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humans (20) 20
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female (17) 17
life sciences (17) 17
urology & nephrology (14) 14
kidney - pathology (13) 13
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nephrology (12) 12
kidneys (10) 10
adult (9) 9
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medicine (7) 7
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blotting, western (6) 6
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fibrosis (6) 6
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immunohistochemistry (5) 5
ischemia (5) 5
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preservation (5) 5
reperfusion injury - pathology (5) 5
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research (5) 5
research article (5) 5
retrospective studies (5) 5
[sdv.mhep]life sciences [q-bio]/human health and pathology (4) 4
aged, 80 and over (4) 4
dose-response relationship, drug (4) 4
human health and pathology (4) 4
kidney diseases (4) 4
mitochondria (4) 4
nephrectomy (4) 4
nephrotic syndrome (4) 4
reverse transcriptase polymerase chain reaction (4) 4
transplants & implants (4) 4
trimetazidine - pharmacology (4) 4
[ sdv ] life sciences [q-bio] (3) 3
acute-renal-failure (3) 3
adrenal cortex hormones - administration & dosage (3) 3
amyloidosis (3) 3
apoptosis (3) 3
cancer (3) 3
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damage (3) 3
delayed graft function (3) 3
drug therapy, combination (3) 3
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expression (3) 3
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fibrillary glomerulonephritis (3) 3
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heart (3) 3
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acute interstitial nephritis (2) 2
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1. Full Text Eculizumab for Treatment of Rapidly Progressive C3 Glomerulopathy
by Le Quintrec, Moglie, MD, PhD and Lionet, Arnaud, MD and Kandel, Christine, MD and Bourdon, Franck, MD and Gnemmi, Viviane, MD and Colombat, Magali, MD, PhD and Goujon, Jean-Michel, MD and Frémeaux-Bacchi, Véronique, MD, PhD and Fakhouri, Fadi, MD, PhD
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 65, Issue 3, pp. 484 - 489
C3 glomerulopathy (C3G) is a prototypic complement-mediated kidney disease. Rapidly progressive forms of C3G usually respond poorly to conventional treatments.... 
Nephrology | acute kidney injury (AKI) | rapidly progressing glomerulonephritis | C3 glomerulopathy (C3G) | eculizumab | complement | complement alternative pathway | C5b-9 deposits | MPGN TYPE | HEMOLYTIC-UREMIC SYNDROME | COMPLEMENT ABNORMALITIES | FACTOR-H | DEFICIENT | UROLOGY & NEPHROLOGY | MICE | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Complement C3 - analysis | Complement C3 - metabolism | Humans | Middle Aged | Male | Treatment Outcome | Glomerulonephritis, Membranoproliferative - drug therapy | Antibodies, Monoclonal, Humanized - administration & dosage | Adult | Female | Glomerulonephritis, Membranoproliferative - diagnosis | Infusions, Intravenous | Glomerulonephritis, Membranoproliferative - metabolism | Life Sciences
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2. Full Text Insights From the Use in Clinical Practice of Eculizumab in Adult Patients With Atypical Hemolytic Uremic Syndrome Affecting the Native Kidneys: An Analysis of 19 Cases
by Fakhouri, Fadi, MD, PhD and Delmas, Yahsou, MD and Provot, François, MD and Barbet, Christelle, MD and Karras, Alexandre, MD and Makdassi, Raifah, MD and Courivaud, Cécile, MD, PhD and Rifard, Khair, MD, PhD and Servais, Aude, MD, PhD and Allard, Catherine, MD and Besson, Virginie, MD and Cousin, Maud, MD and Châtelet, Valérie, MD and Goujon, Jean-Michel, MD, PhD and Coindre, Jean-Philippe, MD and Laurent, Guillaume, MD, PhD and Loirat, Chantal, MD and Frémeaux-Bacchi, Véronique, MD, PhD
American Journal of Kidney Diseases, ISSN 0272-6386, 2013, Volume 63, Issue 1, pp. 40 - 48
Background Atypical hemolytic uremic syndrome (aHUS) is a devastating form of renal thrombotic microangiopathy. Despite plasma exchange, the standard treatment... 
Nephrology | Atypical hemolytic uremic syndrome | eculizumab | thrombotic microangiopathy | complement | UROLOGY & NEPHROLOGY | THROMBOTIC THROMBOCYTOPENIC PURPURA | Biopsy - methods | Kidney - pathology | Humans | Kidney Function Tests - methods | Male | Drug Monitoring - methods | Hemolytic-Uremic Syndrome - epidemiology | Antibodies, Monoclonal, Humanized - administration & dosage | Hemolytic-Uremic Syndrome - physiopathology | Adult | Female | Hemolytic-Uremic Syndrome - diagnosis | Retrospective Studies | France | Kidney - physiopathology | Antibodies, Monoclonal, Humanized - adverse effects | Hemolytic-Uremic Syndrome - complications | Risk Assessment | Acute Kidney Injury - pathology | Immunologic Factors - administration & dosage | Renal Dialysis - statistics & numerical data | Treatment Outcome | Biopsy - statistics & numerical data | Acute Kidney Injury - physiopathology | Platelet Count - methods | Kidney Failure, Chronic - epidemiology | Creatinine - blood | Immunologic Factors - adverse effects | Atypical Hemolytic Uremic Syndrome | Hemolytic-Uremic Syndrome - drug therapy | Kidney Failure, Chronic - etiology | Life Sciences | Human health and pathology
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3. Full Text Kidney Diseases Associated With Monoclonal Immunoglobulin M–Secreting B-Cell Lymphoproliferative Disorders: A Case Series of 35 Patients
by Chauvet, Sophie, MD and Bridoux, Frank, MD, PhD and Ecotière, Laure, MD and Javaugue, Vincent, MD and Sirac, Christophe, PhD and Arnulf, Bertrand, MD, PhD and Thierry, Antoine, MD, PhD and Quellard, Nathalie and Milin, Serge, MD and Bender, Sébastien and Goujon, Jean-Michel, MD, PhD and Jaccard, Arnaud, MD, PhD and Fermand, Jean-Paul, MD and Touchard, Guy, MD
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 66, Issue 5, pp. 756 - 767
Background Kidney diseases associated with immunoglobulin M (IgM) monoclonal gammopathy are poorly described, with few data for patient outcomes and renal... 
Nephrology | tubular disorder | membranoproliferative glomerulonephritis | amyloidosis | kidney biopsy | cast nephropathy | Monoclonal gammopathy | renal response | Waldenström magroglobulinemia | Fanconi syndrome | B-cell lymphoproliferative disorder | IgM | MULTIPLE-MYELOMA | IMMUNOLOGICAL FEATURES | WALDENSTROMS MACROGLOBULINEMIA | RENAL SIGNIFICANCE | DEPOSITION DISEASE | Waldenstrom magroglobulinemia | HEAVY-CHAIN | INTERNATIONAL WORKSHOP | LIGHT-CHAIN AMYLOIDOSIS | UNDETERMINED SIGNIFICANCE | UROLOGY & NEPHROLOGY | NEPHROTIC SYNDROME | Lymphoma, B-Cell - complications | Humans | Middle Aged | Kidney Neoplasms - immunology | Lymphoproliferative Disorders - complications | Male | Lymphoproliferative Disorders - immunology | Immunoglobulin M - immunology | Waldenstrom Macroglobulinemia - pathology | Nephritis, Interstitial - pathology | Paraproteinemias - pathology | Waldenstrom Macroglobulinemia - complications | Amyloidosis - immunology | Lymphoma, B-Cell - immunology | Aged, 80 and over | Adult | Female | Amyloidosis - etiology | Retrospective Studies | Acute Kidney Injury - immunology | Kidney Neoplasms - complications | Acute Kidney Injury - etiology | Antibodies, Monoclonal - immunology | Paraproteinemias - immunology | Glomerulonephritis, Membranoproliferative - pathology | Amyloidosis - pathology | Acute Kidney Injury - pathology | Kidney Diseases - pathology | Nephrotic Syndrome - pathology | Paraproteinemias - complications | Kidney Diseases - immunology | Nephrotic Syndrome - immunology | Waldenstrom Macroglobulinemia - immunology | Glomerulonephritis, Membranoproliferative - immunology | B-Lymphocytes - immunology | Lymphoma, B-Cell - pathology | Amyloid - immunology | Nephritis, Interstitial - etiology | Kidney Neoplasms - pathology | Aged | Kidney Diseases - etiology | Glomerulonephritis, Membranoproliferative - etiology | Nephritis, Interstitial - immunology | Nephrotic Syndrome - etiology | Cohort Studies | Life Sciences | Immunology
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4. Full Text Long-term Kidney Disease Outcomes in Fibrillary Glomerulonephritis: A Case Series of 27 Patients
by Javaugue, Vincent, MD and Karras, Alexandre, MD and Glowacki, François, MD and McGregor, Brigitte, MD and Lacombe, Corinne and Goujon, Jean-Michel, MD, PhD and Ragot, Stéphanie, PharmD, PhD and Aucouturier, Pierre, PhD and Touchard, Guy, MD and Bridoux, Frank, MD, PhD
American Journal of Kidney Diseases, ISSN 0272-6386, 2013, Volume 62, Issue 4, pp. 679 - 690
Background Fibrillary glomerulonephritis (GN) is a rare disorder with poor renal prognosis. Therapeutic strategies, particularly the use of immunosuppressive... 
Nephrology | cyclophosphamide | immunosuppressive drugs | outcomes | Fibrillary glomerulonephritis | rituximab | RITUXIMAB TREATMENT | GLOMERULOPATHY | IGG | CLINICAL-FEATURES | RENAL-FAILURE | UROLOGY & NEPHROLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | C VIRAL-INFECTION | Humans | Immunosuppressive Agents - therapeutic use | Middle Aged | Male | Treatment Outcome | Glomerulonephritis - drug therapy | Time Factors | Glomerulonephritis - pathology | Aged, 80 and over | Adult | Female | Renin-Angiotensin System - drug effects | Aged | Retrospective Studies | Glomerulonephritis | Immunosuppressive Agents | Life Sciences | Immunology | Renin-Angiotensin System
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5. Full Text Distal Angiopathy and Atypical Hemolytic Uremic Syndrome: Clinical and Functional Properties of an Anti–Factor H IgAλ Antibody
by Rigothier, Claire, MD, PhD and Delmas, Yahsou, MD, PhD and Roumenina, Lubka T., PhD and Contin-Bordes, Cécile, PhD and Lepreux, Sébastien, MD, PhD and Bridoux, Frank, MD, PhD and Goujon, Jean Michel, MD, PhD and Bachelet, Thomas, MD, PhD and Touchard, Guy, MD, PhD and Frémeaux-Bacchi, Véronique, MD, PhD and Combe, Christian, MD, PhD
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 66, Issue 2, pp. 331 - 336
Abnormal regulation of the alternative pathway of the complement system is a well-described trigger of microangiopathy leading to atypical hemolytic uremic... 
Nephrology | complement activation | atypical hemolytic uremic syndrome (aHUS) | distal angiopathy | autoantibody | factor H antibody | alternative complement pathway | Gammopathy | complement factor H (CFH) | macroangiopathy | microvascular injury | Index Words Gammopathy | Immunoglobulin Light Chains - immunology | Paraproteinemias - immunology | Complement Factor H - immunology | Kidney Failure, Chronic - immunology | Antibodies, Monoclonal, Humanized - therapeutic use | Atypical Hemolytic Uremic Syndrome - therapy | Humans | Middle Aged | Paraproteinemias - therapy | Atypical Hemolytic Uremic Syndrome - etiology | Male | Atypical Hemolytic Uremic Syndrome - immunology | Paraproteinemias - complications | Raynaud Disease - etiology | Plasma Exchange | Kidney Failure, Chronic - therapy | Raynaud Disease - therapy | Immunoglobulin A - immunology | Thrombotic Microangiopathies - therapy | Raynaud Disease - immunology | Thrombotic Microangiopathies - etiology | Thrombotic Microangiopathies - immunology | Kidney Failure, Chronic - etiology
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6. Full Text FR167653 improves renal recovery and decreases inflammation and fibrosis after renal ischemia reperfusion injury
by Cau, Jerome, MD and Favreau, Frederic, PhD and Zhang, Keqiang, PhD and Febrer, Guillaume, MD and de la Motte, Gilles Regnault, MD and Ricco, Jean-Baptiste, MD and Goujon, Jean-Michel, MD, PhD and Hauet, Thierry, PhD
Journal of Vascular Surgery, ISSN 0741-5214, 2009, Volume 49, Issue 3, pp. 728 - 740
Objective Acute tubular necrosis (ATN) secondary to induced warm ischemia (WI) results in inflammatory and delayed fibrotic processes and remains a common... 
Surgery | SURGERY | ISCHEMIA/REPERFUSION INJURY | ACTIVATED PROTEIN-KINASE | PERITUBULAR CAPILLARIES | WARM ISCHEMIA | PERIPHERAL VASCULAR DISEASE | ACUTE TUBULAR-NECROSIS | PIG-KIDNEY MODEL | HYPOXIA | INHIBITOR | FAILURE | DAMAGE | Inflammation - pathology | Phosphorylation | Kidney - blood supply | Interleukin-1 - blood | Kidney - pathology | Tumor Necrosis Factor-alpha - blood | Male | Renal Insufficiency - prevention & control | Recovery of Function | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Kidney Function Tests | Necrosis | Proteinuria - prevention & control | Renal Insufficiency - physiopathology | Nephrectomy | Time Factors | Swine | p38 Mitogen-Activated Protein Kinases - metabolism | Kidney - physiopathology | Disease Models, Animal | Pyrazoles - pharmacology | Kidney - drug effects | Pyridines - administration & dosage | Constriction | Proteinuria - immunology | Reperfusion Injury - pathology | Pyrazoles - administration & dosage | Animals | Reperfusion Injury - prevention & control | Renal Insufficiency - pathology | Warm Ischemia - adverse effects | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Fibrosis | Reperfusion Injury - physiopathology | Inflammation - prevention & control | Pyridines - pharmacology | Infusions, Intravenous | Inflammation - physiopathology | Inflammation | Reperfusion injury
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7. Full Text Trimetazidine reduces early and long-term effects of experimental renal warm ischemia: A dose effect study
by Cau, Jerome, MD and Favreau, Frederic, PhD and Tillement, Jean Paul, MD, PhD and Lerman, Lilach O., MD, PhD and Hauet, Thierry, MD, PhD and Goujon, Jean Michel, MD, PhD
Journal of Vascular Surgery, ISSN 0741-5214, 2008, Volume 47, Issue 4, pp. 852 - 860.e4
Objective Renal ischemia reperfusion (IR) injury (IRI) is an important mechanism of acute renal failure (ARF) and a crucial factor of tissue damage during... 
Surgery | SURGERY | PERMEABILITY TRANSITION PORE | ISCHEMIA/REPERFUSION INJURY | TRANSPLANTS | REPERFUSION INJURY | COLD ISCHEMIA/REPERFUSION | MITOCHONDRIA | PERIPHERAL VASCULAR DISEASE | AUTOTRANSPLANTED PIG KIDNEYS | BINDING-SITES | HYPOXIA | FAILURE | Hypoxia-Inducible Factor 1, alpha Subunit - analysis | Immunohistochemistry | Trimetazidine - therapeutic use | Vasodilator Agents - pharmacology | Kidney - pathology | Reperfusion Injury - pathology | Vasodilator Agents - therapeutic use | Renal Circulation - physiology | Tissue Survival - drug effects | Blotting, Western | Dose-Response Relationship, Drug | Vimentin - analysis | Animals | Reperfusion Injury - prevention & control | Nephrectomy | Swine | Warm Ischemia - adverse effects | Trimetazidine - pharmacology | Fibrosis - pathology | Apoptosis | Ischemia | Analysis
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8. Full Text The CTX-M-15-Producing Escherichia coli Clone O25b: H4-ST131 Has High Intestine Colonization and Urinary Tract Infection Abilities
by Vimont, Sophie and Boyd, Anders and Bleibtreu, Alexandre and Bens, Marcelle and Goujon, Jean-Michel and Garry, Louis and Clermont, Olivier and Denamur, Erick and Arlet, Guillaume and Vandewalle, Alain
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e46547
Increasing numbers of pyelonephritis-associated uropathogenic Escherichia coli (UPEC) are exhibiting high resistance to antibiotic therapy. They include a... 
UNITED-STATES | STRAINS | COMPLETE GENOME SEQUENCE | PHYLOGENETIC GROUP | COMMUNITY | MULTIDISCIPLINARY SCIENCES | CTX-M-15 | SPECTRUM BETA-LACTAMASES | VIRULENCE | SEQUENCE TYPE ST131 | E. COLI | Kidney Diseases - microbiology | Gene Expression | beta-Lactam Resistance | Cytokines - metabolism | Kidney Diseases - pathology | Humans | Kidney - immunology | Uropathogenic Escherichia coli - growth & development | Escherichia coli Infections - microbiology | Male | beta-Lactamases - biosynthesis | Mice, Inbred C3H | Kidney Diseases - immunology | Kidney - microbiology | Kidney - metabolism | Animals | Intestines - microbiology | Uropathogenic Escherichia coli - pathogenicity | Escherichia coli Proteins - biosynthesis | Female | Urinary Tract Infections - microbiology | Mice | Uropathogenic Escherichia coli - enzymology | Cytokines - genetics | Urinary tract infections | Care and treatment | Dosage and administration | Research | Antibiotics | Escherichia coli | Growth rate | Bladder | Inflammatory response | Urinary tract | Phylogeny | Inoculation | Microbiota | E coli | Urinary bladder | Intestine | Phylogenetics | Leukocytes (polymorphonuclear) | Bacteria | Nosocomial infections | Strains (organisms) | Urogenital system | Urine | Enzymes | Kidneys | Streptomycin | Bacterial infections | Cytokines | Cloning | High resistance | Leukocytes (neutrophilic) | Inflammation | Urinary tract diseases | Strain | Biofilms | Plasmids | Pyelonephritis | Colonization | Chemokines
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9. Full Text Febrile abdominal pain revealing Horton's disease
by Chaudet, Arnaud and Goujon, Jean-Michel and Ghazali, Aiham Daniel
American Journal of Emergency Medicine, ISSN 0735-6757, 10/2017, Volume 35, Issue 10, pp. 1583.e3 - 1583.e5
Horton's disease is a systemic inflammatory vasculitis, usually found in persons over 50 years old. It affects medium and large-sized arteries stemming from... 
Corticosteroid | Aortitis | Horton's disease | Emergency | Temporal artery biopsy | CT scan | MANAGEMENT | AORTIC-ANEURYSM | POLYMYALGIA-RHEUMATICA | INVOLVEMENT | BIOPSY | EMERGENCY MEDICINE | DISSECTION | TEMPORAL ARTERITIS | OF-THE-LITERATURE | GIANT-CELL ARTERITIS | Abdominal Pain - diagnosis | Diagnosis, Differential | Glucocorticoids - administration & dosage | Abdominal Pain - etiology | Giant Cell Arteritis - diagnosis | Humans | Temporal Arteries - diagnostic imaging | Giant Cell Arteritis - complications | Giant Cell Arteritis - drug therapy | Fever - diagnosis | Tomography, X-Ray Computed | Dose-Response Relationship, Drug | Biopsy | Fever - etiology | Female | Aged | Aneurysms | CT imaging | Diagnostic imaging | Corticosteroids | Emergency medical services | Elderly people | Corticoids | Laboratories | Stenosis | Headaches | Arthritis | Family medical history | Arteries | Blood | Pain | Vasculitis | Computed tomography | Etiology | Aorta | Localization | Dissection | Drug dosages | Thickening | Endarteritis | Complications | Abnormalities | Rheumatology | Blood vessels | Patients | Fever | Abdomen | Coronary vessels | Sensitivity enhancement | Elderly | Geriatrics | Veins & arteries
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10. Full Text AL Amyloidosis
by Desport, Estelle and Bridoux, Frank and Sirac, Christophe and Delbes, Sébastien and Bender, Sébastien and Fernandez, Béatrice and Quellard, Nathalie and Lacombe, Corinne and Goujon, Jean-Michel and Lavergne, David and Abraham, Julie and Touchard, Guy and Fermand, Jean-Paul and Jaccard, Arnaud and Ctr Natl Reference Pour Amylose AL and Centre national de référence pour l’amylose AL et les autres maladies par dépôts d’immunoglobulines monoclonales
Orphanet Journal of Rare Diseases, ISSN 1750-1172, 2012, Volume 7, Issue 1, pp. 54 - 54
Definition of the disease: AL amyloidosis results from extra-cellular deposition of fibril-forming monoclonal immunoglobulin (Ig) light chains (LC) (most... 
AL amyloidosis | Primary» amyloidosis | Immunoglobulinic amyloidosis | ORGAN INVOLVEMENT | MEDICINE, RESEARCH & EXPERIMENTAL | PRIMARY SYSTEMIC AMYLOIDOSIS | LOCALIZED AMYLOIDOSIS | RENAL-TRANSPLANTATION | NATRIURETIC PEPTIDE | INTENSIVE MELPHALAN | LIGHT-CHAIN AMYLOIDOSIS | GENETICS & HEREDITY | IMMUNOGLOBULIN DEPOSITION DISEASE | HIGH-DOSE MELPHALAN | STEM-CELL TRANSPLANTATION | " Primary " amyloidosis | Diagnosis, Differential | Prognosis | Amyloidosis - epidemiology | Amyloidosis - pathology | Humans | Middle Aged | Amyloidosis - diagnosis | Aged | Amyloidosis - drug therapy | Care and treatment | Amyloidosis | Research | Diagnosis | Risk factors | Immunohistochemistry | Viral antibodies | Heart | Cardiac patients | Multiple myeloma | Antibodies | Kidney diseases | Heart failure | Plasma | Carpal tunnel syndrome | Chemotherapy | Heart attacks | Mortality | Rodents | Cardiovascular disease | Diabetic neuropathy | Life Sciences | Immunology | Primary » amyloidosis
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11. Full Text Successful heart transplantation following melphalan plus dexamethasone therapy in systemic AL amyloidosis
by Mignot, Aude and Bridoux, Frank and Thierry, Antoine and Varnous, Shaida and Pujo, Myriam and Delcourt, Annick and Gombert, Jean Marc and Goujon, Jean-Michel and Favreau, Frederic and Touchard, Guy and Herpin, Daniel and Jaccard, Arnaud
Haematologica, ISSN 0390-6078, 03/2008, Volume 93, Issue 3, pp. e32 - e35
1 Department of Nephrology and Renal Transplantation, CHU de Poitiers; Université de Poitiers, France. Centre de référence des amyloses primitives et des... 
AL amyloidosis | Cardiac transplantation | Dexamethasone | Melphalan | Heart Failure - surgery | Peripheral Nervous System Diseases - etiology | Dexamethasone - administration & dosage | Graft Rejection - prevention & control | Humans | Middle Aged | Heart Transplantation | Male | Paraproteinemias - complications | Secondary Prevention | Melphalan - therapeutic use | Immunoglobulin kappa-Chains - analysis | Dexamethasone - therapeutic use | Melphalan - administration & dosage | Amyloidosis - surgery | Amyloidosis - etiology | Drug Therapy, Combination | Immunoglobulin lambda-Chains - analysis | Heart Failure - etiology | Nephrotic Syndrome - etiology | Recurrence | Immunoglobulin kappa-Chains | Nephrotic Syndrome | Life Sciences | Immunology | Peripheral Nervous System Diseases | Heart Failure | Paraproteinemias | Graft Rejection | Amyloidosis | Immunoglobulin lambda-Chains
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12. Full Text TLR4- and TLR2-Mediated B Cell Responses Control the Clearance of the Bacterial Pathogen, Leptospira interrogans
by Chassin, Cecilia and Picardeau, Mathieu and Goujon, Jean-Michel and Bourhy, Pascale and Quellard, Nathalie and Darche, Sylvie and Badell, Edgar and d'Andon, Martine Fanton and Winter, Nathalie and Lacroix-Lamande, Sonia and Buzoni-Gatel, Dominique and Vandewalle, Alain and Werts, Catherine
The Journal of Immunology, ISSN 0022-1767, 08/2009, Volume 183, Issue 4, pp. 2669 - 2677
Leptospirosis is a widespread zoonosis caused by pathogenic Leptospira interrogans that are transmitted by asymptomatic infected rodents. Leptospiral... 
IMMUNE-RESPONSE | LETHAL INFECTION | IFN-GAMMA PRODUCTION | EPITHELIAL-CELLS | LIPID-A | ESCHERICHIA-COLI | LIPOPOLYSACCHARIDE | MICE | IMMUNOLOGY | SEROVAR ICTEROHAEMORRHAGIAE | T-CELLS | Toll-Like Receptor 2 - genetics | Signal Transduction - immunology | Toll-Like Receptor 2 - deficiency | Leptospira interrogans - growth & development | Toll-Like Receptor 4 - deficiency | Inflammation Mediators - metabolism | B-Lymphocyte Subsets - immunology | Female | Leptospirosis - microbiology | Inflammation Mediators - physiology | Leptospirosis - immunology | Genetic Predisposition to Disease | B-Lymphocyte Subsets - pathology | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Mice, Transgenic | Signal Transduction - genetics | Leptospira interrogans - immunology | Mice, Knockout | Toll-Like Receptor 4 - physiology | Animals | B-Lymphocyte Subsets - microbiology | Leptospirosis - pathology | Toll-Like Receptor 2 - physiology | Mice
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13. Full Text Defining the optimal duration for normothermic regional perfusion in the kidney donor: A porcine preclinical study
by Kerforne, Thomas and Allain, Geraldine and Giraud, Sebastien and Bon, Delphine and Ameteau, Virginie and Couturier, Pierre and Hebrard, William and Danion, Jerome and Goujon, Jean‐Michel and Thuillier, Raphael and Hauet, Thierry and Barrou, Benoit and Jayle, Christophe
American Journal of Transplantation, ISSN 1600-6135, 03/2019, Volume 19, Issue 3, pp. 737 - 751
Kidneys from donation after circulatory death (DCD) are highly sensitive to ischemia‐reperfusion injury and thus require careful reconditioning, such as... 
ischemia‐reperfusion injury (IRI) | animal models | donors and donation: donation after circulatory death (DCD) | extracorporeal membrane oxygenation (ECMO) | kidney transplantation/nephrology | basic (laboratory) research/science | ischemia-reperfusion injury (IRI) | SURGERY | CIRCULATORY DEATH | NITRIC-OXIDE SYNTHASE | RENAL ISCHEMIA | REPERFUSION | TRANSPLANTATION | LIVER | CARDIAC DEATH | HEME OXYGENASE-1 | IN-SITU | ORGAN DONATION | EXTRACORPOREAL MEMBRANE-OXYGENATION | Transplantation of organs, tissues, etc | Hemoglobin | Analysis | Donation of organs, tissues, etc | Creatinine | Kidneys | CD40 antigen | Pyruvic acid | Nitric-oxide synthase | Blood levels | Reperfusion | Ischemia | Perfusion | Cerebral blood flow | Tumor necrosis factor | Lactic acid | Kidney transplantation | Life Sciences | Human health and pathology
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14. Full Text Renal failure in pediatric Castleman disease: Four French cases with thrombotic microangiopathy
by Cousin, Elie and Flodrops, Hugues and Boyer, Olivia and Hogan, Julien and Ruin, Mahe and Couderc, Anne and Goujon, Jean‐Michel and Taque, Sophie
Pediatric Blood & Cancer, ISSN 1545-5009, 07/2018, Volume 65, Issue 7, pp. e27045 - n/a
Pediatric Castleman disease (CD) is an uncommon and poorly understood disorder of the lymph nodes. Renal failure has not been described in pediatric... 
pediatric Castleman disease | acute renal failure | thrombotic microangiopathy | ONCOLOGY | PEDIATRICS | OF-THE-LITERATURE | HEMATOLOGY | Pediatrics | Kidney failure | Lymphatic diseases | Children | Diseases | Hypertension | Edema | Thrombotic microangiopathy | Therapy | Kidneys | Rituximab | Lymph nodes | Computed tomography | Biopsy | Renal failure | Blood pressure | Failure | Lymph
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