2016, 1st ed. 2017, ISBN 9783319418384, 1 volume (x, 330 pages)
More than two third area of our planet is covered by oceans and assessment of marine biodiversity is a challenging task. With the increasing global population,...
Marine biodiversity conservation | marine plants | marine fish | Conservation | DNA barcoding | Empreintes génétiques | DNA fingerprinting | marine invertebrates | Biodiversité marine | Biology, life sciences | Bioinformatics | Life Sciences | Biochemistry, general | Nature Conservation | Ecology | Marine & Freshwater Sciences | Animal Genetics and Genomics
Marine biodiversity conservation | marine plants | marine fish | Conservation | DNA barcoding | Empreintes génétiques | DNA fingerprinting | marine invertebrates | Biodiversité marine | Biology, life sciences | Bioinformatics | Life Sciences | Biochemistry, general | Nature Conservation | Ecology | Marine & Freshwater Sciences | Animal Genetics and Genomics
Book
2007, ISBN 9780849327940, 232
Book
1974, 1st ed., with documents. --, ISBN 0883865688, xxiv, 301
Book
1986, ISBN 9788170183730, x, 141 p., [54] p. of plates
Book
Book
Cold Spring Harbor perspectives in biology, 2009, Volume 1, Issue 4, p. a000034
Nuclear factor-kappaB (NF-kappaB) consists of a family of transcription factors that play critical roles in inflammation, immunity, cell proliferation,...
Transcription Factors - metabolism | Animals | Cell Proliferation | Immune System | Signal Transduction | Models, Biological | Humans | Gene Expression Regulation | Cytosol - metabolism | NF-kappa B - metabolism | Protein Processing, Post-Translational | Dimerization
Transcription Factors - metabolism | Animals | Cell Proliferation | Immune System | Signal Transduction | Models, Biological | Humans | Gene Expression Regulation | Cytosol - metabolism | NF-kappa B - metabolism | Protein Processing, Post-Translational | Dimerization
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 01/2017, Volume 101, Issue 1, pp. 107 - 119
LPS is a potent trigger of macrophage‐mediated inflammation. However, prolonged exposure to LPS induces a state of tolerance that reprograms the inflammatory...
lipopolysaccharide | chromatin | inflammation | epigenetics | endotoxin | Epigenetics | Lipopolysaccharide | Chromatin | Inflammation | Endotoxin | LONG NONCODING RNAS | DNA METHYLATION | PERITONEAL-MACROPHAGES | IMMUNOLOGY | LIPOPOLYSACCHARIDE TOLERANCE | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | INDUCED EPIGENETIC CHANGES | ENDOTOXIN TOLERANCE | GENE-EXPRESSION | TNF-ALPHA | HEMATOLOGY | NF-KAPPA-B | Animals | Immune Tolerance - immunology | Humans | Immunologic Memory | DNA Methylation - genetics | Toll-Like Receptors - metabolism | Immunity, Innate | Lipopolysaccharides - immunology | Signal transduction | Signaling | Molecular modelling | Disseminated infection | Inflammatory response | Transduction | Exposure | Gene expression | Macrophages | Lipopolysaccharides | Chromatin remodeling
lipopolysaccharide | chromatin | inflammation | epigenetics | endotoxin | Epigenetics | Lipopolysaccharide | Chromatin | Inflammation | Endotoxin | LONG NONCODING RNAS | DNA METHYLATION | PERITONEAL-MACROPHAGES | IMMUNOLOGY | LIPOPOLYSACCHARIDE TOLERANCE | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | INDUCED EPIGENETIC CHANGES | ENDOTOXIN TOLERANCE | GENE-EXPRESSION | TNF-ALPHA | HEMATOLOGY | NF-KAPPA-B | Animals | Immune Tolerance - immunology | Humans | Immunologic Memory | DNA Methylation - genetics | Toll-Like Receptors - metabolism | Immunity, Innate | Lipopolysaccharides - immunology | Signal transduction | Signaling | Molecular modelling | Disseminated infection | Inflammatory response | Transduction | Exposure | Gene expression | Macrophages | Lipopolysaccharides | Chromatin remodeling
Journal Article
Langmuir, ISSN 0743-7463, 10/2006, Volume 22, Issue 22, pp. 9322 - 9328
Recombinant Escherichia coli (E. coli) bacteria expressing green fluorescent protein (GFP) was used as a model system to investigate the antimicrobial...
CHEMISTRY, PHYSICAL | ANTIBACTERIAL PROPERTIES | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Microscopy, Electron, Transmission | Silver - pharmacology | Gene Expression | Escherichia coli - drug effects | Metal Nanoparticles - chemistry | Silver - chemistry | Green Fluorescent Proteins - genetics | Recombinant Proteins - genetics | Escherichia coli - genetics | Models, Biological | Anti-Bacterial Agents - chemistry | Escherichia coli - metabolism | Plasmids - genetics | Anti-Bacterial Agents - pharmacology | Metal Nanoparticles - ultrastructure
CHEMISTRY, PHYSICAL | ANTIBACTERIAL PROPERTIES | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Microscopy, Electron, Transmission | Silver - pharmacology | Gene Expression | Escherichia coli - drug effects | Metal Nanoparticles - chemistry | Silver - chemistry | Green Fluorescent Proteins - genetics | Recombinant Proteins - genetics | Escherichia coli - genetics | Models, Biological | Anti-Bacterial Agents - chemistry | Escherichia coli - metabolism | Plasmids - genetics | Anti-Bacterial Agents - pharmacology | Metal Nanoparticles - ultrastructure
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0129808
Background Epigenetic and genetic alteration plays a major role to the development of head and neck squamous cell carcinoma (HNSCC). Consumption of tobacco...
POLYMORPHISMS | CPG ISLAND METHYLATION | DNA METHYLATION | BIOMARKER | BRAF MUTATION | TUMOR-RELATED GENES | MICROSATELLITE INSTABILITY | MULTIDISCIPLINARY SCIENCES | SQUAMOUS-CELL CARCINOMA | PHENOTYPE | RISK | Epigenomics | Carcinoma, Squamous Cell - genetics | Humans | Middle Aged | Male | DNA Methylation - genetics | DNA Repair - genetics | Multiplex Polymerase Chain Reaction | Promoter Regions, Genetic - genetics | Papillomavirus Infections | India | Young Adult | Squamous Cell Carcinoma of Head and Neck | Carcinoma, Squamous Cell - mortality | Polymorphism, Restriction Fragment Length | Tobacco Use | Aged, 80 and over | Adult | Female | Head and Neck Neoplasms - genetics | Aged | Polymorphism, Single Nucleotide | Head and Neck Neoplasms - mortality | Cluster analysis | Transcription | Genes | Viruses | Chewing | Kinases | Inactivation | Tissues | Gene polymorphism | Human papillomavirus | Glutathione transferase | DNA methylation | CpG islands | Phenotypes | Squamous cell carcinoma | Head | Deactivation | MLH1 protein | Cytochrome P450 | Mastication | GSTM1 protein | Gene expression | Clustering | Survival | Loci | Polymerase chain reaction | Tobacco | Restriction fragment length polymorphism | Epigenetics | Head and neck cancer | Mutation | Methylation | Polymorphism | Tumors | Smoking
POLYMORPHISMS | CPG ISLAND METHYLATION | DNA METHYLATION | BIOMARKER | BRAF MUTATION | TUMOR-RELATED GENES | MICROSATELLITE INSTABILITY | MULTIDISCIPLINARY SCIENCES | SQUAMOUS-CELL CARCINOMA | PHENOTYPE | RISK | Epigenomics | Carcinoma, Squamous Cell - genetics | Humans | Middle Aged | Male | DNA Methylation - genetics | DNA Repair - genetics | Multiplex Polymerase Chain Reaction | Promoter Regions, Genetic - genetics | Papillomavirus Infections | India | Young Adult | Squamous Cell Carcinoma of Head and Neck | Carcinoma, Squamous Cell - mortality | Polymorphism, Restriction Fragment Length | Tobacco Use | Aged, 80 and over | Adult | Female | Head and Neck Neoplasms - genetics | Aged | Polymorphism, Single Nucleotide | Head and Neck Neoplasms - mortality | Cluster analysis | Transcription | Genes | Viruses | Chewing | Kinases | Inactivation | Tissues | Gene polymorphism | Human papillomavirus | Glutathione transferase | DNA methylation | CpG islands | Phenotypes | Squamous cell carcinoma | Head | Deactivation | MLH1 protein | Cytochrome P450 | Mastication | GSTM1 protein | Gene expression | Clustering | Survival | Loci | Polymerase chain reaction | Tobacco | Restriction fragment length polymorphism | Epigenetics | Head and neck cancer | Mutation | Methylation | Polymorphism | Tumors | Smoking
Journal Article
COLD SPRING HARBOR PERSPECTIVES IN BIOLOGY, ISSN 1943-0264, 10/2009, Volume 1, Issue 4, pp. 5 - 18
Nuclear factor-kappa B (NF-kappa B) consists of a family of transcription factors that play critical roles in inflammation, immunity, cell proliferation,...
KINASE-ALPHA | DNA-BINDING | BETA-TRCP | UBIQUITIN LIGASE | CRYSTAL-STRUCTURE | INDUCIBLE NUCLEAR-PROTEIN | GENE-EXPRESSION | IKK-ALPHA | SIGNALING MODULE | DEPENDENT DEGRADATION | CELL BIOLOGY | 040
KINASE-ALPHA | DNA-BINDING | BETA-TRCP | UBIQUITIN LIGASE | CRYSTAL-STRUCTURE | INDUCIBLE NUCLEAR-PROTEIN | GENE-EXPRESSION | IKK-ALPHA | SIGNALING MODULE | DEPENDENT DEGRADATION | CELL BIOLOGY | 040
Journal Article
Nature Reviews Immunology, ISSN 1474-1733, 06/2011, Volume 11, Issue 6, pp. 389 - 402
The innate immune system has a key role in the mammalian immune response. Recent research has demonstrated that mitochondria participate in a broad range of...
FORMYL PEPTIDE RECEPTORS | OXYGEN SPECIES PRODUCTION | REACTIVE OXYGEN | NLRP3 INFLAMMASOME ACTIVATION | ANTIVIRAL SIGNALING PROTEIN | HEPATITIS-C-VIRUS | INTERFERON REGULATORY FACTOR-3 | RIG-I | UNCOUPLING PROTEIN-2 | IMMUNOLOGY | NF-KAPPA-B | Reactive Oxygen Species - metabolism | Virus Diseases - immunology | Humans | Virus Diseases - virology | Mitochondria - immunology | Mitochondria - metabolism | Immunity, Innate - immunology | Signal Transduction - immunology | Models, Immunological | Animals | Bacterial Infections - immunology | Mitochondrial Proteins - metabolism | Reactive Oxygen Species - immunology | Bacterial Infections - microbiology | Mitochondrial Proteins - immunology | Physiological aspects | Natural immunity | Mitochondrial DNA | Research | Apoptosis
FORMYL PEPTIDE RECEPTORS | OXYGEN SPECIES PRODUCTION | REACTIVE OXYGEN | NLRP3 INFLAMMASOME ACTIVATION | ANTIVIRAL SIGNALING PROTEIN | HEPATITIS-C-VIRUS | INTERFERON REGULATORY FACTOR-3 | RIG-I | UNCOUPLING PROTEIN-2 | IMMUNOLOGY | NF-KAPPA-B | Reactive Oxygen Species - metabolism | Virus Diseases - immunology | Humans | Virus Diseases - virology | Mitochondria - immunology | Mitochondria - metabolism | Immunity, Innate - immunology | Signal Transduction - immunology | Models, Immunological | Animals | Bacterial Infections - immunology | Mitochondrial Proteins - metabolism | Reactive Oxygen Species - immunology | Bacterial Infections - microbiology | Mitochondrial Proteins - immunology | Physiological aspects | Natural immunity | Mitochondrial DNA | Research | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60996
Background: Esophageal squamous cell carcinoma (ESCC) develops as a result of complex epigenetic, genetic and environmental interactions. Epigenetic changes...
PROMOTER HYPERMETHYLATION | RISK-FACTORS | POLYMORPHISMS | DNA METHYLATION | ALCOHOL | MULTIDISCIPLINARY SCIENCES | CANCER-RISK | CIGARETTE-SMOKE | BETEL QUID | TUMORS | TOBACCO | Esophageal Neoplasms - etiology | Promoter Regions, Genetic | Risk Assessment | Epigenesis, Genetic | Humans | Middle Aged | Risk Factors | Male | Gene-Environment Interaction | Case-Control Studies | India | Carcinoma, Squamous Cell - etiology | DNA Methylation | Glutathione Transferase - genetics | Aged, 80 and over | Adult | Female | Aged | Polymorphism, Single Nucleotide | Esophageal Squamous Cell Carcinoma | Smoking | Epigenetic inheritance | Squamous cell carcinoma | Genes | Development and progression | Risk factors | Genetic polymorphisms | Tobacco chewing | Glutathione transferase | Analysis | Physiological aspects | Genetic aspects | Methylation | Esophageal cancer | Biotechnology | Alcohol | Environmental changes | Family medical history | Chewing | Kinases | Gene polymorphism | GSTT1 protein | Carcinogens | Cell cycle | DNA methylation | Population | Genotypes | Deoxyribonucleic acid--DNA | Mastication | Regression analysis | GSTM1 protein | Risk analysis | Metabolism | Cigarettes | Environmental effects | Esophagus | Tobacco | Epigenetics | Alcoholic beverages | Tumor suppressor genes | Aberration | Health risk assessment | Habits | Tumors | Polymorphism | Deoxyribonucleic acid | DNA
PROMOTER HYPERMETHYLATION | RISK-FACTORS | POLYMORPHISMS | DNA METHYLATION | ALCOHOL | MULTIDISCIPLINARY SCIENCES | CANCER-RISK | CIGARETTE-SMOKE | BETEL QUID | TUMORS | TOBACCO | Esophageal Neoplasms - etiology | Promoter Regions, Genetic | Risk Assessment | Epigenesis, Genetic | Humans | Middle Aged | Risk Factors | Male | Gene-Environment Interaction | Case-Control Studies | India | Carcinoma, Squamous Cell - etiology | DNA Methylation | Glutathione Transferase - genetics | Aged, 80 and over | Adult | Female | Aged | Polymorphism, Single Nucleotide | Esophageal Squamous Cell Carcinoma | Smoking | Epigenetic inheritance | Squamous cell carcinoma | Genes | Development and progression | Risk factors | Genetic polymorphisms | Tobacco chewing | Glutathione transferase | Analysis | Physiological aspects | Genetic aspects | Methylation | Esophageal cancer | Biotechnology | Alcohol | Environmental changes | Family medical history | Chewing | Kinases | Gene polymorphism | GSTT1 protein | Carcinogens | Cell cycle | DNA methylation | Population | Genotypes | Deoxyribonucleic acid--DNA | Mastication | Regression analysis | GSTM1 protein | Risk analysis | Metabolism | Cigarettes | Environmental effects | Esophagus | Tobacco | Epigenetics | Alcoholic beverages | Tumor suppressor genes | Aberration | Health risk assessment | Habits | Tumors | Polymorphism | Deoxyribonucleic acid | DNA
Journal Article
Genes and Development, ISSN 0890-9369, 02/2012, Volume 26, Issue 3, pp. 203 - 234
The ability to sense and adjust to the environment is crucial to life. For multicellular organisms, the ability to respond to external changes is essential not...
IKK | NF-κB | Signaling | Inflammation | Cancer | signaling | TOLL-LIKE RECEPTOR | P105 REQUIRES PHOSPHORYLATION | MESSENGER-RNA STABILIZATION | INDUCIBLE NUCLEAR-PROTEIN | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | SELECTIVE GENE ACTIVATION | CELL BIOLOGY | SIGNAL-INDUCED UBIQUITINATION | NF-kappa B | UNANCHORED POLYUBIQUITIN CHAINS | inflammation | GENETICS & HEREDITY | cancer | CANDIDATE PROTOONCOGENE BCL-3 | UBIQUITIN-PROTEASOME PATHWAY | Animals | NF-kappa B - genetics | Signal Transduction | Immune System - metabolism | Humans | Gene Expression Regulation | NF-kappa B - metabolism | Enzyme Activation | Review
IKK | NF-κB | Signaling | Inflammation | Cancer | signaling | TOLL-LIKE RECEPTOR | P105 REQUIRES PHOSPHORYLATION | MESSENGER-RNA STABILIZATION | INDUCIBLE NUCLEAR-PROTEIN | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | SELECTIVE GENE ACTIVATION | CELL BIOLOGY | SIGNAL-INDUCED UBIQUITINATION | NF-kappa B | UNANCHORED POLYUBIQUITIN CHAINS | inflammation | GENETICS & HEREDITY | cancer | CANDIDATE PROTOONCOGENE BCL-3 | UBIQUITIN-PROTEASOME PATHWAY | Animals | NF-kappa B - genetics | Signal Transduction | Immune System - metabolism | Humans | Gene Expression Regulation | NF-kappa B - metabolism | Enzyme Activation | Review
Journal Article
Immunological Reviews, ISSN 0105-2896, 03/2013, Volume 252, Issue 1, pp. 41 - 51
Summary The nuclear factor‐κB (NF‐κB) family of transcription factors plays important roles in various biological processes including apoptosis, stress...
NF‐κB | T‐cell development | T‐cell subsets | T‐cell signaling | NF-κB | T-cell development | T-cell subsets | T-cell signaling | IMMUNE-RESPONSE | AIRWAY INFLAMMATION | RECEPTOR | C-REL | IMMUNOLOGY | NF-kappa B | FOLLICULAR-HELPER-CELLS | IN-VIVO | GENE-EXPRESSION | GENERATION | FOXP3 GENE | TRANSCRIPTION FACTOR | Adaptive Immunity | T-Lymphocyte Subsets - immunology | T-Lymphocyte Subsets - cytology | Gene Expression Regulation - immunology | Lymphocyte Activation | CD4-Positive T-Lymphocytes - cytology | Humans | NF-kappa B - immunology | Transcription Factors - genetics | CD4-Positive T-Lymphocytes - immunology | Signal Transduction - immunology | Animals | NF-kappa B - genetics | Transcription, Genetic | Cell Differentiation | Mice | Transcription Factors - immunology | Cytokines - biosynthesis | Cytokines - immunology | Pathogens | Transcription factors | Cytokines | Immunomodulation | Immunoregulation | Gene regulation | Helper cells | Inflammation | Lymphocytes T | Stress | CD4 antigen | Thymus | Signal transduction | NF- Kappa B protein | Microenvironments | Chemokines | Apoptosis
NF‐κB | T‐cell development | T‐cell subsets | T‐cell signaling | NF-κB | T-cell development | T-cell subsets | T-cell signaling | IMMUNE-RESPONSE | AIRWAY INFLAMMATION | RECEPTOR | C-REL | IMMUNOLOGY | NF-kappa B | FOLLICULAR-HELPER-CELLS | IN-VIVO | GENE-EXPRESSION | GENERATION | FOXP3 GENE | TRANSCRIPTION FACTOR | Adaptive Immunity | T-Lymphocyte Subsets - immunology | T-Lymphocyte Subsets - cytology | Gene Expression Regulation - immunology | Lymphocyte Activation | CD4-Positive T-Lymphocytes - cytology | Humans | NF-kappa B - immunology | Transcription Factors - genetics | CD4-Positive T-Lymphocytes - immunology | Signal Transduction - immunology | Animals | NF-kappa B - genetics | Transcription, Genetic | Cell Differentiation | Mice | Transcription Factors - immunology | Cytokines - biosynthesis | Cytokines - immunology | Pathogens | Transcription factors | Cytokines | Immunomodulation | Immunoregulation | Gene regulation | Helper cells | Inflammation | Lymphocytes T | Stress | CD4 antigen | Thymus | Signal transduction | NF- Kappa B protein | Microenvironments | Chemokines | Apoptosis
Journal Article
Nature Immunology, ISSN 1529-2908, 08/2011, Volume 12, Issue 8, pp. 695 - 708
NF-kappa B transcription factors are critical regulators of immunity, stress responses, apoptosis and differentiation. A variety of stimuli coalesce on...
TRANSCRIPTION FACTORS | INDUCED GENE-EXPRESSION | INNATE IMMUNE-RESPONSES | TOLL-LIKE RECEPTORS | NECROTIC CELL-DEATH | NECROSIS-FACTOR-ALPHA | IKK-ALPHA | IMMUNOLOGY | NF-KAPPA-B1 P105 | INSULIN-RECEPTOR SUBSTRATE-1 | TRAF-INTERACTING PROTEIN | Animals | NF-kappa B - genetics | Signal Transduction | Humans | Transcriptional Activation | NF-kappa B - physiology | Cell Communication - physiology | NF-kappa B - metabolism
TRANSCRIPTION FACTORS | INDUCED GENE-EXPRESSION | INNATE IMMUNE-RESPONSES | TOLL-LIKE RECEPTORS | NECROTIC CELL-DEATH | NECROSIS-FACTOR-ALPHA | IKK-ALPHA | IMMUNOLOGY | NF-KAPPA-B1 P105 | INSULIN-RECEPTOR SUBSTRATE-1 | TRAF-INTERACTING PROTEIN | Animals | NF-kappa B - genetics | Signal Transduction | Humans | Transcriptional Activation | NF-kappa B - physiology | Cell Communication - physiology | NF-kappa B - metabolism
Journal Article