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Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, pp. e49152 - e49152
Objective: In addition to improve glucose intolerance, recent studies suggest that glucagon-like peptide-1 (GLP-1) receptor agonism also decreases triglyceride... 
THERAPY | GLUCAGON-LIKE PEPTIDE-1 | DE-NOVO LIPOGENESIS | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | OB/OB MICE | EXENATIDE | LIVER-DISEASE | LIPOPROTEINS | BETA-CELL FUNCTION | TRANSGENIC MICE | Apolipoproteins B - metabolism | Liver - pathology | Fatty Liver - metabolism | Glucagon-Like Peptide 1 - metabolism | Peptides - chemistry | Fatty Liver - therapy | Receptors, Glucagon - metabolism | Male | Mice, Transgenic | Apolipoprotein E3 - metabolism | Apolipoprotein E3 - genetics | Diabetes Mellitus, Type 2 - blood | Dyslipidemias - blood | Insulin - metabolism | Animals | Peptides - metabolism | Glucagon-Like Peptide-1 Receptor | Mice | Blood Glucose - metabolism | Lipogenesis | Venoms - metabolism | Type 2 diabetes | Production management | Glucagon | Liver | Low density lipoproteins | Lipids | Genetic engineering | Glucose | Apolipoproteins | Insulin | Dextrose | Peptides | Genes | Gene regulation | Dyslipidemia | Phospholipids | Experiments | High fat diet | Fatty liver | Metabolites | Apolipoprotein E | Rodents | Down-regulation | Sterol regulatory element-binding protein | Lipid metabolism | Glucagon-like peptide 1 | Food | Medical research | Fasting | Liver diseases | Internal medicine | Research & development--R&D | Diabetes mellitus | Transgenic mice | Gene expression | Metabolism | Cholesterol | Lipoproteins (very low density) | Steatosis | Medicine | Glucose tolerance | Intolerance | Lipoproteins | Brain research | Diet | Reagents | Receptor density | Insulin resistance | Diabetes | Receptor mechanisms | Binding sites | Endocrinology | Metabolic disorders | Index Medicus | Research & development | R&D
Journal Article
Atherosclerosis (Supplements) (Component), ISSN 1567-5688, 06/2018, Volume 32, pp. 7 - 7
Journal Article
PLoS ONE, ISSN 1932-6203, 2013, Volume 8, Issue 2, pp. e55217 - e55217
Central neuropeptide Y (NPY) administration stimulates food intake in rodents. In addition, acute modulation of central NPY signaling increases hepatic... 
HYPOTHALAMIC CONTROL | NERVOUS-SYSTEM | PREPRONEUROPEPTIDE-Y | MULTIDISCIPLINARY SCIENCES | TRIGLYCERIDE SECRETION | SERUM TRIGLYCERIDE | RECEPTOR GENE | POLYMORPHISM | ENERGY-METABOLISM | EXPRESSION | ASSOCIATION | Consciousness - drug effects | Cerebral Ventricles - metabolism | Liver - metabolism | Mice, Inbred C57BL | Injections, Intraventricular | Neuropeptide Y - administration & dosage | Rats | Male | Neuropeptide Y - pharmacology | Cerebral Ventricles - drug effects | Lipoproteins, VLDL - biosynthesis | Eating - drug effects | Animals | Liver - drug effects | Signal Transduction - drug effects | Anesthesia | Mice | Neuropeptide Y | Type 2 diabetes | Analysis | Low density lipoproteins | Body weight | Atherosclerosis | Triglycerides | Statistics | Brain | Animal models | Intravenous administration | Peptides | Liver | Ventricles (cerebral) | Fuel consumption | Cardiovascular disease | Neuropeptides | Cerebrospinal fluid | Metabolic syndrome | Risk factors | Energy | Rodents | Tubes | Food | Internal medicine | Food production | Injection | Gene expression | Apolipoproteins | Low density lipoprotein | Lipoproteins (very low density) | Medicine | Signaling | Medical materials | Brain research | Food intake | Hypertriglyceridemia | Arteriosclerosis | Insulin resistance | Research design | Ventricle | Diabetes | Endocrinology | Metabolic disorders | Index Medicus
Journal Article
Journal Article
FASEB journal, ISSN 0892-6638, 2013, Volume 27, Issue 8, pp. 3354 - 3362
Journal Article
The FASEB Journal, ISSN 0892-6638, 08/2013, Volume 27, Issue 8, pp. 3354 - 3362
Mutations in apolipoprotein A5 (APOA5) have been associated with hypertriglyceridemia in humans and mice. This has been attributed to a stimulating role for... 
Journal Article
Journal of lipid research, ISSN 0022-2275, 2011, Volume 52, Issue 9, pp. 1712 - 1722
Insulin signaling in the central nervous system (CNS) is required for the inhibitory effect of insulin on glucose production. Our aim was to determine whether... 
Insulin - pharmacology | CD36 Antigens - genetics | Central Nervous System - metabolism | Mice, Inbred C57BL |