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1. Full Text BCL-2 family proteins as regulators of mitochondria metabolism
by Gross, Atan
Biochimica et biophysica acta. Bioenergetics, ISSN 0005-2728, 08/2016, Volume 1857, Issue 8, pp. 1243 - 1246
BCL-2 | MTCH2 | Mitochondria metabolism | BID | Stem cells | Apoptosis | Mitochondrial Membrane Transport Proteins - metabolism | Cell Proliferation | Ataxia Telangiectasia Mutated Proteins - metabolism | Calcium - metabolism | Humans | Gene Expression Regulation | bcl-2 Homologous Antagonist-Killer Protein - genetics | bcl-2-Associated X Protein - metabolism | Apoptosis - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | Mitochondria - metabolism | Stem Cells - cytology | Stem Cells - metabolism | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Mitochondrial Membrane Transport Proteins - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Differentiation | Ataxia Telangiectasia Mutated Proteins - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | BH3 Interacting Domain Death Agonist Protein - metabolism | Calcium Signaling | bcl-2-Associated X Protein - genetics | Proteins | Physiological aspects
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2. Full Text A ROS rheostat for cell fate regulation
by Maryanovich, Maria and Gross, Atan
Trends in cell biology, ISSN 0962-8924, 2012, Volume 23, Issue 3, pp. 129 - 134
Pathology | stem cell biology | mitochondria | reactive oxygen species | ATM | BID | cell cycle regulation | hematopoietic lineage | Mitochondria | Reactive oxygen species | Cell cycle regulation | Stem cell biology | Hematopoietic lineage | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Cycle - genetics | Reactive Oxygen Species - metabolism | Humans | NF-kappa B - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Mitochondria - genetics | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Hematopoiesis - genetics | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | NF-kappa B - genetics | Hematopoietic Stem Cells - cytology | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Mitochondrial DNA | Stem cells | Index Medicus
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3. Full Text Mitochondrial plasticity in cell fate regulation
by Bahat, Amir and Gross, Atan
The Journal of biological chemistry, ISSN 0021-9258, 09/2019, Volume 294, Issue 38, pp. 13852 - 13863
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Reactive Oxygen Species - metabolism | DNA, Mitochondrial - metabolism | Signal Transduction | Humans | Homeostasis | Mitochondria - metabolism | Stem Cells - metabolism | Mitochondrial Dynamics - physiology | Epigenesis, Genetic - physiology | Animals | Cell Nucleus - metabolism | Mitochondria - physiology | Cell Differentiation - physiology | Cell Plasticity - physiology | Index Medicus | stem cells | metabolic cross-talk | JBC Reviews | histone modification | mitochondria | ROS signaling | nucleus | molecular dynamics | self-renewal | epigenetics | mitochondrial DNA (mtDNA) | cell signaling | metabolism | mitochondrial metabolism
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4. Full Text Non-apoptotic functions of BCL-2 family proteins
by Gross, Atan and Katz, Samuel G
Cell death and differentiation, ISSN 1350-9047, 08/2017, Volume 24, Issue 8, pp. 1348 - 1358
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Regulators | Fracture mechanics | Bcl-2 protein | DNA damage | Homeostasis | Cracking (fracturing) | Proteins | Electron transport chain | Glucose metabolism | Mitochondria | Protein folding | Cell cycle | Physiology | Lipid metabolism | Calcium homeostasis | Deoxyribonucleic acid--DNA | Cell survival | Metabolism | Survival | Calcium permeability | Electron transport | Endoplasmic reticulum | Calcium (reticular) | Phagocytosis | Apoptosis | Index Medicus | Review
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5. Full Text MTCH2: A new player in mitochondria biology
by Gross, Atan
Biochimica et biophysica acta. Bioenergetics, ISSN 0005-2728, 08/2016, Volume 1857, pp. e14 - e14
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6. Full Text Aven-uing between nuclear export and cell cycle arrest
by Gross, Atan
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 10/2010, Volume 9, Issue 20, pp. 4043 - 4051
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Index Medicus
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7. Full Text M(a)TCH-ing up mitochondrial apoptosis
by Gross, Atan
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 08/2010, Volume 9, Issue 15, pp. 2925 - 2926
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Genome-Wide Association Study | Liver - pathology | Animals | Mitochondrial Proteins - metabolism | Models, Biological | Humans | Liver - metabolism | Mice | Mitochondria - metabolism | Apoptosis | Mitochondrial Membranes - metabolism | Index Medicus
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8. Full Text Balancing glycolysis and mitochondrial OXPHOS: Lessons from the hematopoietic system and exercising muscles
by Haran, Michal and Gross, Atan
Mitochondrion, ISSN 1567-7249, 11/2014, Volume 19, pp. 3 - 7
Glycolysis | Exercise | Hematopoiesis | Oxidative phosphorylation (OXPHOS) | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Glycolysis - physiology | Hematopoietic System - physiology | Animals | Oxidative Phosphorylation | Mitochondria - metabolism | Muscle, Skeletal - physiology | Energy Metabolism - physiology | Glucose metabolism | Index Medicus
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9. Full Text An MTCH2 pathway repressing mitochondria metabolism regulates haematopoietic stem cell fate
by Maryanovich, Maria and Zaltsman, Yehudit and Ruggiero, Antonella and Goldman, Andres and Shachnai, Liat and Zaidman, Smadar Levin and Porat, Ziv and Golan, Karin and Lapidot, Tsvee and Gross, Atan
Nature communications, ISSN 2041-1723, 07/2015, Volume 6, Issue 1, pp. 7901 - 7901
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cell Cycle - genetics | Reactive Oxygen Species - metabolism | Apoptosis - radiation effects | Apoptosis - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | Mitochondria - ultrastructure | Mitochondrial Membrane Transport Proteins - genetics | Glycolysis - genetics | Cell Differentiation - genetics | Flow Cytometry | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial | Colony-Forming Units Assay | Real-Time Polymerase Chain Reaction | BH3 Interacting Domain Death Agonist Protein - metabolism | Microscopy, Electron, Transmission | Mitochondrial Membrane Transport Proteins - metabolism | Oxidative Phosphorylation | Radiation Tolerance - genetics | Hematopoiesis - genetics | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Blotting, Western | Animals | Hematopoietic Stem Cells - cytology | Mice | Mitochondrial Size | Repressing | Phosphorylation | Reactive oxygen species | Hypersensitivity | Radiation | Homeostasis | Homology | Metabolism | Mitochondria | Oxidative phosphorylation | Cell fate | Stem cells | Bone marrow | Irradiation | Glycolysis | ATP | Deficient mutant | Apoptosis | Index Medicus
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10. Full Text BID as a Double Agent in Cell Life and Death
by Gross, Atan
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 03/2006, Volume 5, Issue 6, pp. 582 - 584
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Nucleus | Mitochondria | Phosphorylation | DNA damage | Cell cycle | ATM | S-phase | BID | Apoptosis | BH3-only | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Humans | Protein-Serine-Threonine Kinases - genetics | Apoptosis - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | Mitochondria - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Tumor Suppressor Protein p53 - genetics | Animals | Mitochondria - genetics | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Genes, cdc - physiology | Index Medicus
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11. Full Text Aven-uing between nuclear export and cell cycle arrest
by Gross, Atan
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 10/2010, Volume 9, Issue 20, pp. 4043 - 4044
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Active Transport, Cell Nucleus - physiology | Membrane Proteins - genetics | Humans | Adaptor Proteins, Signal Transducing - genetics | Apoptosis Regulatory Proteins - genetics | Cell Cycle - physiology | Membrane Proteins - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Apoptosis Regulatory Proteins - metabolism
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12. Full Text The ATM-BID pathway regulates quiescence and survival of haematopoietic stem cells
by Maryanovich, Maria and Oberkovitz, Galia and Niv, Hagit and Vorobiyov, Lidiya and Zaltsman, Yehudit and Brenner, Ori and Lapidot, Tsvee and Jung, Steffen and Gross, Atan
Nature cell biology, ISSN 1465-7392, 05/2012, Volume 14, Issue 5, pp. 535 - 541
Life Sciences & Biomedicine | Science & Technology | Cell Biology | DNA-Binding Proteins - physiology | Phosphorylation | BH3 Interacting Domain Death Agonist Protein - physiology | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Tumor Suppressor Proteins - physiology | Animals | Hematopoietic Stem Cells - cytology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Mice | DNA Damage | Cell Cycle Proteins - physiology | Cell Survival - physiology | DNA damage | Physiological aspects | Ataxia telangiectasia | Genetic aspects | Research | Risk factors | Hematopoietic stem cells | Apoptosis | Index Medicus
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13. Full Text The IFN-1 > BID > ROS pathway: Linking DNA damage with HSPC malfunction
by Gross, Atan and Tasdogan, Alpaslan and Fehling, Hans Joerg
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 05/2017, Volume 16, Issue 9, pp. 819 - 820
MTCH2 | HSPC malfunction | DNA damage | mitochondria | ROS | ATM | BID | interferon | MLL5 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Animals | Reactive Oxygen Species - metabolism | Signal Transduction | Humans | Hematopoietic Stem Cells - pathology | Interferon Type I - metabolism | Hematopoietic Stem Cells - metabolism | DNA Damage | BH3 Interacting Domain Death Agonist Protein - metabolism | Index Medicus
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14. Full Text MTCH2-mediated mitochondrial fusion drives exit from naïve pluripotency in embryonic stem cells
by Bahat, Amir and Goldman, Andres and Zaltsman, Yehudit and Khan, Dilshad H and Halperin, Coral and Amzallag, Emmanuel and Krupalnik, Vladislav and Mullokandov, Michael and Silberman, Alon and Erez, Ayelet and Schimmer, Aaron D and Hanna, Jacob H and Gross, Atan
Nature communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 5132 - 11
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Nanog Homeobox Protein - genetics | Dynamins - metabolism | Gene Expression | Mitochondrial Membrane Transport Proteins - metabolism | Pluripotent Stem Cells - cytology | Mitochondrial Dynamics - genetics | Mice, Inbred C57BL | Cells, Cultured | Dynamins - genetics | Mouse Embryonic Stem Cells - cytology | Mitochondria - metabolism | Mouse Embryonic Stem Cells - metabolism | Mice, Knockout | Mitochondrial Membrane Transport Proteins - genetics | Pluripotent Stem Cells - metabolism | Microscopy, Confocal | Animals | GTP Phosphohydrolases - metabolism | Mitochondria - genetics | GTP Phosphohydrolases - genetics | Mice | Nanog Homeobox Protein - metabolism | Regulators | Embryo cells | Stem cell transplantation | Metabolism | Embryos | Proteins | Mitochondria | Stem cells | Dynamin | Acetylation | Fusion protein | Pluripotency | Elongation | Glutamine | Index Medicus
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