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1. Interview with Professor Samy Hadjadj (CHU of Poitiers)
by Hadjadj, S
CORRESPONDANCES EN METABOLISMES HORMONES DIABETES ET NUTRITION, ISSN 2100-9619, 04/2014, Volume 18, Issue 4-5, pp. 9 - 9
NUTRITION & DIETETICS | DAPAGLIFLOZIN | GLUCOSE | ENDOCRINOLOGY & METABOLISM | COTRANSPORTER 2 INHIBITION
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2. Diabetic nephropathy and physical activity
by Hadjadj, Samy
Correspondances en MHND, ISSN 2100-9619, 04/2019, Volume 23, Issue 2, pp. 26 - 29
Physical activity | Diabetic nephropathy | Renal outcomes
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3. Full Text 67-OR: Plasma Copeptin and Risk for Lower Extremity Amputation in People with Type 1 and Type 2 Diabetes
by POTIER, LOUIS and ROUSSEL, RONAN and MOHAMMEDI, KAMEL and SAULNIER, PIERRE and MARRE, MICHEL and HADJADJ, SAMY and VELHO, GILBERTO
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 67
Recently, an increased risk of lower extremity amputation (LEA) was observed with canagliflozin, a SGLT2 inhibitor, in the CANVAS trial although observational... 
Diabetes mellitus | Amputation | Diabetes mellitus (insulin dependent) | Diabetes | Diabetes mellitus (non-insulin dependent) | Reclassification | Contraction | Vasopressin | Glomerular filtration rate
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4. Full Text Initial combination of Empagliflozin and Metformin in patients with type 2 diabetes
by Hadjadj, Samy and Rosenstock, Julio and Meinicke, Thomas and Woerle, Hans J and Broedl, Uli C
Diabetes Care, ISSN 0149-5992, 10/2016, Volume 39, Issue 10, pp. 1718 - 1728
OBJECTIVE This study compared the efficacy and safety of initial combinations of empagliflozin + metformin with empagliflozin and metformin monotherapy in... 
GLYCEMIC CONTROL | ADD-ON | MONOTHERAPY | ENDOCRINOLOGY & METABOLISM | DOUBLE-BLIND | PEOPLE | INFECTION | MELLITUS | INHIBITORS | WEIGHT | PRACTICE RESEARCH DATABASE | Glycated Hemoglobin A - analysis | Benzhydryl Compounds - administration & dosage | Double-Blind Method | Humans | Middle Aged | Blood Glucose | Male | Treatment Outcome | Dose-Response Relationship, Drug | Diabetes Mellitus, Type 2 - blood | Glucosides - administration & dosage | Hypoglycemic Agents - administration & dosage | Adult | Female | Metformin - administration & dosage | Diabetes Mellitus, Type 2 - drug therapy | Drug Therapy, Combination | Type 2 diabetes | Dosage and administration | Metformin | Drug therapy | Comparative studies | Pharmacology | Diabetes | Drug dosages | Erythrocytes | Index Medicus
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5. Full Text A genome-wide association study identifies novel risk loci for type 2 diabetes
by Meyre, David and Balding, David J and Rung, Johan and Polychronakos, Constantin and Vincent, Daniel and Hadjadj, Samy and Balkau, Beverley and Serre, David and Froguel, Philippe and Hudson, Thomas J and Posner, Barry I and Sladek, Robert and Rocheleau, Ghislain and Boutin, Philippe and Belisle, Alexandre and Prentki, Marc and Montpetit, Alexandre and Pshezhetsky, Alexey V and Shen, Lishuang and Dina, Christian and Heude, Barbara and Charpentier, Guillaume and Teknisk-naturvetenskapliga vetenskapsområdet and Institutionen för fysikalisk och analytisk kemi and Uppsala universitet and Kemiska sektionen
Nature, ISSN 0028-0836, 02/2007, Volume 445, Issue 7130, pp. 881 - 885
Type 2 diabetes mellitus results from the interaction of environmental factors with a combination of genetic variants, most of which were hitherto unknown. A... 
POLYMORPHISMS | TRANSCRIPTION-FACTOR-7-LIKE-2 TCF7L2 GENE | ENZYME IDE GENE | POPULATION STRATIFICATION | GLUCOSE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | SUSCEPTIBILITY | LARGE-SCALE | INSULIN-RESISTANCE SYNDROME | NUCLEAR FACTOR-4-ALPHA GENE | Chromosomes, Human, Pair 8 - genetics | Genetic Predisposition to Disease - genetics | Zinc Transporter 8 | Diabetes Mellitus, Type 2 - genetics | Humans | Cation Transport Proteins - genetics | Chromosomes, Human, Pair 10 - genetics | France | Genome, Human | Case-Control Studies | Linkage Disequilibrium | Diabetes | Genomics | Risk factors | Polymorphism | Index Medicus | Genetic Predisposition to Disease | Chromosomes, Human, Pair 8 | Life Sciences | Genetics | Cation Transport Proteins | Diabetes Mellitus, Type 2 | Chromosomes, Human, Pair 10 | Human genetics | Chemistry | Naturvetenskap | Natural Sciences | Kemi | Chemical Sciences
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6. Full Text Sodium and Cardiovascular Disease
by Graudal, N and SURDIAGENE Study Group
NEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 11/2014, Volume 371, Issue 22, pp. 2136 - 2137
MEDICINE, GENERAL & INTERNAL | Diet | Sodium - urine | Humans | Mortality | Potassium - urine | Cardiovascular Diseases - epidemiology | Female | Male | Sodium, Dietary - administration & dosage
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7. Full Text Rare MTNR1B variants impairing melatonin receptor 1B function contribute to type 2 diabetes
by Bonnefond, Amélie and Clément, Nathalie and Fawcett, Katherine and Yengo, Loic and Vaillant, Emmanuel and Guillaume, Jean-Luc and Dechaume, Aurélie and Payne, Felicity and Roussel, Ronan and Czernichow, Sébastien and Hercberg, Serge and Hadjadj, Samy and Balkau, Beverley and Marre, Michel and Lantieri, Olivier and Langenberg, Claudia and Bouatia-Naji, Nabila and Charpentier, Guillaume and Vaxillaire, Martine and Rocheleau, Ghislain and Wareham, Nicholas J and Sladek, Robert and McCarthy, Mark I and Dina, Christian and Barroso, Ins and Jockers, Ralf and Froguel, Philippe and Meta-Anal Glucose Insulin-Related and Meta-Analysis of Glucose and Insulin-Related Traits Consortium (MAGIC) and The Meta-Analysis of Glucose and Insulin-Related Traits Consortium (MAGIC)
Nature Genetics, ISSN 1061-4036, 03/2012, Volume 44, Issue 3, pp. 297 - 301
Genome-wide association studies have revealed that common noncoding variants in MTNR1B (encoding melatonin receptor 1B, also known as MT2) increase type 2... 
EXCESS | FASTING PLASMA-GLUCOSE | GENERAL-POPULATION | DISEASES | GENETICS & HEREDITY | RISK | LOCI | GENOME-WIDE ASSOCIATION | Body Mass Index | Receptor, Melatonin, MT2 - genetics | Amino Acid Sequence | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Diabetes Mellitus, Type 2 - genetics | Gene Frequency | Humans | Models, Molecular | Molecular Sequence Data | Exons - genetics | Logistic Models | Diabetes Mellitus, Type 2 - metabolism | Sequence Analysis, DNA | Genetic Variation | Base Sequence | Odds Ratio | Amino Acid Substitution | Type 2 diabetes | Melatonin | Physiological aspects | Genetic aspects | Research | Health aspects | Risk factors | Drug receptors | Confidence intervals | Medical research | Biomedical research | Disease | Circadian rhythm | Diabetes | Metabolic disorders | Binding sites | Index Medicus
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8. Full Text 535-P: Relationship between Renal Capacities to Reabsorb Glucose and Kidney Disease in Patients with Diabetes
by MATAR, ODETTE and POTIER, LOUIS and ABOULEKA, YAWA and FUMERON, FRÉDÉRIC and MOHAMMEDI, KAMEL and HADJADJ, SAMY and ROUSSEL, RONAN and VELHO, GILBERTO and MARRE, MICHEL
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 535
Hyperglycemia can impair tubulo-glomerular balance and cause kidney disease by opening pre-glomerular resistances. Susceptibility to kidney disease is... 
Creatinine | Hyperglycemia | Kidneys | Epidermal growth factor receptors | Diabetes mellitus | Reabsorption | Glucose | Diabetes | Kidney diseases | Glomerular filtration rate
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9. Full Text 550-P: Age at Diabetes Onset and Risk for Diabetic Kidney Disease
by CARPENTIER, CHARLYNE and DUBOIS, SEVERINE and ABOULEKA, YAWA and DULY-BOUHANICK, BEATRICE and HADJADJ, SAMY and COUTANT, REGIS and ROUSSEL, RONAN and VELHO, GILBERTO and MARRE, MICHEL
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 550
Diabetic kidney disease drives premature mortality among patients with type 1 diabetes. The role of age at diabetes onset is controversial. We analyzed risk... 
Creatinine | Kidneys | Epidermal growth factor receptors | Diabetes mellitus | Diabetes mellitus (insulin dependent) | Diabetes | Kidney diseases | Multivariate analysis | Risk factors | Age | Glomerular filtration rate
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10. Full Text Twelve type 2 diabetes susceptibility loci identified through large-scale association analysis
by Voight, Benjamin and Scott, Laura and Steinthorsdottir, Valgerdur and Morris, Anew and Dina, Christian and Welch, Ryan and Zeggini, Eleftheria and Huth, Cornelia and Aulchenko, Yurii and Thorleifsson, Gudmar and McCulloch, Laura and Ferreira, Teresa and Grallert, Harald and Amin, Najaf and Wu, Guanming and Willer, Cristen and Raychaudhuri, Soumya and McCarroll, Steven and Langenberg, Claudia and Hofmann, Oliver and Dupuis, Josée and Qi, Lu and Segrè, Ayellet and Hoek, Mandy and Navarro, Pau and Ardlie, Kristin and Balkau, Beverley and Benediktsson, Rafn and Bennett, Amanda and Blagieva, Roza and Boerwinkle, Eric and Bonnycastle, Lori and Boström, Kristina Bengtsson and Bravenboer, Bert and Bumpstead, Suzannah and Burtt, Noël and Charpentier, Guillaume and Chines, Peter and Cornelis, Marilyn and Couper, David and Crawford, Gabe and Doney, Alex and Elliott, Katherine and Erdos, Michael and Fox, Caroline and Franklin, Christopher and Ganser, Martha and Gieger, Christian and Grarup, Niels and Green, Todd and Griffin, Simon and Groves, Christopher and Guiducci, Candace and Hadjadj, Samy and Hassanali, Neelam and Herder, Christian and Isomaa, Bo and Jackson, Anne and Johnson, Paul and Jørgensen, Torben and Kao, Wen and Klopp, Norman and Kong, Augustine and Kraft, Peter and Kuusisto, Johanna and Lauritzen, Torsten and Li, Man and Lieverse, Aloysius and Lindgren, Cecilia and Lyssenko, Valeriya and Marre, Michel and Meitinger, Thomas and Midthjell, Kristian and Morken, Mario and Narisu, Narisu and Nilsson, Peter and Owen, Katharine and Payne, Felicity and Perry, John and Petersen, A.K and Platou, Carl and Proença, Christine and Prokopenko, Inga and Rathmann, Wolfgang and Rayner, Nigel William and Robertson, Neil and Rocheleau, Ghislain and Roden, Michael and Sampson, Michael and Saxena, Richa and Shields, Beverley and Shrader, Peter and Sigurdsson, Gunnar and Sparsø, Thomas and Strassburger, Klaus and Stringham, Heather and Sun, Qi and Swift, Amy and Thorand, Barbara and Tichet, Jean and ... and GIANT Consortium and MAGIC Investigators and MAGIC investigators and The GIANT Consortium and The MAGIC investigators and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för genetik och patologi and Uppsala universitet
Nature Genetics, ISSN 1061-4036, 07/2010, Volume 42, Issue 7, pp. 579 - 589
textabstractBy combining genome-wide association data from 8,130 individuals with type 2 diabetes (T2D) and 38,987 controls of European descent and following... 
C-REACTIVE PROTEIN | COMMON VARIANTS | FASTING PLASMA-GLUCOSE | CROHNS-DISEASE | INSULIN-RESISTANCE | HNF1-ALPHA GENE | GENETICS & HEREDITY | GENE-EXPRESSION | COMPLEX DISEASES | QT INTERVAL DURATION | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Meta-Analysis as Topic | KCNQ1 Potassium Channel - genetics | Hepatocyte Nuclear Factor 1-alpha - genetics | Diabetes Mellitus, Type 2 - genetics | Humans | Gene Dosage | Gene Expression Profiling | Mitogen-Activated Protein Kinase Phosphatases - genetics | Genetic Heterogeneity | Genome, Human - genetics | Diabetes Mellitus, Type 2 - blood | Fasting - blood | Dual-Specificity Phosphatases - genetics | Polymorphism, Single Nucleotide | Blood Glucose - metabolism | Type 2 diabetes | Genetic aspects | Research | Genetic susceptibility | Risk factors | Medical research | Diabetes | Meta-analysis | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
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11. Full Text To the editor
by Saulnier, Pierre-Jean and Gand, Elise and Hadjadj, Samy
New England Journal of Medicine, ISSN 0028-4793, 11/2014, Volume 371, Issue 22, pp. 2036 - 2136
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12. Full Text New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk
by Dupuis, Josée and Langenberg, Claudia and Prokopenko, Inga and Saxena, Richa and Soranzo, Nicole and Jackson, Anne and Wheeler, Eleanor and Glazer, Nicole and Bouatia-Naji, Nabila and Gloyn, Anna and Lindgren, Cecilia and Mägi, Reedik and Morris, Anew and Randall, Joshua and Johnson, Toby and Elliott, Paul and Rybin, Denis and Thorleifsson, Gudmar and Steinthorsdottir, Valgerdur and Henneman, Peter and Grallert, Harald and Dehghan, Abbas and JanHottenga, Jouke and Franklin, Christopher and Navarro, Pau and Song, Kijoung and Goel, Anuj and Perry, John and Egan, Josephine and Lajunen, Taina and Grarup, Niels and Sparsø, Thomas and Doney, Alex and Voight, Benjamin and Stringham, Heather and Li, Man and Kanoni, Stavroula and Shrader, Peter and Cavalcanti-Proença, Christine and Kumari, Meena and Qi, Lu and Timpson, Nicholas and Gieger, Christian and Zabena, Carina and Rocheleau, Ghislain and Ingelsson, Erik and An, Ping and O´Connell, J.R and Luan, J and McCarroll, Steven and Payne, Felicity and Roccasecca, R.M and Pattou, François and Sethupathy, Praveen and Ardlie, Kristin and Ariyurek, Yavuz and Balkau, Beverley and Barter, Phil and Beilby, John and Ben-Shlomo, Y and Benediktsson, Rafn and Bennett, Amanda and Bergmann, Sven and Bochud, Murielle and Boerwinkle, Eric and Bonnefond, Amélie and Bonnycastle, Lori and Borch-Johnsen, K and Böttcher, Yvonne and Brunner, Eric and Bumpstead, Suzannah and Charpentier, Guillaume and IdaChen, Yii and Chines, Peter and Clarke, R and McOin, Lachlan and Cooper, Matthew and Cornelis, Marilyn and Crawford, Gabe and Crisponi, Laura and Day, Ian and Geus, Eco and Delplanque, Jerome and Dina, Christian and Erdos, Michael and Fedson, Annette and Fischer-Rosinsky, Antje and Forouhi, Nita and Fox, Caroline and Frants, Rune and GraziaFranzosi, Maria and Galan, Pilar and Goodarzi, Mark and Graessler, Jürgen and Groves, Christopher and Grundy, Scott and Gwilliam, Rhian and Gyllensten, Ulf and Hadjadj, Samy and Hallmans, Göran and ... and DIAGRAM Consortium and Global BPgen Consortium and Anders Hamsten Procardis Consortiu and GIANT Consortium and MAGIC Investigators and Anders Hamsten on behalf of Procardis Consortium and MAGIC investigators and the MAGIC investigators and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Allmänmedicin and Näringsforskning and Umeå universitet and Medicin
Nature Genetics, ISSN 1061-4036, 02/2010, Volume 42, Issue 2, pp. 105 - 116
textabstractLevels of circulating glucose are tightly regulated. To identify new loci influencing glycemic traits, we performed meta-analyses of 21 genome-wide... 
CIRCADIAN CLOCK | DISEASE RISK | TRIGLYCERIDE LEVELS | INSULIN-RESISTANCE | GENETICS & HEREDITY | BETA-CELL DYSFUNCTION | PLASMA-GLUCOSE | MODEL ASSESSMENT | FOLLOW-UP | GENOME-WIDE ASSOCIATION | ESSENTIAL COMPONENTS | Genetic Predisposition to Disease | Genome-Wide Association Study | Meta-Analysis as Topic | Reproducibility of Results | Quantitative Trait, Heritable | Diabetes Mellitus, Type 2 - genetics | Humans | Gene Expression Regulation | Genetic Loci - genetics | Databases, Genetic | DNA Copy Number Variations - genetics | Fasting - blood | Adolescent | Alleles | Polymorphism, Single Nucleotide - genetics | Adult | Blood Glucose - genetics | Blood Glucose - metabolism | Child | Homeostasis - genetics | Quantitative Trait Loci - genetics | Type 2 diabetes | Homeostasis | Physiological aspects | Genetic aspects | Research | Insulin | Health aspects | Risk factors | Studies | Hyperglycemia | Diabetes | Sample size | Meta-analysis | Index Medicus | Clinical Medicine | Medical and Health Sciences | Endokrinologi och diabetes | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
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13. Full Text Genetic variant near IRS1 is associated with type 2 diabetes, insulin resistance and hyperinsulinemia
by Elliott, Paul and Järvelin, Marjo-Riitta and Charpentier, Guillaume and Lauritzen, Torsten and Poulsen, Pernille and Polychronakos, Constantin and Wojtaszewski, Jørgen F P and Belisle, Alexandre and Hadjadj, Samy and Durand, Emmanuelle and Rocheleau, Ghislain and Ribel-Madsen, Rasmus and Cauchi, Stéphane and Dina, Christian and Tichet, Jean and Sladek, Robert and Mazur, Alexander and Borch-Johnsen, Knut and Rung, Johan and Bacot, François and Froguel, Philippe and Meyre, David and Ruokonen, Aimo and Pedersen, Oluf and Pisinger, Charlotta and Prentki, Marc and Albrechtsen, Anders and Sandbaek, Anelli and Posner, Barry and Vaag, Allan and Marre, Michel and Laitinen, Jaana and Montpetit, Alexandre and Shen, Lishuang and Vaxillaire, Martine and Hansen, Torben and Pouta, Anneli and Balkau, Beverley and Serre, David and Cavalcanti-Proença, Christine
Nature Genetics, ISSN 1061-4036, 10/2009, Volume 41, Issue 10, pp. 1110 - 1115
Genome-wide association studies have identified common variants that only partially explain the genetic risk for type 2 diabetes (T2D). Using genome-wide... 
RISK LOCI | DISRUPTION | PROSTATE-CANCER | GENETICS & HEREDITY | MUSCLE GLYCOGEN-SYNTHASE | SUSCEPTIBILITY LOCI | BETA-CELL | MICE | MELLITUS | GENOME-WIDE ASSOCIATION | RECEPTOR SUBSTRATE-1 GENE | Hyperinsulinism - metabolism | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Hyperinsulinism - complications | Diabetes Mellitus, Type 2 - genetics | Humans