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PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0133923 - e0133923
Calorie restriction (CR) enhances longevity and mitigates aging phenotypes in numerous species. Physiological responses to CR are cell-type specific and... 
PROFILE | MEMORY | MULTIDISCIPLINARY SCIENCES | NEUROTROPHIN EXPRESSION | MOUSE MODEL | GENE-EXPRESSION | LTP | NEURODEGENERATION | MICE | AMYLOID PRECURSOR PROTEIN | PLASTICITY | CA1 Region, Hippocampal - cytology | Synaptic Transmission - genetics | Caloric Restriction | RNA, Messenger - genetics | Gene Expression Profiling | Longevity - genetics | Neuroprotection - genetics | RNA, Messenger - metabolism | Body Weight - genetics | Animals | Aging - genetics | Aging - physiology | CA1 Region, Hippocampal - metabolism | Female | Transcription, Genetic | Mice | RNA sequencing | Aging | Genetic aspects | Genetic transcription | Research | Gene expression | Alzheimer's disease | Potentiation | Brain | Neuroprotection | Neurosciences | Transcription | Calcium | Memory | Genes | Genomes | Physiological responses | Gene sequencing | Proteins | Calcium signalling | Learning | Calreticulin | Neurodegeneration | Synaptic transmission | Rodents | Calcium buffering | Signatures | Age | Nutrient deficiency | Neurodegenerative diseases | Neurons | Long-term potentiation | GRP78 protein | Transthyretin | Heat shock proteins | Cyclic AMP response element-binding protein | Feeding | Pathology | Hypotheses | Brain research | Brain-derived neurotrophic factor | Life span | Klotho protein | Neural networks | Quality control | Protein disulfide-isomerase | Molecular biology | Hippocampus | Heat shock | Animal cognition | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 04/2006, Volume 97, Issue 2, pp. 475 - 487
Dysfunction of cholinergic basal forebrain (CBF) neurons of the nucleus basalis (NB) is a cardinal feature of Alzheimer's disease (AD) and correlates with... 
microarray | trkA | mild cognitive impairment | RNA amplification | neurotrophin | nucleus basalis | Neurotrophin | Nucleus basalis | Microarray | Mild cognitive impairment | Humans | Cognition Disorders - metabolism | Male | Receptors, Nerve Growth Factor - metabolism | RNA, Messenger - metabolism | Choline O-Acetyltransferase - metabolism | Mental Status Schedule - statistics & numerical data | Alzheimer Disease - pathology | Reverse Transcriptase Polymerase Chain Reaction - methods | Aged, 80 and over | Female | Neurons - metabolism | In Situ Hybridization - methods | Prosencephalon - pathology | Severity of Illness Index | Oligonucleotide Array Sequence Analysis - methods | Cognition Disorders - pathology | Gene Expression Regulation - physiology | Nerve Tissue Proteins - genetics | Disease Progression | Receptors, Nerve Growth Factor - genetics | Immunohistochemistry - methods | Nerve Tissue Proteins - metabolism | Receptor, trkA - metabolism | Alzheimer Disease - metabolism | Receptor, trkA - genetics | Aged | Alzheimer Disease - genetics | Postmortem Changes | Cohort Studies | Neurosciences | Alzheimers disease | Gene expression | Neurons | Cell survival | Neurodegenerative diseases | Cognitive ability | Nerve growth factor | Forebrain (basal) | Neurotrophin receptors | Polymerase chain reaction | DNA microarrays | Cerebral blood flow | Degeneration | TrkA receptors | TrkB receptors | Alzheimer's disease | Index Medicus
Journal Article
Journal Article
Annals of neurology, ISSN 0364-5134, 2/2018, Volume 83, Issue 2, pp. 406 - 417
Journal Article
Annals of Neurology, ISSN 0364-5134, 02/2018, Volume 83, Issue 2, pp. 406 - 417
Journal Article
Developmental neurobiology, ISSN 1932-8451, 05/2019
Down syndrome (DS), trisomy 21, is marked by intellectual disability and a premature aging profile including degeneration of the basal forebrain cholinergic... 
Journal Article
Hippocampus, ISSN 1050-9631, 04/2018, Volume 28, Issue 4, pp. 251 - 268
Although there are changes in gene expression and alterations in neuronal density and afferent inputs in the forebrain of trisomic mouse models of Down... 
microarray | choline supplementation | hippocampus | trisomic | laser capture microdissection | ENDOTHELIN-CONVERTING ENZYME | INSULIN-DEGRADING ENZYME | TERMINAL CONTINUATION | AMYLOID PRECURSOR PROTEIN | PERINATAL CHOLINE | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | PERFORANT-PATH TRANSECTION | TC RNA AMPLIFICATION | MOUSE MODEL | BASAL FOREBRAIN | Pyramidal Cells - metabolism | Gene Expression | Maternal Nutritional Physiological Phenomena | Down Syndrome - metabolism | Mice, Inbred C57BL | Male | Mice, Transgenic | CA1 Region, Hippocampal - growth & development | Mice, Inbred C3H | Animals | Down Syndrome - prevention & control | Choline - administration & dosage | Alzheimer Disease - prevention & control | Alzheimer Disease - metabolism | CA1 Region, Hippocampal - metabolism | Female | Neuroprotective Agents - administration & dosage | Dietary Supplements | Aging - metabolism | Disease Models, Animal | Enzymes | Neurons | Choline | Analysis | Genes | Down syndrome | Gene expression | Alzheimer's disease | Animal models | Endothelin | Spatial discrimination | Forebrain | Cognition | Kinases | Calcium signalling | Down's syndrome | Neurodegeneration | Rodents | Calcium-binding protein | Sensory neurons | Calmodulin | Mosaics | Translation | Cell survival | Neurodegenerative diseases | Ribonucleic acid--RNA | Forebrain (basal) | Insulin | Plasticity (synaptic) | Protein kinase | Pyramidal cells | β-Amyloid | Alzheimers disease | Index Medicus
Journal Article
Journal Article