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by Balachandran, Vinod P and Luksza, Marta and Zhao, Julia N and Makarov, Vladimir and Moral, John Alec and Remark, Romain and Herbst, Brian and Askan, Gokce and Bhanot, Umesh and Senbabaoglu, Yasin and Wells, Daniel K and Cary, Charles Ian Ormsby and Grbovic-Huezo, Olivera and Attiyeh, Marc and Medina, Benjamin and Zhang, Jennifer and Loo, Jennifer and Saglimbeni, Joseph and Abu-Akeel, Mohsen and Zappasodi, Roberta and Riaz, Nadeem and Smoragiewicz, Martin and Kelley, Z. Larkin and Basturk, Olca and Gönen, Mithat and Levine, Arnold J and Allen, Peter J and Fearon, Douglas T and Merad, Miriam and Gnjatic, Sacha and Iacobuzio-Donahue, Christine A and Wolchok, Jedd D and DeMatteo, Ronald P and Chan, Timothy A and Greenbaum, Benjamin D and Merghoub, Taha and Leach, Steven D and Australian Pancreatic Canc Genom and Garvan Institute of Medical Research and Bankstown Hospital and St Vincent’s Hospital and Royal Adelaide Hospital and Royal North Shore Hospital and Envoi Pathology and QIMR Berghofer Medical Research Institute and Prince of Wales Hospital and University of Melbourne, Centre for Cancer Research and University of Queensland, Institute for Molecular Bioscience and Royal Prince Alfred Hospital, Chris O’Brien Lifehouse and Princess Alexandria Hospital and Fremantle Hospital and Liverpool Hospital and Austin Hospital and University of Glasgow and Johns Hopkins Medical Institutes and Flinders Medical Centre and Westmead Hospital and St John of God Healthcare and ARC-Net Centre for Applied Research on Cancer and Australian Pancreatic Cancer Genome Initiative
Nature, ISSN 0028-0836, 11/2017, Volume 551, Issue 7681, pp. S12 - S16
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 9, p. e0220892
A comprehensive, accurate, and revisable alpha taxonomy is crucial for biodiversity studies, but is challenging when data from reference specimens are... 
Xenopus | Genetic aspects | Phylogeny | Identification and classification | Analysis | Distribution | Geographical distribution | Frogs | Rainforests | Endemic species | Taxonomy | Zoology | Genomes | Biodiversity | Amphibians | Gene sequencing | Biological diversity | Mitochondria | Morphology | Aridity | Phylogenetics | Polymorphism
Journal Article
Nature, ISSN 0028-0836, 11/2017, Volume 551, Issue 7681, pp. 517 - 520
Checkpoint blockade immunotherapies enable the host immune system to recognize and destroy tumour cells(1). Their clinical activity has been correlated with... 
CELL LUNG-CANCER | CTLA-4 BLOCKADE | MELANOMA | THERAPY | LANDSCAPE | PD-1 BLOCKADE | MULTIDISCIPLINARY SCIENCES | SENSITIVITY | RESISTANCE | CLINICAL-RESPONSE | REPERTOIRES | Humans | Lung Neoplasms - pathology | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Antigen Presentation | Melanoma - genetics | Immunotherapy | Cell Cycle Checkpoints - genetics | Cell Cycle Checkpoints - immunology | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Antigens, Neoplasm - genetics | Antigens, Neoplasm - immunology | Carcinoma, Non-Small-Cell Lung - genetics | Lymphocyte Activation | CTLA-4 Antigen - genetics | Lung Neoplasms - therapy | Melanoma - pathology | CTLA-4 Antigen - immunology | Carcinoma, Non-Small-Cell Lung - immunology | Carcinoma, Non-Small-Cell Lung - therapy | Lung Neoplasms - immunology | Models, Immunological | Melanoma - immunology | Survival Analysis | T-Lymphocytes - immunology | Programmed Cell Death 1 Receptor - immunology | CTLA-4 Antigen - antagonists & inhibitors | Melanoma - therapy | Programmed Cell Death 1 Receptor - genetics | Cohort Studies | Evolution, Molecular | Antigens | Care and treatment | Patient outcomes | Models | Health aspects | Tumors | Medical research | Peptides | PD-1 protein | Lung cancer | Melanoma | Cytotoxicity | Lymphocytes T | Patients | Proteins | Cell recognition | CTLA-4 protein | Major histocompatibility complex | Immune checkpoint | Lymphocytes | Evolution | Mutation | Histocompatibility | Cancer | Immune system | Fitness
Journal Article
Journal Article
Science, ISSN 0036-8075, 02/2018, Volume 359, Issue 6375, pp. 582 - 587
CD8(+) T cell-dependent killing of cancer cells requires efficient presentation of tumor antigens by human leukocyte antigen class I (HLA-I) molecules.... 
HIV-1 | METASTATIC MELANOMA | CTLA-4 BLOCKADE | THERAPY | PD-1 BLOCKADE | MULTIDISCIPLINARY SCIENCES | MUTATIONAL LANDSCAPE | RESISTANCE | IDENTIFICATION | HISTOCOMPATIBILITY ANTIGENS | MOLECULES | Immunotherapy - methods | Humans | Middle Aged | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Young Adult | Cancer Vaccines - therapeutic use | Antigen Presentation | Melanoma - genetics | Skin Neoplasms - mortality | Adult | Skin Neoplasms - immunology | Skin Neoplasms - therapy | Antigens, Neoplasm - immunology | Treatment Outcome | Histocompatibility Antigens Class I - genetics | Histocompatibility Antigens Class I - chemistry | Cancer Vaccines - immunology | Melanoma - immunology | Skin Neoplasms - genetics | Protein Conformation | Aged | CD8-Positive T-Lymphocytes - immunology | CTLA-4 Antigen - antagonists & inhibitors | Melanoma - therapy | Genetic Carrier Screening | Cohort Studies | Melanoma - mortality | Care and treatment | Histocompatibility antigens | Immunotherapy | HLA histocompatibility antigens | Genotype | Genetic aspects | Health aspects | Cancer | Peptides | CD8 antigen | Genes | Molecular dynamics | Cytotoxicity | Lymphocytes T | Vaccines | Homozygosity | Cancer vaccines | Cell recognition | Genotype & phenotype | Lymphocytes | Genotypes | Immune system | Antigen presentation | Antigens | Melanoma | Epitopes | Patients | Survival | Loci | Heterozygosity | White blood cells | Immune checkpoint | Cell death | Antigen (tumor-associated) | Loss of heterozygosity | Histocompatibility antigen HLA | Mutation | Recognition | Apoptosis
Journal Article