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1. Full Text Anthrax lethal toxin rapidly reduces c-Jun levels by inhibiting c-Jun gene transcription and promoting c-Jun protein degradation
by Ouyang, Weiming and Guo, Pengfei and Fang, Hui and Frucht, David M
The Journal of biological chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 43, pp. 17919 - 17927
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Transcription, Genetic - drug effects | Nitriles - pharmacology | Humans | MAP Kinase Kinase 1 - genetics | Proteolysis - drug effects | MAP Kinase Signaling System - genetics | Mitogen-Activated Protein Kinase 1 - genetics | MAP Kinase Kinase 2 - genetics | Bacillus anthracis - chemistry | Butadienes - pharmacology | Antigens, Bacterial - chemistry | Mitogen-Activated Protein Kinase 3 - genetics | Proto-Oncogene Proteins c-jun - genetics | Antigens, Bacterial - pharmacology | MAP Kinase Kinase 1 - metabolism | MAP Kinase Kinase 2 - metabolism | Bacterial Toxins - chemistry | Hep G2 Cells | Animals | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Bacterial Toxins - pharmacology | Proto-Oncogene Proteins c-jun - metabolism | Mice | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | anthrax toxin | transcription factor | Signal Transduction | cell proliferation | c-Jun transcription factor | MAPK | protein degradation
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2. Jie fang jun shi jian
2013, ISBN 9787543689091, 1 v.
Book
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3. Full Text The oncogene c-Jun impedes somatic cell reprogramming
by Liu, Jing and Han, Qingkai and Peng, Tianran and Peng, Meixiu and Wei, Bei and Li, Dongwei and Wang, Xiaoshan and Yu, Shengyong and Yang, Jiaqi and Cao, Shangtao and Huang, Kaimeng and Hutchins, Andrew Paul and Liu, He and Kuang, Junqi and Zhou, Zhiwei and Chen, Jing and Wu, Haoyu and Guo, Lin and Chen, Yongqiang and Chen, You and Li, Xuejia and Wu, Hongling and Liao, Baojian and He, Wei and Song, Hong and Yao, Hongjie and Pan, Guangjin and Chen, Jiekai and Pei, Duanqing
Nature cell biology, ISSN 1465-7392, 07/2015, Volume 17, Issue 7, pp. 856 - 867
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Embryonic Stem Cells - metabolism | Humans | Repressor Proteins | Immunoblotting | Male | Gene Expression Profiling | Mice, 129 Strain | Epithelial-Mesenchymal Transition - genetics | Octamer Transcription Factor-3 - genetics | RNA Interference | HEK293 Cells | Mice, Inbred CBA | Female | Cellular Reprogramming - genetics | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Cell Proliferation - genetics | Proto-Oncogene Proteins c-jun - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Mice, Inbred ICR | Microscopy, Confocal | Animals | Embryo, Mammalian - cytology | Proto-Oncogene Proteins c-jun - metabolism | Cell proliferation | Transcription factors | Genetic aspects | Properties | Carcinogenesis | Index Medicus
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4. Bei yang hai jun xin tan : bei yang hai jun cheng jun 120 zhou nian guo ji xue shu yan tao hui lun wen ji
by Qi, Junjie and Guo, Yang
2012, ISBN 9787101086744
Book
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5. Full Text Anxa5 mediates the in vitro malignant behaviours of murine hepatocarcinoma Hca-F cells with high lymph node metastasis potential preferentially via ERK2/p-ERK2/c-Jun/p-c-Jun(Ser73) and E-cadherin
by Sun, Xujuan and Wei, Bin and Liu, Shuqing and Guo, Chunmei and Wu, Na and Liu, Qinlong and Sun, Ming-Zhong
Biomedicine & pharmacotherapy, ISSN 0753-3322, 2016, Volume 84, pp. 645 - 654
Internal Medicine | Medical Education | Anxa5 | c-Jun | Hepatocarcinoma | ERK2 | Malignant behaviour | E-cadherin | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Phosphorylation | Cell Proliferation | Cadherins - metabolism | Signal Transduction | Gene Expression Regulation, Neoplastic | Annexin A5 - genetics | Carcinoma, Hepatocellular - secondary | Lymphatic Metastasis | Carcinoma, Hepatocellular - enzymology | Cell Adhesion | Annexin A5 - metabolism | Animals | Transfection | RNA Interference | Time Factors | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - genetics | Cell Line, Tumor | Liver Neoplasms, Experimental - enzymology | Liver Neoplasms, Experimental - genetics | Mice | Liver Neoplasms, Experimental - pathology | Cell Movement | Mitogen-Activated Protein Kinase 1 - metabolism | Annexins | Development and progression | Metastasis | Measuring instruments | Index Medicus
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6. Full Text miR200c attenuates P-gp-mediated MDR and metastasis by targeting JNK2/c-Jun signaling pathway in colorectal cancer
by Sui, Hua and Cai, Guo-Xiang and Pan, Shu-Fang and Deng, Wan-Li and Wang, Yu-Wei and Chen, Zhe-Sheng and Cai, San-Jun and Zhu, Hui-Rong and Li, Qi
Molecular cancer therapeutics, ISSN 1535-7163, 12/2014, Volume 13, Issue 12, pp. 3137 - 3151
Life Sciences & Biomedicine | Oncology | Science & Technology | Colorectal Neoplasms - genetics | Humans | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Male | Antineoplastic Agents - therapeutic use | Mitogen-Activated Protein Kinase 9 - genetics | Cell Movement - genetics | Matrix Metalloproteinase 9 - metabolism | Neoplasm Metastasis | RNA Interference | Base Sequence | Colorectal Neoplasms - drug therapy | Proto-Oncogene Proteins c-jun - chemistry | Antineoplastic Agents - pharmacology | 3' Untranslated Regions | Binding Sites | Colorectal Neoplasms - metabolism | Disease Models, Animal | Mitogen-Activated Protein Kinase 9 - chemistry | Gene Expression | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Mitogen-Activated Protein Kinase 9 - metabolism | Proto-Oncogene Proteins c-jun - genetics | RNA, Messenger - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Mice | MicroRNAs - genetics | Colorectal Neoplasms - pathology | MicroRNAs - chemistry | Index Medicus
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7. Jin xian dai Riben she Hua mi dang
: Hai jun sheng juan
by Guo, Hongmao, 1955 and Li, Li and Japan. Kaigunshō
2012, Di 1 ban., ISBN 9787512005471, 68 v.
Russo-Japanese War, 1904-1905 | Sino-Japanese War, 1894-1895 | China Foreign relations | Japan Foreign relations
Book
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8. Jin xian dai Riben she Hua mi dang
: Lu jun sheng juan
by Li, Li and Guo, Hongmao, 1955 and Japan. Rikugunshō. Meiji Tennō ondenki shiryō : Meiji gunjishi and Japan. Rikugunshō
2012, Di 1 ban., ISBN 9787512005846, 152 v.
Russo-Japanese War, 1904-1905 | Sino-Japanese War, 1937-1945 | China Foreign relations | Japan Foreign relations | Korean resistance movements, 1905-1945
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9. Full Text Cyanidin 3-glucoside protects 3T3-L1 adipocytes against H2O2- or TNF-α-induced insulin resistance by inhibiting c-Jun NH2-terminal kinase activation
by Guo, Honghui and Ling, Wenhua and Wang, Qing and Liu, Chi and Hu, Yan and Xia, Min
Biochemical pharmacology, ISSN 0006-2952, 03/2008, Volume 75, Issue 6, pp. 1393 - 1401
Anthocyanin | c-Jun NH2-terminal kinase | Cyanidin 3-glucoside | Insulin resistance | 3T3-L1 adipocyte | terminal kinase | c-Jun NH | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Biological and medical sciences | Pharmacology. Drug treatments | Medical sciences | RNA, Small Interfering - genetics | Anthocyanins - pharmacology | Glucosides - pharmacology | Reactive Oxygen Species - metabolism | Hydrogen Peroxide | Mitogen-Activated Protein Kinase 8 - metabolism | Insulin Resistance | Adipocytes - drug effects | 3T3-L1 Cells | Tumor Necrosis Factor-alpha | Insulin - metabolism | Animals | Protective Agents - pharmacology | Adipocytes - metabolism | Glucose - metabolism | Mitogen-Activated Protein Kinase 8 - genetics | Mice | Index Medicus
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10. Full Text A novel function of B‐cell translocation gene 1 (BTG1) in the regulation of hepatic insulin sensitivity in mice via c‐Jun
by Xiao, Fei and Deng, Jiali and Yu, Junjie and Guo, Yajie and Chen, Shanghai and Guo, Feifan
The FASEB journal, ISSN 0892-6638, 01/2016, Volume 30, Issue 1, pp. 348 - 359
insulin signaling | high‐carbohydrate diet | liver | leucine deprivation | high-carbohydrate diet | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Receptors, Leptin - genetics | Humans | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Insulin Resistance | Leucine - deficiency | JNK Mitogen-Activated Protein Kinases - metabolism | Receptors, Retinoic Acid - metabolism | Neoplasm Proteins - metabolism | Hep G2 Cells | Insulin - metabolism | Animals | HEK293 Cells | Mice | Neoplasm Proteins - genetics | Second Messenger Systems | Index Medicus
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11. Full Text Emerging role of the Jun N‐terminal kinase interactome in human health
by Guo, Xiao‐Xi and An, Su and Yang, Yang and Liu, Ying and Hao, Qian and Tang, Tao and Xu, Tian‐Rui
Cell biology international, ISSN 1065-6995, 07/2018, Volume 42, Issue 7, pp. 756 - 768
neurodegenerative disease | insulin receptor substrate | MAPK cascade | amyloid precursor protein | insulin resistance | c‐Jun N‐terminal kinases | c-Jun N-terminal kinases | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell fate | Protein kinase | Tumorigenesis | Kinases | Insulin | Alzheimer's disease | Amyloid precursor protein | Index Medicus
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