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by Yan, CG and Chen, X and Li, L and Castellanos, FX and Bai, TJ and Bo, QJ and Cao, J and Chen, GM and Chen, NX and Chen, W and Cheng, C and Cheng, YQ and Cui, XL and Duan, J and Fang, YR and Gong, QY and Guo, WB and Hou, ZH and Hu, L and Kuang, L and Li, F and Li, KM and Li, T and Liu, YS and Liu, ZN and Long, YC and Luo, QH and Meng, HQ and Peng, DH and Qiu, HT and Qiu, J and Shen, YD and Shi, YS and Wang, CY and Wang, F and Wang, K and Wang, L and Wang, X and Wang, Y and Wu, XP and Wu, XR and Xie, CM and Xie, GR and Xie, HY and Xie, P and Xu, XF and Yang, H and Yang, J and Yao, JS and Yao, SQ and Yin, YY and Yuan, YG and Zhang, AX and Zhang, H and Zhang, KR and Zhang, L and Zhang, ZJ and Zhou, RB and Zhou, YT and Zhu, JJ and Zou, CJ and Si, TM and Zuo, XN and Zhao, JP and Zang, YF
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 04/2019, Volume 116, Issue 18, pp. 9078 - 9083
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e65702 - e65702
Background: In a swine model of acute myocardial infarction (AMI), Statins can enhance the therapeutic efficacy of mesenchymal stem cell (MSCs)... 
ISCHEMIA-REPERFUSION INJURY | HEARTS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | TRACKING | IMPROVES SURVIVAL | STATINS | CARDIAC REPAIR | TRANSPLANTATION | Hydroxymethylglutaryl-CoA Reductase Inhibitors - administration & dosage | Inflammation - pathology | Myocardial Infarction - mortality | Apoptosis - drug effects | Tomography, Emission-Computed, Single-Photon | Myocardial Infarction - therapy | Inflammation - metabolism | C-Reactive Protein - metabolism | Pyrroles - administration & dosage | Swine | Myocardium - metabolism | Heptanoic Acids - administration & dosage | Myocardial Infarction - physiopathology | Nitric Oxide Synthase Type III - metabolism | Heptanoic Acids - pharmacology | Disease Models, Animal | Myocardial Infarction - diagnosis | Ventricular Function, Left - drug effects | Myocardium - pathology | Myocardial Infarction - metabolism | Enzyme Activation - drug effects | Atorvastatin Calcium | Pyrroles - pharmacology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Fibrosis | Nitric Oxide Synthase - metabolism | Nitric Oxide - metabolism | Mesenchymal Stem Cell Transplantation | Heart | PET imaging | Nitric oxide | Stem cells | Bone marrow | Transplantation | SPECT imaging | Heart attack | Antilipemic agents | Endothelium | Myocardial infarction | Mesenchyme | Cardiovascular disease | Activation | Kinases | Heart function | Defects | Reperfusion | Allografts | Ischemia | Computed tomography | Arginine | Rodents | Surgery | Statins | Photon emission | Autografts | Cell survival | Coronary artery | Positron emission | Metabolism | Survival | Nitric-oxide synthase | Emissions | Inhibitors | Magnetic resonance imaging | Perfusion | Atorvastatin | Infarction | Livestock | Positron emission tomography | Ejection | Apoptosis | Index Medicus
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 06/2018, Volume 102, pp. 1209 - 1220
Rheumatoid arthritis (RA) is a long-term autoimmune disorder that primarily results in warm, swollen, and painful joints. In this study, we investigate the... 
PI3K/AKT/mTOR signaling pathway | Chondrocyte | Artesunate | Rheumatoid arthritis | Proliferation | Autophagy | Apoptosis | TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | CANCER | GLYCOLYSIS | INHIBITION | PI3K | RECOMMENDATIONS | PHARMACOLOGY & PHARMACY | EXPRESSION | Inflammation - pathology | Liver - pathology | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Cartilage - pathology | Apoptosis - genetics | Male | Cartilage - drug effects | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Collagen Type II - metabolism | Chondrocytes - drug effects | Artemisinins - pharmacology | Arthritis, Rheumatoid - drug therapy | Cell Cycle Checkpoints - genetics | Autophagy - genetics | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Chondrocytes - metabolism | Disease Models, Animal | Chondrocytes - pathology | RNA, Messenger - genetics | Down-Regulation - drug effects | Rats, Sprague-Dawley | Arthritis, Rheumatoid - pathology | Up-Regulation - drug effects | Arthritis, Rheumatoid - genetics | Animals | Artemisinins - therapeutic use | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Chondrocytes - ultrastructure | Cell Proliferation - drug effects | Immunohistochemistry | Rheumatoid factor | Arthritis | RNA | Transmission electron microscopes | Index Medicus
Journal Article
Journal Article
Journal Article
BMC Pediatrics, ISSN 1471-2431, 09/2018, Volume 18, Issue 1, pp. 305 - 305
Background: Exposure to air pollutants has been related to preterm birth, but little evidence can be available for PM2.5, O-3 and CO in China. This study aimed... 
Air pollution | Time-series analysis | Preterm birth | METAANALYSIS | DAILY OUTPATIENT VISITS | RISK | ADVERSE PREGNANCY OUTCOMES | POLLUTION | PM2.5 | SO2 | PM10 | PEDIATRICS | DAILY MORTALITY | FINE PARTICULATE MATTER | WEIGHT | EXPOSURE | Analysis | Premature birth | Research | Health risk assessment | Pediatric research | Risk factors | Studies | Systematic review | Womens health
Journal Article
Journal Article