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PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110291
Thiosemicarbazones (TSCs) are an interesting class of ligands that show a diverse range of biological activity, including antifungal, anti-viral and... 
METASTASIS SUPPRESSOR | SELECTIVE ANTINEOPLASTIC ACTIVITY | ANTIPROLIFERATIVE ACTIVITY | ATOMIC PHYSICOCHEMICAL PARAMETERS | RIBONUCLEOTIDE REDUCTASE INHIBITOR | PHASE-I TRIAL | MULTIDISCIPLINARY SCIENCES | 3-AMINOPYRIDINE-2-CARBOXALDEHYDE THIOSEMICARBAZONE | REDOX ACTIVITY | IRON OVERLOAD DISEASE | ANTITUMOR-ACTIVITY | Antineoplastic Agents - chemical synthesis | Humans | Iron - chemistry | Thiosemicarbazones - chemical synthesis | Structure-Activity Relationship | Antineoplastic Agents - chemistry | Ascorbic Acid - metabolism | Thiosemicarbazones - chemistry | Drug Design | Oxidation-Reduction - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Biological Transport - drug effects | Cell Proliferation - drug effects | Thiosemicarbazones - pharmacology | Care and treatment | Nitrogen | Surgery | Cancer | Reactive oxygen species | Ascorbic acid | Fungicides | Clinical trials | Iron | Structure-activity relationships | Kinases | Cancer therapies | Anticancer properties | Biological effects | Immunology | Ribonucleotide reductase | Antitumor agents | Piperazine | Oxidation | Sulfur | Coordination compounds | Antiviral agents | Enzymes | Fragments | Interdisciplinary aspects | Metabolism | Biological activity | Pathology | Morpholine | Chemistry | Pharmacy | In vivo methods and tests | Chelating agents | Alzheimers disease
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, pp. e0155893 - e0155893
Resistance to glucocorticosteroids (GCs) is a major adverse prognostic factor in B-ALL, but the molecular mechanisms leading to GC resistance are not... 
CANCER-CELLS | COMPLEX | PHOSPHORYLATION | MONITORING AUTOPHAGY | MULTIDISCIPLINARY SCIENCES | GLUCOCORTICOID-INDUCED APOPTOSIS | VITRO DRUG-SENSITIVITY | RESISTANCE | TUBEROUS SCLEROSIS | EXPRESSION PROFILES | INDUCTION | MAP Kinase Kinase Kinase 1 - metabolism | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - pathology | TOR Serine-Threonine Kinases - metabolism | Humans | Computational Biology | Autophagy | MAP Kinase Signaling System | Gene Expression Regulation, Enzymologic | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Dexamethasone - pharmacology | Flow Cytometry | Cell Death | Cell Line, Tumor | Benzimidazoles - pharmacology | MAP Kinase Kinase Kinase 1 - antagonists & inhibitors | Microscopy, Fluorescence | Apoptosis | RNA, Small Interfering - metabolism | Complications and side effects | Dexamethasone | Physiological aspects | Dosage and administration | Genetic aspects | Acute lymphocytic leukemia | Research | B cells | Drug therapy | TOR protein | Phosphorylation | Transplants & implants | Toxicity | Leukemia | Oncology | Kinases | Cancer therapies | Proteins | Signal transduction | Immunology | Modulation | Cell cycle | Inhibition | Acute lymphatic leukemia | Hematology | MEK inhibitors | Mortality | Markers | Extracellular signal-regulated kinase | MAP kinase | Lymphatic leukemia | Gene expression | Patients | Medicine | Signaling | Molecular modelling | Inhibitors | Lymphocytes B | Cell death | Cell lines | Phagocytosis | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2012, Volume 31, Issue 5, pp. 1062 - 1079
Journal Article