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1. Ri zhi lu xuan yi
by Zhang, Yanyun and Gu, Yanwu, 1613-1682 and Duan, Tali
1994, Di 1 ban., Gu dai wen shi ming zhu xuan yi cong shu, ISBN 9787805236377, 4, 3, 10, 304
Gu, Yanwu, 1613-1682. Ri zhi lu
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2. Full Text Gut microbiome and serum metabolome alterations in obesity and after weight-loss intervention
by Liu, Ruixin and Hong, Jie and Xu, Xiaoqiang and Feng, Qiang and Zhang, Dongya and Gu, Yanyun and Shi, Juan and Zhao, Shaoqian and Liu, Wen and Wang, Xiaokai and Xia, Huihua and Liu, Zhipeng and Cui, Bin and Liang, Peiwen and Xi, Liuqing and Jin, Jiabin and Ying, Xiayang and Wang, Xiaolin and Zhao, Xinjie and Li, Wanyu and Jia, Huijue and Lan, Zhou and Li, Fengyu and Wang, Rui and Sun, Yingkai and Yang, Minglan and Shen, Yuxin and Jie, Zhuye and Li, Junhua and Chen, Xiaomin and Zhong, Huanzi and Xie, Hailiang and Zhang, Yifei and Gu, Weiqiong and Deng, Xiaxing and Shen, Baiyong and Xu, Xun and Yang, Huanming and Xu, Guowang and Bi, Yufang and Lai, Shenghan and Wang, Jian and Qi, Lu and Madsen, Lise and Wang, Jiqiu and Ning, Guang and Kristiansen, Karsten and Wang, Weiqing
Nature Medicine, ISSN 1078-8956, 07/2017, Volume 23, Issue 7, pp. 859 - 868
Emerging evidence has linked the gut microbiome to human obesity. We performed a metagenome-wide association study and serum metabolomics profiling in a cohort... 
MEDICINE, RESEARCH & EXPERIMENTAL | THETAIOTAOMICRON | RICHNESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | Y GASTRIC BYPASS | MARKERS | DIET-INDUCED OBESITY | CELL BIOLOGY | MONOSODIUM GLUTAMATE | IMPACT | GLUCOSE | GENES | BARIATRIC SURGERY | Humans | Male | Dysbiosis - microbiology | Obesity - microbiology | Bacteroides thetaiotaomicron - genetics | Case-Control Studies | Young Adult | Adiposity | Adult | DNA, Bacterial - analysis | Female | Weight Gain | Bariatric Surgery | Obesity - surgery | Metabolome | Fusobacterium - genetics | Gastrectomy | Gastrointestinal Microbiome - genetics | Dysbiosis - metabolism | Obesity - metabolism | Bacteroides - genetics | Animals | Glutamic Acid - blood | Mice | Metagenome | Obesity | Usage | Microbiota (Symbiotic organisms) | Patient outcomes | Analysis | Surgery | Weight loss | Glutamate | Health aspects | Intervention | Glutamic acid | Metabolomics | Stomach | Profiling | Adipose tissue | Amino acids | Abundance | Alterations | Microorganisms | Weight control | Metabolites | Intestine | Human performance | Rodents | Weight reduction | Links | Gastrointestinal surgery | Intestinal microflora | Digestive tract
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3. Full Text Analyses of gut microbiota and plasma bile acids enable stratification of patients for antidiabetic treatment
by Gu, Yanyun and Wang, Xiaokai and Li, Junhua and Zhang, Yifei and Zhong, Huanzi and Liu, Ruixin and Zhang, Dongya and Feng, Qiang and Xie, Xiaoyan and Hong, Jie and Ren, Huahui and Liu, Wei and Ma, Jing and Su, Qing and Zhang, Hongmei and Yang, Jialin and Wang, Xiaoling and Zhao, Xinjie and Gu, Weiqiong and Bi, Yufang and Peng, Yongde and Xu, Xiaoqiang and Xia, Huihua and Li, Fang and Xu, Xun and Yang, Huanming and Xu, Guowang and Madsen, Lise and Kristiansen, Karsten and Ning, Guang and Wang, Weiqing
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1785 - 12
Antidiabetic medication may modulate the gut microbiota and thereby alter plasma and faecal bile acid (BA) composition, which may improve metabolic health.... 
METFORMIN | ENTEROTYPES | METAGENOME | INSULIN-RESISTANCE | GLUCOSE | MULTIDISCIPLINARY SCIENCES | FARNESOID-X-RECEPTOR | BARIATRIC SURGERY | RANDOMIZED-TRIAL | SERUM METABOLOME | ACARBOSE | Hypoglycemic Agents - therapeutic use | Acarbose - therapeutic use | Bile Acids and Salts - blood | Diabetes Mellitus, Type 2 - microbiology | Humans | Middle Aged | Bacteroides - physiology | Glycoside Hydrolase Inhibitors - therapeutic use | Male | Glipizide - therapeutic use | Diabetes Mellitus, Type 2 - metabolism | Feces - chemistry | Gastrointestinal Microbiome - drug effects | Female | Lactobacillus - physiology | Diabetes Mellitus, Type 2 - drug therapy | Bifidobacterium - physiology | Population Dynamics | Drugs | Acarbose | Bile acids | Diabetes mellitus | Abundance | Metabolism | Patients | Microorganisms | Microbiota | Acids | Relative abundance | Plasma levels | Bile
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4. Full Text P1-22-6 Establishing the profile of differential expression proteins in diabetic arteries and using it in the study of diabetic macrovascular diseases
by Gu Yanyun and Rui Jin Hospital and Shanghai Jiaotong University School of Medicine and Shanghai Institute of Endocrine and Metabolic Diseases
ENDOCRINE JOURNAL, ISSN 0918-8959, 2010, Volume 57, Issue suppl.2-1, pp. S398 - S398
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5. Full Text Manipulating the Stacking of Triplet Chromophores in the Crystal Form for Ultralong Organic Phosphorescence
by Gan, Nan and Wang, Xuan and Ma, Huili and Lv, Anqi and Wang, He and Wang, Qian and Gu, Mingxing and Cai, Suzhi and Zhang, Yanyun and Fu, Lishun and Zhang, Meng and Dong, Chaomin and Yao, Wei and Shi, Huifang and An, Zhongfu and Huang, Wei
Angewandte Chemie International Edition, ISSN 1433-7851, 10/2019, Volume 58, Issue 40, pp. 14140 - 14145
Provided here is evidence showing that the stacking between triplet chromophores plays a critical role in ultralong organic phosphorescence (UOP) generation... 
phosphorescence | noncovalent interactions | fluorescence | crystal engineering | solid-state structures | Carbazoles | Stacking | Fluorescence | Chromophores | Phosphorescence | Carbazole | Crystal structure
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6. Full Text Mesenchymal stem cells alleviate bacteria‐induced liver injury in mice by inducing regulatory dendritic cells
by Zhang, Yi and Cai, Wei and Huang, Qingrong and Gu, Yuting and Shi, Yufang and Huang, Jiefang and Zhao, Fang and Liu, Qiang and Wei, Xunbin and Jin, Min and Wu, Changping and Xie, Qing and Zhang, Yi and Wan, Bing and Zhang, Yanyun
Hepatology, ISSN 0270-9139, 02/2014, Volume 59, Issue 2, pp. 671 - 682
Fulminant hepatic failure (FHF) is a clinical syndrome characterized by sudden and severe impairment of liver function. Mesenchymal stem cells (MSCs) have been... 
HEPATOCELLULAR-CARCINOMA | ACTIVATION | IN-VIVO | DISEASE | GENERATION | FULMINANT HEPATIC-FAILURE | GASTROENTEROLOGY & HEPATOLOGY | PRECURSORS | PIVOTAL ROLE | TRANSPLANTATION | ANTIGEN | Th1 Cells - pathology | T-Lymphocytes, Regulatory - metabolism | Liver - pathology | Liver Failure, Acute - microbiology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Dendritic Cells - pathology | Liver Failure, Acute - chemically induced | Cell- and Tissue-Based Therapy | Liver Failure, Acute - therapy | Lipopolysaccharides - adverse effects | T-Lymphocytes, Regulatory - pathology | Th1 Cells - metabolism | Animals | Mice | Mesenchymal Stromal Cells - pathology | Gram-Positive Bacterial Infections - complications | Propionibacterium acnes | Transforming Growth Factor beta - metabolism | Dendritic Cells - metabolism | Mesenchymal Stem Cell Transplantation | Disease Models, Animal | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Rodents | Liver | Hepatology | Stem cells | Regeneration | Liver Injury
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7. Full Text Essential role for TrpC5-containing extracellular vesicles in breast cancer with chemotherapeutic resistance
by Xin Ma and Zhen Chen and Dong Hua and Dongxu He and Linjun Wang and Peng Zhang and Junqi Wang and Yanfei Cai and Caiji Gao and Xiaodong Zhang and Fangfang Zhang and Teng Wang and Tingting Hong and Linfang Jin and Xiaowei Qi and Shuxian Chen and Xiaoting Gu and Dangtong Yang and Qiongxi Pan and Yifei Zhu and Yun Chen and Daozhen Chen and Liwen Jiang and Xiaofeng Han and Yanyun Zhang and Jian Jin and Xiaoqiang Yao
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 04/2014, Volume 111, Issue 17, pp. 6389 - 6394
A critical challenge for chemotherapy is the development of chemoresistance in breast cancer. However, the underlying mechanisms and validated predictors... 
PROMOTE TUMOR-GROWTH | CELLS | MEMBRANE-VESICLES | MULTIDISCIPLINARY SCIENCES | EXOSOMES | CA15.3 | MICROVESICLES | GENERATION | MEDIATORS | MICROPARTICLES | P-GLYCOPROTEIN | Breast Neoplasms - ultrastructure | Cytoplasmic Vesicles - drug effects | Doxorubicin - therapeutic use | Humans | TRPC Cation Channels - blood | Treatment Outcome | Breast Neoplasms - drug therapy | Breast Neoplasms - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Mice, Nude | Cytoplasmic Vesicles - ultrastructure | Cytoplasmic Vesicles - metabolism | TRPC Cation Channels - metabolism | Cell Line, Tumor | Female | Mice | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Pharmaceutical research | Chemotherapy | Oncology, Experimental | Breast cancer | Research | Drug therapy | Drug resistance | Cancer | Biological Sciences
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8. Structure and luminescence investigations on the chromophore intercalated layered rare-earth hydroxides hybrids
by Qingyang Gu and Xia Liu and Yingxi Hu and Lifang Zhang and Yanyun Li
Journal of Photochemistry and Photobiology. A, Chemistry, ISSN 1010-6030, 02/2017, Volume 335, p. 268
Niflumic acid (NFC) and surfactant 1-octane sulfonic acid sodium (OS) are intercalated into the gallery of NO3- type layered europium hydroxide (NO3-LEuH) by... 
Rare earth metals | Hybrids | Hydroxides | Luminescence | Atoms & subatomic particles | Emission | Layered materials | Thermal stability | Sulfonic acid | Emissions | Studies | Fabrication | Diffraction | Sodium | Acids | Synergistic effect | Ion exchange | Europium
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9. Full Text Dual Effect of Raptor on Neonatal beta-Cell Proliferation and Identity Maintenance
by Wang, YQ and Sun, JJ and Ni, QC and Nie, AF and Gu, YY and Wang, S and Zhang, WZ and Ning, G and Wang, WQ and Wang, QD
DIABETES, ISSN 0012-1797, 10/2019, Volume 68, Issue 10, pp. 1950 - 1964
Immature pancreatic beta-cells are highly proliferative, and the expansion of beta-cells during the early neonatal period largely determines functional... 
ACTIVATION | PANCREATIC-ISLETS | REPLICATION | ALPHA-CELLS | REGENERATION | MTOR PATHWAY | ENDOCRINOLOGY & METABOLISM | CYCLE PROGRESSION | MASS EXPANSION | MATURATION | GLUCAGON
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10. Full Text Relationships of circular RNA with diabetes and depression
by Jiang, Guangjian and Ma, Yue and An, Tian and Pan, Yanyun and Mo, Fangfang and Zhao, Dandan and Liu, Yufei and Miao, Jia-Nan and Gu, Yu-Jie and Wang, Yangang and Gao, Si-Hua
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 7285 - 8
Type 2 diabetes mellitus (T2DM) is closely related to depression; however, the exact molecular mechnisms of this association are unknown. Here, we investigated... 
DISORDER | ANXIETY | TYPE-2 | ASSOCIATION | MULTIDISCIPLINARY SCIENCES | Wnt protein | Mental disorders | ErbB protein | Diabetes mellitus | MiRNA | Reverse transcription | MAP kinase | Mental depression | Gene expression | Kinases | Polymerase chain reaction | Signal transduction | DNA microarrays | Protein kinase | Circular RNA | Diabetes | Thyroid
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11. Full Text Raptor regulates functional maturation of murine beta cells
by Ni, Qicheng and Gu, Yanyun and Xie, Yun and Yin, Qinglei and Zhang, Hongli and Nie, Aifang and Li, Wenyi and Wang, Yanqiu and Ning, Guang and Wang, Weiqing and Wang, Qidi
Nature communications, ISSN 2041-1723, 06/2017, Volume 8, Issue 1, p. 15755
Diabetes is associated with beta cell mass loss and islet dysfunctions. mTORC1 regulates beta cell survival, proliferation and function in physiological and... 
LACTATE-DEHYDROGENASE | MAMMALIAN TARGET | RAPAMYCIN | ENDOCRINE PANCREAS | MESSENGER-RNA TRANSLATION | MULTIDISCIPLINARY SCIENCES | HEXOKINASE-I | MICE | INDUCED INSULIN-SECRETION | GLUCOSE-TOLERANCE | MTOR | Diabetes Mellitus, Type 2 - genetics | Humans | Mice, Inbred C57BL | Male | Diabetes Mellitus, Type 2 - metabolism | Mechanistic Target of Rapamycin Complex 1 - metabolism | Mice, Knockout | DNA (Cytosine-5-)-Methyltransferases - metabolism | DNA (Cytosine-5-)-Methyltransferases - genetics | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Mechanistic Target of Rapamycin Complex 1 - genetics | Animals | Glucose - metabolism | Female | Cell Differentiation | Insulin-Secreting Cells - cytology | Mice | Regulatory-Associated Protein of mTOR - genetics | Regulatory-Associated Protein of mTOR - metabolism
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12. Full Text Leonurine suppresses neuroinflammation through promoting oligodendrocyte maturation
by Jin, Min and Li, Qian and Gu, Yuting and Wan, Bing and Huang, Jiefang and Xu, Xuanbai and Huang, Rui and Zhang, Yanyun
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2019, Volume 23, Issue 2, pp. 1470 - 1485
Focal inflammation and remyelination failure are major hallmarks of multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE).... 
EAE | CNS inflammation | oligodendrocyte differentiation | leonurine | remyelination | MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | STEM-CELLS | ACTIVATION | THYROID-HORMONE | CNS | CELL BIOLOGY | MULTIPLE-SCLEROSIS | INFLAMMATION | GENE-EXPRESSION | CUPRIZONE | Multiple sclerosis | Disease susceptibility | T cells | Analysis | Animal models | Encephalomyelitis | Maturation | Myelin | Pathogenesis | Central nervous system | Nervous system | Lymphocytes T | Inflammation | Experimental allergic encephalomyelitis | Microglia | Proteins | Myelination | Cuprizone | Demyelination | Lysine | Lymphocytes | Oligodendrocytes | Methylation | Histone H3 | Original
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13. Full Text Analysis of Plasma EBV-DNA and Soluble Checkpoint Proteins in Nasopharyngeal Carcinoma Patients after Definitive Intensity-Modulated Radiotherapy
by Ruan, YY and Hu, W and Li, WH and Lu, HS and Gu, HM and Zhang, Y and Zhu, CM and Chen, Q
BIOMED RESEARCH INTERNATIONAL, ISSN 2314-6133, 2019, Volume 2019, pp. 3939720 - 9
Background. Tumor immunotherapy and immunological checkpoint-related proteins are research hotspots. Intensity-modulated radiotherapy (IMRT) is the main... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | Interleukin 22 | CD223 antigen | Metastasis | Gender | Interleukin 6 | Proteins | Nasopharyngeal carcinoma | Immunology | Interleukin 5 | Immunotherapy | Deoxyribonucleic acid--DNA | Age | Target detection | Medical research | Statistical analysis | Cytokines | Interleukin 12 | Radiation therapy | Tumor necrosis factor-α | Gene expression | IL-1β | Survival | Patients | Cell death | Pancreatic cancer | Medical prognosis | γ-Interferon | PD-L1 protein | Interleukin 10 | Biomarkers | Ligands | Software | Apoptosis | Tumors
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14. Full Text IgG Galactosylation status combined with MYOM2-rs2294066 precisely predicts anti-TNF response in ankylosing spondylitis
by Liu, Jing and Zhu, Qi and Han, Jing and Zhang, Hui and Li, Yuan and Ma, Yanyun and He, Dongyi and Gu, Jianxin and Zhou, Xiaodong and Reveille, John D and Jin, Li and Zou, Hejian and Ren, Shifang and Wang, Jiucun
Molecular Medicine, ISSN 1076-1551, 06/2019, Volume 25, Issue 1, pp. 25 - 7
Background: Tumor necrosis factor (TNF) blockers have a high efficacy in treating Ankylosing Spondylitis (AS), yet up to 40% of AS patients show poor or even... 
TNF blocker | Ankylosing spondylitis | Drug response prediction | IgG-gal ratio | MYOM2-rs2294066 | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOSYLATION | CELL BIOLOGY | ALPHA BLOCKERS | POLYMORPHISMS | DISEASE | ASSOCIATION
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15. Full Text Rictor/mTORC2 is essential for maintaining a balance between β-cell proliferation and cell size
by Gu, Yanyun and Lindner, Jill and Kumar, Anil and Yuan, Weiping and Magnuson, Mark A
Diabetes, ISSN 0012-1797, 03/2011, Volume 60, Issue 3, pp. 827 - 837
OBJECTIVE-We examined the role of Rictor/mammalian target of rapamycin complex 2 (mTORC2), a key component of the phosphotidylinositol-3-kinase... 
MAMMALIAN TARGET | SURVIVAL | DISRUPTION | GENE | ISLET MASS | TRANSCRIPTION FACTOR FOXO1 | GROWTH | EXPANSION | ENDOCRINOLOGY & METABOLISM | MICE | PDK1 | Glucose Intolerance - metabolism | Phosphorylation - physiology | Cell Proliferation | Hyperglycemia - genetics | Proto-Oncogene Proteins c-akt - genetics | Insulin-Secreting Cells - metabolism | Insulin-Secreting Cells - cytology | Proto-Oncogene Proteins c-akt - metabolism | Rapamycin-Insensitive Companion of mTOR Protein | PTEN Phosphohydrolase - genetics | Glucose Intolerance - genetics | PTEN Phosphohydrolase - metabolism | Cell Size | Genotype | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Hyperglycemia - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Analysis of Variance | Alleles | Fluorescent Antibody Technique | Glucose - metabolism | Signal Transduction - physiology | Mice | Apoptosis - physiology | Islet Studies
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16. Full Text JMJD5 cleaves monomethylated histone H3 N‐tail under DNA damaging stress
by Shen, Jing and Xiang, Xueping and Chen, Lihan and Wang, Haiyi and Wu, Li and Sun, Yanyun and Ma, Li and Gu, Xiuting and Liu, Hong and Wang, Lishun and Yu, Ying‐nian and Shao, Jimin and Huang, Chao and Chin, Y Eugene
EMBO reports, ISSN 1469-221X, 12/2017, Volume 18, Issue 12, pp. 2131 - 2143
The histone H3 N‐terminal protein domain (N‐tail) is regulated by multiple posttranslational modifications, including methylation, acetylation,... 
N‐tail cleavage | H3K9me1 | Histone H3 | JMJD5 | N-tail cleavage | CHROMATIN | DOMAIN-CONTAINING PROTEIN | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLIPPING ACTIVITY | PROTEOLYSIS | IDENTIFICATION | H2AX PHOSPHORYLATION | CELL BIOLOGY | DEMETHYLASE | CATHEPSIN-L | EMBRYONIC STEM-CELLS | A549 Cells | Phosphorylation | Humans | Protein Processing, Post-Translational - genetics | Gene Expression Regulation | Histone Demethylases - genetics | Histone Demethylases - metabolism |