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Diabetes, ISSN 0012-1797, 02/2019, Volume 68, Issue 2, pp. 324 - 336
The Takeda-G-protein-receptor-5 (TGR5) mediates physiological actions of bile acids. Since it was shown that TGR5 is expressed in pancreatic tissue, a direct... 
Protein kinase A | Stimulus-secretion coupling | Calcium channels | Bile acids | Glucagon | Secretion | Cyclic AMP | Tauroursodeoxycholic acid | Kinases | Insulin | Calcium influx | Oleanolic acid | Phospholipase C | Proteins | Adenylate cyclase | Phospholipase | Cell activation | Membrane potential | Pancreas | Glucagon-like peptide 1 | Bile
Journal Article
Scientific reports, ISSN 2045-2322, 07/2019, Volume 9, Issue 1, pp. 10261 - 11
Isolated human islets do not always meet the quality standards required for transplant survival and reliable functional in vitro studies. The formation of... 
PATHWAYS | HUMAN PANCREATIC-ISLETS | HUMAN BETA-CELLS | INHIBITION | RELEVANCE | MULTIDISCIPLINARY SCIENCES | ARCHITECTURE | RECEPTOR | RAT ISLETS | RELEASE | HEALTH | Epinephrine | Cell culture | Glucagon | Secretion | Forskolin | Islet cells | Glucose | Gene expression | Palmitic acid | Insulin | Glucagon-like peptide 1 | Endothelial cells
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, p. e0198016
Journal Article
Diabetologia, ISSN 0012-186X, 11/2017, Volume 60, Issue 11, pp. 2240 - 2251
Obesity-linked ectopic fat accumulation is associated with the development of type 2 diabetes. Whether pancreatic and liver steatosis impairs insulin secretion... 
Beta cells | Fetuin-A | Medicine & Public Health | Human Physiology | Palmitate | Metabolic Diseases | Internal Medicine | Inflammation | Islets | Adipocytes | TLR4 | ISLET INFLAMMATION | MACROPHAGES | HUMANS | ALPHA | BETA-CELL FUNCTION | INHIBITION | ENDOCRINOLOGY & METABOLISM | RESISTANCE | CONTRIBUTES | ADIPOSE-TISSUE | Immunohistochemistry | Inflammation - pathology | Interleukin-8 - genetics | Fatty Liver - metabolism | Toll-Like Receptor 4 | Interleukin-6 - genetics | Fatty Liver - pathology | Humans | Cells, Cultured | Pancreas - pathology | Chemokine CCL2 - genetics | Diabetes Mellitus, Type 2 - metabolism | Pancreas - metabolism | Blotting, Western | Inflammation - metabolism | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | alpha-2-HS-Glycoprotein - metabolism | Mice | Palmitates - metabolism | Insulin Secretion | Type 2 diabetes | Transplantation of organs, tissues, etc | Fatty liver | Insulin | Liver | Transcription factors | Calcium | Glucagon | Cytotoxicity | Confocal microscopy | Palmitic acid | Kinases | Macrophages | Western blotting | Adiponectin | Interleukin 6 | Toll-like receptors | Pancreas | Enzyme-linked immunosorbent assay | Cytokines | Secretion | Diabetes mellitus | c-Jun protein | Metabolism | Gene expression | Polymerase chain reaction | Blindness | Islet cells | Islets of Langerhans | Monocyte chemoattractant protein 1 | Apoptosis
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92238
Journal Article
Journal Article
Journal Article
Molecular Metabolism, ISSN 2212-8778, 07/2019, Volume 25, pp. 1 - 10
Journal Article
Experimental and Clinical Endocrinology & Diabetes, ISSN 0947-7349, 04/2019
Abstract Glucose-stimulated insulin secretion (GSIS) is the gold standard for β-cell function. Both experimental and clinical diabetology, i. e., preceding... 
Article
Journal Article
Journal Article
Journal Article