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1. Full Text Exploring the anti-cancer activity of novel thiosemicarbazones generated through the combination of retro-fragments: Dissection of critical structure-activity relationships
by Serda, Maciej and Kalinowski, Danuta S and Rasko, Nathalie and Potůčková, Eliška and Mrozek-Wilczkiewicz, Anna and Musiol, Robert and Małecki, Jan G and Sajewicz, Mieczysław and Ratuszna, Alicja and Muchowicz, Angelika and Goła¸b, Jakub and Šimůnek, Tomáš and Richardson, Des R and Polanski, Jaroslaw
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110291
Thiosemicarbazones (TSCs) are an interesting class of ligands that show a diverse range of biological activity, including antifungal, anti-viral and... 
METASTASIS SUPPRESSOR | SELECTIVE ANTINEOPLASTIC ACTIVITY | ANTIPROLIFERATIVE ACTIVITY | ATOMIC PHYSICOCHEMICAL PARAMETERS | RIBONUCLEOTIDE REDUCTASE INHIBITOR | PHASE-I TRIAL | MULTIDISCIPLINARY SCIENCES | 3-AMINOPYRIDINE-2-CARBOXALDEHYDE THIOSEMICARBAZONE | REDOX ACTIVITY | IRON OVERLOAD DISEASE | ANTITUMOR-ACTIVITY | Antineoplastic Agents - chemical synthesis | Humans | Iron - chemistry | Thiosemicarbazones - chemical synthesis | Structure-Activity Relationship | Antineoplastic Agents - chemistry | Ascorbic Acid - metabolism | Thiosemicarbazones - chemistry | Drug Design | Oxidation-Reduction - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Biological Transport - drug effects | Cell Proliferation - drug effects | Thiosemicarbazones - pharmacology | Care and treatment | Nitrogen | Surgery | Cancer | Reactive oxygen species | Ascorbic acid | Fungicides | Clinical trials | Iron | Structure-activity relationships | Kinases | Cancer therapies | Anticancer properties | Biological effects | Immunology | Ribonucleotide reductase | Antitumor agents | Piperazine | Oxidation | Sulfur | Coordination compounds | Antiviral agents | Enzymes | Fragments | Interdisciplinary aspects | Metabolism | Biological activity | Pathology | Morpholine | Chemistry | Pharmacy | In vivo methods and tests | Chelating agents | Alzheimers disease
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2. Full Text Photodynamic therapy of cancer: An update
by Agostinis, Patrizia and Berg, Kristian and Cengel, Keith A and Foster, Thomas H and Girotti, Albert W and Gollnick, Sandra O and Hahn, Stephen M and Hamblin, Michael R and Juzeniene, Asta and Kessel, David and Korbelik, Mladen and Moan, Johan and Mroz, Pawel and Nowis, Dominika and Piette, Jacques and Wilson, Brian C and Golab, Jakub
CA: A Cancer Journal for Clinicians, ISSN 0007-9235, 07/2011, Volume 61, Issue 4, pp. 250 - 281
Photodynamic therapy (PDT) is a clinically approved, minimally invasive therapeutic procedure that can exert a selective cytotoxic activity toward malignant... 
HEAT-SHOCK PROTEINS | PHASE-II TRIAL | FLUORESCENCE-GUIDED RESECTION | HIGH-GRADE DYSPLASIA | MOUSE-TUMOR MODEL | ONCOLOGY | BASAL-CELL CARCINOMA | ENDOPLASMIC-RETICULUM STRESS | RANDOMIZED CONTROLLED-TRIAL | RECURRENT PROSTATE-CANCER | TOPICAL METHYL AMINOLEVULINATE | Photosensitizing Agents - therapeutic use | Photochemotherapy - methods | Humans | Photochemotherapy - instrumentation | Neoplasms - drug therapy | Usage | Care and treatment | Research | Photochemotherapy | Health aspects | Methods | Cancer | Cancer therapies | Cells | Tumors | Apoptosis | Quality of life | Index Medicus | Abridged Index Medicus | cancer | laser | singlet oxygen | photodynamic therapy | photosensitizer
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3. Full Text MEK inhibition sensitizes precursor B-cell acute lymphoblastic leukemia (B-ALL) cells to dexamethasone through modulation of mTOR activity and stimulation of autophagy
by Polak, Anna and Kiliszek, Przemysław and Sewastianik, Tomasz and Szydłowski, Maciej and Jabłońska, Ewa and Białopiotrowicz, Emilia and Górniak, Patryk and Markowicz, Sergiusz and Nowak, Eliza and Grygorowicz, Monika A and Prochorec-Sobieszek, Monika and Nowis, Dominika and Gołab, Jakub and Giebel, Sebastian and Lech-Marańda, Ewa and Warzocha, Krzysztof and Juszczyński, Przemysław
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, pp. e0155893 - e0155893
Resistance to glucocorticosteroids (GCs) is a major adverse prognostic factor in B-ALL, but the molecular mechanisms leading to GC resistance are not... 
CANCER-CELLS | COMPLEX | PHOSPHORYLATION | MONITORING AUTOPHAGY | MULTIDISCIPLINARY SCIENCES | GLUCOCORTICOID-INDUCED APOPTOSIS | VITRO DRUG-SENSITIVITY | RESISTANCE | TUBEROUS SCLEROSIS | EXPRESSION PROFILES | INDUCTION | MAP Kinase Kinase Kinase 1 - metabolism | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - pathology | TOR Serine-Threonine Kinases - metabolism | Humans | Computational Biology | Autophagy | MAP Kinase Signaling System | Gene Expression Regulation, Enzymologic | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Dexamethasone - pharmacology | Flow Cytometry | Cell Death | Cell Line, Tumor | Benzimidazoles - pharmacology | MAP Kinase Kinase Kinase 1 - antagonists & inhibitors | Microscopy, Fluorescence | Apoptosis | RNA, Small Interfering - metabolism | Complications and side effects | Dexamethasone | Physiological aspects | Dosage and administration | Genetic aspects | Acute lymphocytic leukemia | Research | B cells | Drug therapy | TOR protein | Phosphorylation | Transplants & implants | Toxicity | Leukemia | Oncology | Kinases | Cancer therapies | Proteins | Signal transduction | Immunology | Modulation | Cell cycle | Inhibition | Acute lymphatic leukemia | Hematology | MEK inhibitors | Mortality | Markers | Extracellular signal-regulated kinase | MAP kinase | Lymphatic leukemia | Gene expression | Patients | Medicine | Signaling | Molecular modelling | Inhibitors | Lymphocytes B | Cell death | Cell lines | Phagocytosis | Index Medicus
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4. Full Text A novel pathway combining calreticulin exposure and ATP secretion in immunogenic cancer cell death
by Garg, Abhishek D and Krysko, Dmitri V and Verfaillie, Tom and Kaczmarek, Agnieszka and Ferreira, Gabriela B and Marysael, Thierry and Rubio, Noemi and Firczuk, Malgorzata and Mathieu, Chantal and Roebroek, Anton J M and Annaert, Wim and Golab, Jakub and de Witte, Peter and Vandenabeele, Peter and Agostinis, Patrizia
The EMBO Journal, ISSN 0261-4189, 03/2012, Volume 31, Issue 5, pp. 1062 - 1079
Surface‐exposed calreticulin (ecto‐CRT) and secreted ATP are crucial damage‐associated molecular patterns (DAMPs) for immunogenic apoptosis. Inducers of... 
calreticulin | cancer | immunogenic apoptosis | photodynamic therapy | DAMPs | MEMBRANE-PERMEABILITY | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | UNFOLDED PROTEIN RESPONSE | ANTICANCER THERAPY | NITRIC-OXIDE | ANTITUMOR IMMUNITY | MEDIATED PHOTODYNAMIC THERAPY | FIND-ME SIGNAL | MOLECULAR EFFECTORS | Cell Line | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Dendritic Cells - immunology | Humans | Adenosine Triphosphate - secretion | B7-1 Antigen - metabolism | Reactive Oxygen Species - toxicity | Antigens, CD - metabolism | Immunoglobulins - metabolism | B7-2 Antigen - metabolism | Photosensitizing Agents - pharmacology | Neoplasms - immunology | Endoplasmic Reticulum - drug effects | Cell Death | Calreticulin - metabolism | Histocompatibility Antigens Class II - metabolism | Signal transduction | Immunology | Photodynamic therapy | Cellular biology | Kinases | Apoptosis | Cancer
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5. Full Text Photodynamic therapy: illuminating the road from cell death towards anti-tumour immunity
by Garg, Abhishek D and Nowis, Dominika and Golab, Jakub and Agostinis, Patrizia
Apoptosis, ISSN 1360-8185, 9/2010, Volume 15, Issue 9, pp. 1050 - 1071
Photodynamic therapy (PDT) utilizes the destructive power of reactive oxygen species generated via visible light irradiation of a photosensitive dye... 
Biochemistry, general | Biomedicine | Photodynamic therapy | Immunology | Immunotherapy | Neutrophils | Oncology | Cancer Research | Antitumour immunity | Virology | Cell Biology | DAMPs | EXTRACORPOREAL PHOTOCHEMOTHERAPY | TOPICAL 5-AMINOLEVULINIC ACID | COLONY-STIMULATING FACTOR | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | VERSUS-HOST-DISEASE | CELL BIOLOGY | HEAT-SHOCK PROTEINS | ACUTE-PHASE RESPONSE | IN-VIVO | APOPTOTIC CELLS | NF-KAPPA-B | Immunity - drug effects | Animals | Neoplasms - immunology | Humans | Neoplasms - physiopathology | Photochemotherapy | Apoptosis | Neoplasms - drug therapy | Medical colleges | Cell death | Heat shock proteins | Biochemistry | Cancer | Tumors
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6. Full Text Natural mechanisms protecting against cancer
by Jakóbisiak, Marek and Lasek, Witold and Gołąb, Jakub
Immunology Letters, ISSN 0165-2478, 12/2003, Volume 90, Issue 2-3, pp. 103 - 122
Carcinogenesis is a multistage process. At each step of this process, there are natural mechanisms protecting against development of cancer. The majority of... 
Tumor | Immunosurveillance | Tumor suppressor genes | Oncogenes | NKT CELLS | METHYLCHOLANTHRENE-INDUCED SARCOMAS | COLONY-STIMULATING FACTOR | oncogenes | IMMUNOLOGY | immunosurveillance | FACTOR-ALPHA | tumor suppressor genes | TUMOR-NECROSIS-FACTOR | GROWTH-FACTOR-BETA | KILLER-CELL-ACTIVITY | APOPTOSIS-INDUCING LIGAND | tumor | IN-VIVO | T-CELLS | Neoplasms - metabolism | Cytokines - metabolism | Humans | Monitoring, Immunologic | Neoplasms - prevention & control | Cell Transformation, Neoplastic - metabolism | Immunity, Innate - immunology | Cell Transformation, Neoplastic - immunology | Animals | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Neoplasms - immunology | Genes, Tumor Suppressor | Cytokines - immunology
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