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The New England Journal of Medicine, ISSN 0028-4793, 07/2013, Volume 369, Issue 4, pp. 351 - 361
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2012, Volume 185, Issue 3, pp. 311 - 321
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 04/2010, Volume 181, Issue 8, pp. 851 - 861
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 07/2011, Volume 184, Issue 1, pp. 116 - 123
Journal Article
Respiratory Research, ISSN 1465-9921, 06/2010, Volume 11, Issue 1, pp. 73 - 73
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 08/2018, Volume 198, Issue 4, pp. 537 - 541
V/Q lung scintigraphy was normal, as was right heart catheterization (RHC) at rest: mean pulmonary artery pressure (mPAP), 17 mm Hg; pulmonary artery wedge... 
HYPERTENSION | VENOOCCLUSIVE DISEASE | RESPIRATORY SYSTEM | CRITICAL CARE MEDICINE | Heart | Genetic counseling | Dyspnea | Transplants & implants | Lungs | Pulmonary arteries | Mutation | Patients | Natural history | Pulmonary hypertension
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 11/2016, Volume 194, Issue 10, pp. 1273 - 1285
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1416 - 16
Pulmonary arterial hypertension (PAH) is a rare disorder with a poor prognosis. Deleterious variation within components of the transforming growth factor-beta... 
SOX17 | GENE | ANGIOGENESIS | CRYSTAL-STRUCTURE | MULTIDISCIPLINARY SCIENCES | BMP9 | ASSOCIATION | EXPRESSION | NONSENSE MUTATION | BETA-RECEPTOR | CAVEOLIN-1 | Prognosis | Humans | Male | Familial Primary Pulmonary Hypertension - diagnosis | SOXF Transcription Factors - metabolism | Aquaporin 1 - metabolism | Case-Control Studies | Familial Primary Pulmonary Hypertension - metabolism | Membrane Transport Proteins - genetics | Base Sequence | HEK293 Cells | Adult | Female | Growth Differentiation Factors - genetics | Membrane Transport Proteins - metabolism | Familial Primary Pulmonary Hypertension - pathology | Genetic Predisposition to Disease | Bone Morphogenetic Protein Receptors, Type II - genetics | Signal Transduction | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Models, Molecular | Familial Primary Pulmonary Hypertension - genetics | Bone Morphogenetic Protein Receptors, Type II - metabolism | Membrane Transport Proteins - chemistry | Whole Genome Sequencing | Aquaporin 1 - chemistry | Aquaporin 1 - genetics | Growth Differentiation Factors - metabolism | Transforming Growth Factor beta - genetics | Adenosine Triphosphatases - chemistry | Adenosine Triphosphatases - genetics | Mutation | SOXF Transcription Factors - genetics | Transforming Growth Factor beta - metabolism | SOXF Transcription Factors - chemistry | Growth Differentiation Factors - chemistry | Hypertension | Bone morphogenetic protein receptor type II | Secretion | Lung | Transforming growth factor | Genes | Genomes | Heritability | Aquaporin 1 | Gene sequencing | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 05/2012, Volume 125, Issue 17, pp. 2128 - 2137
Background-The French pulmonary hypertension (PH) registry allows the survey of epidemiological trends. Isolated cases of precapillary PH have been reported in... 
Dasatinib | Chronic myeloid leukemia | Vascular complications | Adverse drug events | Pulmonary hypertension | CHRONIC MYELOGENOUS LEUKEMIA | LONG-TERM | SURVIVAL | CARDIAC & CARDIOVASCULAR SYSTEMS | MYELOPROLIFERATIVE DISORDERS | INHIBITORS IMATINIB | pulmonary hypertension | TYROSINE KINASE | CELL-GROWTH | FAILURE | NILOTINIB | CHRONIC MYELOID-LEUKEMIA | adverse drug events | vascular complications | dasatinib | chronic myeloid leukemia | PERIPHERAL VASCULAR DISEASE | Hypertension, Pulmonary - diagnosis | Follow-Up Studies | Humans | Middle Aged | Calcium Channel Blockers - therapeutic use | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Male | Antineoplastic Agents - therapeutic use | Protein Kinase Inhibitors - adverse effects | Thiazoles - therapeutic use | Hypertension, Pulmonary - chemically induced | Thiazoles - adverse effects | Antineoplastic Agents - adverse effects | Adult | Female | Registries | Antineoplastic Agents - pharmacology | Hypertension, Pulmonary - drug therapy | Drug Utilization - statistics & numerical data | Endothelin Receptor Antagonists | Bone Morphogenetic Protein Receptors, Type II - genetics | Adverse Drug Reaction Reporting Systems | Treatment Outcome | Piperazines - therapeutic use | Hypertension, Pulmonary - epidemiology | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - adverse effects | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Adolescent | Pyrimidines - adverse effects | Hydroxyurea - therapeutic use | Aged | Hemodynamics - drug effects | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Care and treatment | Dosage and administration | Drug therapy | Index Medicus | Abridged Index Medicus | Life Sciences | Biochemistry, Molecular Biology
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 03/2017, Volume 56, Issue 3, pp. 402 - 405
[...]we hypothesized that bacterial translocation from gut lumen to bloodstream occurs in human and experimental PH, and that LPS translocation activates TLR4... 
CHRONIC HEART-FAILURE | CYTOKINES | RESPIRATORY SYSTEM