 4, pp. 458 - 471
To more fully characterize the clinical and pathological spectrum of a recently described tumor entity of the sinonasal tract characterized by loss of nuclear... 
sinonasal tract | SMARCB1-deficient sinonasal carcinoma | basaloid carcinoma | SMARCB1 | rhabdoid carcinoma | ARID1A | SMARCA2 | INI1 | SMARCA4
Journal Article