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Gut, ISSN 0017-5749, 03/2017, Volume 66, Issue 3, pp. 507 - 518
ObjectivePatients with liver cirrhosis suffer from increased susceptibility to life-threatening bacterial infections that cause substantial... 
Hepatic fibrosis | liver immunology | bacterial infection | immune response | liver cirrhosis | LISTERIA-MONOCYTOGENES | IMMUNITY | REACTIVE OXYGEN | COMMENSAL BACTERIA | I INTERFERONS | INTERLEUKIN-10 | INDUCTION | GASTROENTEROLOGY & HEPATOLOGY | MACROPHAGE ACTIVATION | HEPATIC-FIBROSIS | T-CELLS | Toll-Like Receptor 2 - genetics | Carbon Tetrachloride | Immunity, Innate - genetics | Toll-Like Receptor 9 - genetics | Receptor, Interferon alpha-beta - antagonists & inhibitors | Myeloid Cells - immunology | Listeriosis - immunology | Interferon Type I - metabolism | Receptor, Interferon alpha-beta - genetics | Toll-Like Receptor 7 - genetics | Liver Cirrhosis, Experimental - complications | Listeriosis - metabolism | Liver Cirrhosis, Experimental - immunology | Signal Transduction | Listeriosis - complications | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Mice, Transgenic | Bacterial Translocation | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Myeloid Cells - metabolism | Receptors, Interleukin-10 - antagonists & inhibitors | Receptors, Pattern Recognition - genetics | Interleukin-10 - biosynthesis | Mice | Myxovirus Resistance Proteins - genetics | Liver Cirrhosis, Experimental - metabolism | Myeloid Cells - microbiology | Bacterial infections | Microbiota (Symbiotic organisms) | Myelocytic leukemia | Genetic aspects | Nonlymphoid leukemia | Research | Liver cirrhosis
Journal Article
Journal Article
Glia, ISSN 0894-1491, 10/2018, Volume 66, Issue 10, pp. 2246 - 2261
Chemokines are important signaling molecules in the immune and nervous system. Using a fluorescence reporter mouse model, we demonstrate that the chemokine... 
CCL22 | hippocampus | microglia morphology | LPS‐induced inflammation | neuromodulatory chemokines | neuron–glia cross‐talk | CCL17 | neuron–glia cross-talk | LPS-induced inflammation | SEIZURE SUSCEPTIBILITY | CHEMOKINE RECEPTOR 4 | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | neuron-glia cross-talk | DENDRITIC CELLS | DEFICIENT MICE | NEUROSCIENCES | MICROGLIA | INFLAMMATION | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | PLASTICITY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Neurons - pathology | Synaptic Transmission - physiology | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Chemokine CCL17 - genetics | Male | Neuroimmunomodulation - physiology | Green Fluorescent Proteins - genetics | Monocytes - immunology | Chemokine CCL22 - metabolism | Lipopolysaccharides | Receptors, CCR4 - metabolism | Inflammation - metabolism | Microglia - immunology | Monocytes - pathology | Microglia - pathology | Female | Green Fluorescent Proteins - metabolism | Gene Expression | Hippocampus - immunology | Mice, Inbred C57BL | Neurons - immunology | Mice, Transgenic | Hippocampus - pathology | Animals | Homeostasis - physiology | Chemokine CCL17 - metabolism | Neurons | Neuroimaging | Brain | Cytology | Fluorescence | Nervous system | Cerebrospinal fluid | Macrophages | Cell surface | Neuromodulation | CCL22 protein | Synaptic transmission | Rodents | Tumor necrosis factor-TNF | CCL17 protein | Trees | Inflammation | Microglia | Signaling | Tumor necrosis factor | Morphology | Mice | Leukocytes (granulocytic) | Surface area | Hippocampus | Chemokines
Journal Article
Journal Article
Immunology, ISSN 0019-2805, 05/2015, Volume 145, Issue 1, pp. 150 - 160
Summary Sepsis initially starts with a systemic inflammatory response (SIRS phase) and is followed by a compensatory anti‐inflammatory response syndrome (CARS)... 
compensatory anti‐inflammatory response syndrome | T‐cell cytotoxicity | helminth | immune paralysis | sepsis | compensatory anti-inflammatory response syndrome | Helminth | Sepsis | T-cell cytotoxicity | Immune paralysis | APOPTOSIS | HUMAN DENDRITIC CELLS | LITOMOSOIDES-SIGMODONTIS | IMPROVES SURVIVAL | IMMUNOLOGY | IL-10 | HELMINTH INFECTION | NONOBESE DIABETIC MICE | INFLAMMATION | REGULATORY T-CELLS | SEVERE SEPSIS | Transforming Growth Factor beta - immunology | CD8-Positive T-Lymphocytes - pathology | Filariasis - immunology | Escherichia coli - immunology | CD4-Positive T-Lymphocytes - pathology | Systemic Inflammatory Response Syndrome - genetics | CD4-Positive T-Lymphocytes - immunology | Escherichia coli Infections - genetics | Systemic Inflammatory Response Syndrome - pathology | Animals | Transforming Growth Factor beta - genetics | Escherichia coli Infections - pathology | Filarioidea - immunology | Filariasis - pathology | Interferon-gamma - immunology | Systemic Inflammatory Response Syndrome - immunology | Filariasis - genetics | Escherichia coli Infections - immunology | Female | Mice | Mice, Inbred BALB C | Interferon-gamma - genetics | CD8-Positive T-Lymphocytes - immunology | Chronic Disease | Bacterial infections | Escherichia coli | Bone morphogenetic proteins | Development and progression | Paralysis | Biological response modifiers | T cells | Health aspects | Original
Journal Article
PLoS Pathogens, ISSN 1553-7366, 01/2015, Volume 11, Issue 1
  Helminths immunomodulate their hosts and induce a regulatory, anti-inflammatory milieu that prevents allergies and autoimmune diseases. Helminth... 
Antigens | Animals | Cytokines | E coli | Bacteria | Sepsis | Infections | Inflammation | Autoimmune diseases | Gene expression | Experiments | Allergies
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2012, Volume 188, Issue 2, pp. 559 - 568
Leading hypotheses to explain helminth-mediated protection against autoimmunity postulate that type 2 or regulatory immune responses induced by helminth... 
Journal Article
Journal of Immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 1/2012, Volume 188, Issue 2, pp. 559 - 568
Leading hypotheses to explain helminth-mediated protection against autoimmunity postulate that type 2 or regulatory immune responses induced by helminth... 
Type I diabetes | regulatory T cells | Litomosoides sigmodontis | autoimmune | interleukin 4 (IL-4) | nonobese diabetic (NOD) | IL-17 | TGFβ | filaria | helminth | FoxP3 | IL-10
Journal Article
PLoS ONE, 03/2014, Volume 9, Issue 3
Background Interactions of the Th2 cytokine IL-33 with its receptor ST2 lead to amplified Type 2 immune responses. As Type 2 immune responses are known to... 
Journal Article
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