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American journal of respiratory cell and molecular biology, 04/2019
Inflammation and vascular smooth muscle cell (VSMC) phenotypic switching are causally linked to pulmonary arterial hypertension (PAH) pathogenesis. Carbonic... 
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 03/2001, Volume 88, Issue 6, pp. 555 - 562
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2009, Volume 54, Issue 1, pp. S32 - S42
Journal Article
PLoS Computational Biology, ISSN 1553-734X, 08/2014, Volume 10, Issue 8
  Errors in sample annotation or labeling often occur in large-scale genetic or genomic studies and are difficult to avoid completely during data generation... 
Disease | Genomics | Genomes | Gender | Gene expression | Studies | Datasets | Genotype & phenotype | Annotations | Proteomics | Epigenetics | Software | Chronic obstructive pulmonary disease | Labeling | Cancer
Journal Article
American Journal of Physiology: Lung Cellular and Molecular Physiology, ISSN 1040-0605, 07/2016, Volume 311, Issue 1, pp. L124 - L124
Epigenetic mechanisms, including DNA methylation and histone acetylation, regulate gene expression in idiopathic pulmonary arterial hypertension (IPAH). These... 
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1217 - 1223
Sepsis, a systemic inflammatory response to infection, commonly progresses to acute lung injury (ALI), an inflammatory lung disease with high morbidity. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ICAM-1 | MODEL | MOLECULES | CELL BIOLOGY | LEUKOCYTES | IN-VIVO | HEPARAN-SULFATE | CELL | UNFRACTIONATED HEPARIN | Intestinal Perforation - complications | Humans | Heparitin Sulfate - antagonists & inhibitors | Male | Adoptive Transfer | Heparitin Sulfate - metabolism | Pulmonary Alveoli - enzymology | Glucuronidase - deficiency | Glucuronidase - analysis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Glucuronidase - physiology | Respiratory Insufficiency - enzymology | Ventilator-Induced Lung Injury - enzymology | Acute Lung Injury - prevention & control | Endotoxemia - physiopathology | Intercellular Adhesion Molecule-1 - biosynthesis | Endothelium - enzymology | Disease Models, Animal | Acute Lung Injury - physiopathology | Lipopolysaccharides - toxicity | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Neutrophils - physiology | Ventilator-Induced Lung Injury - pathology | Lung - physiopathology | Mice, Knockout | Gene Expression Regulation - drug effects | Respiratory Insufficiency - pathology | Glycocalyx - physiology | Animals | Cell Adhesion - physiology | Endotoxemia - complications | Receptors, Tumor Necrosis Factor, Type I - physiology | Intercellular Adhesion Molecule-1 - genetics | Tumor Necrosis Factor-alpha - physiology | Mice | Enzyme Activation | Acute Lung Injury - etiology | Intestinal Perforation - microbiology | Endothelium - physiology | Care and treatment | Glycocalyx | Cellular control mechanisms | Neutrophils | Cell adhesion | Physiological aspects | Sepsis | Development and progression | Research | Properties | Respiratory physiology | Endothelium | Leucocytes | Glycoproteins | Lung diseases | Cell adhesion & migration | Immune system
Journal Article
COPD: Journal of Chronic Obstructive Pulmonary Disease, ISSN 1541-2555, 01/2004, Volume 1, Issue 2, pp. 153 - 153
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2000, Volume 97, Issue 24, pp. 13275 - 13280
There is evidence from both genetic and pharmacologic studies to suggest that the cyclooxygenase-2(COX-2) enzyme plays a causal role in the development of... 
Biological Sciences | Receptors | Messenger RNA | RNA | Epithelial cells | Cell lines | Colorectal cancer | Ligands | Colorectal neoplasms | Transactivation | Tumors | INHIBITION | PROTEIN | EPITHELIAL-CELLS | CARCINOMA CELLS | LIGANDS | MULTIDISCIPLINARY SCIENCES | HUMAN COLON-CANCER | PPAR-GAMMA | CYCLOOXYGENASE-2 EXPRESSION | MICE | TERMINAL DIFFERENTIATION | Adenocarcinoma | Up-Regulation | Intestinal Mucosa - metabolism | Colorectal Neoplasms - genetics | Cyclooxygenase 2 | Humans | Recombinant Fusion Proteins - metabolism | Epoprostenol - physiology | In Situ Hybridization | Transfection | Intestinal Mucosa - enzymology | Epoprostenol - pharmacology | Transcription, Genetic | Colon | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Nuclear Proteins - genetics | Gene Expression Regulation, Neoplastic - physiology | Prostaglandin-Endoperoxide Synthases - genetics | Colorectal Neoplasms - enzymology | Transcription Factors - physiology | Signal Transduction | Isoenzymes - genetics | Receptors, Cytoplasmic and Nuclear - physiology | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | Membrane Proteins | Epoprostenol - analogs & derivatives | Rectal Neoplasms | Animals | Mice | Nuclear Proteins - physiology | Colorectal Neoplasms - pathology | Colonic Neoplasms | Proteins | Enzymes | Ribonucleic acid--RNA | Ribonucleic acid
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 11/2010, Volume 182, Issue 9, pp. 1153 - 1160
Journal Article
Journal Article