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Molecular Neurodegeneration, ISSN 1750-1326, 2009, Volume 4, Issue 1, pp. 13 - 13
Journal Article
Neuron, ISSN 0896-6273, 10/2016, Volume 92, Issue 2, pp. 383 - 391
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 05/2016, Volume 90, Issue 3, pp. 535 - 550
Journal Article
Neuron, ISSN 0896-6273, 12/2015, Volume 88, Issue 5, pp. 892 - 901
Noncoding expansions of a hexanucleotide repeat (GGGGCC) in the gene are the most common cause of familial amyotrophic lateral sclerosis and frontotemporal... 
DEMENTIA | EXPANDED C9ORF72 | DIPEPTIDE-REPEAT PROTEINS | EXPANSIONS | AMYOTROPHIC-LATERAL-SCLEROSIS | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSLATION | HEXANUCLEOTIDE REPEAT | NEUROSCIENCES | GGGGCC REPEAT | RNA FOCI | Muscle Strength - genetics | Spinal Cord - metabolism | Age Factors | Amyotrophic Lateral Sclerosis - physiopathology | DNA Repeat Expansion - genetics | Humans | Psychomotor Performance - physiology | Brain - metabolism | Chromosomes, Artificial, Bacterial - genetics | Spinal Cord - pathology | Chromosomes, Artificial, Bacterial - metabolism | Neuromuscular Junction - genetics | C9orf72 Protein | Neurons - drug effects | Neuromuscular Junction - pathology | Disease Models, Animal | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Frontotemporal Dementia - physiopathology | Glutamic Acid - pharmacology | Amyotrophic Lateral Sclerosis - genetics | Cells, Cultured | Mice, Transgenic | Proteins - genetics | Motor Activity - genetics | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain - pathology | Mice | Neurosciences | Genetic engineering | RNA | Dementia | Medical colleges | Peptides | Proteins | Brain research | Transgenic animals | Neurodegeneration | Laboratories | Rodents | Amyotrophic lateral sclerosis | Genomes | Artificial chromosomes | Alzheimers disease | Experiments | Patients | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 09/2014, Volume 83, Issue 5, pp. 1043 - 1050
Journal Article
Acta neuropathologica, ISSN 0001-6322, 2015, Volume 130, Issue 6, pp. 845 - 861
Journal Article
Neuron, ISSN 0896-6273, 12/2015, Volume 88, Issue 5, pp. 902 - 909
A non-coding hexanucleotide repeat expansion in the gene is the most common mutation associated with familial amyotrophic lateral sclerosis (ALS) and... 
RAN translation | transgenic mice | Amyotrophic lateral sclerosis (ALS) | frontotemporal dementia (FTD) | neurodegeneration | microRNA | repeat expansions | C9ORF72 | RNA foci | Repeat expansions | MicroRNA | Neurodegeneration | Transgenic mice | Frontotemporal dementia (FTD) | DEMENTIA | ALS | AMYOTROPHIC-LATERAL-SCLEROSIS | SPECTRUM | MODEL | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSLATION | SENSE | NEUROSCIENCES | PATHOLOGICAL FEATURES | Age Factors | Amyotrophic Lateral Sclerosis - physiopathology | DNA Repeat Expansion - genetics | Humans | MicroRNAs - metabolism | Frontotemporal Dementia - mortality | Cerebral Cortex - cytology | Brain - metabolism | Chromosomes, Artificial, Bacterial - genetics | Chromosomes, Artificial, Bacterial - metabolism | Neurons - physiology | C9orf72 Protein | Dipeptides - metabolism | Neurons - drug effects | Disease Models, Animal | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Frontotemporal Dementia - physiopathology | Gene Expression Regulation - genetics | Amyotrophic Lateral Sclerosis - genetics | Cells, Cultured | Genotype | Mice, Transgenic | Amyotrophic Lateral Sclerosis - mortality | Nerve Tissue Proteins - metabolism | Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain - pathology | In Vitro Techniques | Dipeptides - genetics | Proteins | Neurosciences | Proline | Genetic engineering | Glycine | Dementia | Analysis | Genetic translation | Plasmids | Neurons | Rodents | Cloning | Amyotrophic lateral sclerosis | Behavior | Artificial chromosomes | Expansion | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2009, Volume 106, Issue 18, pp. 7607 - 7612
Journal Article