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Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 298 - 305
Journal Article
PLoS Genetics, ISSN 1553-7390, 09/2014, Volume 10, Issue 9, pp. e1004566 - e1004566
Journal Article
Science, ISSN 0036-8075, 12/2015, Volume 350, Issue 6266, pp. 1391 - 1396
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, pp. e0179762 - e0179762
During rheumatoid arthritis (RA), Tumor Necrosis Factor (TNF) activates fibroblast-like synoviocytes (FLS) inducing in a temporal order a constellation of... 
ACTIVATED PROTEIN-KINASE | TRISTETRAPROLIN | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | GENE-EXPRESSION | BINDING | Humans | RNA Stability - drug effects | RNA, Messenger - metabolism | Synoviocytes - drug effects | Arthritis, Rheumatoid - metabolism | RNA - isolation & purification | Synoviocytes - metabolism | Cyclooxygenase 2 - genetics | Cytokines - genetics | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | RNA - metabolism | Cytokines - metabolism | Interleukin-6 - genetics | Synoviocytes - cytology | Cells, Cultured | Matrix Metalloproteinase 3 - metabolism | RNA - chemistry | Chemokines - genetics | Arthritis, Rheumatoid - pathology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Sequence Analysis, RNA | Cyclooxygenase 2 - metabolism | Chemokines - metabolism | Fibroblasts - cytology | Matrix Metalloproteinase 3 - genetics | RNA sequencing | Usage | Messenger RNA | Tumor necrosis factor | Rheumatoid arthritis | Analysis | Genetic aspects | Inflammation | Research | Genetic transcription | Synovitis | Regulations | Transcription | Stabilization | Genes | Genomics | Switches | Genomes | Biology | Arthritis | Kinases | Gene sequencing | Interleukin 6 | Proteins | Surgery | Fibroblasts | Tumor necrosis factor-TNF | Temporal variations | Bioinformatics | Interleukin 8 | Cytokines | mRNA turnover | MAP kinase | Gene expression | Synoviocytes | Studies | Molecular modelling | Ribonucleic acids | Decay | Dynamic stability | Monocyte chemoattractant protein 1 | mRNA stability | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 09/2017, Volume 18, Issue 10, pp. 1104 - 1116
Cross-regulation of Toll-like receptor (TLR) responses by cytokines is essential for effective host defense, avoidance of toxicity and homeostasis, but the... 
TRANSCRIPTION FACTORS | RESPONSES | CHROMATIN | LANDSCAPE | TOLERANCE | CELL IDENTITY | ENVIRONMENT | ENDOTOXIN | IMMUNOLOGY | ENHANCERS | POLARIZATION | Tumor Necrosis Factor-alpha - metabolism | Epigenesis, Genetic | Humans | Lupus Erythematosus, Systemic - metabolism | Monocytes - metabolism | Gene Expression Profiling | Monocytes - immunology | Lipopolysaccharides - immunology | Inflammation - metabolism | Chromatin Immunoprecipitation | Epigenomics - methods | Nucleotide Motifs | Lupus Erythematosus, Systemic - immunology | Interferon Type I - metabolism | Binding Sites | Cytokines - genetics | Macrophages - immunology | Promoter Regions, Genetic | Computational Biology - methods | Cytokines - metabolism | Signal Transduction | Gene Expression Regulation | Chromatin Assembly and Disassembly | Inflammation - etiology | Toll-Like Receptor 4 - metabolism | Protein Transport | Transcription Factors - metabolism | Macrophages - metabolism | Lupus Erythematosus, Systemic - genetics | Protein Binding | High-Throughput Nucleotide Sequencing | Cluster Analysis | Interferon | Genetic aspects | Immune response | Tumor necrosis factor | Health aspects | Chromatin | Cytokines | Toxicity | Genes | Crosstalk | Homeostasis | Priming | Inflammation | TLR4 protein | Macrophages | Immune systems | Proteins | Gene silencing | Cell activation | Coding | Cascades | Toll-like receptors | Index Medicus
Journal Article