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Cell Reports, ISSN 2211-1247, 02/2016, Volume 14, Issue 6, pp. 1555 - 1566
Journal Article
Nature, ISSN 0028-0836, 10/2014, Volume 514, Issue 7524, pp. 628 - 632
Journal Article
Oncotarget, ISSN 1949-2553, 06/2019, Volume 10, Issue 40, p. 3979
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2012, Volume 18, Issue 10, pp. 1503 - 1510
Journal Article
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7411, pp. 337 - 342
Inactivation of tumour-suppressor genes by homozygous deletion is a prototypic event in the cancer genome, yet such deletions often encompass neighbouring... 
ENOLASE DEFICIENCY | CELLS | INHIBITION | MULTIDISCIPLINARY SCIENCES | REGIONS | Neoplasm Transplantation | RNA, Small Interfering - genetics | Enzyme Inhibitors | Phosphopyruvate Hydratase - deficiency | Cell Proliferation | Biomarkers, Tumor - deficiency | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Phosphonoacetic Acid - analogs & derivatives | Phosphopyruvate Hydratase - metabolism | Antineoplastic Agents - therapeutic use | DNA-Binding Proteins - deficiency | Gene Knockdown Techniques | Glioblastoma - genetics | Phosphopyruvate Hydratase - genetics | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Antineoplastic Agents - pharmacology | Chromosomes, Human, Pair 1 - genetics | Genes, Tumor Suppressor | Hydroxamic Acids - pharmacology | Molecular Targeted Therapy - methods | Genes, Essential - genetics | Brain Neoplasms - genetics | Phosphopyruvate Hydratase - antagonists & inhibitors | Brain Neoplasms - drug therapy | DNA-Binding Proteins - genetics | Phosphonoacetic Acid - therapeutic use | Phosphonoacetic Acid - pharmacology | Homozygote | Animals | Glioblastoma - pathology | Cell Line, Tumor | Hydroxamic Acids - therapeutic use | Biomarkers, Tumor - genetics | Mice | Glioblastoma - drug therapy | Sequence Deletion - genetics | Gene mutations | Physiological aspects | Tumor suppressor genes | Genetic aspects | Research | Health aspects | Risk factors | Cancer | Proteins | Genetics | Kinases | Genes | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 02/2017, Volume 542, Issue 7639, pp. 119 - 123
The genome of pancreatic ductal adenocarcinoma (PDAC) frequently contains deletions of tumour suppressor gene loci, most notably SMAD4, which is homozygously... 
SREBP ACTIVITY | PATHWAYS | AMINO-ACID-METABOLISM | ACTIVATED PROTEIN-KINASE | GLUTAMINE | GENE | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | BIOLOGY | GROWTH | TARGETS | Mitochondria - enzymology | AMP-Activated Protein Kinases - metabolism | Reactive Oxygen Species - metabolism | Amino Acids, Branched-Chain - metabolism | Humans | Pregnancy Proteins - genetics | Male | Carcinoma, Pancreatic Ductal - genetics | Malate Dehydrogenase - genetics | Carcinoma, Pancreatic Ductal - psychology | Gene Deletion | Minor Histocompatibility Antigens - genetics | NADP - metabolism | Ketoglutaric Acids - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Transaminases - biosynthesis | Biocatalysis | Transaminases - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - therapy | Malate Dehydrogenase - deficiency | Mitochondria - pathology | NADP - biosynthesis | Pregnancy Proteins - biosynthesis | Carcinoma, Pancreatic Ductal - enzymology | Minor Histocompatibility Antigens - biosynthesis | Animals | Mice | Pancreatic Neoplasms - therapy | Enzymes | Complications and side effects | Pancreatic cancer | Genetic aspects | Chromosome deletion | Health aspects | Risk factors | Cell growth | Mitochondria | Metabolites | Homeostasis | Collateral | Biosynthesis | Tumors | Cancer | Apoptosis
Journal Article
Journal of Experimental and Clinical Cancer Research, ISSN 1756-9966, 2012, Volume 31, Issue 1, pp. 71 - 71
Journal Article
Nature, ISSN 0028-0836, 02/2017, Volume 542, Issue 7641, pp. 362 - 366
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 03/2019, Volume 37, Issue 7_suppl, pp. TPS678 - TPS678
Journal Article
Cancer Research, ISSN 0008-5472, 05/2018, Volume 78, Issue 10 Supplement, pp. A03 - A03
Journal Article
Genes and Development, ISSN 0890-9369, 2012, Volume 26, Issue 8, pp. 756 - 784
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 71 - 71
The comprehensive and coordinated efforts of The Cancer Genome Atlas (TCGA) and the International Cancer Genome Consortium (ICGC) identified the somatic... 
Journal Article
Journal of Crohn's and Colitis, ISSN 1873-9946, 04/2019, Volume 13, Issue 5, pp. 659 - 668
Abstract Intestinal fibrosis is an inevitable complication in patients with inflammatory bowel disease [IBD], occurring in its two major clinical... 
CELLS | TISSUE | Crohn's disease | MECHANISMS | INDUCTION | EMT | inflammatory bowel disease | epithelial-to-mesenchymal transition | PATHOGENESIS | IN-VITRO | FIBROBLASTS DERIVE | ulcerative colitis | INFECTION | intestinal fibrosis | GASTROENTEROLOGY & HEPATOLOGY | EndMT
Journal Article
by Brennan, Cameron W and Verhaak, Roel G.W and McKenna, Aaron and Campos, Benito and Noushmehr, Houtan and Salama, Sofie R and Zheng, Siyuan and Chakravarty, Debyani and Sanborn, J. Zachary and Berman, Samuel H and Beroukhim, Rameen and Bernard, Brady and Wu, Junyuan and Wu, Chang-Jiun and Genovese, Giannicola and Shmulevich, Ilya and Barnholtz-Sloan, Jill and Zou, Lihua and Vegesna, Rahulsimham and Shukla, Sachet A and Ciriello, Giovanni and Yung, W.K and Yung, W.K. Alfred and Zhang, Zhaobin and Zhang, Wei and Zhang, Jianhua and Zhang, Hailei and Sougnez, Carrie and Mikkelsen, Tom and Aldape, Kenneth and Bigner, Darell D and Bigner, Darell and Van Meir, Erwin G and Prados, Michael and Sloan, Andrew and Sloan, Andrew E and Black, Keith L and Eschbacher, Jennifer and Finocchiaro, Gaetano and Friedman, William and Andrews, David W and Guha, Abhijit and Iacocca, Mary and O’Neill, Brian P and Foltz, Greg and Myers, Jerome and Weisenberger, Daniel J and Penny, Robert and Kucherlapati, Raju and Perou, Charles M and Hayes, D. Neil and Gibbs, Richard and Marra, Marco and Mills, Gordon and Mills, Gordon B and Lander, Eric S and Lander, Eric and Spellman, Paul and Wilson, Richard K and Wilson, Richard and Sander, Chris and Weinstein, John N and Weinstein, John and Meyerson, Matthew and Gabriel, Stacey B and Gabriel, Stacey and Laird, Peter W and Haussler, David and Getz, Gad and Chin, Lynda and Benz, Christopher and Barrett, Wendi and Ostrom, Quinn and Wolinsky, Yingli and Bose, Bikash and Boulos, Madgy and Boulos, Paul T and Brown, Jenn and Czerinski, Christine and Eppley, Matthew and Kempista, Thelma and Kitko, Teresa and Koyfman, Yakov and Rabeno, Brenda and Rastogi, Pawan and Sugarman, Michael and Swanson, Patricia and Yalamanchii, Kennedy and Otey, Ilana P and Liu, Yuexin and Liu, Yingchun Spring and Liu, Wenbin and Xiao, Yonghong and Auman, J.Todd and Chen, Qing-Rong and Chen, Peng-Chieh and Chen, Ken and Hadjipanayis, Angela and Lee, Eunjung and Lee, Semin and ... and TCGA Res Network
Cell, ISSN 0092-8674, 10/2013, Volume 155, Issue 2, pp. 462 - 477
We describe the landscape of somatic genomic alterations based on multidimensional and comprehensive characterization of more than 500 glioblastoma tumors... 
PATHWAYS | SURVIVAL | GRADE GLIOMA | MELANOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN CANCERS | COPY-NUMBER ALTERATION | TUMORS | EXPRESSION | DRIVER MUTATIONS | EGFR | CELL BIOLOGY | Platelet-derived growth factor | RNA | Gene mutations | Analysis | Methylation | Glioblastoma multiforme | Telomerase
Journal Article