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eLife, ISSN 2050-084X, 11/2016, Volume 5
Colorectal cancer (CRC) organoids can be derived from almost all CRC patients and therefore capture the genetic diversity of this disease. We assembled a panel... 
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1064 - 1072.e5
Background & Aims Lynch syndrome, a nonpolyposis form of hereditary colorectal cancer, is caused by inherited defects in DNA mismatch repair (MMR) genes. Most... 
Gastroenterology and Hepatology | Carcinogen | Defective Mismatch Repair | Cancer Risk | Dietary Factors | STEM-CELLS | NONPOLYPOSIS COLORECTAL-CANCER | COLON | MLH1 | INACTIVATION | DNA | MICE | AGENTS | MUTATIONS | METHYLATION TOLERANCE | GASTROENTEROLOGY & HEPATOLOGY | Intestines - drug effects | Adenocarcinoma - pathology | Adenoma - genetics | Neoplastic Stem Cells - drug effects | Adenocarcinoma - chemically induced | Colorectal Neoplasms, Hereditary Nonpolyposis - pathology | Male | Cell Transformation, Neoplastic - chemically induced | Intestines - metabolism | Adenoma - metabolism | Adenocarcinoma - metabolism | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | MutS Homolog 2 Protein - deficiency | Dacarbazine - analogs & derivatives | Neoplastic Stem Cells - pathology | Female | Adenocarcinoma - genetics | Disease Models, Animal | Intestines - pathology | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Risk Factors | DNA Mismatch Repair - drug effects | MutS Homolog 2 Protein - genetics | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Colorectal Neoplasms, Hereditary Nonpolyposis - metabolism | Animals | Adenoma - pathology | Cell Proliferation - drug effects | Colorectal Neoplasms, Hereditary Nonpolyposis - chemically induced | Cell Transformation, Neoplastic - pathology | Dacarbazine - toxicity | Adenoma - chemically induced | Temozolomide | Statistics | Analysis | Colorectal cancer | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
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