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1. Full Text Meta-Analysis of Genome-Wide Association Studies in >80 000 Subjects Identifies Multiple Loci for C-Reactive Protein Levels
by Dehghan, A and Dupuis, J and Barbalic, M and Bis, J.C and Eiriksdottir, G and Lu, C and Pellikka, N and Wallaschofski, H and Kettunen, J and Henneman, P and Baumert, J and Strachan, D.P and Fuchsberger, C and Vitart, V and Wilson, J.F and Paré, G and Naitza, S and Rudock, M.E and Surakka, I and de Geus, E.J.C and Alizadeh, BZ and Guralnik, J and Shuldiner, A and Tanaka, T and Zee, R.Y.L and Schnabel, R.B and Nambi, V and Kavousi, M and Ripatti, S and Nauck, M and Smith, N.L and Smith, A.V and Sundvall, J and Scheet, P and Liu, Y and Ruokonen, A and Rose, L.M and Larson, M.G and Hoogeveen, R.C and Freimer, N.B and Teumer, A and Tracy, R.P and Launer, L.J and Buring, J.E and Yamamoto, J.F and Folsom, A.R and Sijbrands, E.J.G and Pankow, J and Elliott, P and Keaney, J.F and Sun, W and Sarin, A.-P and Fontes, J.D and Badola, S and Astor, B.C and Hofman, A and Pouta, A and Werdan, K and Greiser, K.H and Meyer zu Schwabedissen, H.E and Thiery, J and Jamshidi, Y and Nolte, I.M and Soranzo, N and Spector, T.D and Völzke, H and Aspelund, T and Young, L and Siscovick, D.S and Rotter, J.I and Schlessinger, D and Rudan, I and Hicks, A.A and Penninx, B.W.J.H and Gieger, C and Willemsen, G and Harris, T.B and Uitterlinden, A.G and Järvelin, M.J and Rice, K and Salomaa, V and Boerwinkle, E and Ferrucci, L and Snieder, H and Boomsma, D.I and Hayward, C and Pramstaller, P.P and van Duijn, C.M and Peltonen, L and Psaty, B.M and Gudnason, V and Ridker, P.M and Homuth, G and Koenig, W and Ballantyne, C.M and Witteman, J.C.M and Benjamin, E.J and Perola, M and Chasman, D.I
Circulation, ISSN 0009-7322, 2011, Volume 123, Issue 7, pp. 731 - 738
Background-C-reactive protein (CRP) is a heritable marker of chronic inflammation that is strongly associated with cardiovascular disease. We sought to... 
meta-analysis | genetics | inflammation | genome-wide association study | myocardial infarction | EPIDEMIOLOGIC APPLICATIONS | FRAMINGHAM | COMMON VARIANTS | CRP GENE | DESIGN | CARDIAC & CARDIOVASCULAR SYSTEMS | GENETICALLY ISOLATED POPULATION | NETHERLANDS TWIN REGISTER | PERIPHERAL VASCULAR DISEASE | CORONARY-HEART-DISEASE | HEMATOLOGY | ALPHA-GENE | Index Medicus | Abridged Index Medicus | epidemiology | coronary heart disease | C-reactive protein | genome-wide association
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2. Full Text Genetics of the thrombomodulin-endothelial cell protein C receptor system and the risk of early-onset ischemic stroke
by Cole, John W and Xu, Huichun and Ryan, Kathleen and Jaworek, Thomas and Dueker, Nicole and McArdle, Patrick and Gaynor, Brady and Cheng, Yu-Ching and O'Connell, Jeffrey and Bevan, Steve and Malik, Rainer and Ahmed, Naveed Uddin and Amouyel, Philippe and Anjum, Sheraz and Bis, Joshua C and Crosslin, David and Danesh, John and Engelter, Stefan T and Fornage, Myriam and Frossard, Philippe and Gieger, Christian and Giese, Anne-Katrin and Grond-Ginsbach, Caspar and Ho, Weang Kee and Holliday, Elizabeth and Hopewell, Jemma and Hussain, M and Iqbal, W and Jabeen, S and Jannes, Jim and Kamal, Ayeesha and Kamatani, Yoichiro and Kanse, Sandip and Kloss, Manja and Lathrop, Mark and Leys, Didier and Lindgren, Arne and LongstrethJr, W.T and Mahmood, Khalid and Meisinger, Christa and Metso, Tiina M and Mosley, Thomas and Müller-Nurasyid, Martina and Norrving, Bo and Parati, Eugenio and Peters, Annette and Pezzini, Alessandro and Quereshi, I and Rasheed, Asif and Rauf, A and Salam, T and Shen, Jess and Slowik, Agnieszka and Stanne, Tara and Strauch, Konstantin and Tatlisumak, Turgut and Thijs, Vincent N and Tiedt, Steffen and Traylor, Matthew and Waldenberger, Melanie and Walters, Matthew and Zhao, Wei and Boncoraglio, Giorgio and Debette, Stephanie and Jern, Christina and Levi, Christopher and Markus, Hugh and Meschia, James and Rolfs, Arndt and Rothwell, Peter and Saleheen, Danish and Seshadri, Sudha and Sharma, Pankaj and Sudlow, Cathie and Worrall, Bradford and Stine, O. Colin and Kittner, Steven J and Mitchell, Braxton D and WTCCC-2 Consortium and METASTROKE Consortium ISGC and METASTROKE Consortium of the ISGC and Stroke policy och kvalitetsregisterforskning and Clinical Stroke Research Group and Lund University and Neurologi, Lund and Stroke policy and quality register research and Klinisk strokeforskning and EpiHealth: Epidemiology for Health and Lunds universitet and Neurology, Lund
PLoS ONE, ISSN 1932-6203, 11/2018, Volume 13, Issue 11, pp. e0206554 - e0206554
Background and purpose Polymorphisms in coagulation genes have been associated with early-onset ischemic stroke. Here we pursue an a priori hypothesis that... 
YOUNG-ADULTS | MORTALITY | POPULATION | FACTOR-V-LEIDEN | POLYMORPHISMS | METAANALYSIS | MYOCARDIAL-INFARCTION | MULTIDISCIPLINARY SCIENCES | CLASSIFICATION | MECHANISMS | GENOME-WIDE ASSOCIATION | Brain Ischemia - epidemiology | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genetic Association Studies | Humans | Middle Aged | Brain Ischemia - genetics | African Americans - genetics | Male | Endothelial Protein C Receptor - genetics | Case-Control Studies | Stroke - genetics | Young Adult | Adolescent | Age of Onset | Adult | Female | Stroke - epidemiology | Polymorphism, Single Nucleotide | Thrombomodulin - genetics | Stroke (Disease) | Genetic variation | African Americans | Genetic aspects | Research | Health aspects | Endothelium | Membrane proteins | Heart attacks | Population studies | Cardiovascular disease | Genomes | Single-nucleotide polymorphism | Risk factors | Proteins | Receptors | Ischemia | Pruning | Genetics | Thrombomodulin | Age | Stroke | Health risks | Protein C | Genetic diversity | Risk analysis | Minority & ethnic groups | Endothelial cells | Medicine | Hospitals | Replication | Health risk assessment | Age composition | Index Medicus | Neurologi | Clinical Medicine | Neurology | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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3. Full Text Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis
by Paternoster, Lavinia and Standl, Marie and Waage, Johannes and Baurecht, Hansjoerg and Hotze, Melanie and Strachan, David P and Curtin, John A and Bonnelykke, Klaus and Tian, Chao and Takahashi, Atsushi and Esparza-Gordillo, Jorge and Alves, Alexessander Couto and Thyssen, Jacob P and den Dekker, Herman T and Ferreira, Manuel A and Altmaier, Elisabeth and Sleiman, Patrick M. A and Xiao, Feng Li and Gonzalez, Juan R and Marenholz, Ingo and Kalb, Birgit and Pino-Yanes, Maria and Xu, Chengjian and Carstensen, Lisbeth and Groen-Blokhuis, Maria M and Venturini, Cristina and Pennell, Craig E and Barton, Sheila J and Levin, Albert M and Curjuric, Ivan and Bustamante, Mariona and Kreiner-Moller, Eskil and Lockett, Gabrielle A and Bacelis, Jonas and Bunyavanich, Supinda and Myers, Rachel A and Matanovic, Anja and Kumar, Ashish and Tung, Joyce Y and Hirota, Tomomitsu and Kubo, Michiaki and McArdle, Wendy L and Henderson, A. John and Kemp, John P and Zheng, Jie and Smith, George Davey and Rueschendorf, Franz and Bauerfeind, Anja and Lee-Kirsch, Min Ae and Arnold, Aneas and Homuth, Georg and Schmidt, Carsten O and Mangold, Elisabeth and Cichon, Sven and Keil, Thomas and Roiguez, Elke and Peters, Annette and Franke, Ane and Lieb, Wolfgang and Novak, Natalija and Foelster-Holst, Regina and Horikoshi, Momoko and Pekkanen, Juha and Sebert, Sylvain and Husemoen, Lise L and Grarup, Niels and De Jongste, Johan C and Rivadeneira, Fernando and Hofman, Albert and Jaddoe, Vincent W. V and Pasmans, Suzanne G. M. A and Elbert, Niels J and Uitterlinden, Ane G and Marks, Guy B and Thompson, Philip J and Matheson, Melanie C and Robertson, Colin F and Ried, Janina S and Li, Jin and Zuo, Xian Bo and Zheng, Xiao Dong and Yin, Xian Yong and Sun, Liang Dan and McAleer, Maeve A and O'Regan, Grainne M and Fahy, Caoimhe M. R and Campbell, Linda E and Macek, Milan and Kurek, Michael and Hu, Donglei and Eng, Celeste and Postma, Dirkje S and Feenstra, Bjarke and Geller, Frank and Hottenga, Jouke Jan and Middeldorp, Christel M and Hysi, Pirro and Bataille, Veronique and Spector, Tim and Tiesler, Carla M. T and ... and AAGC and EArly Genetics Lifecourse Epidemio and Australian Asthma Genetics Consortium (AAGC) and the EArly Genetics and Lifecourse Epidemiology (EAGLE) Eczema Consortium and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institute of Clinical Sciences, Section for the Health of Women and Children, Department of Obstetrics and Gynecology and Sahlgrenska Academy and Institutionen för kliniska vetenskaper, sektionen för kvinnors och barns hälsa, Avdelningen för obstetrik och gynekologi
Nature Genetics, ISSN 1061-4036, 12/2015, Volume 47, Issue 12, pp. 1449 - +
Genetic association studies have identified 21 loci associated with atopic dermatitis risk predominantly in populations of European ancestry. To identify... 
CELLS | METAANALYSIS | VARIANTS | FILAGGRIN | DISEASE | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | ASTHMA | SKIN | INTERLEUKIN-7 RECEPTOR | IL-7 | Dermatitis, Atopic - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Risk Factors | Immunity, Innate - genetics | Dermatitis, Atopic - pathology | Genetic Loci | Dermatitis, Atopic - ethnology | Case-Control Studies | Ethnic Groups - genetics | T-Lymphocytes - cytology | T-Lymphocytes - metabolism | Genetic Markers - genetics | Polymorphism, Single Nucleotide - genetics | T-Lymphocytes - immunology | Genome-wide association studies | Immune response | Atopic dermatitis | Development and progression | Genotype | Genetic aspects | Identification and classification | Health aspects | Methods | Studies | Confidence intervals | Inflammatory bowel disease | Genealogy | Disease | Psoriasis | Bias | Eczema | Genomes | Mutation | Dermatitis | Quality of life | Index Medicus | Genètica | Grups ètnics | Dermatitis atòpica | GENETIC ARCHITECTURE | ATES OF AMERICA | Genetics & Heredity | PE1761 | Obstetrics, Gynecology and Reproductive Medicine | INFLAMMATORY-BOWEL-DISEASE | JAPANESE POPULATION | Reproduktionsmedicin och gynekologi | V110 | PSORIASIS
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4. Full Text A comprehensive 1000 Genomes-based genome-wide association meta-analysis of coronary artery disease
by Nikpay, Majid and Goel, Anuj and Won, Hong-Hee and Hall, Leanne M and Willenborg, Christina and Kanoni, Stavroula and Saleheen, Danish and Kyriakou, Theodosios and Nelson, Christopher P and CHopewell, Jemma and Webb, Thomas R and Zeng, Lingyao and Dehghan, Abbas and Alver, Maris and MArmasu, Sebastian and Auro, Kirsi and Bjonnes, Andrew and Chasman, Daniel I and Chen, Shufeng and Ford, Ian and Franceschini, Nora and Gieger, Christian and Grace, Christopher and Gustafsson, Stefan and Huang, Jie and Hwang, Shih-Jen and Kim, Yun Kyoung and Kleber, Marcus E and Lau, King Wai and Lu, Xiangfeng and Lu, Yingchang and Lyytikäinen, Leo-Pekka and Mihailov, Evelin and Morrison, Alanna C and Pervjakova, Natalia and Qu, Liming and Rose, Lynda M and Salfati, Elias and Saxena, Richa and Scholz, Markus and Smith, Albert V and Tikkanen, Emmi and Uitterlinden, Andre and Yang, Xueli and Zhang, Weihua and Zhao, Wei and De Andrade, Mariza and De Vries, Paul S and Van Zuydam, Natalie R and Anand, Sonia S and Bertram, Lars and Beutner, Frank and Dedoussis, George and Frossard, Philippe and Gauguier, Dominique and Goodall, Alison H and Gottesman, Omri and Haber, Marc and Han, Bok-Ghee and Huang, Jianfeng and Jalilzadeh, Shapour and Kessler, Thorsten and König, Inke R and Lannfelt, Lars and Lieb, Wolfgang and Lind, Lars and MLindgren, Cecilia and Lokki, Marja-Liisa and Magnusson, Patrik K and Mallick, Nadeem H and Mehra, Narinder and Meitinger, Thomas and Memon, Fazal-Uur-Rehman and Morris, Andrew P and Nieminen, Markku S and Pedersen, Nancy L and Peters, Annette and Rallidis, Loukianos S and Rasheed, Asif and Samuel, Maria and Shah, Svati H and Sinisalo, Juha and EStirrups, Kathleen and Trompet, Stella and Wang, Laiyuan and Zaman, Khan S and Ardissino, Diego and Boerwinkle, Eric and Borecki, Ingrid B and Bottinger, Erwin P and Buring, Julie E and Chambers, John C and Collins, Rory and Cupples, Ladrienne and Danesh, John and Demuth, Ilja and Elosua, Roberto and Epstein, Stephen E and Esko, Tõnu and Feitosa, Mary F and ... and CARDIoGRAMplusC4D Consortium and the CARDIoGRAMplusC4D Consortium and Medicinska fakulteten and Science for Life Laboratory, SciLifeLab and Medicinska och farmaceutiska vetenskapsområdet and Geriatrik and Kardiovaskulär epidemiologi and Uppsala universitet and Institutionen för medicinska vetenskaper and Molekylär epidemiologi and Institutionen för folkhälso- och vårdvetenskap
Nature Genetics, ISSN 1061-4036, 09/2015, Volume 47, Issue 10, pp. 1121 - 1130
Existing knowledge of genetic variants affecting risk of coronary artery disease (CAD) is largely based on genome-wide association study (GWAS) analysis of... 
HEART-DISEASE | MYOCARDIAL-INFARCTION | CELL-MIGRATION | GENETICS & HEREDITY | CARDIOVASCULAR-DISEASE | RISK | GENETIC-VARIANTS | SUSCEPTIBILITY LOCUS | IDENTIFIES 13 | EXPRESSION | MICE LACKING | Genetic susceptibility | Genetic research | Development and progression | Genetic aspects | Single nucleotide polymorphisms | Research | Cardiovascular research | Coronary heart disease | Health aspects | Kardiologi | Basic Medicine | Medical Genetics | Medicinsk genetik | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
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5. Full Text Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
by Jostins, Luke and Ripke, Stephan and Weersma, Rinse K and Duerr, Richard H and McGovern, Dermot P and Hui, Ken Y and Lee, James C and Schumm, L. Philip and Sharma, Yashoda and Anderson, Carl A and Essers, Jonah and Mitrovic, Mitja and Ning, Kaida and Cleynen, Isabelle and Theatre, Emilie and Spain, Sarah L and Raychaudhuri, Soumya and Goyette, Philippe and Wei, Zhi and Abraham, Clara and Achkar, Jean-Paul and Ahmad, Tariq and Amininejad, Leila and Ananthakrishnan, Ashwin N and Andersen, Vibeke and Anews, Jane M and Baidoo, Leonard and Balschun, Tobias and Bampton, Peter A and Bitton, Alain and Boucher, Gabrielle and Brand, Stephan and Büning, Carsten and Cohain, Ariella and Cichon, Sven and D'Amato, Mauro and de Jong, Dirk and Devaney, Kathy L and Dubinsky, Marla and Edwards, Cathryn and Ellinghaus, David and Ferguson, Lynnette R and Franchimont, Denis and Fransen, Karin and Gearry, Richard and Georges, Michel and Gieger, Christian and Glas, Jürgen and Haritunians, Talin and Hart, Ailsa and Hawkey, Chris and Hedl, Matija and Hu, Xinli and Karlsen, Tom H and Kupcinskas, Limas and Kugathasan, Subra and Latiano, Anna and Laukens, Debby and Lawrance, Ian C and Lees, Charlie W and Louis, Edouard and Mahy, Gillian and Mansfield, John and Morgan, Angharad R and Mowat, Craig and Newman, William and Palmieri, Orazio and Ponsioen, Cyriel Y and Potocnik, Uros and Prescott, Natalie J and Regueiro, Miguel and Rotter, Jerome I and Russell, Richard K and Sanderson, Jeremy D and Sans, Miquel and Satsangi, Jack and Schreiber, Stefan and Simms, Lisa A and Sventoraityte, Jurgita and Targan, Stephan R and Taylor, Kent D and Tremelling, Mark and Verspaget, Hein W and de Vos, Martine and Wijmenga, Cisca and Wilson, David C and Winkelmann, Juliane and Xavier, Ramnik J and Zeissig, Sebastian and Zhang, Bin and Zhang, Clarence K and Zhao, Hongyu and Silverberg, Mark S and Annese, Vito and Hakonarson, Hakon and Brant, Steven R and Radford-Smith, Graham and Mathew, Christopher G and Rioux, John D and Schadt, Eric E and ... and Int IBD Genetics Consortium IIBDGC and International IBD Genetics Consortium (IIBDGC) and The International IBD Genetics Consortium (IIBDGC) and Region Örebro län and Örebro universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 2012, Volume 491, Issue 7422, pp. 119 - 124
Crohn's disease and ulcerative colitis, the two common forms of inflammatory bowel disease (IBD), affect over 2.5 million people of European ancestry, with... 
HYPER-IGE SYNDROME | RISK LOCI | NETWORK | NUMBER | METAANALYSIS | TUBERCULOSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MUTATIONS | EXPRESSION | Crohn Disease - genetics | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Mycobacterium tuberculosis - immunology | Inflammatory Bowel Diseases - physiopathology | Colitis, Ulcerative - immunology | Host-Pathogen Interactions - immunology | Mycobacterium tuberculosis - pathogenicity | Haplotypes - genetics | Mycobacterium - pathogenicity | Mycobacterium Infections - genetics | Inflammatory Bowel Diseases - genetics | Crohn Disease - microbiology | Mycobacterium Infections - microbiology | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Reproducibility of Results | Mycobacterium - immunology | Crohn Disease - immunology | Genome, Human - genetics | Phenotype | Colitis, Ulcerative - microbiology | Host-Pathogen Interactions - genetics | Polymorphism, Single Nucleotide - genetics | Inflammatory Bowel Diseases - microbiology | Crohn Disease - physiopathology | Colitis, Ulcerative - physiopathology | Inflammatory bowel disease | Candidates | Tuberculosis | Architecture | Genetics | Genomes | Diabetes | Gene expression | Health risk assessment | Immune system | Index Medicus | Rheumatology and Autoimmunity | Basic Medicine | Medical Genetics | Medicinsk genetik | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
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6. Full Text Genetic studies of body mass index yield new insights for obesity biology
by Locke, A.E and Kahali, B and Berndt, S.I and Justice, A.E and Pers, T.H and Day, F.R and Powell, C and Vedantam, S and Buchkovich, M.L and Yang, J and Croteau-Chonka, D.C and Esko, T and Fall, T and Ferreira, T and Gustafsson, S and Kutalik, Z and Luan, J and Magi, R and Randall, J.C and Winkler, T.W and Wood, A.R and Workalemahu, T and Faul, J.D and Smith, J.A and Zhao, J and Zhao, W and Chen, J and Fehrmann, R and Hedman, A.K and Karjalainen, J and Schmidt, E.M and Absher, D and Amin, N and Anderson, D and Beekman, M and Bolton, J.L and Bragg-Gresham, J.L and Buyske, S and Demirkan, A and Deng, G and Ehret, G.B and Feenstra, B and Feitosa, M.F and Fischer, K and Goel, A and Gong, J and Jackson, A.U and Kanoni, S and Kleber, M.E and Kristiansson, K and Lim, U and Lotay, V and Mangino, M and Leach, I. Mateo and Medina-Gomez, C and Medland, S.E and Nalls, M.A and Palmer, C.D and Pasko, D and Pechlivanis, S and Peters, M.J.W and Prokopenko, I and Shungin, D and Stancakova, A and Strawbridge, R.J and Sung, Y. Ju and Tanaka, T and Teumer, A and Trompet, S and Laan, S.W. van der and Setten, J. van and Vliet-Ostaptchouk, J.V. Van and Wang, Z and Yengo, L and Zhang, W and Isaacs, A and Albrecht, E and Arnlov, J and Arscott, G.M and Attwood, A.P and Bandinelli, S and Barrett, A and Bas, I.N and Bellis, C and Bennett, A.J and Berne, C and Blagieva, R and Bluher, M and Bohringer, S and Bonnycastle, L.L and Bottcher, Y and Boyd, H.A and Bruinenberg, M and Caspersen, I.H and Chen, Y.D and Clarke, R and Daw, E.W and Craen, A.J. de and Delgado, G and Dimitriou, M and ... and ADIPOGen Consortium and PAGE Consortium and LifeLines Cohort Study and MIGen Consortium and CARDIOGRAMplusC4D Consortium and MuTHER Consortium and ICBP and CKDGen Consortium and Int Endogene Consortium and GENIE Consortium and MAGIC Investigators and ReproGen Consortium and AGEN-BMI Working Grp and GLGC and International Endogene Consortium and AGEN-BMI Working Group and The PAGE Consortium and The International Endogene Consortium and The MAGIC Investigators and The ReproGen Consortium and The CKDGen Consortium and The ADIPOGen Consortium and The ICBP and The CARDIOGRAMplusC4D Consortium and The GLGC and The GENIE Consortium and The AGEN-BMI Working Group and The MIGen Consortium and The MuTHER Consortium and The LifeLines Cohort Study and Medicinska fakulteten and Science for Life Laboratory, SciLifeLab and Medicinska och farmaceutiska vetenskapsområdet and Medicinsk genetik och genomik and Geriatrik and Uppsala universitet and Klinisk diabetologi och metabolism and Institutionen för immunologi, genetik och patologi and Molekylär medicin and Kardiovaskulär epidemiologi and Institutionen för medicinska vetenskaper and Molekylär epidemiologi and Institutionen för folkhälso- och vårdvetenskap
Nature, ISSN 0028-0836, 2015, Volume 518, Issue 7538, pp. 197 - 206
Obesity is heritable and predisposes to many diseases. To understand the genetic basis of obesity better, here we conduct a genome-wide association study and... 
INDIVIDUALS | PROVIDES INSIGHTS | METAANALYSIS | VARIANTS | MULTIDISCIPLINARY SCIENCES | HIPPOCAMPAL | GLYCEMIC TRAITS | ARCHITECTURE | TOPIRAMATE | LOCI | GENOME-WIDE ASSOCIATION | Body Mass Index | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Age Factors | Humans | Male | Continental Population Groups - genetics | Adiposity - genetics | Obesity - genetics | Europe - ethnology | Obesity - metabolism | Insulin - metabolism | Synapses - metabolism | Polymorphism, Single Nucleotide - genetics | Female | Glutamic Acid - metabolism | Energy Metabolism - genetics | Adipogenesis - genetics | Insulin Secretion | Quantitative Trait Loci - genetics | Body mass index | Obesity | Genetic research | Genetic aspects | Research | Diabetes | Gene expression | Health aspects | Studies | Genomics | Genetics | Biology | Genomes | Metabolic disorders | Meta-analysis | Index Medicus | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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