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Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 12/2017, Volume 70, Issue 23, pp. 2946 - 2947
Journal Article
BMC Cardiovascular Disorders, ISSN 1471-2261, 02/2013, Volume 13, Issue 1, pp. 12 - 12
Background: Increased levels of cardio-enriched microRNAs (miRNAs) have been described in patients with myocardial infarction (MI). We wanted to evaluate the... 
Myocardial infarction | Biomarker | Prognosis | Acute coronary syndrome | MiRNA | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | ELEVATION | INJURY | BIOMARKERS | DISEASE | EXPRESSION | TROPONIN | Myocardial Infarction - blood | Myocardial Infarction - genetics | Predictive Value of Tests | Up-Regulation | Myocardial Infarction - mortality | Ventricular Function, Left | Area Under Curve | Humans | Middle Aged | Heart Failure - physiopathology | Male | Systole | Genetic Testing - methods | Troponin T - blood | Time Factors | Ventricular Dysfunction, Left - blood | Ventricular Dysfunction, Left - genetics | Aged, 80 and over | Female | Myocardial Infarction - physiopathology | Odds Ratio | Heart Failure - etiology | Real-Time Polymerase Chain Reaction | Myocardial Infarction - surgery | Risk Factors | Heart Failure - genetics | Genetic Markers | Ventricular Dysfunction, Left - physiopathology | Discriminant Analysis | Stroke Volume | Myocardial Infarction - complications | Analysis of Variance | Coronary Artery Bypass | Heart Arrest, Induced | Aged | MicroRNAs - blood | Heart | MicroRNA | Coronary artery bypass | Cardiac patients | Mortality | Health aspects | Heart attack | Heart failure | Plasma | Heart attacks | Disease | Kinases | Gene expression | Ischemia | Coronary vessels | Biomarkers | Standard deviation | Acute coronary syndromes | Thermal cycling | Veins & arteries | Index Medicus
Journal Article
Journal Article
by Smith, J Gustav and Felix, Janine and Morrison, Alanna and Kalogeropoulos, Aneas and Trompet, Stella and Wilk, Jemma and Gidlöf, O and Wang, X and Morley, Michael and Mendelson, M and Joehanes, Roby and Ligthart, Symen and Shan, X and Bis, Joshua and Wang, Ying and Sjögren, Marketa and Ngwa, J.S and Brandimarto, Jeffrey and Stott, David. J and Aguilar, D and Rice, Kenneth and Sesso, H and Demissie, Serkalem and Buckley, Brendan M and Taylor, Kent and Ford, Ian and Yao, Chen and Liu, C and Sotoodehnia, Nona and Harst, Pim and Stricker, Bruno and Kritchevsky, Stephen and Liu, Yongmei and Gaziano, J. Michael and Hofman, Albert and Moravec, C.S and Uitterlinden, Ané and Kellis, Manolis and Meurs, Joyce and Margulies, K.B and Dehghan, Abbas and Levy, Daniel and Olde, B and Psaty, Bruce and Cupples, Aienne and Jukema, Jan Wouter and Djoussé, Luc and Franco, Oscar and Boerwinkle, Eric and Boyer, L.A and Newton-Cheh, Christopher and Butler, J and Vasan, Ramachanan Srini and Cappola, T.P and Smith, N.L and CHARGE-SCD Consortium and EchoGen Consortium and QT-IGC Consortium and CHARGE-QRS Consortium and CHARGE-QRS consortium and CHARGE-SCD consortium and EchoGen consortium and QT-IGC consortium and Kardiologi and Lund University and Heart Failure and Mechanical Support and EpiHealth: Epidemiology for Health and Cardiology and Institutionen för kliniska vetenskaper, Malmö and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden and Department of Clinical Sciences, Malmö
PL o S Genetics (Online), ISSN 1553-7390, 05/2016, Volume 12, Issue 5, pp. e1006034 - e1006034
Journal Article
Journal Article
Journal of Heart and Lung Transplantation, ISSN 1053-2498, 04/2019, Volume 38, Issue 4, pp. S155 - S155
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e37491 - e37491
Background and Purpose: Extracellular adenosine triphosphate (ATP) regulates inflammatory cells by activation of the P2X(7) receptor. We hypothesized that... 
HUMAN P2X RECEPTOR | GAIN-OF-FUNCTION | GENETIC ASSOCIATION | DISEASE | SUSCEPTIBILITY | BIOLOGY | ATHEROSCLEROSIS | POLYMORPHISM | ISCHEMIC-STROKE | BLOOD-PRESSURE | LYMPHOCYTES | Genetic Predisposition to Disease | Gene Frequency | Humans | Middle Aged | Genotype | Male | Risk | Mutation, Missense | Cardiovascular Diseases - genetics | Receptors, Purinergic P2X7 - genetics | Linkage Disequilibrium | Adenosine Triphosphate - metabolism | Aged, 80 and over | Receptors, Purinergic P2X7 - metabolism | Female | Aged | Polymorphism, Single Nucleotide | Gene Order | Quality Control | Stroke (Disease) | Hypertension | Ischemia | Genetic aspects | Muscle proteins | Heart diseases | Risk factors | Adenosine triphosphate | Heart | Haplotypes | Atherogenesis | Neurosciences | Multiple sclerosis | Genes | Cardiovascular disease | Single-nucleotide polymorphism | Kinases | Proteins | Regression models | Ultrasonic imaging | Lymphocytes | Chromosome 12 | Physiology | Stroke | Cytokines | Health risks | Risk reduction | Inflammation | Regression analysis | Risk analysis | Thrombosis | Coronary artery disease | Studies | Signaling | Biomarkers | Diabetes | Rehabilitation | Cardiovascular diseases | ATP | Polymorphism | Index Medicus | atherosclerosis | Clinical Medicine | disease | lymphocytes | susceptibility | gain-of-function | human p2x receptor | genetic association | polymorphism | blood-pressure | Klinisk medicin | ischemic-stroke
Journal Article
Journal Article