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1. Full Text The role of stroma in pancreatic cancer: Diagnostic and therapeutic implications
by Erkan, Mert and Hausmann, Simone and Michalski, Christoph W and Fingerle, Alexander A and Dobritz, Martin and Kleeff, Jãrg and Friess, Helmut
Nature Reviews Gastroenterology and Hepatology, ISSN 1759-5045, 08/2012, Volume 9, Issue 8, pp. 454 - 467
Pancreatic ductal adenocarcinoma (PDAC) is one of the five most lethal malignancies worldwide and survival has not improved substantially in the past 30 years.... 
PAPILLARY MUCINOUS NEOPLASMS | PHASE-III TRIAL | MATRIX METALLOPROTEINASES | RECEPTOR ANTAGONISTS | RENIN-ANGIOTENSIN SYSTEM | DUCTAL ADENOCARCINOMA | STELLATE CELLS | ELLAGIC ACID | GASTROENTEROLOGY & HEPATOLOGY | FIBROBLAST ACTIVATION PROTEIN | TUMOR MICROENVIRONMENT | Immunohistochemistry | Adenocarcinoma - pathology | Humans | Pancreatic Neoplasms - pathology | Stromal Cells - metabolism | Tomography, X-Ray Computed | Pancreatic Stellate Cells - physiology | Adenocarcinoma - drug therapy | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - diagnostic imaging | Pancreatic Neoplasms - drug therapy | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Adenocarcinoma - physiopathology | Stromal Cells - drug effects | Renin-Angiotensin System - drug effects | Carcinoma, Pancreatic Ductal - physiopathology | Adenocarcinoma - diagnostic imaging | Pancreatic Neoplasms - physiopathology | Stromal Cells - physiology | Pancreatic Neoplasms - diagnostic imaging | Care and treatment | Diagnosis | Research | Carcinogenesis | Pancreatic cancer | Index Medicus
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2. Full Text Mesenchymal Wt1 positive stellate cells represent a source of the fibrotic response in pancreatitis
by Regel, Ivonne and Dreyer, Tobias and Beyer, Georg and Teller, Steffen and Wuschek, Alexander and Hausmann, Simone and Benitz, Simone and Steiger, Katja and Esposito, Irene and Mayerle, Julia and Michalski, Christoph and Kleeff, Jörg
Pancreatology, ISSN 1424-3903, 06/2018, Volume 18, Issue 4, p. S89
Fibroblasts | Stellate cells | Population | Mesenchyme | Rodents | Pancreatitis
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3. Full Text Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma
by Mazur, Pawel K and Herner, Alexander and Mello, Stephano S and Wirth, Matthias and Hausmann, Simone and Sánchez-Rivera, Francisco J and Lofgren, Shane M and Kuschma, Timo and Hahn, Stephan A and Vangala, Deepak and Trajkovic-Arsic, Marija and Gupta, Aayush and Heid, Irina and Noël, Peter B and Braren, Rickmer and Erkan, Mert and Kleeff, Jörg and Sipos, Bence and Sayles, Leanne C and Heikenwalder, Mathias and Heßmann, Elisabeth and Ellenrieder, Volker and Esposito, Irene and Jacks, Tyler and Bradner, James E and Khatri, Purvesh and Sweet-Cordero, E. Alejandro and Attardi, Laura D and Schmid, Roland M and Schneider, Guenter and Sage, Julien and Siveke, Jens T
Nature Medicine, ISSN 1078-8956, 10/2015, Volume 21, Issue 10, pp. 1163 - 1171
Pancreatic ductal adenocarcinoma (PDAC) is one of the most lethal human cancers and shows resistance to any therapeutic strategy used. Here we tested... 
SELECTIVE-INHIBITION | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-MYC | INTRAEPITHELIAL NEOPLASIA | CELL BIOLOGY | ENGINEERED MOUSE MODELS | LUNG-CANCER | IN-VIVO | BROMODOMAIN INHIBITION | KRAS | DRUG-COMBINATION | PROGRESSION | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Adenocarcinoma - therapy | Epigenesis, Genetic | Humans | Clustered Regularly Interspaced Short Palindromic Repeats | Carcinoma, Pancreatic Ductal - therapy | Histone Deacetylase Inhibitors - therapeutic use | Mice | Adenocarcinoma - drug therapy | Proteins - antagonists & inhibitors | Adenocarcinoma | Proteins | Physiological aspects | Care and treatment | Research | Pancreatic cancer | Clinical trials | Cancer therapies | Tumors
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4. Full Text Mesenchymal Wt1 positive stellate cells represent a minor source of the fibrotic response in pancreatitis
by Regel, Ivonne and Dreyer, Tobias and Wuschek, Alexander and Hausmann, Simone and Benitz, Simone and Schäffer, Isabell and Raulefs, Susanne and Kong, Bo and Steiger, Katja and Esposito, Irene and Erkan, Mert and Kleeff, Jörg and Michalski, Christoph W
Pancreatology, ISSN 1424-3903, 2015, Volume 15, Issue 3, pp. S2 - S2
  [...]isolated pancreatic fibroblasts from cerulein-treated mice show an elevated number of Wt1 positive cells compared to untreated control mice and a strong... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Fibroblasts | Pancreas | Rodents
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5. Full Text Cellulose‐Based Microparticles for Magnetically Controlled Optical Modulation and Sensing
by Hausmann, Michael K and Hauser, Alina and Siqueira, Gilberto and Libanori, Rafael and Vehusheia, Signe Lin and Schuerle, Simone and Zimmermann, Tanja and Studart, André R
Small, ISSN 1613-6810, 12/2019, p. 1904251
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6. Full Text Pathobiology of pancreatic cancer: implications on therapy
by Regel, Ivonne and Hausmann, Simone and Benitz, Simone and Esposito, Irene and Kleeff, Jörg
Expert Review of Anticancer Therapy, ISSN 1473-7140, 02/2016, Volume 16, Issue 2, pp. 219 - 227
Although the concept of tumor heterogeneity was established several decades ago, the interest in this topic is still unbroken. With the identification of... 
combination therapy | epigenetic alterations | cancer stem cells | tumor microenvironment | Pancreatic cancer | tumor heterogeneity | INITIATING CELLS | STEM-CELLS | SIGNALING PATHWAYS | TARGETING NOTCH | K-RAS | ONCOLOGY | GENE-EXPRESSION | FOCAL ADHESION KINASE | DUCTAL ADENOCARCINORNA | TUMOR-GROWTH | CLINICAL-SIGNIFICANCE | Epigenesis, Genetic | Humans | Pancreatic Neoplasms - pathology | Tumor Microenvironment | Drug Resistance, Neoplasm | Pancreatic Neoplasms - genetics | Antineoplastic Agents - administration & dosage | Molecular Targeted Therapy | Animals | Neoplastic Stem Cells - metabolism | Gene Rearrangement | Antineoplastic Agents - pharmacology | Pancreatic Neoplasms - therapy
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7. Full Text Insulin receptor trafficking steers insulin action
by Hausmann, Simone and Ussar, Siegfried
Molecular Metabolism, ISSN 2212-8778, 2016, Volume 5, Issue 4, pp. 253 - 254
Endocrinology & Metabolism | ENDOCYTOSIS | INTERNALIZATION | ENDOCRINOLOGY & METABOLISM
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8. Full Text GAD autoantibody affinity in adult patients with latent autoimmune diabetes, the study participants of a gad65 vaccination trial
by Krause, Stephanie and Landherr, Ulrike and Agardh, Carl-David and Hausmann, Simone and Link, Katarina and Hansen, Jesse M and Lynch, Kristian F and Powell, Michael and Furmaniak, Jadwiga and Rees-Smith, Bernard and Bonifacio, Ezio and Ziegler, Anette G and Lernmark, Åke and Achenbach, Peter and Diabetes - klinisk obesitasforskning and Lund University and Diabetes and Celiac Unit and Diabetes - Clinical Obesity and Institutionen för kliniska vetenskaper, Malmö and Diabetes och celiaki and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden and Department of Clinical Sciences, Malmö
Diabetes Care, ISSN 0149-5992, 2014, Volume 37, Issue 6, pp. 1675 - 1680
OBJECTIVE Patients with latent autoimmune diabetes in adults (LADA) express autoantibodies against the 65-kDa isoform of GAD (GADA). Intervention with... 
RESPONSES | THERAPY | ENDOCRINOLOGY & METABOLISM | RISK | MELLITUS | ONSET | CHILDREN | Alum Compounds - chemistry | Double-Blind Method | Autoantibodies - blood | Humans | Middle Aged | Vaccination | Male | Vaccines, Synthetic - immunology | Diabetes Mellitus, Type 1 - therapy | Glucose Intolerance | Disease Progression | Insulin-Secreting Cells - immunology | Insulin-Secreting Cells - metabolism | Adult | Female | Aged | Diabetes Mellitus, Type 1 - immunology | Vaccines, Synthetic - administration & dosage | Glutamate Decarboxylase - immunology | C-Peptide - metabolism | Diabetics | Usage | Autoantibodies | Analysis | Clinical trials | Health aspects | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
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9. Full Text The role of inflammation in pancreatic cancer
by Hausmann, Simone and Kong, Bo and Michalski, Christoph and Erkan, Mert and Friess, Helmut
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 2014, Volume 816, pp. 129 - 151
Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with an extremely poor prognosis. Inflammatory processes have emerged as key mediators of... 
Pancreatic Neoplasms - epidemiology | Pancreatic Neoplasms - etiology | Humans | Pancreatitis, Chronic - epidemiology | Carcinoma, Pancreatic Ductal - therapy | Signal Transduction - genetics | Pancreatitis, Chronic - genetics | Cytokines - physiology | Carcinoma, Pancreatic Ductal - epidemiology | Signal Transduction - immunology | Cell Transformation, Neoplastic - immunology | Animals | Cell Transformation, Neoplastic - genetics | Pancreatitis, Chronic - complications | Pancreatitis, Chronic - therapy | Mice | Carcinoma, Pancreatic Ductal - etiology | Pancreatic Neoplasms - therapy
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10. Full Text The role of inflammation in pancreatic cancer
by Hausmann, Simone and Kong, Bo and Michalski, Christoph and Erkan, Mert and Friess, Helmut
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 2014, Volume 816, pp. 129 - 151
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11. Full Text How fibrosis influences imaging and surgical decisions in pancreatic cancer
by Erkan, Mert and Hausmann, Simone and Michalski, Christoph W and Schlitter, Anna M and Fingerle, Alexander A and Dobritz, Martin and Friess, Helmut and Kleeff, Jörg
Frontiers in Physiology, ISSN 1664-042X, 2012, Volume 3, p. 389
Our understanding of pancreatic ductal adenocarcinoma (PDAC) is shifting away from a disease of malignant ductal cells-only, toward a complex system where... 
Angiogenesis | Stellate cells | Molecular diagnostics | Desmoplasia | Periostin | Microenvironment | Cystic pancreatic tumors | molecular diagnostics | PHYSIOLOGY | RISK-FACTORS | desmoplasia | periostin | DUCTAL ADENOCARCINOMA | angiogenesis | stellate cells | CYSTIC NEOPLASMS | ALPHA-V-BETA-6 INTEGRIN | INTERNATIONAL CONSENSUS GUIDELINES | microenvironment | PAPILLARY MUCINOUS NEOPLASMS | POSITRON-EMISSION-TOMOGRAPHY | DISTAL PANCREATECTOMY | IN-VIVO | cystic pancreatic tumors
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12. Full Text Periostin promotes pancreatic carcinogenesis and metastatic spread
by Hausmann, Simone and Regel, Ivonne and Mazur, Pawel and Federlein, Anna and Wagner, Nadine and Steiger, Katja and Michalski, Christoph and Erkan, Mert and Friess, Helmut and Kleeff, Joerg
Pancreatology, ISSN 1424-3903, 2015, Volume 15, Issue 3, pp. S33 - S33
  [...]the development of therapeutic drugs directed against periostin and its downstream targets might be a promising approach to inhibit pancreatic... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Pancreas | Rodents | Metastasis
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13. Full Text Abstract B08: The role of periostin in pancreatic carcinogenesis and metastatic spread
by Hausmann, Simone and Regel, Ivonne and Federlein, Anna and Mazur, Pawel K and Steiger, Katja and Michalski, Christoph W and Erkan, Mert and Kleeff, Jörg
Cancer Research, ISSN 0008-5472, 07/2015, Volume 75, Issue 13 Supplement, pp. B08 - B08
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14. Full Text Analysis of the extracellular matrix protein periostin in early pancreatic carcinogenesis
by Hausmann, Simone and Regel, Ivonne and Michalski, Christoph W and Erkan, Mert and Friess, Helmut and Kleeff, Jörg
Pancreatology, ISSN 1424-3903, 2013, Volume 13, Issue 3, pp. S14 - S15
  Conclusion: The lack of periostin promotes a worse pancreatic regeneration after cerulein induced pancreatic injury. [...]periostin affects the development... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Cell culture | Extracellular matrix | Pancreas | Experiments | Rodents
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15. Full Text The role of inflammation in pancreatic cancer
by Hausmann, Simone and Kong, Bo and Michalski, Christoph and Erkan, Mert and Friess, Helmut
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 2014, Volume 816, pp. 129 - 151
Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with an extremely poor prognosis. Inflammatory processes have emerged as key mediators of... 
PHASE-II TRIAL | ENHANCED EXPRESSION | ONCOLOGY | UROKINASE PLASMINOGEN-ACTIVATOR | DUCTAL ADENOCARCINOMA | EPIDERMAL-GROWTH-FACTOR | BETA SIGNALING PATHWAYS | MATRIX METALLOPROTEINASE-2 | IMMUNOLOGY | CELL-LINES | INTRAEPITHELIAL NEOPLASIA | NF-KAPPA-B
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16. Full Text Chronic d-serine supplementation impairs insulin secretion
by Suwandhi, Lisa and Hausmann, Simone and Braun, Alexander and Gruber, Tim and Heinzmann, Silke S and Gálvez, Eric J.C and Buck, Achim and Legutko, Beata and Israel, Andreas and Feuchtinger, Annette and Haythorne, Elizabeth and Staiger, Harald and Heni, Martin and Häring, Hans-Ulrich and Schmitt-Kopplin, Philippe and Walch, Axel and Cáceres, Cristina García and Tschöp, Matthias H and Rutter, Guy A and Strowig, Till and Elsner, Martin and Ussar, Siegfried
Molecular Metabolism, ISSN 2212-8778, 10/2018, Volume 16, pp. 191 - 202
The metabolic role of -serine, a non-proteinogenic NMDA receptor co-agonist, is poorly understood. Conversely, inhibition of pancreatic NMDA receptors as well... 
Obesity | Diabetes | d-serine | Insulin secretion | D-serine | NMDA RECEPTORS | SEQUENCES | SUSCEPTIBILITY | SCHIZOPHRENIA | RISK | METABOLOMICS | ANTIPSYCHOTICS | ENDOCRINOLOGY & METABOLISM | DOUBLE-BLIND | EXPRESSION | CONTRIBUTES
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17. Full Text METTL13 Methylation of eEF1A Increases Translational Output to Promote Tumorigenesis
by Liu, Shuo and Hausmann, Simone and Carlson, Scott Moore and Fuentes, Mary Esmeralda and Francis, Joel William and Pillai, Renjitha and Lofgren, Shane Michael and Hulea, Laura and Tandoc, Kristofferson and Lu, Jiuwei and Li, Ami and Nguyen, Nicholas Dang and Caporicci, Marcello and Kim, Michael Paul and Maitra, Anirban and Wang, Huamin and Wistuba, Ignacio Ivan and Porco, John Anthony and Bassik, Michael Cory and Elias, Joshua Eric and Song, Jikui and Topisirovic, Ivan and Van Rechem, Capucine and Mazur, Pawel Karol and Gozani, Or
Cell, ISSN 0092-8674, 01/2019, Volume 176, Issue 3, pp. 491 - 504.e21
Increased protein synthesis plays an etiologic role in diverse cancers. Here, we demonstrate that METTL13 (methyltransferase-like 13) dimethylation of eEF1A... 
lung cancer | pancreatic cancer | METTL13 | RAS | protein methylation | translation | eEF1A | lysine methylation | translation elongation | PROTEIN METHYLATION | INHIBITION | K-RAS ONCOGENE | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | CARCINOMAS | VECTORS | PROGRESSION | CELL BIOLOGY | Medical colleges | Lysine | Analysis | Pancreatic cancer | Lung cancer | Transferases | Protein biosynthesis | Methylation | Statistics
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18. Full Text Loss of Periostin Results in Impaired Regeneration and Pancreatic Atrophy after Cerulein-Induced Pancreatitis
by Hausmann, Simone and Regel, Ivonne and Steiger, Katja and Wagner, Nadine and Thorwirth, Manja and Schlitter, Anna M and Esposito, Irene and Michalski, Christoph W and Friess, Helmut and Kleeff, Jörg and Erkan, Mert
American Journal of Pathology, The, ISSN 0002-9440, 2016, Volume 186, Issue 1, pp. 24 - 31
The extracellular matrix molecule periostin (POSTN, encoded by POSTN ), which is secreted by activated pancreatic stellate cells, has important functions in... 
Pathology | FIBROSIS | CELLS | PROTEIN | RESECTION | INFLAMMATION | STROMA | MODEL | PATHOLOGY | EXPRESSION | Immunohistochemistry | Ceruletide - toxicity | Pancreas, Exocrine - pathology | Pancreatitis - chemically induced | Cell Adhesion Molecules - metabolism | Mice, Knockout | Pancreas, Exocrine - physiology | Atrophy | Regeneration | Animals | Pancreatitis - pathology | Mice | Pancreatitis - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal
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19. Full Text MYC-induced epigenetic activation of GATA4 in lung adenocarcinoma
by Castro, Ines C and Breiling, Achim and Luetkenhaus, Katharina and Ceteci, Fatih and Hausmann, Simone and Kress, Sebastian and Lyko, Frank and Rudel, Thomas and Rapp, Ulf R
Molecular Cancer Research, ISSN 1541-7786, 02/2013, Volume 11, Issue 2, pp. 161 - 172
Human lung cancer is a disease with high incidence and accounts for most cancer-related deaths in both men and women. Metastasis is a common event in non-small... 
TRANSCRIPTION FACTORS | METHYLATION | PROTEIN | REPRESSION | ONCOLOGY | GENE-EXPRESSION | BINDING DOMAIN | GRANULOSA-CELL TUMORS | PROMOTER | CANCER | FAMILY | CELL BIOLOGY | Mucin-2 - genetics | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Epigenesis, Genetic | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Cell Growth Processes - physiology | Lung Neoplasms - pathology | Male | Genes, myc | DNA Methylation | Proto-Oncogene Proteins c-myc - biosynthesis | Adenocarcinoma - metabolism | Female | Adenocarcinoma - genetics | GATA4 Transcription Factor - biosynthesis | Lung Neoplasms - genetics | Promoter Regions, Genetic | GATA4 Transcription Factor - genetics | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cell Adhesion - physiology | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics
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20. Full Text Proceedings of the 23rd Paediatric Rheumatology European Society Congress: part two
by Olga Lomakina and Ekaterina Alekseeva and Sania Valieva and Tatiana Bzarova and Irina Nikishina and Elena Zholobova and Svetlana Rodionovskaya and Maria Kaleda and Yasuo Nakagishi and Masaki Shimizu and Mao Mizuta and Akihiro Yachie and Yuko Sugita and Nami Okamoto and Kousuke Shabana and Takuji Murata and Hiroshi Tamai and Eve M. Smith and Peng Yin and Andrea L. Jorgensen and Michael W. Beresford and on behalf of On behalf of the UK JSLE Cohort Study and Eve M. Smith and Antonio Eleuteri and Beatrice Goilav and Laura Lewandowski and Angel Phuti and Dawn Wahezi and Tamar Rubinstein and Caroline Jones and Paul Newland and Stephen Marks and Rachel Corkhill and Diana Ekdawy and Clarissa Pilkington and Kjell Tullus and Chaim Putterman and Chris Scott and Antony C. Fisher and Michael W. Beresford and Eve M. Smith and Laura Lewandowski and Angel Phuti and Andrea Jorgensen and Chris Scott and Michael W. Beresford and Ezgi Deniz Batu and Can Kosukcu and Ekim Taskiran and Sema Akman and Kubra Ozturk and Betul Sozeri and Erbil Unsal and Zelal Ekinci and Yelda Bilginer and Mehmet Alikasifoglu and Seza Ozen and Hanna Lythgoe and Michael W. Beresford and Hermine I. Brunner and Gaurav Gulati and Jordan T. Jones and Mekibib Altaye and Jamie Eaton and Mark Difrancesco and Joo Guan Yeo and Jingyao Leong and Loshinidevi D/O Thana Bathi and Thaschawee Arkachaisri and Salvatore Albani and Nagla Abdelrahman and Michael W Beresford and Valentina Leone and UK JSLE study group supported by the National Institute of Health Research Clinical Research Network and Noortje Groot and D. Shaikhani and I. E. M. Bultink and M. Bijl and R. J. E. M. Dolhain and Y. K. O. Teng and E. Zirkzee and K. de Leeuw and R. Fritsch-Stork and S. S. M. Kamphuis and Rachael D. Wright and Eve M. Smith and Michael W. Beresford and Reem Abdawani and Laila Al Shaqshi and Ibrahim Al Zakwani and Natali W. Gormezano and David Kern and Oriany L. Pereira and Gladys C. C. Esteves and Adriana M. Sallum and Nadia E. Aikawa and Rosa M. Pereira and Clovis A. Silva and Eloisa Bonfa and Jessica Beckmann and ...
PEDIATRIC RHEUMATOLOGY, ISSN 1546-0096, 05/2017, Volume 15, Issue S1, pp. 105 - 201
PEDIATRICS | RHEUMATOLOGY
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