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Nature, ISSN 0028-0836, 01/2016, Volume 529, Issue 7584, pp. 110 - 114
Gain-of-function IDH mutations are initiating events that define major clinical and prognostic classes of gliomas(1,2). Mutant IDH protein produces a new... 
MAINTENANCE | METHYLATION | LANDSCAPE | DEMETHYLATION | MULTIDISCIPLINARY SCIENCES | INTEGRATED GENOMIC ANALYSIS | ARCHITECTURE | PHENOTYPE | EXPRESSION | 2-HYDROXYGLUTARATE | PRINCIPLES | Chromatin - metabolism | Up-Regulation | Humans | Glioma - genetics | Base Sequence | Glioma - pathology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Binding Sites | Chromatin - drug effects | Repressor Proteins - metabolism | Oncogenes - genetics | Insulator Elements - genetics | Glioma - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Isocitrate Dehydrogenase - genetics | DNA Methylation - genetics | Down-Regulation - drug effects | Mutation - genetics | CRISPR-Cas Systems - genetics | Insulator Elements - drug effects | Phenotype | Isocitrate Dehydrogenase - chemistry | Receptor, Platelet-Derived Growth Factor alpha - genetics | CCCTC-Binding Factor | CpG Islands - genetics | Protein Binding | Isocitrate Dehydrogenase - metabolism | Cell Proliferation - drug effects | Enhancer Elements, Genetic - genetics | Glutarates - metabolism | Cell Transformation, Neoplastic - drug effects | Chromatin - genetics | DNA Methylation - drug effects | Glioma - drug therapy | Complications and side effects | Care and treatment | Platelet-derived growth factor | Gliomas | Analysis | Influence | Genetic aspects | Research | Methylation | Oncogenes | DNA methylation | Epigenetics | Genomes | Mutation | Gene expression | Binding sites | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Science, ISSN 0036-8075, 03/2017, Volume 355, Issue 6332, pp. eaai8478 - eaai8478
Journal Article
Cell, ISSN 0092-8674, 04/2014, Volume 157, Issue 3, pp. 580 - 594
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 4, pp. 364 - 370
The identification of activating NOTCH1 mutations in T cell acute lymphoblastic leukemia (T-ALL) led to clinical testing of g-secretase inhibitors (GSIs) that... 
NOTCH1 | INHIBITION | CHROMATIN | PATHWAY | GENES | GENETICS & HEREDITY | C-MYC | ACUTE MYELOID-LEUKEMIA | CANCER | LENTIVIRAL RNAI LIBRARY | GENOME | Chromatin - metabolism | Humans | Flow Cytometry | Chromatin Immunoprecipitation | Gene Expression Regulation, Neoplastic - drug effects | Indoles | Nuclear Proteins - genetics | Real-Time Polymerase Chain Reaction | Gene Expression Regulation, Neoplastic - genetics | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Enzyme-Linked Immunosorbent Assay | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Signal Transduction - genetics | Transcription Factors - genetics | Enzyme Inhibitors - therapeutic use | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Epigenesis, Genetic - genetics | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Histones - metabolism | Receptor, Notch1 - antagonists & inhibitors | Receptor, Notch1 - genetics | Chromatin - genetics | Care and treatment | Pharmacology, Experimental | Acute lymphocytic leukemia | Research | Drug resistance | Proteins | Genotype & phenotype | Leukemia | Epigenetics | Genomes | Kinases | Gene expression | Cancer therapies
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7673, pp. 533 - 537
High-grade gliomas (HGG) are a devastating group of cancers, and represent the leading cause of brain tumour-related death in both children and adults.... 
AUTISM | PEPTIDES | PHOSPHORYLATION | DISINTEGRIN | SUBTYPES | MULTIDISCIPLINARY SCIENCES | OLIGODENDROCYTE PRECURSOR CELLS | MUTATION | HIPPOCAMPAL | SELF-RENEWAL | CLEAVAGE | Neoplasm Transplantation | Neurons - pathology | Oligodendroglia - metabolism | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Nerve Tissue Proteins - deficiency | Brain Neoplasms - pathology | ADAM10 Protein - metabolism | Tumor Microenvironment | Male | Phosphatidylinositol 3-Kinases - metabolism | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Adhesion Molecules, Neuronal - deficiency | Glioma - genetics | Membrane Proteins - deficiency | ADAM10 Protein - antagonists & inhibitors | Heterografts | Glioma - pathology | Adult | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Neurons - metabolism | Oligodendroglia - cytology | Child | Signal Transduction | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Membrane Proteins - genetics | Brain Neoplasms - genetics | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Membrane Proteins - antagonists & inhibitors | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Gliomas | Health aspects | Research | TOR protein | Brain | Phosphorylation | Transcription | Brain tumors | Brain cancer | Genes | Glioblastoma | Genomes | Kinases | Glial stem cells | Proteins | Signal transduction | Cell growth | Proteolysis | Glioma cells | Xenografts | Children | Secretion | Adhesion | 1-Phosphatidylinositol 3-kinase | Oligodendroglioma | Signaling | Proteomics | Focal adhesion kinase | Cancer
Journal Article
Journal Article
Cell Reports, ISSN 2211-1247, 05/2013, Volume 3, Issue 5, pp. 1567 - 1579
Journal Article
Journal Article
Nature, ISSN 0028-0836, 07/2017, Volume 547, Issue 7663, pp. 355 - 359
Journal Article