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Allergy, ISSN 0105-4538, 08/2016, Volume 71, Issue 8, pp. 1103 - 1134
Journal Article
Allergy, ISSN 0105-4538, 04/2016, Volume 71, Issue 4, pp. 443 - 462
Journal Article
Clinical & Experimental Allergy, ISSN 0954-7894, 12/2012, Volume 42, Issue 12, pp. 1772 - 1781
Summary Background To date, genetic studies of hypersensitivity reactions to non‐steroidal anti‐inflammatory drugs (NSAIDs) have been carried out mainly in... 
prostaglandins | skin reactions | drug allergy | mast cells | NSAIDs | leukotrienes | Prostaglandins | Mast cells | Skin reactions | Leukotrienes | Drug allergy | SPANISH POPULATION | ASPIRIN-INTOLERANT ASTHMA | LEUKOTRIENE C-4 SYNTHASE | PROMOTER POLYMORPHISM | IMMUNOLOGY | PERIPHERAL-BLOOD LEUKOCYTES | ALLERGY | KOREAN POPULATION | A-444C POLYMORPHISM | SENSITIVE ASTHMATICS | CYSTEINYL-LEUKOTRIENES | PROSTAGLANDIN E-2 | Arachidonic Acid - metabolism | Humans | Middle Aged | Urticaria - genetics | Male | Receptors, Prostaglandin - metabolism | Case-Control Studies | Receptors, Prostaglandin - genetics | Drug Hypersensitivity - genetics | Mast Cells - metabolism | Adult | Female | Drug Hypersensitivity - etiology | Acute Disease | Genotype | Receptors, Leukotriene - genetics | Arachidonate 15-Lipoxygenase - metabolism | Receptors, Leukotriene - metabolism | Leukotrienes - metabolism | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Arachidonate 15-Lipoxygenase - genetics | Urticaria - chemically induced | Polymorphism, Single Nucleotide | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Prostaglandins - metabolism | Enzymes | Unsaturated fatty acids | Urticaria | Analysis | Genes | Genetic research | Nonsteroidal anti-inflammatory drugs | Arachidonic acid | Cells | Drug therapy | Medical research | Side effects | Index Medicus | Aspirin | Genotyping | Hypersensitivity | Data processing | Population studies | Inflammation | Single-nucleotide polymorphism | Asthma | Nonsteroidal antiinflammatory drugs
Journal Article
Journal Article
Journal Article
Allergy, ISSN 0105-4538, 08/2013, Volume 68, Issue 8, pp. 1001 - 1007
Background Multiple NSAID‐induced urticaria/angioedema (MNSAID‐UA) is an entity well differentiated from aspirin‐exacerbated respiratory disease (AERD),... 
inflammatory mediators | aspirin‐exacerbated cutaneous disease | nonsteroidal anti‐inflammatory drugs | lysine nasal challenge | multiple nonsteroidal anti‐inflammatory drug‐induced urticaria/angioedema | nonsteroidal anti-inflammatory drugs | aspirin-exacerbated cutaneous disease | multiple nonsteroidal anti-inflammatory drug-induced urticaria/angioedema | DIAGNOSIS | TESTS | ACID | INTOLERANT ASTHMA | LYSINE-ASPIRIN | PATTERNS | IMMUNOLOGY | angioedema | multiple nonsteroidal anti-inflammatory drug-induced urticaria | ALLERGY | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | INTRANASAL CHALLENGE | SENSITIVE ASTHMATICS | URTICARIA | Humans | Middle Aged | Respiratory Hypersensitivity - chemically induced | Urticaria - genetics | Male | Urticaria - immunology | Respiratory Hypersensitivity - immunology | Administration, Intranasal | Angioedema - immunology | Angioedema - genetics | Aspirin - administration & dosage | Young Adult | Respiratory Hypersensitivity - genetics | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Aspirin - adverse effects | Adolescent | Inflammation Mediators - metabolism | Urticaria - chemically induced | Adult | Angioedema - chemically induced | Female | Immunophenotyping - methods | Aged | Allergy | Respiratory tract diseases | Aspirin | Genetic aspects | Allergic reaction | Analysis | Genotype & phenotype | Allergies | Nonsteroidal anti-inflammatory drugs | Life Sciences
Journal Article