Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 10/2018, Volume 82, Issue 4, pp. 723 - 732
Tyrosine kinase inhibitors (TKI) that target MET signaling have shown promise in various types of cancer, including lung cancer. Combination strategies have...
Medicine & Public Health | MET phosphorylation | Oncology | Cancer Research | Topotecan | Circulating tumor cells | Pharmacology/Toxicology | Tivantinib | ARQ-197 | ADULT PATIENTS | ARQ 197 MONOTHERAPY | COMBINATION | CELL LUNG-CANCER | DOSE-ESCALATION | HEPATOCELLULAR-CARCINOMA | INHIBITION | THERAPY | ONCOLOGY | C-MET | GROWTH | PHARMACOLOGY & PHARMACY | Antimitotic agents | Care and treatment | Lung cancer | Analysis | Clinical trials | Product development | Antineoplastic agents | Cancer | Tyrosine | Thrombocytopenia | Phosphorylation | Toxicity | Tumor cells | DNA topoisomerase | Pharmacology | Patients | c-Met protein | Granulocyte colony-stimulating factor | Solid tumors | Protein-tyrosine kinase | Tumors | Neutropenia | circulating tumor cells | topotecan
Medicine & Public Health | MET phosphorylation | Oncology | Cancer Research | Topotecan | Circulating tumor cells | Pharmacology/Toxicology | Tivantinib | ARQ-197 | ADULT PATIENTS | ARQ 197 MONOTHERAPY | COMBINATION | CELL LUNG-CANCER | DOSE-ESCALATION | HEPATOCELLULAR-CARCINOMA | INHIBITION | THERAPY | ONCOLOGY | C-MET | GROWTH | PHARMACOLOGY & PHARMACY | Antimitotic agents | Care and treatment | Lung cancer | Analysis | Clinical trials | Product development | Antineoplastic agents | Cancer | Tyrosine | Thrombocytopenia | Phosphorylation | Toxicity | Tumor cells | DNA topoisomerase | Pharmacology | Patients | c-Met protein | Granulocyte colony-stimulating factor | Solid tumors | Protein-tyrosine kinase | Tumors | Neutropenia | circulating tumor cells | topotecan
Journal Article
Cancer Research, ISSN 0008-5472, 11/2015, Volume 75, Issue 22 Supplement 1, pp. A1 - A1-36
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2015, Volume 33, Issue 15_suppl, pp. TPS11110 - TPS11110
Journal Article
Frontiers in Oncology, ISSN 2234-943X, 2019, Volume 9, p. 132
Introduction: Mesenchymal epidermal transition and vascular endothelial growth factor pathways are important in mediating non-small cell lung cancer (NSCLC)...
RET | TKI resistance | MET | VEGF | EGFR | Non-small cell lung cancer | non-small cell lung cancer | GEFITINIB | MULTICENTER | XL184 | ACQUIRED-RESISTANCE | OPEN-LABEL | EGFR MUTATIONS | MET AMPLIFICATION | ONCOLOGY | Erlotinib | Gene mutations | Development and progression | Dosage and administration | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy
RET | TKI resistance | MET | VEGF | EGFR | Non-small cell lung cancer | non-small cell lung cancer | GEFITINIB | MULTICENTER | XL184 | ACQUIRED-RESISTANCE | OPEN-LABEL | EGFR MUTATIONS | MET AMPLIFICATION | ONCOLOGY | Erlotinib | Gene mutations | Development and progression | Dosage and administration | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2015, Volume 33, Issue 15_suppl, pp. 6060 - 6060
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2018, Volume 24, Issue 12, pp. 2771 - 2779
Purpose: Evaluate: safety and determine the recommended phase II dose (RI , 21)) of ensartinib (X-396), a potent anaplastic lymphoma kinase (ALK) tyrosine...
ANAPLASTIC LYMPHOMA KINASE | ONCOLOGY | CERITINIB | EML4-ALK FUSION GENE | CRIZOTINIB | OPEN-LABEL | SINGLE-ARM | INHIBITOR | IDENTIFICATION | CHEMOTHERAPY | Tyrosine | Brain | Toxicity | Exanthema | Lung cancer | Brain tumors | Central nervous system | Non-small cell lung carcinoma | Fatigue | Nausea | Pharmacology | Pruritus | Patients | Lymphoma | Metastases | Anticancer properties | Vomiting | Antitumor activity | Solid tumors | Pharmacokinetics | Protein-tyrosine kinase | Cancer | Index Medicus | non-small cell lung cancer | ALK inhibitor | ensartinib | X-396 | first-in-human study
ANAPLASTIC LYMPHOMA KINASE | ONCOLOGY | CERITINIB | EML4-ALK FUSION GENE | CRIZOTINIB | OPEN-LABEL | SINGLE-ARM | INHIBITOR | IDENTIFICATION | CHEMOTHERAPY | Tyrosine | Brain | Toxicity | Exanthema | Lung cancer | Brain tumors | Central nervous system | Non-small cell lung carcinoma | Fatigue | Nausea | Pharmacology | Pruritus | Patients | Lymphoma | Metastases | Anticancer properties | Vomiting | Antitumor activity | Solid tumors | Pharmacokinetics | Protein-tyrosine kinase | Cancer | Index Medicus | non-small cell lung cancer | ALK inhibitor | ensartinib | X-396 | first-in-human study
Journal Article
Future oncology (London, England), 10/2014, Volume 10, Issue 12, p. 1913
Microtubule antagonists are highly active agents for treatment of metastatic lung cancer, but can lead to significant toxicities and tumor resistance. Eribulin...
Ketones - pharmacology | Lung Neoplasms - drug therapy | Animals | Tubulin Modulators - pharmacology | Furans - pharmacology | Humans | Antineoplastic Agents - pharmacology
Ketones - pharmacology | Lung Neoplasms - drug therapy | Animals | Tubulin Modulators - pharmacology | Furans - pharmacology | Humans | Antineoplastic Agents - pharmacology
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2011, Volume 17, Issue 10, pp. 3304 - 3315
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 2018, Volume 36, Issue 14, pp. 1412 - 1418
PurposeThis multinational study evaluated the antitumor activity of nivolumab in nasopharyngeal carcinoma (NPC). Tumor and plasma-based biomarkers were...
THERAPY | MELANOMA | BARR-VIRUS DNA | ONCOLOGY | PD-L1 EXPRESSION | MUTATIONS | BLOCKADE | CANCER | SUBSET | CELL CARCINOMA | ESCAPE | ORIGINAL REPORTS
THERAPY | MELANOMA | BARR-VIRUS DNA | ONCOLOGY | PD-L1 EXPRESSION | MUTATIONS | BLOCKADE | CANCER | SUBSET | CELL CARCINOMA | ESCAPE | ORIGINAL REPORTS
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 03/2001, Volume 19, Issue 6, pp. 1649 - 1657
Purpose: To integrate stage, grade, and Eastern Cooperative Oncology Group (ECOG) performance status (PS) into a clinically useful fool capable of stratifying...
SURVIVAL | GRADE | ONCOLOGY | PROGNOSIS | MORPHOLOGIC PARAMETERS | Prognosis | Carcinoma, Renal Cell - pathology | Humans | Middle Aged | Kidney Neoplasms - classification | Carcinoma, Renal Cell - classification | Male | Neoplasm Staging - methods | Endpoint Determination | Survival Analysis | Aged, 80 and over | Adult | Female | Kidney Neoplasms - pathology | Aged | Retrospective Studies | Health Status
SURVIVAL | GRADE | ONCOLOGY | PROGNOSIS | MORPHOLOGIC PARAMETERS | Prognosis | Carcinoma, Renal Cell - pathology | Humans | Middle Aged | Kidney Neoplasms - classification | Carcinoma, Renal Cell - classification | Male | Neoplasm Staging - methods | Endpoint Determination | Survival Analysis | Aged, 80 and over | Adult | Female | Kidney Neoplasms - pathology | Aged | Retrospective Studies | Health Status
Journal Article
Cancer Discovery, ISSN 2159-8274, 06/2016, Volume 6, Issue 6, pp. 601 - 611
Here, we report that novel epidermal growth factor receptor (EGFR) gene fusions comprising the N-terminal of EGFR linked to various fusion partners, most...
CETUXIMAB | GEFITINIB | GENE | ONCOLOGY | ADENOCARCINOMA | EPIDERMAL-GROWTH-FACTOR | PHASE-III | OPEN-LABEL | MUTATIONS | FACTOR RECEPTOR | KINASE DOMAIN | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Exons | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Genetic Loci | Molecular Targeted Therapy | Young Adult | Neoplasm Metastasis | Oncogene Proteins, Fusion - chemistry | Adult | Female | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Rad51 Recombinase - genetics | Introns | Models, Molecular | Combined Modality Therapy | Magnetic Resonance Imaging | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Biomarkers | Cell Line, Tumor | Oncogene Proteins, Fusion - antagonists & inhibitors | Protein Conformation | High-Throughput Nucleotide Sequencing | Protein Kinase Inhibitors - pharmacology | Lung Neoplasms - diagnosis | targeted therapy | afatinib | Epidermal growth factor receptor (EGFR) | next-generation sequencing (NGS) | erlotinib | osimertinib | kinase domain | cetuximab | gene rearrangement | kinase fusion | tyrosine kinase inhibitor (TKI) | non-small cell lung cancer (NSCLC)
CETUXIMAB | GEFITINIB | GENE | ONCOLOGY | ADENOCARCINOMA | EPIDERMAL-GROWTH-FACTOR | PHASE-III | OPEN-LABEL | MUTATIONS | FACTOR RECEPTOR | KINASE DOMAIN | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Exons | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Genetic Loci | Molecular Targeted Therapy | Young Adult | Neoplasm Metastasis | Oncogene Proteins, Fusion - chemistry | Adult | Female | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Rad51 Recombinase - genetics | Introns | Models, Molecular | Combined Modality Therapy | Magnetic Resonance Imaging | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Biomarkers | Cell Line, Tumor | Oncogene Proteins, Fusion - antagonists & inhibitors | Protein Conformation | High-Throughput Nucleotide Sequencing | Protein Kinase Inhibitors - pharmacology | Lung Neoplasms - diagnosis | targeted therapy | afatinib | Epidermal growth factor receptor (EGFR) | next-generation sequencing (NGS) | erlotinib | osimertinib | kinase domain | cetuximab | gene rearrangement | kinase fusion | tyrosine kinase inhibitor (TKI) | non-small cell lung cancer (NSCLC)
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 03/2017, Volume 12, Issue 3, pp. 446 - 457
Erb-b2 receptor tyrosine kinase (HER2) transmembrane domain (TMD) mutations (HER2 , HER2 ) have previously been identified in lung adenocarcinomas, but their...
HER2 G660 | Actionable driver mutation | HER2 V659 | NSCLC | Afatinib | Transmembrane mutation | Lung Neoplasms - drug therapy | Prognosis | Follow-Up Studies | Receptor, ErbB-2 - genetics | Humans | Middle Aged | Protein Multimerization | Receptor, ErbB-2 - chemistry | Lung Neoplasms - pathology | Male | Radiation-Sensitizing Agents - therapeutic use | Protein Domains | Adult | Female | Retrospective Studies | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Amino Acid Sequence | Carcinoma, Non-Small-Cell Lung - genetics | Sequence Alignment | Quinazolines - therapeutic use | Protein Conformation | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Neoplasm Staging
HER2 G660 | Actionable driver mutation | HER2 V659 | NSCLC | Afatinib | Transmembrane mutation | Lung Neoplasms - drug therapy | Prognosis | Follow-Up Studies | Receptor, ErbB-2 - genetics | Humans | Middle Aged | Protein Multimerization | Receptor, ErbB-2 - chemistry | Lung Neoplasms - pathology | Male | Radiation-Sensitizing Agents - therapeutic use | Protein Domains | Adult | Female | Retrospective Studies | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Amino Acid Sequence | Carcinoma, Non-Small-Cell Lung - genetics | Sequence Alignment | Quinazolines - therapeutic use | Protein Conformation | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Neoplasm Staging
Journal Article
Future Oncology, ISSN 1479-6694, 10/2014, Volume 10, Issue 12, pp. 1913 - 1924
Microtubule antagonists are highly active agents for treatment of metastatic lung cancer, but can lead to significant toxicities and tumor resistance. Eribulin...
lung cancer | antimicrotubule | halichondrin | eribulin | taxanes | HALICHONDRIN-B | MECHANISM | MESYLATE E7389 | NATURAL-PRODUCTS | PHASE-II | ONCOLOGY | DYNAMICS | TUBULIN | CELL | BINDING
lung cancer | antimicrotubule | halichondrin | eribulin | taxanes | HALICHONDRIN-B | MECHANISM | MESYLATE E7389 | NATURAL-PRODUCTS | PHASE-II | ONCOLOGY | DYNAMICS | TUBULIN | CELL | BINDING
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1801 - 19
The transcribed ultraconserved regions (T-UCRs) encode long non-coding RNAs implicated in human carcinogenesis. Their mechanisms of action and the factors...
MYC MESSENGER-RNA | LUNG-CANCER | CYCLIN-E | LONG NONCODING RNA | MULTIDISCIPLINARY SCIENCES | C-MYC | CELL-PROLIFERATION | CERNA HYPOTHESIS | EXPRESSION | P53 | Carcinogens | Cell survival | p53 Protein | Lung cancer | MicroRNAs | MiRNA | Cell lines | Non-small cell lung carcinoma | Tumor suppressor genes | Ribonucleic acid--RNA | Carcinogenesis | Cancer | Index Medicus
MYC MESSENGER-RNA | LUNG-CANCER | CYCLIN-E | LONG NONCODING RNA | MULTIDISCIPLINARY SCIENCES | C-MYC | CELL-PROLIFERATION | CERNA HYPOTHESIS | EXPRESSION | P53 | Carcinogens | Cell survival | p53 Protein | Lung cancer | MicroRNAs | MiRNA | Cell lines | Non-small cell lung carcinoma | Tumor suppressor genes | Ribonucleic acid--RNA | Carcinogenesis | Cancer | Index Medicus
Journal Article
Lung Cancer, ISSN 0169-5002, 2017, Volume 106, pp. 110 - 114
Highlights • SCLC transformation is a resistance mechanism to ALK inhibitors. • Liquid biopsy missed morphologic examination. • Emergence of Rb1 C706Y and p53...
Hematology, Oncology and Palliative Medicine | Pulmonary/Respiratory | Small cell lung cancer transformation | ALK G1202R solvent front mutation | Lorlatinib | Rb1 C706Y | Circulating tumor DNA | ALK+ NSCLC | ADENOCARCINOMA | EML4-ALK FUSION | ACQUIRED-RESISTANCE | 1ST | ALK plus NSCLC | ONCOLOGY | RESPIRATORY SYSTEM | ALECTINIB | Rbl C706Y | Lung Neoplasms - drug therapy | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - pathology | Male | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Cell Transformation, Neoplastic - genetics | Fatal Outcome | Adult | Female | Lactams, Macrocyclic - administration & dosage | Lactams, Macrocyclic - therapeutic use | Retinoblastoma Binding Proteins - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Pyridines | Neoplastic Cells, Circulating - drug effects | Liquid Biopsy - methods | Pyrazoles | Carcinoma, Non-Small-Cell Lung - metabolism | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Receptor Protein-Tyrosine Kinases - genetics | Protein Kinase Inhibitors - therapeutic use | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Ubiquitin-Protein Ligases - genetics | Tumor proteins | Medical colleges | Genetic aspects | Development and progression | Lung cancer, Small cell | Lung cancer, Non-small cell
Hematology, Oncology and Palliative Medicine | Pulmonary/Respiratory | Small cell lung cancer transformation | ALK G1202R solvent front mutation | Lorlatinib | Rb1 C706Y | Circulating tumor DNA | ALK+ NSCLC | ADENOCARCINOMA | EML4-ALK FUSION | ACQUIRED-RESISTANCE | 1ST | ALK plus NSCLC | ONCOLOGY | RESPIRATORY SYSTEM | ALECTINIB | Rbl C706Y | Lung Neoplasms - drug therapy | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - pathology | Male | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Cell Transformation, Neoplastic - genetics | Fatal Outcome | Adult | Female | Lactams, Macrocyclic - administration & dosage | Lactams, Macrocyclic - therapeutic use | Retinoblastoma Binding Proteins - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Pyridines | Neoplastic Cells, Circulating - drug effects | Liquid Biopsy - methods | Pyrazoles | Carcinoma, Non-Small-Cell Lung - metabolism | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Receptor Protein-Tyrosine Kinases - genetics | Protein Kinase Inhibitors - therapeutic use | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Ubiquitin-Protein Ligases - genetics | Tumor proteins | Medical colleges | Genetic aspects | Development and progression | Lung cancer, Small cell | Lung cancer, Non-small cell
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2016, Volume 34, Issue 15_suppl, pp. 9083 - 9083
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 160 - 160
In the originally published version of this Article, the positions of the final two authors in the author list were inadvertently inverted during the...
Tumor suppressor genes | Carcinogens | Error correction | Carcinogenesis | MiRNA
Tumor suppressor genes | Carcinogens | Error correction | Carcinogenesis | MiRNA
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 02/2012, Volume 18, Issue 4, pp. 1092 - 1100
Purpose: Bosutinib, a potent ATP-competitive, quinolinecarbonitrile Src/Abl kinase inhibitor, was tested in this first-in-human phase I trial in patients with...
SRC INHIBITORS | ACTIVATION | INVASION | AZD0530 | EFFICACY | ONCOLOGY | PROSTATE-CANCER | THERAPEUTIC AGENTS | DASATINIB | SKI-606 BOSUTINIB | IDENTIFICATION | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Quinolines - administration & dosage | Nitriles - administration & dosage | Young Adult | Antineoplastic Agents - adverse effects | Aged, 80 and over | Adult | Female | Aniline Compounds - administration & dosage | Neoplasms - mortality | Treatment Outcome | Neoplasms - drug therapy | Protein Kinase Inhibitors - administration & dosage | Protein Kinase Inhibitors - therapeutic use | Survival Analysis | Aged | Quinolines - therapeutic use | Aniline Compounds - therapeutic use | Aniline Compounds - adverse effects | Neoplasm Staging | Neoplasms - pathology | Nitriles - adverse effects | Quinolines - adverse effects | Nitriles - therapeutic use
SRC INHIBITORS | ACTIVATION | INVASION | AZD0530 | EFFICACY | ONCOLOGY | PROSTATE-CANCER | THERAPEUTIC AGENTS | DASATINIB | SKI-606 BOSUTINIB | IDENTIFICATION | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Quinolines - administration & dosage | Nitriles - administration & dosage | Young Adult | Antineoplastic Agents - adverse effects | Aged, 80 and over | Adult | Female | Aniline Compounds - administration & dosage | Neoplasms - mortality | Treatment Outcome | Neoplasms - drug therapy | Protein Kinase Inhibitors - administration & dosage | Protein Kinase Inhibitors - therapeutic use | Survival Analysis | Aged | Quinolines - therapeutic use | Aniline Compounds - therapeutic use | Aniline Compounds - adverse effects | Neoplasm Staging | Neoplasms - pathology | Nitriles - adverse effects | Quinolines - adverse effects | Nitriles - therapeutic use
Journal Article
The Journal of Urology, ISSN 0022-5347, 2003, Volume 169, Issue 6, pp. 2076 - 2083
We better defined the benefits and morbidity of lymph node dissection in patients with localized renal cell carcinoma using the experience of patients treated...
kidney | lymph node dissection | carcinoma, renal cell | survival | immunotherapy | Lymph node dissection | Carcinoma, renal cell | Survival | Immunotherapy | Kidney | SURGERY | CLASSIFICATION | NATURAL-HISTORY | CANCER | RADICAL NEPHRECTOMY | LYMPHADENECTOMY | GROWTH | UROLOGY & NEPHROLOGY | SPREAD | EXPRESSION | Lymph Node Excision | Recombinant Proteins - therapeutic use | Outcome Assessment (Health Care) | Carcinoma, Renal Cell - pathology | Humans | Interleukin-2 - therapeutic use | Carcinoma, Renal Cell - surgery | Male | Survival Rate | Combined Modality Therapy | Lymphatic Metastasis | Kidney Neoplasms - mortality | Carcinoma, Renal Cell - mortality | Kidney Neoplasms - surgery | Regression Analysis | Analysis of Variance | Nephrectomy | Retroperitoneal Space | Carcinoma, Renal Cell - secondary | Female | Kidney Neoplasms - pathology | Retrospective Studies | Surgery | Carcinoma, Renal cell
kidney | lymph node dissection | carcinoma, renal cell | survival | immunotherapy | Lymph node dissection | Carcinoma, renal cell | Survival | Immunotherapy | Kidney | SURGERY | CLASSIFICATION | NATURAL-HISTORY | CANCER | RADICAL NEPHRECTOMY | LYMPHADENECTOMY | GROWTH | UROLOGY & NEPHROLOGY | SPREAD | EXPRESSION | Lymph Node Excision | Recombinant Proteins - therapeutic use | Outcome Assessment (Health Care) | Carcinoma, Renal Cell - pathology | Humans | Interleukin-2 - therapeutic use | Carcinoma, Renal Cell - surgery | Male | Survival Rate | Combined Modality Therapy | Lymphatic Metastasis | Kidney Neoplasms - mortality | Carcinoma, Renal Cell - mortality | Kidney Neoplasms - surgery | Regression Analysis | Analysis of Variance | Nephrectomy | Retroperitoneal Space | Carcinoma, Renal Cell - secondary | Female | Kidney Neoplasms - pathology | Retrospective Studies | Surgery | Carcinoma, Renal cell
Journal Article
Frontiers in Oncology, ISSN 2234-943X, 2013, Volume 3, p. 135
Epigenetic deregulation of gene function has been strongly implicated in carcinogenesis and is one of the mechanisms contributing to the development of lung...
DNA methylation | Epigenetics | Histone deacetylase inhibitors | Lung cancer | MicroRNAs | Hypomethylating agent | lung cancer | DNA Methylation | Histone deacetylase inhibitor | Hypomethylating Agent | epigenetics
DNA methylation | Epigenetics | Histone deacetylase inhibitors | Lung cancer | MicroRNAs | Hypomethylating agent | lung cancer | DNA Methylation | Histone deacetylase inhibitor | Hypomethylating Agent | epigenetics
Journal Article
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