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Developmental Biology, ISSN 0012-1606, 2016, pp. 1 - 12
Transposable elements are parasitic genomic elements that can be deleterious for host gene function and genome integrity. Heterochromatic histone modifications... 
Journal Article
PeerJ, ISSN 2167-8359, 2016, Volume 4, pp. 10 - 20
Journal Article
Genesis, ISSN 1526-968X, 2011, pp. 1 - 31
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 538, Issue 7625, pp. 336 - 343
Journal Article
The EMBO Journal, ISSN 0261-4189, 2009, Volume 28, Issue 10, pp. 1418 - 1428
We used ChIP‐Seq to map ERα‐binding sites and to profile changes in RNA polymerase II (RNAPII) occupancy in MCF‐7 cells in response to estradiol (E2),... 
SERMs | estrogen receptor α | RNA polymerase II | ChIP‐Seq | Estrogen receptor a | ChIP-Seq | TAMOXIFEN | BIOCHEMISTRY & MOLECULAR BIOLOGY | estrogen receptor alpha | PROLIFERATION | CELL BIOLOGY | BREAST-CANCER CELLS | HUMAN GENOME | PROMOTERS | ESTROGEN-RECEPTOR-ALPHA | MCF-7 CELLS | BINDING-SITES | TRANSCRIPTIONAL REGULATION | EXPRESSION
Journal Article
Bioinformatics, ISSN 1367-4803, 2011, Volume 27, Issue 2, pp. 270 - 271
Journal Article
Plos One, ISSN 1932-6203, 2011, Volume 6, Issue 3, pp. e17574 - e17574
The tumor suppressor p53 plays a crucial role in cellular growth control inducing a plethora of different response pathways. The molecular mechanisms that... 
APOPTOTIC RESPONSE | CHROMATIN | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | PHOSPHORYLATES P53 | TUMOR-SUPPRESSOR | CELL-CYCLE ARREST | INTERACTING PROTEIN KINASE-2 | BINDING-SITES | STRESS | POSTTRANSLATIONAL MODIFICATION | Chromatin - metabolism | Humans | Molecular Sequence Annotation | Tumor Suppressor Protein p53 - metabolism | Etoposide - pharmacology | Structure-Activity Relationship | DNA - metabolism | Phosphoserine - metabolism | Serine - metabolism | Genome, Human - genetics | Sequence Analysis, Protein | Chromatin Immunoprecipitation | Protein Binding - drug effects | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Binding Sites | Dactinomycin - pharmacology | Cluster Analysis | Chromatin | Analysis | Serine | Genes | Genomics | Etoposide | Genomes | Genetic transcription | Tumor proteins | Apoptosis | Actinomycin | Phosphorylation | Transcription | p53 Protein | Gene regulation | Kinases | Proteins | Pathways | Cell fate | Cell cycle | DNA methylation | Post-translation | Life sciences | Tumorigenesis | Trends | Bioinformatics | Deoxyribonucleic acid--DNA | Binding | Medical research | Gene expression | Molecular modelling | MicroRNAs | Tumor suppressor genes | Molecular biology | Binding sites | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article