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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2008, Volume 105, Issue 3, pp. 967 - 972
Journal Article
Immunity, ISSN 1074-7613, 11/2013, Volume 39, Issue 5, pp. 874 - 884
Severe sepsis remains a poorly understood systemic inflammatory condition with high mortality rates and limited therapeutic options in addition to organ... 
ACTIVATION | INFECTIOUS-DISEASES | INFLAMMATION | TOLERANCE | SEPTIC SHOCK | RESISTANCE | MICE | AUTOPHAGY | IMMUNOLOGY | FANCONI-ANEMIA | EPIDEMIOLOGY | Whole-Body Irradiation | Sepsis - prevention & control | Cecum - injuries | Shock, Septic - prevention & control | Epirubicin - administration & dosage | Anti-Bacterial Agents - therapeutic use | Inflammation Mediators - analysis | Anthracyclines - pharmacology | Peritonitis - genetics | Lung - metabolism | Microtubule-Associated Proteins - deficiency | Thienamycins - therapeutic use | Peritonitis - etiology | DNA Repair - drug effects | Mice, Inbred C57BL | Injections, Intraperitoneal | Adenoviridae Infections - immunology | Inflammation | Peritonitis - immunology | Organ Specificity | Ataxia Telangiectasia Mutated Proteins - physiology | Microtubule-Associated Proteins - physiology | Autophagy-Related Protein 7 | Animals | Lung - drug effects | Respiratory Tract Infections - immunology | Epirubicin - therapeutic use | Peritonitis - physiopathology | Anti-Bacterial Agents - pharmacology | Ataxia Telangiectasia Mutated Proteins - deficiency | Fanconi Anemia Complementation Group D2 Protein - physiology | Mice | Anthracyclines - therapeutic use | DNA Damage | Peritonitis - drug therapy | Epirubicin - pharmacology | Anthracyclines | DNA repair | DNA damage | DNA | Flow cytometry | Antibiotics | Mortality | Sepsis | Infections | Viral infections | Deoxyribonucleic acid--DNA | Index Medicus | ATM | Autophagy
Journal Article
Journal of the American Society of Nephrology : JASN, ISSN 1046-6673, 2014, Volume 25, Issue 4, pp. 707 - 716
Podocyte loss is a major determinant of progressive CKD. Although recent studies showed that a subset of parietal epithelial cells can serve as podocyte... 
CELLS CONTRIBUTE | KIDNEY-DISEASE | EPITHELIAL-CELLS | REGENERATION | RATS | UROLOGY & NEPHROLOGY | DIABETIC-NEPHROPATHY | RENAL PROGENITORS | MICE | CAUSES GLOMERULOSCLEROSIS | TRANSPLANTATION | Regeneration | Animals | Flow Cytometry | Podocytes - physiology | Mice | Kidney Glomerulus - pathology | Aging - pathology | Hypertrophy | Index Medicus | Basic Research
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2197 - 2209
Chronic glomerular diseases, associated with renal failure and cardiovascular morbidity, represent a major health issue. However, they remain poorly... 
KIDNEY-DISEASE | RAPAMYCIN | MEDICINE, RESEARCH & EXPERIMENTAL | DE-NOVO | GROWTH-HORMONE | PATHWAY | GLOMERULAR-FILTRATION | IN-VIVO | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | RENAL HYPERTROPHY | TRANSGENIC MICE | Humans | Multiprotein Complexes | Kidney Glomerulus - physiopathology | Mechanistic Target of Rapamycin Complex 1 | Proteinuria - prevention & control | Trans-Activators - physiology | TOR Serine-Threonine Kinases - antagonists & inhibitors | Diabetic Nephropathies - physiopathology | Trans-Activators - genetics | Adult | Regulatory-Associated Protein of mTOR | TOR Serine-Threonine Kinases - physiology | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - physiopathology | Proteinuria - etiology | Rapamycin-Insensitive Companion of mTOR Protein | Carrier Proteins - physiology | Proteins - physiology | Diabetic Nephropathies - pathology | Genetic Predisposition to Disease | Sirolimus - therapeutic use | Mice, Inbred C57BL | Mice, Transgenic | Gene Dosage | Kidney Glomerulus - pathology | Podocytes - pathology | Proteinuria - genetics | Disease Progression | Mice, Inbred ICR | Mice, Knockout | Nephrosis, Lipoid - physiopathology | Proteinuria - physiopathology | Proteins - genetics | Carrier Proteins - genetics | Adaptor Proteins, Signal Transducing | Animals | Trans-Activators - deficiency | Podocytes - drug effects | Podocytes - physiology | Mice | Transcription Factors | Diabetic nephropathies | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2015, Volume 125, Issue 6, pp. 2307 - 2316
Journal Article
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2003, Volume 23, Issue 14, pp. 4917 - 4928
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2009, Volume 106, Issue 21, pp. 8579 - 8584
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 12/2011, Volume 22, Issue 12, pp. 2193 - 2202
The prorenin receptor (PRR) is highly expressed in podocytes, but its role in the maintenance of podocyte function is unknown. Here we generated... 
PHYSIOLOGY | ACTIN | ALZHEIMERS-DISEASE | ATPASE | BIOLOGY | UROLOGY & NEPHROLOGY | ANGIOTENSIN-II | CARDIOMYOCYTES | (PRO)RENIN RECEPTOR | WNT | IDENTIFICATION | Animals | Podocytes - physiology | Cell Survival | Female | Autophagy - physiology | Mice | Receptors, Cell Surface - physiology | Index Medicus | Basic Research
Journal Article
Autophagy, ISSN 1554-8627, 07/2015, Volume 11, Issue 7, pp. 1130 - 1145
The glomerulus is a highly specialized capillary tuft, which under pressure filters large amounts of water and small solutes into the urinary space, while... 
sclerosis | endothelial cells | autophagy | podocytes | proteinuria | diabetic nephropathy | Podocytes | Diabetic nephropathy | Autophagy | Endothelial cells | Sclerosis | Proteinuria | apoptosis-related cysteine peptidase | LC3A | sequestosome 1 | caspase 3 | mechanistic target of rapamycin | CELL BIOLOGY | Cdh5 | MAP1LC3A | CASP3 | STZ | NITRIC-OXIDE | Nphs2 | transmission electron microscopy | GLYCEMIC CONTROL | OXIDATIVE STRESS | GEC | TUBA | cadherin 5 | end-stage renal disease | NEPHROPATHY | streptozotocin | Wilms tumor 1 | DISEASE | GROWTH-FACTOR | SQSTM1 | ESRD | GFB | diabetes mellitus | blood urea nitrogen | microtubule-associated protein 1 light chain 3 | GBM | GENE-EXPRESSION | BUN | glomerular endothelial cells | nephrosis 2 | glomerular filtration barrier | podocin | tubulin | RENAL BIOPSY SPECIMENS | KNOCKOUT MICE | MTOR | WT1 | glomerular basement membrane | TEM | TRANSGENIC MICE | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Autophagy - drug effects | Mesangial Cells - drug effects | Endothelial Cells - ultrastructure | Glomerular Filtration Rate - drug effects | Diabetic Nephropathies - physiopathology | Gene Deletion | Mesangial Cells - ultrastructure | Integrases - metabolism | Microtubule-Associated Proteins - deficiency | Diabetic Nephropathies - prevention & control | Diabetic Nephropathies - pathology | Mice, Inbred C57BL | Cells, Cultured | Glucose - pharmacology | Podocytes - pathology | Phenotype | Podocytes - ultrastructure | Animals | Autophagy-Related Protein 5 | Podocytes - drug effects | Mesangial Cells - pathology | Endothelial Cells - pathology | Endothelial Cells - drug effects | Index Medicus | Life Sciences | Development Biology
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 04/2009, Volume 20, Issue 4, pp. 798 - 806
Atypical protein kinase C (aPKC) is a central component of the evolutionarily conserved Par3-Par6-aPKC complex, one of the fundamental regulators of cell... 
Basic Research
Journal Article