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Diabetes, Obesity and Metabolism, ISSN 1462-8902, 08/2018, Volume 20, Issue 8, pp. 1836 - 1851
Aim We performed acute and chronic studies in healthy and diet‐induced obese animals using mouse‐specific or monkey‐specific dual GLP‐1R/GCGR agonists to... 
mouse | dual agonism, energy expenditure | non‐human primates | diet‐induced obesity | glucagon | GLP‐1 | diabetes | GLP-1 | non-human primates | diet-induced obesity | RODENTS | FOOD-INTAKE | HUMANS | dual agonism | INCREASED ENERGY-EXPENDITURE | energy expenditure | OXYNTOMODULIN | GLUCAGON-LIKE PEPTIDE-1 | REDUCTION | ENDOCRINOLOGY & METABOLISM | FAT-FED MICE | SECRETION | METABOLIC-RATE | Appetite Depressants - adverse effects | Obesity - drug therapy | Body Weight - drug effects | Diet, High-Fat - adverse effects | Macaca fascicularis | Male | Diabetes Mellitus, Type 2 - metabolism | Appetite Depressants - therapeutic use | Obesity - blood | Dose-Response Relationship, Drug | Glucagon-Like Peptide-1 Receptor - genetics | Drug Therapy, Combination - adverse effects | Glucagon-Like Peptide-1 Receptor - metabolism | Hypoglycemic Agents - administration & dosage | Appetite Depressants - administration & dosage | Female | Obesity - etiology | Animals, Outbred Strains | Insulin Secretion - drug effects | Hypoglycemic Agents - therapeutic use | Receptors, Glucagon - agonists | Hyperglycemia - prevention & control | Mice, Inbred C57BL | Receptors, Glucagon - metabolism | Energy Intake - drug effects | Random Allocation | Mice, Knockout | Obesity - metabolism | Diabetes Mellitus, Type 2 - blood | Animals | Diabetes Mellitus, Type 2 - drug therapy | Hypoglycemic Agents - adverse effects | Energy Metabolism - drug effects | Glucagon-Like Peptide-1 Receptor - agonists | Primates | Obesity | Comparative analysis | Blood sugar | Body weight | Energy intake | Secretion | Diabetes mellitus | Energy expenditure | Body weight loss | Glucose | Insulin | Glucose tolerance | Diet | Food intake | Oxidation | Diabetes | Food consumption | Original
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, p. e0178658
Background Diabetic retinopathy is characterized by defects in the retinal neurovascular unit. The underlying mechanisms of impairment including reactive... 
COMPLEMENT | RAT | MULTIDISCIPLINARY SCIENCES | ANIMAL-MODELS | COMPLICATIONS | EXPRESSION | DAMAGE | Retina - metabolism | Complement Activation | Rats, Wistar | Diabetes Mellitus, Type 2 - genetics | Diabetes Mellitus, Experimental - genetics | Hyperglycemia - complications | Male | Gene Expression Profiling | Diabetes Mellitus, Type 2 - metabolism | Hyperglycemia - genetics | Rats, Mutant Strains | Hyperinsulinism - genetics | Eye Proteins - genetics | Diabetes Mellitus, Experimental - metabolism | Hyperinsulinism - metabolism | Hyperinsulinism - complications | Signal Transduction | Acute-Phase Proteins | Gene Expression Regulation | Insulin Resistance | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Hyperglycemia - metabolism | Oncostatin M - biosynthesis | Animals | Diabetes Mellitus, Type 2 - physiopathology | Oncostatin M - genetics | Eye Proteins - biosynthesis | Diabetic retinopathy | Insulin resistance | Genetic aspects | Drug therapy | Hyperinsulinemia | Regulators | Animal models | Retinopathy | Transcription | Interleukin | Impairment | Retina | Activation | Complement | Glucose | Phase transitions | Defects | Interleukin 6 | Genotype & phenotype | Signal transduction | Hyperglycemia | Pathways | Technology | Rodents | Bioinformatics | Growth factors | Age | Obesity | Patterning | Mortality | Diabetes mellitus | Rats | Injection | Group dynamics | Metabolism | Gene expression | Clustering | Insulin | Intermediates | Polymerase chain reaction | Signaling | Complement activation | Interferon | Diabetes | Arrays
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 727 - 8
Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 08/2018, Volume 20, Issue 8, pp. i - i
Journal Article
Obesity, ISSN 1930-7381, 12/2010, Volume 18, Issue 12, pp. 2247 - 2254
Macrophage infiltration into adipose tissue (AT‐MP) is thought to induce insulin resistance and diabetes in obesity. Here, we investigated the effect of the... 
METABOLIC SYNDROME | OBESITY | NUTRITION & DIETETICS | CANNABINOID-1 RECEPTOR BLOCKER | RISK-FACTORS | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | ENDOCANNABINOID SYSTEM | OVERWEIGHT PATIENTS | NITRIC-OXIDE | RIMONABANT | ALTERNATIVE ACTIVATION | Diabetes Mellitus, Experimental - drug therapy | Pyrazoles - therapeutic use | Obesity - drug therapy | Rats, Wistar | Humans | Adipose Tissue - cytology | Male | Anti-Obesity Agents - therapeutic use | Lipopolysaccharides | Adipose Tissue - metabolism | Inflammation - complications | Inflammation - metabolism | Piperidines - pharmacology | Inflammation - drug therapy | Insulin Resistance - physiology | Female | Interleukin-10 - secretion | Phosphorylation - drug effects | Diabetes Mellitus, Experimental - metabolism | Pyrazoles - pharmacology | Hypoglycemic Agents - therapeutic use | Cell Line | Dietary Fats - adverse effects | Adipose Tissue - pathology | Rats | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Obesity - pathology | Rats, Zucker | Culture Media, Conditioned | Insulin - metabolism | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Piperidines - therapeutic use | Adipocytes - metabolism | Macrophages - drug effects | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Blood Glucose - metabolism | Anti-Obesity Agents - pharmacology | Tumor Necrosis Factor-alpha - biosynthesis
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM, ISSN 0193-1849, 08/2019, Volume 317, Issue 2, pp. E212 - E233
Journal Article