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Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2018, Volume 72, Issue 16, p. C80
Journal Article
Journal Article
Journal Article
Journal Article
by Kang, SH and Lee, HA and Kim, M and Lee, E and Sohn, UD and Kim, I
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, pp. E495 - E507
Cushing's syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
glucocorticoid receptor | PHYSIOLOGY | PROTEIN | ANTAGONIST | skeletal muscle atrophy | COMPONENTS | Cushing's syndrome | AUTOPHAGY | FOXO TRANSCRIPTION FACTORS | HYPERTROPHY | GLUCOCORTICOID-RECEPTOR | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | forkhead box O3 | CROSSTALK | UP-REGULATION | Receptors, Glucocorticoid - antagonists & inhibitors | Cushing Syndrome - physiopathology | SKP Cullin F-Box Protein Ligases - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Male | Muscle, Skeletal - metabolism | Receptors, Glucocorticoid - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Cushing Syndrome - metabolism | Promoter Regions, Genetic - drug effects | Receptors, Glucocorticoid - agonists | Forkhead Box Protein O3 - genetics | Chromatin Immunoprecipitation | Cushing Syndrome - pathology | RNA Interference | Muscle, Skeletal - drug effects | Muscle Proteins - metabolism | Forkhead Box Protein O3 - antagonists & inhibitors | Muscle Proteins - antagonists & inhibitors | Muscular Atrophy - etiology | Disease Models, Animal | Cell Line | Tripartite Motif Proteins - antagonists & inhibitors | Ubiquitin-Protein Ligases - metabolism | Tripartite Motif Proteins - agonists | Tripartite Motif Proteins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - antagonists & inhibitors | Rats, Sprague-Dawley | Forkhead Box Protein O3 - metabolism | Hormone Antagonists - pharmacology | Forkhead Box Protein O3 - agonists | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Response Elements - drug effects | Muscle Proteins - agonists | Animals | Active Transport, Cell Nucleus - drug effects | Muscle Fibers, Skeletal - pathology | Glucocorticoids - pharmacology | Genes, Reporter - drug effects | Tripartite Motif Proteins - metabolism | Muscle, Skeletal - pathology | Ubiquitin-Protein Ligases - genetics | Muscles | Physiological aspects | Atrophy, Muscular | Cushing syndrome
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, pp. E495 - E507
Cushing’s syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
Skeletal muscle atrophy | Cushing’s syndrome | Forkhead box O3 | Glucocorticoid receptor
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 01/2018, Volume 314, Issue 1, pp. E39 - E52
Cushing’s syndrome (CS) caused by hypercortisolism is occasionally accompanied by metabolic disorders such as hypertension, diabetes mellitus (DM),... 
Hypertension | Histone deacetylashyperglycemia | Cushing’s syndrome | Glucocorticoid receptor
Journal Article
by Lee, E and Lee, HA and Kim, M and Do, GY and Cho, HM and Kim, GJ and Jung, H and Song, JH and Cho, JM and Kim, I
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY, ISSN 1440-1681, 03/2019, Volume 46, Issue 3, pp. 226 - 236
Journal Article
American Journal of Physiology, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, p. E495
Cushing's syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
Ubiquitin | Transcription | Glucocorticoids | Hormones | Hydrocortisone | Atrophy | Proteins | Infusion | Rodents | Forkhead protein | FOXO3 protein | Enzymes | Nervous system diseases | Pol II | Dexamethasone | Diabetes mellitus | Muscles | Cushing's syndrome | Skeletal muscle | Musculoskeletal system | Adrenocorticotropic hormone | Pituitary | Weight reduction | Protein expression | Sepsis
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 2014, Volume 34, Issue 12, pp. 4309 - 4317
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 01/2019, Volume 109, p. 921
Inhibition of histone deacetylase (HDAC) suppresses inflammation of pancreatic islets and apoptosis of [beta]-cells. However, the underlying molecular... 
Immunohistochemistry | Oxidative stress | Medical colleges | Pancreatic beta cells | Chromatin | RNA | Streptozocin | Superoxide | Biological response modifiers | Macrophages | Antioxidants | Hyperglycemia | Glutathione transferase | Type 1 diabetes | Cell death | Analysis | Interferon | Protein binding
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, p. e0136801
Journal Article
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 11/2018, Volume 19, Issue 11, p. 3539
Type 2 diabetes mellitus (T2DM) is a chronic disease manifested by hyperglycemia. It is essential to effectively control hyperglycemia to prevent complications... 
type 2 diabetes mellitus | FoxO1 acetylation | histone deacetylase | transcriptional regulation | histone deacetylase inhibitors | ACETYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHEMISTRY, MULTIDISCIPLINARY | FOXO1 | PATHOPHYSIOLOGY | METABOLISM | GLUCONEOGENESIS | INSULIN SIGNALING BLOCKADE | GLUCOSE | REVEALS
Journal Article