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1. Full Text Cerebral preconditioning and ischaemic tolerance
by Gidday, Jeffrey M
Nature reviews. Neuroscience, ISSN 1471-003X, 06/2006, Volume 7, Issue 6, pp. 437 - 448
Neurology | Pharmacology. Drug treatments | Biological and medical sciences | Neuroprotective agent | Vascular diseases and vascular malformations of the nervous system | Medical sciences | Neuropharmacology | Brain - blood supply | Animals | Brain - physiopathology | Humans | Brain Ischemia - pathology | Adaptation, Physiological - physiology | Ischemic Preconditioning | Signal Transduction - physiology | Brain Ischemia - physiopathology | Apoptosis - physiology | Index Medicus
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2. Full Text Hypoxic preconditioning protects human brain endothelium from ischemic apoptosis by Akt-dependent survivin activation
by Yunhong Zhang and Tae S. Park and Jeffrey M. Gidday
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 06/2007, Volume 292, Issue 6, pp. 2573 - 2581
Stroke | Cell survival | Phosphatidylinositol 3-kinase | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Reperfusion Injury - etiology | Microcirculation - metabolism | Phosphorylation | Microtubule-Associated Proteins - metabolism | Humans | Repressor Proteins | Brain Ischemia - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Oxygen - metabolism | Cell Hypoxia | Brain - blood supply | Time Factors | Proto-Oncogene Proteins c-akt - metabolism | Reperfusion Injury - metabolism | Brain Ischemia - complications | Inhibitor of Apoptosis Proteins | Reperfusion Injury - pathology | Signal Transduction | Cell Survival | Endothelial Cells - metabolism | Cells, Cultured | Ischemic Preconditioning | Brain Ischemia - physiopathology | Glucose - deficiency | Phenotype | Microcirculation - pathology | Reperfusion Injury - prevention & control | Brain Ischemia - pathology | Reperfusion Injury - physiopathology | Endothelial Cells - pathology | Apoptosis | Brain | Neurology | Hypoxia | Kinases | Cells | Index Medicus
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3. Full Text CCL2 upregulation triggers hypoxic preconditioning-induced protection from stroke
by Stowe, Ann M and Wacker, Bradley K and Cravens, Petra D and Perfater, Jennifer L and Li, Min K and Hu, Ruilong and Freie, Angela B and Stüve, Olaf and Gidday, Jeffrey M
Journal of neuroinflammation, ISSN 1742-2094, 02/2012, Volume 9, Issue 1, pp. 33 - 33
Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Receptors, CCR2 - genetics | Central Nervous System - metabolism | Chemokine CCL2 - immunology | Central Nervous System - pathology | Male | NADH Dehydrogenase - genetics | Phosphopyruvate Hydratase - metabolism | Green Fluorescent Proteins - genetics | Glial Fibrillary Acidic Protein - metabolism | Brain Infarction - prevention & control | Ischemic Preconditioning - methods | Time Factors | Infarction, Middle Cerebral Artery - complications | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Up-Regulation - physiology | Chemokine CCL2 - metabolism | Monocytes - physiology | Disease Models, Animal | Green Fluorescent Proteins - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Immunoglobulin G - therapeutic use | Chemokine CCL2 - genetics | Infarction, Middle Cerebral Artery - pathology | NADH Dehydrogenase - metabolism | Chemokine CCL2 - deficiency | Mice, Knockout | Brain Infarction - etiology | Receptors, CCR2 - metabolism | Animals | Mice | Stroke (Disease) | Prevention | Physiological aspects | Research | Chemokines | Viral antibodies | Brain | RNA | Neurons | Antibodies | B cells | T cells | Kinases | Gene expression | Experiments | Manuscripts | Medicine | Studies | Signal transduction | Genotype & phenotype | Ischemia | Rodents | Epigenetics | Ligands | Hypoxia | Drug dosages | Index Medicus
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4. Full Text Repetitive hypoxia extends endogenous neurovascular protection for stroke
by Stowe, Ann M and Altay, Tamer and Freie, Angela B and Gidday, Jeffrey M
Annals of neurology, ISSN 0364-5134, 06/2011, Volume 69, Issue 6, pp. 975 - 985
Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Biological and medical sciences | Vascular diseases and vascular malformations of the nervous system | Medical sciences | Infarction, Middle Cerebral Artery - prevention & control | Leukocytes - pathology | Cerebrovascular Circulation - physiology | Blood-Brain Barrier - physiopathology | Male | Green Fluorescent Proteins - genetics | L-Selectin - genetics | L-Selectin - metabolism | Time Factors | E-Selectin - genetics | Infarction, Middle Cerebral Artery - complications | Vascular Cell Adhesion Molecule-1 - genetics | Disease Models, Animal | Cerebrovascular Circulation - drug effects | Ischemic Preconditioning | E-Selectin - metabolism | Mice, Transgenic | Hippocampus - pathology | Functional Laterality | Infarction, Middle Cerebral Artery - pathology | Hippocampus - metabolism | Animals | Tumor Necrosis Factor-alpha - adverse effects | Analysis of Variance | Mice | Hematocrit - methods | Vascular Cell Adhesion Molecule-1 - metabolism | Medical research | Hypoxia | Rodents | Index Medicus
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5. Full Text Hypoxic preconditioning induces stroke tolerance in mice via a cascading HIF, sphingosine kinase, and CCL2 signaling pathway
by Wacker, Bradley K and Perfater, Jennifer L and Gidday, Jeffrey M
Journal of neurochemistry, ISSN 0022-3042, 12/2012, Volume 123, Issue 6, pp. 954 - 962
sphingosine‐1‐phosphate | chemokines | neuroprotection | bioactive lipids | sphingosine-1-phosphate | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Fundamental and applied biological sciences. Psychology | Signal transduction | Neurology | Biological and medical sciences | Cell physiology | Vascular diseases and vascular malformations of the nervous system | Molecular and cellular biology | Medical sciences | Male | Ischemic Preconditioning - methods | Chemokine CCL2 - physiology | Female | Hypoxia-Ischemia, Brain - therapy | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Receptors, Chemokine - genetics | Hypoxia-Ischemia, Brain - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Chemokine CCL2 - genetics | Phosphotransferases (Alcohol Group Acceptor) - genetics | Stroke - drug therapy | Chemokine CCL2 - deficiency | Mice, Knockout | Receptors, Chemokine - deficiency | Stroke - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Phenotype | Stroke - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | CX3C Chemokine Receptor 1 | Stroke (Disease) | Gene expression | Sphingosine | Proteins | Neurochemistry | Brain | Stroke | Rodents | Kinases | Index Medicus | Hypoxia-inducible factors | Translation | sphingosine kinase | Data processing | cobalt chloride | Cobalt | epigenetics | Sphingosine 1-phosphate | Ischemia | Feedback | FTY720 | Hypoxia | genomics | Chemokines | Monocyte chemoattractant protein 1
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6. Full Text Extending injury- and disease-resistant CNS phenotypes by repetitive epigenetic conditioning
by Gidday, Jeffrey M
Frontiers in neurology, ISSN 1664-2295, 2015, Volume 6, pp. 42 - 42
Epigenetics | Neuroprotection | Retina | Stroke | Postconditioning | Preconditioning | postconditioning | epigenetics | preconditioning
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7. Full Text Enhancing and Extending Biological Performance and Resilience
by Leak, Rehana K and Calabrese, Edward J and Kozumbo, Walter J and Gidday, Jeffrey M and Johnson, Thomas E and Mitchell, James R and Ozaki, C. Keith and Wetzker, Reinhard and Bast, Aalt and Belz, Regina G and Botker, Hans E and Koch, Sebastian and Mattson, Mark P and Simon, Roger P and Jirtle, Randy L and Andersen, Melvin E
Dose-response, ISSN 1559-3258, 08/2018, Volume 16, Issue 3, pp. 1559325818784501 - 1559325818784501
HYDROGEN-SULFIDE PRODUCTION | adaptation | RANDOMIZED CONTROLLED-TRIAL | endurance | epigenetics | preconditioning | fitness | dose-response | SPINAL-CORD-INJURY | hormesis | tolerance | dietary restriction | ELEVATION MYOCARDIAL-INFARCTION | stress | caloric restriction | GERBIL HIPPOCAMPAL-NEURONS | HORMETIC DOSE RESPONSES | FOCAL CEREBRAL-ISCHEMIA | REPETITIVE INTERMITTENT HYPOXIA | biphasic | ISCHEMIA-REPERFUSION INJURY | J-shaped | PERCUTANEOUS CORONARY INTERVENTION | conditioning | U-shaped | Tolerance | Biphasic | Stress | Response | Dietary restriction | Epigenetics | Endurance | Hormesis | Conditioning | Dose | Preconditioning | Adaptation | Caloric restriction | Fitness | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Human performance | Defense mechanisms | Researchers
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